Final Exam Study Pt. 2 (most recent info) Flashcards

1
Q

Describe substance-related disorders

A

Involve problems associated with using and abusing drugs that alter patterns of thinking, feeling, and behaving.

Include problems with the use of:

  • depressants (alcohol, benzodiazepines)
  • stimulants (amphetamines, nicotine, cocaine)
  • opiates (heroin, codeine, morphine)
  • hallucinogens (cannabis, LSD)
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2
Q

Clinical description of substance-abuse related disorder

A
  • cognitive, behavioral and physiological symptoms indicating the individual continues using the substance despite significant problems
    - impaired control over substance use
    - social impairment (e.g., impact on relationships, work, school)
    - risky use

Underlying change in brain circuits that may persist beyond detoxification
- tolerance and withdrawal

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3
Q

Biological and psychological causes of substance abuse disorder

A

Biological dimensions

      - familial/genetic: twin and family studies indicate that certain people may be genetically vulnerable to drug abuse - neurobiological: drugs affect the reward or pleasure centres of the brain. 

Psychological dimensions

  - positive and negative reinforcement: people are positively reinforced for using drugs and negatively reinforced when the drugs remove unpleasant experiences such as pain or anxiety, or result in withdrawal symptoms. 
     - cognitive factors (e.g., expectations, cognitive implications of substance use)
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4
Q

Social and cultural causes of substance abuse disorder

A

Social dimensions

- exposure to substance: family, friends,  media
 - social experiences

Cultural dimensions
- differences in what is “normal”/”abnormal” or acceptable/unacceptable behavior

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5
Q

List some biological treatments for substance abuse disorder

A

Agonist substitution - provision of safe drug that is chemically similar to the abused drug (e.g., methadone)

Antagonist treatments - block or counteract the positive effects of psychoactive drugs (naloxone)

Aversive treatment - use of drugs that make ingestion of abused substance extremely unpleasant

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6
Q

Psychosocial treatments for substance abuse disorder

A

Psychosocial

 - inpatient facilities
 - alcoholics anonymous (AA)
 - controlled use
 - relapse prevention

Harm reduction
- example of SISs for harm reduction and to minimize risk of overdose

Relapse prevention

- education
- reform of laws regarding drug possession and use
 - community-based interventions
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7
Q

Describe Cluster A personality disorders and a short description of each

A

Cluster A are the odd/eccentric personality disorders

Paranoid - distrusting and suspicious interpretation of the motives of others

Schizoid - social detachment and restricted emotional expression

Schizotypal - social discomfort, cognitive distortions, behavioral eccentricities

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8
Q

Describe Cluster B personality disorders and a short clinical description of each

A

Cluster B are the dramatic/erratic disorders.

Antisocial - disregard for and violation of the rights of others

Borderline - unstable relationships, self-image, affects and impulsivity

Histrionic - excessive emotionality and attention seeking

Narcissistic - grandiosity, need for admiration, lack of empathy

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9
Q

Describe Cluster C personality disorders and give a short clinical description of each

A

Cluster C are the anxious/fearful personality disorders.

Avoidant - socially inhibited feelings of inadequacy, hypersensitivity to negative evaluation

Dependent - submissive behavior, need to be taken care of

Obsessive-compulsive - preoccupation with orderliness, perfectionism, and control

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10
Q

Define delusions, hallucinations and psychosis.

A

Delusions - fixed beliefs that do not change in light of conflicting evidence; may include variety of themes:

  • persecutory (belief that one is going to be harmed)
  • referential (belief that certain gestures and comments are directed at oneself
  • religious
  • grandiose

Hallucinations - vivid and clear perception-like experiences that occur without an external stimulus

Psychosis - loss of touch with reality; involves delusions and/or hallucination

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11
Q

Differentiate positive and negative symptoms

A

positive symptoms - ‘abnormal’ things that start happening, such as delusions and hallucinations

negative symptoms - ‘normal’ things that stop happening, such as lack of pleasure, trouble with speech, flattened affect

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12
Q

Clinical description of schizophrenia

A

Two (or more) of the following, present for a significant portion of time during a 1-month period:

  - delusions
  - hallucinations
  - disorganized speech
  - disorganized or catatonic behavior
  - negative symptoms
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13
Q

50-70% of people with schizophrenia experience positive symptoms (hallucinations, delusions, or both) (true/false)

A

True

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14
Q

Both men and women are affected by schizophrenia at the same rate (true/false)

A

True

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15
Q

Describe the development and onset of schizophrenia

A

Age of onset - greatest in early adulthood and declines with age for males, increases with age for females

Early brain damage during neurodevelopment may contribute to the development of schizophrenia

Relapse rates are very high when treatment is discontinued

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16
Q

Genetic influences of schizophrenia

A
  • multiple gene variances combine to produce vulnerability
  • children of parents with schizophrenia likely to have it too

Gene-environment interaction: interaction with specific environmental pathogens and stimuli at crucial developmental stages

17
Q

Neurobiological influences in schizophrenia

A

Dopamine - clues to the role of dopamine in schizophrenia…

  • neuroleptics (dopamine antagonists) effective in treating schizophrenia
  • neuroleptics produce negative side effects
  • L-dopa (agonist) can produce schizophrenia-like symptoms
  • amphetamines, which activate dopamine, can worsen some symptoms in schizophrenia
18
Q

Neurobiological influences of negative symptoms

A

Hypofrontality (less active frontal lobe) associated with negative symptoms

19
Q

Psychological and Social influences of schizophrenia

A

Stress

  - retrospective and prospective approaches to examine impact of stress
  - note: extreme stress can produce psychotic-like symptoms in otherwise healthy persons
   - tendency for people with schizophrenia to be found in lower social classes
20
Q

Relapse in schizophrenia treatment appears to be triggered by

A

hostile and critical family environments characterized by high expressed emotion

21
Q

Biological treatments for schizophrenia

A

Neuroleptics (dopamine antagonists) reduce or eliminate delusions and hallucinations and are effective in 60-70% of people.

Newer antipsychotics have fewer side effects (like tardive dyskinesia which are involuntary movements of the tongue, face, mouth, or jaw

Transcranial magnetic stimulation (TMS) treatment for hallucinations. TMS also improves auditory hallucinations briefly.

22
Q

Psychosocial interventions for schizophrenia

A

Behavioral family therapy - must be ongoing if patients and families are to benefit from other

Support for improving medication compliance and self care

Multilevel treatments reduce relapses

23
Q

How the DSM-5 defines neurodevelopmental disorders

A

A group of conditions with onset in the developmental period. They are characterized by developmental deficits that produce impairments of personal, social, academic, or occupational functioning.

24
Q

Clinical description of ADHD

A

Inattention (e.g. difficulty listening when spoken to, difficulty organizing tasks and activities, forgetful)

and/or

Hyperactivity-impulsivity (e.g., fidgets, difficulty waiting their turn)

May be combined presentation, predominantly inattentive, or predominantly hyperactive/impulsive

25
Q

Causes of ADHD

A

Hereditary factors; there are multiple genes responsible.

Inhibition of the dopamine transporter gene.

Exposure to lead, infections, or alcohol in utero.

Family members with ADHD more likely to have children with ADHD

26
Q

Biological treatment of ADHD

A

Medication
Aim: to reduce impulsivity and hyperactivity, and to improve attention
- typically, use of prescription of stimulant medication (i.e., Ritalin, Dexedrine)
- effects non long-term

Stimulant medication: increases dopamine levels in brain

27
Q

Psychological treatments for ADHD

A

Goal setting and reinforcement. Cognitive behavioral intervention for ADHD also appears to be helpful for adults with ADHD. Most clinicians focus on using a combination of treatment approaches.

28
Q

Clinical description of ASD

A
  • persistent difficulties with social communication and social interaction across multiple contexts
  • restricted, repetitive patterns of behavior, interests, or activities
  • impairments present in early childhood
  • effect/limit daily functioning
29
Q

Describe the social difficulties of those with ASD

A
  • challenge with developing age-appropriate social relationships
  • inability to engage in joint attention
  • disinterest in social situations
  • deficits in nonverbal communication
  • lack of prosody
30
Q

Describe the restricted and repetitive patterns of ASD

A

Restricted, repetitive patterns of behavior, interests, or activities

  - stereotyped and ritualistic behaviors
   - rituals often complex
   - if rituals are interrupted or prevented, person has a severe emotional reaction or tantrum
31
Q

According to the DSM-5, there are three levels of severity in ASD

A
  1. requiring support
  2. requiring substantial support
  3. requiring very substantial support
32
Q

Genetic influences of ASD

A
  • genetic heritability
  • families that have one child with ASD have about a 20% chance of having another child with the disorder
  • children with ASD often have older parents (gene mutations may occur in sperm of fathers or ovum of mothers)
33
Q

Neurobiological and psychological/social influences of ASD

A
  • fewer neurons in amygdala in adults. Larger amygdala in children with ASD which may contribute to fear and social withdrawal.
  • lower levels of oxytocin in blood, contributing to children not being able to form attachments/social bonds

Parents of individuals with ASD may not differ substantially from parents of children without

34
Q

Treatment of ASD

A

Psychosocial treatments:
- behavioral focus: communication, socialization, living skills

Early Intensive Behavioral Intervention (EIBI)
This is a type of ABA for very young children with ASD, usually younger than 5 and often younger than 3. EIBI uses a highly structured teaching approach to build positive behaviors (such as social communication) and reduce unwanted behaviors (such as tantrums, aggression, and self-injury). EIBI takes place in a one-on-one adult-to-child environment under the supervision of a trained professional
- inclusive schooling

Medical intervention has not been successful.

35
Q

Describe neurocognitive disorders

A

a group of disorders in which the primary clinical deficit is cognitive function.

   - cognitive function deficits are not present at birth or early life.
   - decline from a previous level of cognitive functioning. 

Have consequences for behavior and personality (e.g., paranoia, agitation, aggression)

36
Q

Clinical description of major and mild neurocognitive disorder.

A

Major neurocognitive disorder

    - gradual deterioration of brain functioning
    - affects judgment, memory, language, other advance processes

Mild neurocognitive disorder
- early stages of cognitive decline

37
Q

Describe the initial stage symptoms of neurocognitive symptoms

A

Memory and visuospatial skills are affected. Agnosia may occur, the inability to recognize and name objects. Facial agnosia may occur, in which the person has the inability to recognize even familiar faces.

38
Q

Subtypes of neurocognitive disorders

A

Alzheimer’s disease, frontotemporal lobar degeneration, Lewy body disease, vascular disease, traumatic brain injury, substance-medication-induced, HIV infection, Prion disease, Parkinson’s disease, Huntington’s disease, other medical condition, multiple etiologist