Final Stretch Flashcards
What is a self limiting state of disequilibrium/ struggle for equilibrium & adaptation
Crisis
Crisis is experienced as what?
An overwhelming emotional reaction
How long does a crisis last?
4-6 weeks
What is the goal crisis intervention?
Return individual to pre-crisis, level of functioning or higher
Crisis intervention is focused on what?
Current situation
Types of crisis
Maturational
Situational
Adventitious
Existential
What is a maturational crisis?
Difficulty in a new developmental stage
Example:
- having a child leave home to attend college
- Birth of a child
- Death of your aged parents
- Teenagers confused about what college to get into
What is a situational crisis?
External “situation” causes crisis- happens to self
Example:
- job loss, death of a loved one, a divorce, physical or mental illness
What is an adventitious crisis?
Crisis of disaster: Larger in scale than situational: group
Example:
- natural disasters; floods, fires, earthquakes, etc.
- Mass shootings
- COVID
What is a existential crisis?
Questioning life’s purpose or spirituality
Seven stages of crisis intervention?
- Plan and conduct crisis management: SI assessment
- Establish rapport
- Identify major problems: what is the #1 problem/ trigger
- Deal with feelings/ emotions
- Explore alternative: reframe cognitive distortions
- Develop/ formulate an action: tell pt what to do/ what’s important
- Crisis resolution
- Follow up
How many phases of crisis are there?
4
Phase 1 of crisis
- Conflict threatens self-concept
- Increased anxiety
- Stimulates Defense Mechanisms/ problem solving skills to lower anxiety.
Phase 2 of crisis
- defensive response fails/threat persists
- Anxiety escalates.
- Disorganized; can’t focus
- trial-and-error attempts to solve problems begin.
Phase 3 of crisis
- Trial-and-error attempts fail
- severe and panic levels anxiety
Phase 4 of crisis
- Problem is not solved after some time
- new coping skills: ineffective
- Anxiety overwhelms pt
- Serious illness: depression, hopelessness, violence, assess for suicidal thoughts.
Crisis
Pt assessment
- SI/ HI/ plans
- Trigger
- Ability to identify trigger; perception
- Understanding of the situation
- Coping skills
- Support system; local support systems
- Religious and cultural beliefs: churches provide food, housing, clothing.
- Need for psychiatric treatment or hospitalization.
- Need for primary, secondary, or tertiary intervention.
Crisis:
How do you assess pt’s perception of the trigger?
- Has anything upsetting happened to you within the past few days or weeks?
- What was happening in your life before you started to feel this way?
- Has anything traumatic happened in the past and is still bothering you?
- What leads you to seek help now?
- Describe how you are feeling right now
- How does this situation affect your life?
- What would need to be done to resolve this situation?
- What type of help do you think you need?
Crisis interventions
- Empathy
- Listen
- Calming (therapeutic communication)
- Hope (instill hope)
- Empowerment
- lower anxiety
- Connectedness (support systems)
- assist in crisis coping
- acceptance of the situation
- Set realistic goals
- Help pt formulate a plan
- Strategies to build up resilience
- Pt safety
- Schedule regular follow-up to assess progress.
Crisis:
Strategies to build up resilience
- Build social support
- CBT to reframe perception
- Teach pt change is part of life, it does not=powerlessness
- Empower pt to identify goals and take action
- Spiritual support (MSHP)
- Self-care:
—Cultivate insight/self-awareness
—encourage positive view of self,skills developed, gain self—-worth, strength, resilience hope, see big picture
Crisis intervention techniques
Goal: quick resolution
- Catharsis: release of feelings
- Clarification
- Suggestion: use CBT to reframe perception
- Reinforcement of behavior: (positive reinforcement)
- Support of defenses: gently encouraged or discouraged
- Raising self-esteem: empowers pt to problem solve
- Exploration of solutions: to immediate crisis
Phases of Critical Incident Stress Debriefing (CISD)
If tr s tr
Introductory
Fact
Thought
Reaction
Symptom
Teaching
Reentry
Crisis:
What happens in the introductory phase of CISD
overview of what happened;
- process
- confidentiality
- key members are identified
Crisis:
What happens in the fact phase CISD
what happened; facts only
Crisis:
What happens in the thought phase of CISD
what were you thinking when event happened
Crisis:
What happens in the reaction phase of CISD
what was your reaction; discuss feelings; most painful/ hardest to process
Crisis:
What happens in the symptom phase of CISD
what is everyone’s s/s, how’s it affecting your work
Crisis:
What happens in the teaching phase of CISD
Teach:
- resources; additional therapy
- s/s of PTSD
Crisis:
What happens in the reentry phase of CISD?
are you ok to go back to work?
being a paramedic, nurse, caretaker
Agnosia
Ability to name/recognize objects
Confabulation
Making up stories to fill in gaps of memory
Aphasia
Loss of ability to understand or express speech
Apraxia
Not being able to complete physical tasks because you don’t remember
Hypermetamorphosis
Having to touch things
Perseveration
Mind is on a hamster wheel; continuous thought
Delirium risk factors
DAMP SNIDS
- Dementia
- Age; young children/older adults
- Medication; polypharmacy; anesthesia
- Pain; uncontrolled/ perfusion problems
- Sleep disturbances
- Number of co-occurring conditions; comorbidity
- Infection
- Depression
- Substance/alcohol use
Delirium onset
Sudden; hours- days
What causes Delirium
- Hypoglycemia
- Infection
- Fever
- Dehydration
- Hypotension
- Head injury
- Pain
- Change in environment
- Emotional stress
- B12 and folate deficiencies
- meds
Delirium S/S
- Altered LOC
- Mood swings
- Anxiety
- Paranoid
- Aggression
- Hallucinations/delusions
- Speech: rapid, inappropriate, incoherent, rambling
- Activity: fluctuates
—Sundowners - cognition: fluctuates
—Impaired - memory
- Judgment
- Calculations
- Attention span
Delirium, prognosis
Reversible; with proper in timely treatment
Delirium intervention
- Consistent caretakers
- Monitor continuously
- Assess LOC every 2-4 hours
- Reorient
- Distract (redirect) pt from harmful behaviors
- Use short, simple, concrete phrases to communicate
- Keep room well lit
- Explain procedures, simply
- Comfort needs: pain, thirst, hunger, elimination
- Glasses & hearing aides
- Provide clocks & calendars
What is Dementia
Characterized by multiple cognitive deficits, including memory, and develops over a long period of time. May be secondary, due to a medical condition, or primary.
Dementia onset
Slow; months - years
What causes Dementia
Neurological disease
Alzheimer’s disease
Vascular disease
HIV
Chronic alcoholism
Vitamin B12 deficiency
Folate deficiency
Head trauma
Dementia S/S
LOC: not altered
Impaired:
- memory
- judgment
- calculations
- attention span
- thinking
- Agnosia: Doesn’t recognize family or objects
- Anxiety
- Anger
- Aggression
- Delusions
- Paranoid
- Catastrophic reactions
- Flat affect
Activity: not altered; (sundowners)
- Impaired motor ability
- Wandering
Speech:
- incoherent
- Slow
- Inappropriate
- Rambling
- Repetitious
Dementia prognosis
not reversible; progressive
Dementia interventions
Monitor client closely
Reorient Client
Positive feedback
Explain simply
Discourage suspiciousness
Avoid cultivation of false ideas
What is Huntington’s disease?
Progressive brain disorder
defective dominant gene on a chromosome 4
Huntington’s disease S/S
- Chorea: Uncontrolled movements of the arms, legs, head, face and upper body
- Impaired Judgement
- Impaired attention span
- Impaired Memory
- Impaired planning and organization
- anxiety
- anger
- irritability
- Obsessive compulsive behavior: person repeats same question and activity over and over
Huntington’s disease intervention
Interventions: manage symptoms; Meds
No Cure
Support group: pts feel normal/ not alone
Huntington’s Disease Society of America (HDSA)
HDSA at 800.345.4372
Huntington’s disease
Chorea meds
Atypical antipsychotics:
- Olanzapine
- Baclofin (muscle relaxer)
Huntington’s disease
Irritability Meds
Atypical antipsychotic
SSRI (less severe nonthreatening irritability)
Huntington’s disease
OCD thoughts- Meds
SSRIs
Creutzfeldt-Jakob Disease
Viral protein (Prion) found in CSF causes brain degeneration
- Spread by infected meat: MAD Cow disease
Creutzfeldt-Jakob Disease Labs
CSF proteins
EEG
MRI
Creutzfeldt-Jakob Disease S/S
Early stage
memory loss
visual impairment
dysphagia; speech disfunction
Creutzfeldt-Jakob Disease S/S
Within weeks
progressive dementia set in
Creutzfeldt-Jakob Disease S/S
1-2 years
Death
Creutzfeldt-Jakob Disease S/S
Rapid progression is one of the most important clue
A MAD DR. - MJ-TP
- apathy
- mood swings
- Agitation
- Depression
- Disorientation
- Rapidly worsening confusion
Impaired:
- memory
- judgment
- thinking
- planning
Activity:
- Difficulty walking
- Muscle stiffness
- Muscle twitches; involuntary jerky movements
Eyes:
- double vision
- hallucinations
Creutzfeldt-Jakob Disease intervention
No treatment can slow or stop brain cell destruction
Vascular Dementia
Reduce blood flow/ oxygen/ nutrients to the brain causes a decline in thinking skills/ multiple small strokes
Vascular Dementia S/S
- Confusion
- Disorientation
- Trouble speaking / understanding speech
- Physical stroke symptoms; sudden headache
- Difficulty walking; poor balance
- Numbness or paralysis on one side of the face or the body
Vascular Dementia
Interventions
keep circulation good
- Don’t smoke
- Keep BP/ cholesterol/ blood sugar within limits
- Healthy, balanced diet
- Exercise
- Healthy weight
- Limit alcohol
Picks diseases
frontotemporal neurocognitive disorder
Picks disease (FTD) prognosis
5 years average lifespan after diagnosis
Picks disease (FTD) S/S
reflect area of brain affected Behavior, Personality, Language
- Memory loss; advanced FTD
- Behavior changes; first noticeable s/s
- Speech difficulties
- difficulty making sense when they speak
- Difficulty understanding the speech of others
- Difficulty reading
What causes Picks disease (FTD)
Unknown
Picks disease (FTD) onset
40s- early 60s
What is Lewy Body Disease
faster acting than Alzheimer’s
- Protein deposits (Lewy bodies) in the brain stem (and other parts of the brain) deplete Dopamine and Acetylcholine causing Parkinson’s-like physical symptoms and Alzheimer’s-like cognitive symptoms.
Lewy Body Disease Prognosis
Average lifespan 5-7 years
Lewy Body Disease S/S
- Changes in thinking and reasoning.
- Memory loss- less prominent than in Alzheimer’s
- Fluctuating cognition that is delirium-like.
- Trouble interpreting visual information
- visual hallucinations.
- REM sleep behavior disorder; acting out dreams.
- slowness of movement
- resting tremor/ rigidity.
Malfunctioning (“automatic” (autonomic) nervous system): - sweating, BP/ HR, digestion and sexual response
Lewy Body Disease interventions
None other than:
- self-care deficit
- meds to treat s/s
Lewy Body Disease Meds:
Treat thinking changes in Alzheimer’s
Cholinesterase inhibitor drugs
Lewy Body Disease Meds:
Treat behavioral symptoms in Alzheimer’s
Adverse affects of meds?
Antipsychotic drugs; use with caution. May cause:
- sudden changes in consciousness
- impaired swallowing
- acute confusion
- delusions/ hallucinations
- appearance or worsening of Parkinson’s symptoms.
Lewy Body Disease Meds:
- depression in Alzheimer’s disease
SSRIs
Most common type of dementia
Alzheimer’s disease
Alzheimer’s disease prognosis
Death 4-8 years after dx
Alzheimer’s disease risk factors
- Age: >65
- diabetes
- hypertension
- Hx: TBI
Alzheimer’s disease
Modifiable risk factors
behaviors that can be changed
- physical activity
- smoking
- education
- staying socially and mentally active
- blood pressure and diet
What causes Alzheimer’s disease?
- Microscopic changes in the brain begin long before the first signs of memory loss
- Role of plaques and tangles in damaging and killing nerve cells
— Protein fragments called beta-amyloids deposit plaques that build up in the spaces between nerve cells.
—Twisted fibers of a protein called tau build up into Tangles inside cells.
Alzheimer’s disease labs
CSF: for tau
MRI
CT scan
Beta-amyloid pet scan
Early s/s Alzheimer’s disease
- Memory loss that disrupts daily life
- Challenges in Planning or solving problems
- Difficulty completing family tasks
- Confusion with time or place
- Trouble understanding visual images and spatial relationships
- New problems with words in speaking or writing
- Misplacing things and losing the ability to retrace steps
- Decreased or poor judgment
- Withdrawal from work or social activities
- mood/ personality Changes
Stages of Alzheimer’s disease
Mild
Moderate
Moderate to severe
Late: end stage
Alzheimer’s disease: S/S
Stage: mild
Forgetfulness
symptoms not easily noticeable
- short-term memory loss
- puts things in odd places; loses them
- Uses memory aids: sticky notes
- Denial of problems common
Alzheimer’s disease: intervention
Stage: mild
Discuss estate planning in this phase
Alzheimer’s disease: s/s
Stage: moderate
Confusion
- Memory losses apparent – may forget address &/or date
- ADL’s suffer
- Problems managing money, legal affairs
- Labile mood
- Withdraw from activities- socially isolate
Alzheimer’s disease: Interventions
Stage: Moderate
Needs day care / in-home assistance
Alzheimer’s disease: S/S
Stage: Moderate to Severe
Ambulatory dementia
- Doesn’t recognize family or objects (agnosia)
- Forgets how to do activities such as walking, eating (apraxia).
- Shows ADL deficits:
— Wandering
— Depression resolves as awareness of losses diminish
Alzheimer’s disease: Interventions
Stage: Moderate to Severe
- May need placement in skilled care facility
- Needs total assistance for all activities of daily living
- Take away car keys- assess ability to drive
- dont ask open ended questions; Ask yes no questions
- Limit options
- Avoid arguing
- Talk to them in front of them
- Don’t have multiple conversations
- Limit outside noises
- Sundowners: appropriate rest periods, go outside to burn off energy, increase lighting in home in the evening, sleep routine
- Meds
Meds for Moderate to severe stage Alzheimer’s disease
NMDA blocker: Memantine (Namenda)
- blocks NMDA receptors to block excessive glutamate, which has a toxic effect on neurons.
Alzheimer’s disease: S/S
Stage: Late: End Stage
- Exhibits hypermetamorphosis & hyperorality
- Does not recognize self anymore
- Forgets how to eat, swallow, chew
- Loses ability to talk & walk
- Prone to complications of immobility like pneumonia
Alzheimer’s disease: Interventions
Stage: Late: End Stage
- Require around-the-clock assistance with daily personal care
- Needs total care; incontinence common
Alzheimer’s disease prognosis
DX-death: 10 years, some people may live up to 20 years.
Alzheimer’s disease first line treatment medication
Cholinesterase inhibitors: increase ACh
- Donepezil (Aricept)
- Rivastigmine (Exelon)
- Galantamine (Razadyne)
Nursing implications:
- Take with food
- Bradycardia
- Fall risk
Antidote: Atropine
New Alzheimer’s disease med?
S/S?
Aducanumab
S/S:
- Headache
- Fall risk
- dizziness
- nausea
- confusion
- vision changes
- potential for allergic reactions
Addiction:
Prior to admission you need to know what 3 things
What was taken?
how much was taken?
when was it last taken?
What are the CNS depressants?
alcohol
benzodiazepines
barbiturates
One drink =
A 12 ounce can/ bottle of beer 5% Alcohol
A 5-ounce glass of wine 12% alcohol
A shot of Hard Liquor 1.5 ounces 40% Alcohol
Low risk drinking amount
MEN: 18-65
- 4 per day
- 17 per week
WOMEN: 18-65 & AGES 66+
- 3 per day
- 7 per week
Blood Alcohol Levels: .05= S/S
X Drinks
1-2 drinks
changes in mood/ behavior
Legal blood alcohol content
.08 %
Blood Alcohol Levels: .10= S/S
X Drinks
5-6 drinks
voluntary actions, clumsy
Blood Alcohol Levels: .20= S/S
X Drinks
10-12 drinks
staggering, labile emotions
Blood Alcohol Levels: .30= S/S
X Drinks
15-18 drinks
confusion, stupor
Blood Alcohol Levels: .40= S/S
X Drinks
20-24 drinks
coma
Blood Alcohol Levels: .50= S/S
X Drinks
25-30 drinks
Death due to respiratory depression
Alcohol INTOXICATION S/S
- slurred speech
- incoordination
- staggering; unsteady gait
- drowsiness
- Decreased vital signs
- inhibitions lowered resulting in impulsive sex and aggression
- impaired judgment; role function
- Impaired attention/memory
- Irritability
- social problems
- occupational problems
When do Alcohol Withdrawal S/S start
Within a few hours after last drink
When do alcohol withdrawal s/s peak and disappear?
24-48 hours
Alcohol withdrawal s/s
DIC I SAID SHCVATTAANV 😮💨
- Delirium
- illusions
- confusion
- Increased BP & HR
- Seizures
- anorexia
- insomnia
- Disorientation
- Sweating
- Headache
- Clouded senses
- Visual disturbances
- Audio disturbances
- Tactile disturbances
- Tremors
- Anxiety
- Agitation
- Nausea
- Vomiting
Alcohol withdrawal interventions
Calm, quiet, reassuring atmosphere
Frequent reorientation
- Assess CIWA
- Monitor for seizures
- VS Q 2-4 hours
- Psycho-social support
- Medication
— Benzodiazepines
— clonidine
— thiamine/ multivitamins
- relapse prevention
— Antabuse (Disulfiram)
— Naltrexone (ReVia)
Tool developed to monitor withdrawal symptoms
CIWA scale
If the CIWA score is ______ for _____ long you should contact the HCP
> 17 for >4 hours
CIWA categories
- Visual Disturbances
- Auditory disturbances
- Nausea
- Clouding of the sensorium
- Headache
- Agitation
- Tactile Disturbances
- Tremor
- Vomiting
- Paroxysmal Sweats
- Anxiety
Write out the CIWA med/reassessment table
Gold standard medication: alcohol withdrawal
Benzodiazepines:
Short acting : lorazepam (Ativan)
Long acting : Chlordiazepoxide (Librium)