FISDAP TEST 1 REPORT STUDY GUIDE Flashcards
Effects of Hyperventilation
- Respiratory alkalosis (blows off too much CO2)
- Decreases O2 sats
- Leads to hypocalcemia b/c it increases bound calcium in the blood
- The hypocalcemia leads to carpopedal spasms aka cramping/tingling in hands and feel
- Promotes air trapping, damage in the lungs, and bronchoconstriction b/c it prevents full expiration
- Increases intrathoracic pressure reducing cardiac output and increasing cardiac work load (bad in CHF and cardiac pts)
- Other symptoms may include fatigue, nervousness, dizziness, dyspnea, chest pain, and tachycardia. If the pt has a history of seizures it can also precipitate one.
Nasal Intubation Techniques
Blind Nontracheal Intubation Technique:
- Use standard Precautions
- Using basic manual and adjunctive maneuvers, open the airway and ventilate the pt with 100% O2
- Prepare your equipment
- Place pt in his position of comfort. If the pt is unconscious or if you suspect cervical spine injury place the pt supine and use manual inline stabilization as needed.
- Inspect the nose and select the larger nostril as your passageway
- Select the correct size tube, (normally 1 to 1 1/2 sizes smaller than oral tube)
- Lubricate the tube (lidocaine can be used for longer term comfort
- Insert the ETT into the nostril with the bevel along the floor of the nostril or facing the septum, directed posterior.
- As you feel the tube drop to the posterior pharynx, listen at its proximal end for the pts breath sounds and observe for end tidal CO2. Sounds will be loudest when you reach the glottic opening.
- When you are at the glottic opening, the next time the pt takes a breath in you must quickly and gently advance the ETT tube into the glottic opening. This usually occurs at 26 or 28 if male or female. Coughing or anterior displacement of the larynx indicates correct placement. Gagging or vocal chord sounds indicates esophageal placement.
- Once placement is indicated, inflate the cuff and oxygenate the pt
- Now secure the ETT tube and reconfirm the tube depth did not change.
Nontracheal intubation is indicated for patients who are breathing spontaneously but require definitive airway management to prevent further deterioration of their condition. Responsive patients and patients with an altered mental status and an intact gag reflex who are in respiratory failure because of conditions such as COPD, asthma, or pulmonary edema.
Contraindications include:
- Suspected Nasal Fractures
- Suspected basilar skull fractures
- Suspected increase in ICP
- Combative/Uncooperative pt
- Coagulopathy (pt on heparin or warfarin)
- Significant deviated nasal septum or other obstruction
- Hypoxemia
- Cardiac or Respiratory arrest (absolute contraind.)
Disadvantages:
- more difficult and time consuming
- big risk of epistaxis
- smaller diameter tubes must be used
- more risk of sinusitis
Signs and Symptoms of ARDS
ARDS is a form of pulmonary edema that is caused by fluid accumulation in the interstitial space within the lungs. A pt with ARDS accumulates fluids as the result of increased vascular permeability and decreased fluid removal from the lung tissue as the result of a large range of lung insults. As opposed to cardiogenic pulmonary edema which is caused by a poorly functioning left Ventricle.
The cause of the S&Ss is because the fluid accumulation in the interstitial spaces causes an increase in the thickness of the respiratory membrane, reducing the ability for O2 to diffuse across it. In advanced cases, the fluids can accumulate in the alveoli as well causing surfactant washout.
S&Ss:
- There is usually symptoms present that are related to the cause of the insult to the lungs
- Tachypnea
- Tachycardia
- Crackles (rales) in both lungs
- Possible wheezing
- Severe cases may cause severe tachypnea, central cyanosis, and low O2 sats
Since ARDS is always caused by something else, treatment includes treating the underlying cause. Also CPAP can be very useful for pts approaching resp failure and may prevent need for intubation
Techniques for Removing an Advanced Airway
Extubating is very rare b/c it can cause aspiration, laryngospasms, negative pressure pulmonary edema, or the pt may even deteriorate again and be difficult to re-intubate. However, if the pt is clearly able to maintain his own airway and is intolerant to the tube and ventilator, no meds are available to make him comfortable and reassessment indicates that the problem that led to the pt being intubated is resolved (such as narc overdose), extubating may be indicated.
The steps are:
- Use PPE
- Ensure adequate O2. One way to do this is to ensure adequate signs of Oxygenation on room air
- Prepare intubation and suction equipment
- Confirm pt responsiveness
- Position on his side if possible
- Suction the oropharynx
- Deflate the ETT cuff
- Remove the ETT upon coughing or expiration
- Provide supp O2 as indicated
- Reassess the adequacy of the pts ventilation and oxygenation
Techniques for successful insertion of Dual Lumen Airway
Go over on pg 551-553 there are 2 types
Treating a pt with an obstructed airway (adult)
CONSCIOUS:
- Determine if the obstruction is complete or causing poor air exchange. If the pt can speak they can try to cough up obstruction.
- If they cannot speak that indicates complete obstruction or poor air exchange and you should provide rapid abdominal thrusts until obstruction is relieved. If abd thrusts are ineffective or pt is obese try using chest thrusts.
UNCONSCIOUS:
- Open air way with appropriate technique
- Begin CPR
- Each time you open the airway during CPR, look for an object in the victim’s mouth to remove it
- If obstruction persists and ventilation cannot be provided, use a laryngoscope to visualize the airway and if you can se the foreign body, try to remove it with Magill forceps.
If the upper airway obstruction is caused by laryngeal edema and you have tried and failed to provide O2 and BVM ventilations then next admin crystalloid solution with IV or IM epi and diphenhydramine
Treating a Vomiting pt
Calculating Drip Rates
LOOK UP IN MOODLE
Identifying and Treating a Bradycardic Rhythm
RULES: (if sinus) regularity - regular rate - less than 60 P waves - 1 per QRS PRI - 0.12-0.20sec QRS - less than 0.12sec
ACLS:
- Must be under 50bpm
- SYMPTOMATIC?
- if NO then monitor and observe
- if YES then Atropine (1mg every 3-5min up to 3mg); if Atropine is ineffective then move to TCPacing and/or dopamine (5-20mcg/kg/min) or epinephrine infusion (2-10mcg/min)
Risk Factors Associated with Strokes
From Book:
- Hypertension (the biggest one)
- A-Fib (second biggest one)
- Middle aged and older patients due to disability
- Atherosclerosis
- Heart disease
From Online:
- Diabetes
- Smoking
- Birth Control Pills
- History of TIAs
- High counts of RBCs, cholesterol, or lipids
Signs and Symptoms of Myocardial Infarction
- Most common is retrosternal chest discomfort, this can be pressure or tightness with or without pain
- Elderly, Women, and Diabetics present differently
- Radiation to shoulders, in-between shoulders, back, neck, one or both arms or jaw
- Light headed/dizziness
- fainting/sycnope
- sweating
- N/V
- Unexplained, sudden SOB W/ or W/out chest discomfort
- Less common is indigestion or acid reflux type feeling
Signs and Symptoms of Uncontrolled A-Fib
- Sensations of a fast, fluttering or pounding heartbeat (palpitations)
- Chest pain
- Dizziness
- Fatigue
- Lightheadedness
- Reduced ability to exercise
- Shortness of breath
- Weakness
A-Fib can be:
- Occasional (paroxysmal atrial fibrillation): A-fib symptoms come and go, usually lasting for a few minutes to hours.
- Persistent. With this type of atrial fibrillation, the heart rhythm doesn’t go back to normal on its own. If a person has A-fib symptoms, cardioversion or treatment with medications may be used to restore and maintain a normal heart rhythm.
- Long-standing persistent. This type of atrial fibrillation is continuous and lasts longer than 12 months.
- Permanent. In this type of atrial fibrillation, the irregular heart rhythm can’t be restored. Medications are needed to control the heart rate and to prevent blood clots.
Treating a pt in Cardiogenic Shock
- If the pt shows signs and symptoms of shock, poor perfusion, CHF, or acute Pulmonary Edema then…
- Treat hypoxia, place pt on ECG, oximeter, capno, insert an IV, obtain baseline vitals
- Look for what is causing the symptoms (treatments based off cause):
- VOLUME LOSS:
1. infuse normal saline or LR solution - VASODILATION:
1. Infuse normal saline or LR solution
2. consider vasopressor - PUMP FAILURE: is there signs of Acute Pulm Edema?
1. If NO then maintain BP with dopamine
2. If YES then:
a) apply CPAP
b) admin Nitro
c) admin ACE inhibitor
d) consider benzo for any anxiety
e) maintain BP with dopamine - ABNORMAL HEART RATE:
1. If greater than 150 then go to Tachycardic Algorithm
2. If less than 50 go to Bradycardic Algorithm
Treating a pt with Cardiac Origin Chest Pain
- Place them in a position of rest
- Given O2
- TRANSPORT
- Get IV and 12 lead but do not delay transport for this
- Give Aspirin and Nitro if possible
- If pain is unrelieved consider CCB and then Morphine
If the pt has new onset angina, unstable angina, or has a history of angina but it is not relieved by O2, rest, and Nitro then TRANSPORT IMMEDIATELY.
Treating a pt with Heart Failure
- DO NOT have the pt exert themselves in any way or have them lay supine. DO sit them on the caught sitting up with their feet dangling.
- Admin O2, Nitro, CPAP, if needed an anxiolytic, and if available an ACE inhibitor such as Captopril or Enalapril
Antidotes for Alcohol Ingestion
- Establish and maintain airway
- determine whether other drugs are involved
- start an IV of NS or LR
- Admin 25g of D50W if hypoglycemic
- 100mg Thiamine IV
- Consider benzos
Communicating with a patient having a behavioral emergency