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Toxicology > Final notes > Flashcards

Final notes Flashcards

1
Q

Produced by incomplete oxidation of fossil fuels

A

CO

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2
Q

what are Oxidizing Agents

A 
Nitrogen oxide
nitrates
nitrites
chlorates
some medications
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3
Q

Strong acids are

A

inorganic acids
HCL
H2SO4
HNO3

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4
Q

Strong Bases are

A

inorganic bases
NaOH
KOH
NH4OH

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5
Q

competes with O2 for Fe2+ binding site in here of reduced Hb to decrease oxygen saturation of arterial blood –> tissue hypoxia

A

CO

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6
Q

Forms carboxy-Hb

A

CO

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7
Q

Cherry red cyanosis

A

CO

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8
Q

primary target organ for CO poisoning

A

brain–> increases risk of Parkinsons Disease

Myocardium–> Angina/MI

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9
Q

initial s/s for CO poisoning

A

HA

confusion

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10
Q

how do you manage CO poisoning?

A

use 100% O2 or hyperbaric O2 chamber

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11
Q

Heme Fe2+–> Heme Fe3+ this process is called

A

oxidization

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12
Q

oxidized Hb aka

A

methmeoblobin

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13
Q

cannot transport O2–> dec O2 saturation of arterial blood–> tissue hypoxia

A

oxidizing agents

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14
Q

normally kept

A

oxidizing agents

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15
Q

developmental deficiency of this enzyme in neonates can result in methemglobinemia if fertilizer contaminated well water is used to mix baby formula

A

oxidizing agents

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16
Q

neonate developmental deficiency of neonates resulting in methemoglobinemia is called

A

blue baby syndrome

17
Q

ammonium nitrate

A

fertilizer

18
Q

cause chocolate cyanosis

A

oxidizing agents

19
Q

precipitative necrosis at site of contact with Eschar formation that prevents deep tissue damage

A

strong acid

20
Q

wash with cold running water for no less than 15 min

A

strong acid/strong base

21
Q

dissolution necrosis at site of contact

A

strong base

22
Q

no scar–> progressive penetrative damage to underlying tissues possible

A

strong base

23
Q

salicylates

A

aka NSAD acetylsalicylic acid- aspirin, sodium salicylate

24
Q

analgesic, antipyretic, and anti inflammatory uses

A

salicylates

25
Q

COX=

A

cycloxyginase

26
Q

drug of choice for OA

A

salicylates

27
Q

major adverse effect of NSAD

A

Gastric ulcers

28
Q

COX1 presents where

A

gastric mucosa

29
Q

inhibits Cox enzymes–> decrease prostaglandin synthesis

A

salicylates

30
Q

inhibition Cox 1 in gastric mucosa decreases mucous and can cause gastric ulcers

A

salicylates

31
Q

cardinal signs of inflammation

A

rubor
calor
dolor
tumor (edema)

Toxicology (3 decks)

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