Final Exam Study Guide Flashcards
Define fatigue as it relates to speech processing.
feeling of being tired after sustained listening of speech
Differentiate between listening effort and fatigue as it relates to speech processing.
effort is something you do in the moment, allocation of resources to do the listening and fatigue is how you feel at the end of the day
What are some potential consequences for a person who experiences listening-related fatigue?
bad attitude, irritable, shorter attention span, can affect cognitive processing, how you make decisions
How is listening-related fatigue measured?
Self-report
Physical, emotional, cognitive
Effects of fatigue on
Physiologic
Salivary cortisol levels
Behavioral
WRS, word recall, response time while completing sustained speech dual-task
What situations might lead to listening-related fatigue?
noise and hearing to hear over it like being outside, riding on a train
What are some coping strategies that people with hearing loss use to reduce listening-related fatigue?
rest, isolation, skipping school, use of hearing aids or taking them out, proactive engagement
Explain how people with hearing loss can experience listening-related fatigue (physically, cognitively, socially, emotionally) with examples.
physical: feeling tired
cognitive: decreased ability to remember things
social: avoidance
emotional: stress, cranky, tired
what are external situational determinants of listening-related fatigue
Noisy conditions
Large groups
Listening for a long time
Difficulty with certain talkers
what are internalsituational determinants of listening-related fatigue
Urgency or importance of situation
Relationship of listener to talker
Perceived effort to participate
Required participation
What are some effects of listening-related fatigue?
Physical
Feeling tired
Low energy
Exhausted
Social
Avoidance
Withdrawal
Isolation
Emotional
Frustration
Anger
Sadness
Stress
Disappointment
Cognitive
Difficulty concentrating
Difficulty remembering
Define efficacy and effectiveness and explain why the difference is important. Be able to recognize examples of each
Efficacy
Does it work in the lab?
Controlled conditions
SNR, speech material, task, degree and type of hearing loss, age and cognitive status of participant
“ideal” conditions
Effectiveness
Does it work outside of the lab?
Anything goes!
Efficacy- the treatment works under controlled conditions
Effectiveness- the treatment works outside of the lab
Treatment may work under controlled conditions but not outside of the lab. Lab-based studies may tell us that a specific treatment works but outside of the lab it may not.
How is the Vanderbilt fatigue scale administered?
it is a questionnaire
they rate how often each of these situations apply
There are two factors that come into play when considering cochlear hearing loss and speech perception, sensitivity and distortion. Define each of these.
sensitivity is caused by pure tone threshold hearing loss; the audibility (did they hear it yes or not). no sensitivity will cause you to not here the signal
HA’s address this well
distortion is impairment or disorder within the auditory or cognitive system
loss of nonlinearity of cochlea, age, neuropathology, cognitive impairment
disorder in the cochlea or beyond
broadening of filters etc.
what causes sensitivity loss?
noise exposure, ototoxicity, (damage to hair cells
would fluid in the ear cause sensitivity loss?
yes but will not cause a distortion lsos because this happens in cochlea and beyond
conductive pathology only causes sensitivity loss but not distortion
Restoring or providing audibility addresses which of these? Does providing audibility completely take care of any speech perception difficulty? Why or why not?
sensitivity
if you make sounds louder, just audibility alone can it take care of speech issues? no because it only addresses the sensitivity part
What does the ear (auditory system) do?
amplifies, compresses and anaylzes frequencies
it is a nonlinear signal processors (ear, cochlear)
when it gets interrupted then distortion and sensitivity occurs
by amplifying to make up for sensitivity only addresses audibility and not distortion
what interactions across the BM result from nonlinear processing
two tone suppression
distortion products
when the signal youre hearing is different than what is in your long term memory. what addresses this?
ELU model - applies to someone who has acquired HL over time and what is in their long term memory is different than what they are getting due to the loss of OHC
Define and describe two frequency interactions across the basilar membrane that result from nonlinear signal processing.
two tone suppression & distortion product
Discuss at least three consequences of loss of nonlinear processing on the basilar membrane.
reduce frequency selectivity (reduced ability to ell two sounds apart), reduced of tonotopic (lose HF definition, whole bm becomes sensitive to lf and not hf) & loss of two tone suppression (lose peaks, everything becomes a flat line)
There are two factors that come into play when considering cochlear hearing loss and speech perception, sensitivity and distortion. Define each of these. What causes sensitivity loss? What causes distortion in the auditory system?
Sensitivity (loss of audibility)- Caused by pure tone threshold loss
Distortion (distortion within the listener’s auditory system)- Can be caused by outer hair cell loss (focus this week is effects of loss of nonlinearity because of OHC loss)- leads to reduced frequency selectivity, reduced tonotopic organization of the BM, loss of 2-tone suppression and distortion products
Cochlear synaptopathy is another cause of distortion in the auditory system
Restoring or providing audibility addresses which of these (from the previous question)? Does providing audibility completely take care of any speech perception difficulty? Why or why not?
No.
It comes close in quiet but not completely, particularly if there is a lot of distortion within the auditory-cognitive system. Addressing audibility does not address distortion.
two tone suppression
when two tones are close together in frequency, the one with the greater intensity will be amplified and the one with the lesser intensity suppressed
what are 2 frequency interactions across the basilar membrane that result from nonlinear signal processing.
2 tone suppression
distortion products
distortion products
a third tone is generated when two tones are presented. They have to be mathematically related for this to occur which you probably learned in OAE class. Enough for this class to describe that a third tone is created in the presence of two tones, in a nonlinear system.
Discuss at least three consequences of loss of nonlinear processing on the basilar membrane.
Tonotopy shifts, best frequency shifts to lower frequency
Loss of 2-tone suppression and distortion products as discussed in (4). This results in a completely different pattern of neural firing that is not restored with amplification.
Loss of or reduction of frequency selectivity and frequency resolution. This makes it more difficult to tell speech sounds apart (vowels differing only in F1, for example) and more difficulty identifying speech sounds in the presence of noise.
Which type of auditory nerve fiber has a low (level) threshold, high spontaneous rate or low spontaneous rate? Which type of auditory nerve fiber has a high (level) threshold, high spontaneous rate or low spontaneous rate? Which of these is affected in cochlear synaptopathy?
Low threshold = high spontaneous firing rate
High threshold = low spontaneous firing rate
What do we mean when we say that hearing loss causes distortions in neural activity patterns? What does this mean for amplification? How does this fit into our understanding of the ELU model of speech/language processing?
Because of the nonlinear cochlea, responses to sound inputs of varying frequencies results in patterns of excitation that are different from the auditory nerve fiber excitation that would be expected for a single-frequency stimulus. These patterns of excitation are what the brain has come to associate with certain speech sounds. If these patterns are changed due to loss of cochlear nonlinearity, a mismatch between the signal and what is stored in long term memory will occur. This will lead to explicit, effortful processing of speech/language.
How does cochlear synaptopathy affect speech processing? In a very broad sense, how would you measure degeneration of these synapses (threshold measures or supra-threshold measures)? Is threshold testing that we do in the clinic sensitive to degeneration of these synapses?
Cochlear synaptopathy makes it difficult to perceive changes in amplitude (very important for speech understanding) especially at higher levels and in noise.
This would be measured at a supra-threshold level, not at threshold. Pure tone threshold testing is not sensitive to this.
Explain why someone with cochlear synaptopathy might be able to process speech in quiet with minimal difficulty but would have significant difficulty processing speech in noise.
Cochlear synaptopathy affects hearing for high level inputs but does not adversely affect hearing for low level inputs. In a quiet situation, there is generally enough information contained in the low-level inputs for speech understanding to occur. In noisy situations, the low-level sounds are masked by the noise. The only sounds available to the auditory system, then, are the high-level sounds, which are affected by cochlear synaptopathy.
Which value of d’ (d-prime) indicates a more sensitive test, a higher value or a lower value?
higher value
What is the advantage of using d’ as a measure of test sensitivity?
d-prime is independent of pass-fail criteria
which is to identify cochlear dead regions?
The TEN noise is designed to mask off-frequency listening