Fluids & Electrolytes Flashcards

1
Q

what is a normal range of K

A

3.3-5

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2
Q

renal failure is most likely to cause which type of imbalance with K

A

hyperkalemia (can’t rid dietary K)

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3
Q

tissue destruction such as rhabdomyolysis, hemolysis, leukemia blast crisis can cause which type of electrolyte imbalance

A

hyperkalemia

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4
Q

For every .1 shift in pH, how is K shifted?

A

.5-1.0

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5
Q

acidosis causes which type of imbalance with K and why?

A

hyperkalemia, because a decrease in pH shifts K extracellularly (.1 in pH=.5-1.0 K)

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6
Q

what are the most common signs/symptoms of hyperkalemia

A

EKG changes, arrhythmias (both atrial & ventricle) PVC’s,

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7
Q

what are you likely to see in the EKG when K >7

A

ventricular fibrillation

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8
Q

How does the EKG progress w/ increasing K

A

tall peaked T waves>wide QRS>sine wave appearance> asystole

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9
Q

how would you treat a nonemergency hyperkalemia

A

discontinue any causes, loop diuretics, *Kayexalate-binds K in gut excreted in stool.

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10
Q

how would you treat emergency hyperkalemia

A

glucose with insulin drive K into cell, Alkanizing serum with sodium bicarbonate, albuterol, and Ca gluconate (stabilizes myocardium) Dialysis if severe..

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11
Q

what are some common causes of hypokalemia

A

diuretics, GI loss, **Burns, shift due to insulin/alkalosis, dietary intake

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12
Q

signs symptoms of hypokalemia

A

mild >3…moderate < 3 primarily EKG changes, flattened T waves, prominent U waves, ectopy, arrhythmias, muscle weakness (including resp.) in severe low K

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13
Q

tx hypokalemia

A

orally: 20-40 KCL parenterally- 10-20 mEq in adults & .25 in kids x2 hrs.

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14
Q

what amount of KCL given IV is damaging to veins & may be painful

A

> 40 mEq

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15
Q

which problem is often associated with hypokalemia that must be addressed first

A

hypomagnesmia

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16
Q

what are normal Ca levels in seru

A

8.5-10.5 total and ionized 4.65-5.25

17
Q

what percent Ca is bound to proteins vs. ionized (free)

A

60% bound, 40% free

18
Q

what are the 3 factors regulating serum calcium

A

PTH, Vitamin D, and Calcium+phosphate

19
Q

how does PTH respond to lowered level of serum Ca

A
  1. signal kidney to reabsorb more 2. signal gut to absorb more from diet 3. increase resorption of Ca from bone
20
Q

how does hypoalbuminemia affect calcium levels (both total and ionized)

A

lowers total, ionized may be normal

21
Q

how does alkalosis affect calcium levels

A

it lowers free calcium by increasing the amount bound to albumin

22
Q

how does carpal pedal spasms occur

A

alkalosis from hyperventilation, resulting in hypocalcemia

23
Q

what are some causes of hypoparathyroidsim

A

surgical removal, autoimmune disorder effecting gland, or radiation destruction, others.

24
Q

in a pt with normal PTH, albumin, and no acid-base disorders… what are some other causes of hypocalcemia

A

Vitamin D deficiency, hyperphosphatemia (renal failure can’t rid Phosphate, hence binds to free Ca), massive transfusions, hypomagnesium

25
Q

signs/sx of hypocalcemia

A

tetany, Trosseaus sign, Chvostek’s sign, seizures, myocardial dysfunction

26
Q

Tx hypocalcemia

A

correct cause: renal dialysis, Vit D or Ca supp. Mg. supp. correct ventilation

27
Q

what are two primary causes of hypercalcemia

A

hyperPTH, and malignancy

28
Q

how does milk alkali syndrome manifest itself with calcium levels..

A

causes hypercalcemia; too much dietary intake or Ca supp. for osteoporosis

29
Q

signs/sx of hypercalcemia

A

constipation, polydipsia, polyuria, lethargy, coma, anorexia, nausea, muscle weakness, recurrent stones, renal failure

30
Q

tx hypercalcemia

A

mild none, moderate- Aggressive saline hydration, Calcitonin (counteracts PTH, decreases bone resorption, increase renal excretion) **Bisphophonates **dialysis

31
Q

causes of hypermg

A

meds, renal insufficiency (urine 20-50)

32
Q

this method is used to monitor Mg levels in an eclamptic pt on a drip to ensure they don’t develop hypermg

A

hyporeflexia

33
Q

what most often associated with hypomg

A

alcoholics