Fractures Flashcards

1
Q

Fracture description

A
  1. Name of injured bone
  2. Integrity of skin/soft tissue
    Closed, open
  3. Location
    epiphyseal, metaphyseal, diaphyseal (proximal, middle, distal), physis (growth plate)
  4. Orientation/fracture pattern
    • transverse: fracture line perpendicular (<30° of angulation) to long axis of bone; result of direct high energy force
    • oblique: angular fracture line (30°- 60° of angulation); result of angulation and compressive force, high energy
    • butterfly: fracture site fragment which looks like a butterfly
    • segmental: a separate segment of bone bordered by fracture lines; result of high energy force
    • spiral: complex, multi-planar fracture line; result of rotational force, low energy
    • comminuted/multi-fragmentary: >2 fracture fragments
    • intra-articular: fracture line crosses articular cartilage and enters joint
    • avulsion: tendon or ligament tears/pulls off bone fragment; often in children, high energy
    • compression/impacted: impaction of bone; typical sites are vertebrae or proximal tibia
    • torus: a buckle fracture of one cortex, often in children
    • greenstick: an incomplete fracture of one cortex, often in children
    • pathologic: fracture through bone weakened by disease/tumour
  5. Alignment of fracture fragments
    • non-displaced: fracture fragments are in anatomic alignment
    • displaced: fracture fragments are not in anatomic alignment
    • distracted: fracture fragments are separated by a gap (opposite of impacted)
    • impacted: fracture fragments are compressed, resulting in shortened bone
    • angulated: direction of fracture apex (e.g. varus/valgus)
    • translated/shifted: percentage of overlapping bone at fracture site
    • rotated: fracture fragment rotated about long axis of bone
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2
Q

Signs of open fracture

A

continuous bleeding from puncture site or fat droplets in blood are suggestive of an open fracture

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3
Q

How to describe displacement

A

Refers to position of the distal fragment relative to the proximal fragment

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4
Q

Varus angulation

A

Apex away from midline

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5
Q

Valgus angulation

A

Apex toward midline

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6
Q

Xray rule of 2s

A

2 sides - bilateral

2 views - AP and lat

2 joints - joint above and below

2 times - before and after reduction

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7
Q

Reasons for spinting

A

Pain control

Reduces further damage to vessels, nerves and skin and may improve vascular status

Decreases risk of inadvertently converting closed to open fracture

Facilitates patient transport

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8
Q

Approach to fractures

A
  1. Clinical assessment
    - ABCs
    - R/o other fractures/injuries
    - rule out open fracture
    - SAMPLE
    - Physical exam (deformity, soft tissue integrity, maximal tenderness, NVS, avoid ROM/moving injured area to prevent exacerbation)
  2. Analgesia
  3. Imaging
  4. Splint extremity
  5. Management: Closed vs. Open Reduction
  6. obtain the reduction (see appropriate IV sedation)
    ■ closed reduction
    ◆ apply traction in the long axis of the limb
    ◆ reverse the mechanism that produced the fracture
    ◆ reduce with IV sedation and muscle relaxation (fluoroscopy can be used if available)
    ■ indications for open reduction
    ◆ “NO CAST”
    ◆ other indications include – failed closed reduction – not able to cast or apply traction due to site (e.g. hip fracture) – pathologic fractures – potential for improved function with ORIF
    ■ ALWAYS re-check and document NVS after reduction and obtain post-reduction x-ray
  7. maintain the reduction
    ■ external stabilization: splints, casts, traction, external fixator
    ■ internal stabilization: percutaneous pinning, extramedullary fixation (screws, plates, wires), IM fixation (rods)
    ■ follow-up: evaluate bone healing
  8. rehabilitate to regain function and avoid joint stiffness
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9
Q

Indications for open reduction

A

NO CAST

Non-union 
Open fracture 
Neurovascular Compromise 
Displaced intra-Articular fracture 
Salter-Harris 3,4,5 
PolyTrauma
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10
Q

What is Buck’s Traction

A

system of weights, pulleys, and ropes that are attached to the end of a patient’s bed exerting a longitudinal force on the distal end of a fracture, improving its length, alignment, and rotation

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11
Q

Normal progression of fracture healing

A

Weeks 0-3
Hematoma, macrophages surround fracture site

Weeks 3-6
Osteoclasts remove sharp edges, callus forms within hematoma

Weeks 6-12
Bone forms within the callus, bridging fragments

Months 6-12
Coritcal gap is brdiged by bone

Years 1-2
Normal architecture is achieved through remodelling

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12
Q

Evaluation of fracture healing

A

Tests of union

  • clinical: no longer tender to palpation or stressing on physical exam
  • x-ray: trabeculae cross fracture site, visible callus bridging site on at least 3 of 4 cortices
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13
Q

Local early fracture complications

A

Compartment syndrome

Neurological injury

Vascular injury

Infection

Implant failure

Fracture blisters

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14
Q

Local late fracture complications

A

Mal/non-union

AVN

Osteomyelitis

Heterotopic ossification

Post traumatic OA

Joint stiffness/adhesive capsulitis

CRPS type I/RSD

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15
Q

Systemic early fracture complications

A

sepsis

DVT

PE

ARDS secondary to fat embolism

Hemorrhagic shock

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16
Q

What is heterotopic ossification

A

The formation of bone in abnormal locations (e.g. in muscle), secondary to patho

17
Q

What is Wolff’s Law

A

Bone remodels itself to over time in response to mechanical load to better withstand loading stressors placed upon it

18
Q

Articular cartilage properties

A
  • 2-4 mm layer covering ends of articulating bones, provides nearly frictionless surface
  • avascular (nutrition from synovial fluid), aneural, alymphatic
19
Q

Avascular necrosis definition and commonly affected areas

A

Avascular Necrosis Ischemia of bone due to disrupted blood supply; most commonly affecting the femoral neck, talus neck, or proximal scaphoid

20
Q

Fracture blister definition

A

Formation of vesicles or bullae that occur on edematous skin overlying a fractured bone

21
Q

Osteochondritis dissecans

A

Osteonecrosis of subchondral bone most often occurring in children and adolescents and causing pain and potentially hindering joint motion

22
Q

Articular cartilage defects etiology

A
  • overt trauma, repetitive minor trauma (such as repetitive ankle sprains or patellar maltracking); common sports injury
  • degenerative conditions such as early stage OA or osteochondritis dissecans
23
Q

Articular cartilage defects clinical features

A
  • similar to symptoms of OA (joint line pain with possible effusion, etc.)
  • often have predisposing factors, such as ligament injury, malalignment of the joint (varus/valgus), obesity, bone deficiency (AVN, osteochondritis dissecans, ganglion bone cysts), inflammatory arthropathy, and familial osteoarthropathy
  • may have symptoms of locking or catching related to the torn/displaced cartilage
24
Q

Articular cartilage defects investigations

A
  • x-ray (to rule out bony defects and check alignment)
  • MRI
  • diagnostic arthroscopy (treatment is often guided by what is seen during arthroscopy)
25
Q

Outerbridge Classification of Chrondral Defects

A

Grade 1 - Softening and swelling of cartilage

II - Fragmentation and fissuring <1/2” in diameter

III- fragmentation and fissuring >1/2” in diameter

IV- Erosion of cartilage down to bone

26
Q

Articular cartilage defects treatment

A

• individualized
■ patient factors (age, skeletal maturity, activity level, etc.)
■ defect factors (Outerbridge Classification, subchondral bone involvement, etc.)

• non-operative
■ rest, NSAIDs, bracing

• operative
■ microfracture, osteochondral grafting (autograft or allograft) autologous chondrocyte implantation