GA of the critical patient Flashcards

1
Q

What should be done before GA on a critical patient?

A

Correct dehydration, hypovolaemia, BP, elctrolyte abnormalities
Ideally at least 2 catheters- ideally then place an arterial one once asleep

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2
Q

Aside from analgesia, what are some other useful properties of lidocaine?

A

Retard the effects of compromised viscera, reperfusion injury, venricular arrhythmias
Due to free radical scavenging abilities, analgesic effects, antiarrhythmic properties

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3
Q

WHat effects can anticholinergic agents (atropine/ glycopyrolate) have?

A

May make secretions more viscous
Increase anatomic dead space
Increase HR
Increase myocardial work and 02 consumption

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4
Q

What affects may dissociative agens (ketamine) have?

A

Salivation
Increase HR/ ICP/ intraoccular pressure
Analgesia
Renal elimination in cats

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5
Q

What effects can benzodiazepines have?

A

decrease other drug dosages
mild sedation and muscle relaxation
anticonvulsant
not analgesic

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6
Q

What effects can propofol have

A
rapid acting, short duration
resp depression
decreased ICP IOP
caution with volume depletion or CV compromise, can be significant depression
Peripheral vasodilation
myocardial depressant
heinz body anaemia in cats
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7
Q

When are arrythmias more prevalent during a GA?

A

Splenic disease
GDV
septic peritonitis
hypoxia

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8
Q

What levels should you act on BP?

A

< 60 mean, <90 systolic

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9
Q

What are the steps for low BP during GA?

A

decrease inhalational anaesthetic
fluid bolus
CRI of dobutamine or dopamine - monitor for tachycardia
If still low then inhalational GA may have to be swapped and switched to CRI

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10
Q

What id the use of monitoring lactate?

A

Indicator of poor perfusion as is a product of anaerobic respiration
Best to assess serial lactates to get an impression of lactate clearance - good lactate clearance suggests improving perfusion and better prognosis

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11
Q

How can lactate help give a prognosis for GVD?

A

Over 6 initially suggestive of gastric necrosis

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12
Q

WHat is the imporatance of a ventricular arrhythmia?

A

Normally a non perfusing rhythm
Can deteriorate to ventricular fibrillation
Tx when BP low, multiform complexes, R on T phenomenom
Tx with lidocaine

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13
Q

WHat are the types of capnography?

A

More common = side stream, delay of 1-2 sec

Less common = main stream, more expensive, instant, less prone to errors d/t condensation

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14
Q

What are the stages of a capnograph wave?

A
A-B = inspiratory pause
B-C start of exhalation
C-D alveolar plateau
D - end tidal CO2
D-E start of inhalation
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15
Q

What happens in hypo or hyperventilation?

A

Hypoventilation - >45mmHg - vasodilation, hypotension, poor perfusion
Hyperventilation - <35mmHg, vasoconstriction, poor perfusion, resp alkalosis

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16
Q

What does it mean if the inspiratory flow is not O?

A

Leak or fresh gas flow not high enough

17
Q

What does it mean if there is no alveolar plateau?

A

Obstruction (e.g. mucous in tube) or bronchoconstriction

18
Q

What can it mean related to the heart if there is low CO2

A

Poor cardiac output - can suggest impending crash

19
Q

How do you calculate BP?

A

BP = CO x vascular resistance

20
Q

Compare oscillometric and doppler BP measures

A

Doppler - systolic, good for small patients
can under estimate BP when low, or over estimate it when high

Oscillometric - Systolic, mean, diastolic
>5kg

21
Q

Compare warm and cold shock

A

Cold - classic shock signs, due to low volume

Warm - low vascular tone, when crt time v quick, injected mm

22
Q

What SPO2 should you aim for in a hypoxaemic patient?

A

90-95%

23
Q

What is the importance of glucosuria with normoglycaemia?

A

Suggestive of renal tubular injury
This can be due to ischaemia, toxins, drugs
Can be present days before azotaemia