Gait Cycle, IPJ Conditions, STJ, Statistics Flashcards

1
Q

Why is muscle testing important?

A
  • To assess whether muscles acting as supposed to.

- Weak muscles affect gait & stance, can be strengthened if recognized.

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2
Q

What are the 9 principles/guidelines of muscle testing?

A
  • Knowledge of muscle origin & insertions, lever arms & position relative to foot axes to understand function
  • Pt & tester comfortable as possible
  • Fixate proximal to area being tested for specificity
  • Apply pressure directly against muscle’s action
  • Apply pressure gradually
  • One joint muscle tested at end ROM
  • Two or more joint muscle tested mid range (most foot muscles)
  • Muscles tested 3 times & compared to opposite side
  • Isolate muscle being tested
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3
Q

What is the name of the grading system used?

A

Kendall’s System

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4
Q

Describe pre-heel contact phase of gait (5)

A
  • Limb internally rotates
  • knee extended
  • Ankle slightly dorsiflexed
  • STJ & MTJ slightly supinated (tib anterior)
  • 1st Ray & Hallux dorsiflexes (tib anterior & EHL)
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5
Q

Describe Contact phase of gait

A
  • Limb continues to internally rotate
  • Ankle joint begins to plantar flex
  • Knee starts to flex
  • STJ pronates to absorb leg rotations & foot becomes mobile adaptor, adjusting to terrain & absorbing shock
  • Tib post, soleus, gastrocs, FHL & FDL decelerate STJ pronation
  • MTJ remains pronated at oblique axis & starts to pronate at LA with forefoot loading. (GRF & Tib Ant)
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6
Q

Describe end of contact phase

A
  • Limb externally rotate
  • Knee extends to accept weight from contralateral leg
  • AJ starts to dorsiflex
  • STJ begins to supinate
  • MTJ remains pronated at the oblique axis
  • 1st ray dorsiflexes
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7
Q

Describe Midstance of gait

A
  • Leg externally rotate
  • Knee extended
  • AJ dorsiflexion as the trunk moves over the foot until 10 degrees of dorsiflextion
  • STJ supination
  • MTJ pronated & achieves max pronated position
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8
Q

Describe Heel lift

A
  • forward trunk momentum (COG has passed over foot) & deceleration of the tibia by the calf muscles
  • Continuing gastroc contraction which flexes knee & Pf ankle & flexes the hip indirectly
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9
Q

Describe Propulsion

A
  • Leg externally rotate
  • knee flexed
  • STJ supinates past neutral= rigid lever position, stabalise forefoot joints
  • MTJ remains maximally pronated & OA starts to supinate, inc ach heigh & foot stability
  • 1st ray plantarflexion allows 90 degrees of dorsiflexion at MPJ. Propel of hallux
  • dorsiflexion of the toes post heel lift, plantar fascia is tightened, pulling forefoot to rear foot- ‘windlass mechanism’
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10
Q

Describe swing phase

A
  • Leg internally rotates
  • Knee flexed for ground clearance, then extending for heel strike
  • STJ pronates in 1st half to allow ground clearance, supinates for heel strike
  • AJ rapidly dorsiflexes to 5 degree position at heel strike

Note: Running has ‘airborne (float) phase’ & shorter times

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11
Q

Name 4 specific gait conditions

A
  • Abductory twist: ffoot abd, heel add at HL if foot pronation occurs longer in midstance
  • Too many toes: occurs due to excessively abd gait pattern or excess STJ pronation causing inc ffoot abd
  • Resupination: lack of notes by midfoot collapse, heel ev in late midstance, ffoot splay & rolling off hallux
  • Propulsion: (lack of) noted by not pushing off hallux & hip lift
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12
Q

What are the 7 causes of Claw Toes?

What is the pathology of claw toes?

A
  • Forefoot adductus (phalanges offline with metatarsals)
  • Plantarflexed 1st ray (rigid) - OA supination occurs during midstance due to rearfoot inversion compensation increasing forefoot adduction
  • Arthritis (joints unstable, flexors overpower)
  • Spasm of digital flexors (constant flexed position, i.e, thongs)
  • Weakness of gastrocnemius muscle
  • Forefoot supinatus - addiction & plantarflexion of forefoot to toes (upset alignment= joint instability)
  • Congenital plantarflexed foot (sagittal plane alignment incorrect)
  • Instability in transverse plane
  • Flexors greater pull & ROM
  • Usually mets 2-5 affected
  • Bending at both proximal & distal interphalangeal joints (flexion at joints)
  • Joint unstable, flexor digitorum longus & flexor hallucis longus working harder than intrinsic muscles holding phalanges
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13
Q

What are the 8 causes of Hammer Toes?

What is the pathology of hammer toes?

A
  • Plantarflexed met
  • Loss of lumbrical function (main stabilisers of phalanges)
  • Imbalance between medial/lateral interossei (also stabilisers)
  • Flaccid paralysis of extensor digitorum brevis & extensor digitorum longus (injury to top of foot)
  • Short metatarsal - plantarflexion of met (to try & reach ground)
  • Forefoot valgus- grasping of toes or plantarflexion of 4th & 5th in prop (lateral side of footinverted to ground
  • Abd pressure of hallux (HAV, 2nd met unstable)
  • Trauma (fractured joints are unstble)

*Affects single toes, 2nd most common

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14
Q

Mallet toe is seen clinically as?

A

Normal proximal interphalangeal joint &

flexed distal interphalangeal joint

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15
Q

Claw toes is seen clinically as?

A

MTP hyperextension, Flexion at PIP (proximal interphalangeal joints) & DIP (distal phalangeal joints)
(middle & end joints in the toe)

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16
Q

Hallux rigidus & limitus is seen clinically as?

A

Restricted dorsiflexion of hallux & degenerative changes within first MPJ
Pain & stiffness of hallux

17
Q

What is Hallux Limitus?

A

Pathology of hallux, reduced dorsiflexion (sagittal plane motion) at first MPJ during propulsive phase of gait
- 60 degrees or less of dorsiflexion at MPJ

18
Q

What is Hallux Rigidus?

A

Total absence of dorsiflexion at first MPJ of hallux

- 5 degrees or less of dorsiflexion at MPJ

19
Q

What is the normal ROM of hallux dorsiflexion?

A

65-70 degrees

20
Q

Hallux Limitus/ Rigidus causes? (6)

A
  • Dorsiflexed 1st ray
    (can’t contact ground properly, cant Pf enough & glide on sesamoids)
  • Ankylosed 1st ray (joined to medial cuneiform)
  • Metatarsal parabola variations (2nd toe longer/shorter than 1st= doesn’t achieve proper plantar flexion)
  • Ankylosed sesamoid/ met head (ppl with bunion have hallux out of alignment with metatarsals. Causes sesamoids to get stuck & doesn’t move freely, slliding motion doesn’t occur= joint jamming)
  • Ankylosed 1st MPJ (could be why 1st MPJ not moving properly in first place) could be due to gout, joint restricted for period of time
  • Eversion in propulsion (eversion walking= pronation of foot, 1st ray pushed into ground)

*Easiest to control if pain in MPJ can control forces & minimise pain using shoe inserts etc

21
Q

How is the Subtalar Joint a mobile adaptor during pronation? (in gait)

A

Opens the medial column of the foot, to adapt to uneven terrain.

22
Q

How is the Subtalar Joint a mobile adaptor during supination? (in gait)

A

Foot becomes rigid & more stable.

Becomes a rigid lever for propulsion

23
Q

Describe the Subtalar Joint Axis

A
  • Triplanar axis, with triplanar motion (large individual variation in ROM)
  • Average 42° from Transverse Plane & 16° from Sagittal Plane
  • Recent literature suggest axis not fixed
    [Lundberg 1989]
  • 10° rotation= 7.1 FP, 6.7 TP & 2.0 SP
  • > the angle between the axis & plane of motion=
    the motion
  • More vertical the axis (> angle to TP) >TP motion
    (More further away from TP= more TP movement)
    -More medial axis (< angle to SP)=
24
Q

Discuss Subtalar Joint Neutral

A
  • Theorised position where the STJ is neither pronated or supinated & there is a 2:1 ratio of motion of inversion to eversion (supination:pronation)
  • Neutral is where the foot functions at its greatest efficiency
  • Varies from person to person
25
Q

Discuss pros vs cons of 2:1 ratio

A

Pros:
Cons: Low reproducibility amongst ppl, everyone diff.

26
Q

What is the aim of biomechanical (orthotic) treatment of STJ?

A

Aim of orthotics is to align STJ as close to neutral as possible

27
Q

What is the motion of the STJ during gait?

A
  • STJ pronates at heel contact (to absorb shock & transverse plane motion) & supinates at propulsion.
  • Root suggests foot achieves neutral postn at MTJ & into heel lift.
28
Q

What is the difference between an experimental group & a control (non-experimental) group?

A
  • Experimental group (treatment group) receives the variable being tested in an experiment.
  • One variable is tested at a time.
  • The experimental group (treatment group) is compared to a control group (did not receive treatment) which does not receive the test variable.
29
Q

What is an ICC & what does it measure?

A
  • Intraclass Correlation Coefficient measures the reliability of numerical data
  • The reliability of raters
  • The reproducibility of numerical measurements made by different people measuring the same thing
  • ICC ranges from 0-1
  • It is calculated in SPSS software as a respective 95% Confidence Interval
30
Q

What does a a high Intraclass Correlation Coefficient (ICC) close to 1 indicate?

A

High similarity between values from the same group

31
Q

What does a low Intraclass Correlation Coefficient ICC close to zero mean?

A

Values from the same group are not similar.

32
Q

Name the levels of evidence in Lloyd Smith’s

‘Hierarchy of Evidence’ (6)

A
  1. Meta-analysis of RCTs (& other studies in a systematic review)
  2. RCT
  3. Well designed non-randomised controlled study (Rolf’s never found one of these)
  4. Well designed quasi experimental study (Commonly seen, non controlled group RCT)
  5. Non experimental descriptive study- case study, comparative study (measures groups of ppl, collecting data, making comparisons, no intervention/ not experiment)
  6. Respectable opinion