FINAL EXAM Flashcards

1
Q

The degree of cortical impairment depends on __

A

Location of damage
Extent of damage
Type of damage (trauma vs blunt force)
Unilateral vs. bilateral

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2
Q

what swallowing deficits would a lower brainstem stroke exhibit?

A

difficulty triggering the pharyngeal swallow
absent pharyngeal swallow
delayed pharyngeal swallow
reduced laryngeal elevation
reduced UES opening
medulla is affected

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3
Q

What is oral agnosia?

A

inability to visually recognize food or liquid

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4
Q

What oral agnosia may be exhibited in Dementia and Alzheimer’s patients?

A

The patient may not eat the food because they don’t realize it is food
May not know what to do with it or refuse to eat the food
Patient may need a feeding tube to maintain nutrition

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5
Q

why can patients with TBI be difficult to treat for dysphagia?

A

behavioral issues such as impulsive, poor attention and awareness
cognitive issues such as in and out of alertness, making it hard to counsel them and feed them
cannot eat or swallow if not alert

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6
Q

What swallowing deficits are associated with Parkinson’s?

A

oropharyngeal - poor bolus control
random tongue movement
tongue pumping/lingual rolling
delayed swallow initiation
pharyngeal residue
drooling (increases risk of silent aspiration)
incoordination of swallow and respiration
swallow as progressive as disease

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7
Q

Generalized treatments for Parkinson’s

A

Early Parkinsons’: exercises
Moderate Parkinson’s: sensory changes/input
Severe Parkinson’s: counseling on enteral nutrition/maximizing nutrition
quality of life as it’s a progressive, non-curable disease

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8
Q

Neurological disorders causing dysphagia: Dementia

A

causes dysphagia due to structural changes and chemical changes affecting neurological control

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9
Q

Neurological disorders causing dysphagia: TBI

A

external physical trauma to head
causes dysphagia due to changes in posture and muscular tone in muscles needed for swallowing
cognitive and behavior changes

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10
Q

Neurological disorders causing dysphagia: Parkinson’s disease

A

neurodegenerative disorder that occurs due to a disorder basal ganglia and causes slowed movements (bradykinesia) rigidity and tremors
rigidity and bradykinesia impacts swallow initiation time, UES relaxation, and pharyngeal residue

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11
Q

Neurological disorders causing dysphagia: Brain Tumor

A

causes dysphagia due to neurogenic changes, cranial nerve deficits, sensory/motor changes, cognitive changes

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12
Q

Neurological disorders causing dysphagia: ALS

A

progressive neuromuscular degenerative disease

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13
Q

Side effects of radiation; how does it effect swallowing

A

the scatter effect
mucositis - inflammation of mucous membranes
xerostomia - dry mouth in relation to decreased production of saliva
odynophagia - painful swallowing
edema - swelling
dental changes
fibrosis - scarring of tissue; changes into excessive fibrous connective tissue
may cause patient a lot of pain or discomfort when swallowing which leads to feeding tubes

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14
Q

why is dry mouth/xerostomia a problem in head and neck cancer patients?

A

causes patient to swallow less and less, which can lead to lack of nutrition, energy, weight loss, and insertion of feeding tube
this in turn can cause atrophy of pharyngeal muscles
this can also cause psychological and emotional problems as well such as depression

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15
Q

when should swallow treatment begin for a head and neck cancer patient?

A

the sooner therapy is initiated after cancer treatment, the better the outcome.
there is no consensus regarding the optimal time after treatment to begin dysphagia therapy

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16
Q

head and neck cancer before, during and after treatment

A

pre-treatment - establish baseline, protocols, START exercises and counseling
during - monitor, exercises, counseling
post - monitor for change, continue exercises, counseling

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17
Q

Why is it important to keep a head and neck cancer patient swallowing EVEN if they are aspirating?

A

Helps limit this buildup of residue and allows them to continue to exercise the structures to avoid worsening swallow inefficiency and atrophy of pharyngeal muscles

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18
Q

What is trismus?

A

Problems opening mouth, lockjaw
Reduced ability to open the mouth secondary to tonic/tight contraction of the muscle

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19
Q

What is a total laryngectomy-how does this affect swallowing?

A

physical separation of the GI tract from the respiratory tract; removal of the larynx, OR separation of the airway from the esophagus
This affects swallowing because there is no elevation because there is no larynx or hyoid bone

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20
Q

What specific swallow changes would you see in an oral cancer patient?

A

Limit mastication, bolus formation and containment, bolus control, and bolus transport from the front to the back of the mouth.

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21
Q

What specific swallow changes would you see in a laryngeal cancer patient?

A

Reduced laryngeal elevation
Reduced glottal and laryngeal closure
Reduced UES or PES opening
Reduced pharyngeal wall contraction

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22
Q

True/False. SLPs can diagnose esophageal dysphagia

A

FALSE. SLPs cannot diagnose – but they can perform esophageal SCREEN during MBS

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23
Q

Esophageal dysphagia can impact….

A

oropharyngeal function

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24
Q

The esophagus is……

A

Innervated by CN X (Vagus)
Proximal 1/3 - striated muscle; Distal 2/3 - smooth muscle
Hollowed muscular tube; Collapsed at rest; distends when food/liquid/air is swallowed

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25
Q

what is peristalsis?

A

a series of muscular contractions

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26
Q

what are the three different types of peristalsis?

A

primary - rapid movement of cervical esophagus; slow movement of mid/distal esophagus; shortens
secondary - distends
tertiary wave - not peristaltic; disordered

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27
Q

symptoms of esophageal dysphagia

A

Food sticking in throat (globus sensation), coughing during/after meals – Common
Chest pain, shortness of breath, respiratory symptoms, odynophagia – less common
Patient sensation is often inaccurate
Referred sensation – sensation of something stuck in throat, but actually stuck in esophagus 2/2 esophageal innervation by vagus nerve

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28
Q

different kinds of structural disorders of the esophagus

A

esophageal stenosis/stricture
schatzki’s ring
esophageal web
malignancy/obstruction
diverticulum

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29
Q

what is esophageal stenosis/stricture?

A

narrowing of esophagus

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30
Q

schatzki’s ring

A

distal/lower esophagus and usually symmetric
see a slow progression of symptoms

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31
Q

esophageal web

A

upper 1/3; usually asymmetric

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32
Q

malignancy/obstruction

A

usually from esophageal tumors or Barrett’s esophagus (premalignant condition 2/2 severe/chronic GERD)
progresses rapidly - usually advance by time detected

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33
Q

diverticulum

A

pouch/sac branches off esophagus 2/2 bulging from esophageal pressure
regurgitation
zenker’s diverticulum - common and develops at UES
cricopharyngeal bar - failure of muscle to distend (stretch)

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34
Q

motility disorders of esophagus

A
  1. GERD - LES muscle relaxation results in backflow of stomach acid into the esophagus
  2. Laryngopharyngeal Reflux (LPR) - backflow of stomach acid into laryngopharynx
  3. Achalasia - incomplete/absent relaxation of LES and bolus can’t move into stomach; absent peristalsis
  4. Eosinophilic Esophagitis - allergic inflammatory disease
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35
Q

treatments of esophageal disorders

A

myotomy
dilation
fundoplication
botox

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36
Q

supplemental oxygen types for respiratory dysphagia

A

nasal cannula
BiPap
CPAP
non-rebreather

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37
Q

what is the endotracheal tube?

A

oral intubation; long/plastic tube placed through mouth - vocal folds - trachea; cuff on the end can be inflated/deflated; connected to ventilator; patient sedated

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38
Q

What are the risks associated with the Endotracheal tube?

A
  • granuloma
  • hematoma
  • ulcers
  • edema
  • vocal fold paralysis
  • deconditioning
  • delayed swallow response time
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39
Q

what is a tracheostomy tube?

A

a hole created in neck to the trachea
allows patient to wean sedation, initiate mobility, possibility for speaking/swallowing, deflated to allow air to flow to oral cavity

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40
Q

risks of tracheostomy tube

A
  1. decreased taste/smell
  2. increased risk of aspiration
  3. 2/2 deconditioning from medical condition requiring trach placement
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41
Q

what is a cuffed trach and associated risks?

A
  • inflated to maintain air from vent to lungs, prevent aspiration
  • increased risk of silent aspiration if cuff inflated
42
Q

speaking valve

A
  • Allows air in through trach during inhale and closes during exhale; moves air around trach tube through vocal folds and into oral cavity
  • Improves upper airway secretions, ability to cough/clear secretions and improves speech
43
Q

what is COPD?

A

group of progressive lung diseases (emphysema and chronic bronchitis)
chronic obstructive pulmonary disease

44
Q

COPD risks

A

Increased risk of aspiration 2/2 decreased hyolaryngeal excursion, delayed oral/pharyngeal initiation, deconditioning, swallow on inhale cycle, and earlier and longer apneic periods

45
Q

what is iatrogenic dysphagia?

A

difficulty swallowing that is caused by medical treatment or intervention (surgeries)

46
Q

True or false: head and neck surgeries increase risk of dysphagia

47
Q

what surgeries can cause dysphagia?

A

thyroidectomy
carotid endarterectomy (CEA)
cardiovascular surgery
skull base surgery

48
Q

Possible complications of surgeries

A

damage to vagus nerve
unilateral vocal fold paralysis
intubation
deconditioning

49
Q

what are possible complications of cervical spine surgery?

A

prevertebral edema, esophageal injury, vagus nerve injury

50
Q

what is cervical osteophytes and possible complications?

A
  • Bony outgrowth of the cervical vertebrae; narrows pharyngeal space
  • Possible complications: Obstructive dysphagia, most symptomatic at C3 and C6
51
Q

what is an esophagectomy and possible complications?

A
  • removal of esophagus or portion of esophagus
  • Possible complications: vagus nerve injury, changes to esophageal motility, stricture, pharyngeal/esophageal dysphagia
52
Q

what are possible complications of thermal burn trauma?

A

inflammation
intubation/trach
anoxic BI
sedation

53
Q

Postural maneuvers & compensation techniques for dysphagia: head tilt

A

directs bolus to good/strong side

54
Q

Postural maneuvers & compensation techniques for dysphagia: head turn

A

closes weak side

55
Q

Postural maneuvers & compensation techniques for dysphagia: chin up

A

improves anterior-posterior oral transit; use only with good pharyngeal phase/airway closure

56
Q

Postural maneuvers & compensation techniques for dysphagia: chin tuck

A

improves oral containment, increases BOT/PPW contact, decreases laryngeal vestibule diameter

57
Q

Postural maneuvers & compensation techniques for dysphagia: supraglottic swallow

A

improves laryngeal vestibule closure

58
Q

Postural maneuvers & compensation techniques for dysphagia: super-supraglottic swallow

A

tighter laryngeal vestibule closure

59
Q

Postural maneuvers & compensation techniques for dysphagia: effortful swallow

A

increases BOT/PPW pressure, pharyngeal pressure, improves UES opening and hyolaryngeal excursion

60
Q

Postural maneuvers & compensation techniques for dysphagia: repeat swallows

A

can clear pharyngeal residue

61
Q

Postural maneuvers & compensation techniques for dysphagia: mendelsohn maneuver

A

improves hyolaryngeal excursion/UES opening, improved coordination but difficulty to teach

62
Q

associated risks with chin tucks

A

can increase risk of aspiration with delayed initiation, pyriform sinus residue

63
Q

what are compensation techniques that are modifications?

A

Small bites/sips, slow rate, alternate liquids/solids, no straws, etc.
Liquid modifications – thin, nectar/mildly thick, honey thick/moderately thick liquids
Carbonated/sour/cold bolus – immediate effects of timing, but no long-term improvement
Frazier free water protocol – thickened liquids (as indicated) at meals, free water between meals (Strong oral care required)
Diet modifications – national dysphagia diets, IDDSI, clear or full liquids

64
Q

benefits of liquid modifications?

A

slows bolus transit
reduces aspiration; but no strong evidence it decreases pneumonia rates
possible decreased fluid intake

65
Q

exercises principles for swallowing rehabilitation include..

A
  1. gradual progression of intensity
  2. frequency (Number Of Training Sessions Per Unit Of Time)
  3. load
66
Q

head and neck muscular swallowing rehabilitation is specialized for

A

speech > force

67
Q

effortful swallow

A

targets lingual/palatal pressure, lingual strength, oral manipulation, pharyngeal pressure, hyolaryngeal excursion/UES opening, BOT/PPW pressure

68
Q

tongue-hold/masako

A

works on BOT/PPW contact and pharyngeal constriction

69
Q

head-lift/shaker

A

targets laryngeal elevation, hyolaryngeal excursion/UES opening

70
Q

CTAR (chin tuck against resistance)

A

targets laryngeal elevation, hyolaryngeal excursion/UES opening

71
Q

mendelsohn maneuver

A

targets laryngeal elevation

72
Q

EMST (expiratory muscle strength training)

A

targets buccinator strength, BOT/PPW contact, hyolaryngeal excursion, laryngeal vestibule closure, cough strength, breath support

73
Q

MDTP (McNeil Dysphagia therapy program)

A

possibly targets pharyngeal response and hyolaryngeal excursion

74
Q

jaw opening

A

targets laryngeal elevation, UES opening

75
Q

Effortful pitch glide

A

targets laryngeal elevation, hyolaryngeal excursion, pharyngeal contraction/shortening

76
Q

sEMG (device)

A

adjunctive therapy and biofeedback

77
Q

NMES (neuromuscular electrical stimulation)

A

possibly targets pharyngeal response time, pharyngeal transit time, hyolaryngeal excursion

78
Q

IOPI (device)

A

targets lingual/palatal pressures, bolus/pharyngeal transit, BOT/PPW contact

79
Q

swallow strong/tongueometer

A

targets lingual/palatal pressures; biofeedback

80
Q

true or false: there is no evidence to support thermal-tactile stim

81
Q

biofeedback

A

sEMG - visual representation of muscular effort placed during swallow response; patient can visualize effort and increase/decrease with feedback provided

82
Q

what are two swallowing prevention techniques?

A

Pharyngocise – various exercises included to be completed prior to or during XRT for head/neck cancer

Therabite – device used to improve/maintain jaw opening
Targets muscle preservation, trismus, saliva production, taste

83
Q

Head/neck development in normal infant/child development

A
  • oral cavity/jaw smaller
  • oral cavity filled by tongue
  • large/fat buccal pads
  • uvula/epiglottis in contact at rest
  • larynx/hyoid bone higher in neck
  • Eustachian tubes shorter and run horizontal
84
Q

gut development for infant/child

A

anatomic completed by 20 weeks; physiologic function late in gestation

85
Q

lung development for infant/child

A

latest to develop; 28 weeks - surfactant development

86
Q

neurological development for infant/child

A
  • 1st trimester - spinal cord beings to develop
  • 2nd trimester - brainstem matures, breathing/sucking/swallowing being to emerge
  • 3rd trimester - brainstem most highly developed, primitive cerebral cortex
87
Q

fetal development

A
  • 7 weeks lips form
  • 13 weeks swallowing
  • 18 weeks sucking
  • 32 weeks suck/swallow coordination
  • 37 weeks suck/swallow/breathe coordination
88
Q

infant swallowing phases

A

oral prep
oral transit
initiation of pharyngeal swallow
pharyngeal phase
esophageal phase

89
Q

what is the purpose of adaptive reflexes and what are they?

A

direct feeds to gut
rooting, suckling, sucking

90
Q

nutritive vs nonnutritive suck

A

Nonnutritive suck – suck/swallow ratio 6:1 to 8: 1, twice as fast as nutritive suck

Nutritive suck – integrates suck/swallow/breathe; suck/swallow 1:1 ratio
(Immature (3-5 sucks/burst), transitional (5-10 sucks/burst), mature (10-30 sucks/burst))

91
Q

what is the purpose of protective reflexes and what are they?

A

airway protection
tongue protrusion, phasic bite, gag, tongue lateralization, cough, laryngeal chemoreflex

92
Q

infant/child disorders that cause dysphagia

A
  1. Respiratory disorders (newborn apnea, pulmonary hypoplasia, RDS, BPD, laryngomalacia)
  2. Cardiac disorders (tetralogy of fallot, transposition of great arteries, VSD, patent ductus arteriosus)
  3. GI deficits (NEC, Hirschsprung’s disease, esophageal atresia, GERD, EoE)
  4. Neurological disorders (hydrocephalus, TBI, CP, intraventricular hemorrhage, seizures)
  5. Cleft Lip/Palate (lip only, lip and palate, hard and/or soft palate, velopharyngeal insufficiency)
  6. Congenital (Pierre Robin sequence, Moebius syndrome, Down syndrome)
  7. Material Conditions (FAS, NAS)
  8. Prematurity (difficulty with state control, stress, postural control, oral motor control, gut maturity/health, physiological control, respiratory rate, heart rate, endurance, suck/swallow/breathe)
  9. Tongue Tie (tight lingual frenulum, creates heart shaped tongue when protruded, can affect breast feeding (poor latch), treatment with frenulotomy)
  10. Sensory processing (hypersensitivity, hyposensitivity, oral sensitivity)
  11. Autism Spectrum (usually demonstrate oral motor delay, sensory sensitivity or desire for sameness)
  • Any of these deficits can result in nutrition/hydration/energy concerns
93
Q

child treatment for dysphagia

A
  1. Oral sensory – motor therapy; determine if skill deficits, learned behavior or both
  2. Special Feeding equipment – teething toys, gum brushes, food nets, nosey cups
  3. Oral motor toys – chewy tubes, bite blocks, tongue depressors
  4. Specialty spoons – textured, maroon
  5. Positioning – important, feed/back/ trunk/head support
  6. Behavioral Feeding Therapy
94
Q

what is the purpose of behavioral feeding therapy?

A
  • Increase desirable behavior - accepting foods offered, eat acceptable amount, eat variety of foods, quality over quantity
  • Decrease undesirable behavior - refuse foods, verbal/physical protests, withdrawal or refusal
95
Q

infant treatment for dysphagia

A
  1. Side-lying – changes direction of gravity, milk diverted to cheek
  2. Flow rate – slow flow to improve coordination of suck/swallow/breathe, improves ability to control flow
  3. External Pacing – tip bottle downward to allow fluid into bottle, but leave nipple in baby’s mouth
  4. Thickened liquids – slows flows, decreases regurgitation, use caution with commercial thickeners
  5. Chin/Cheek support – improves seal on nipple; used as a cue not a crutch
  6. Quality over Quantity – support positive experiences; negative experiences can create stress and eventually learn to avoid eating or develop bad patterns
96
Q

what is cue based feeding?

A

involves following the infants cues to drive feeding

97
Q

what does quality vs quantity mean in infant feeding?

A

priority is feeding performance over amount taken in

98
Q

what are the signs of stress in infant feeding?

A
  • change in state/alertness
  • change in tone/postural control
  • raised/furrowed brows
  • pull head back
  • turn head away
  • hand/arm extension
  • finger splay
  • gulping, gurgling, milk spilling out of mouth
  • coughing/choking, gagging
  • Change in cardio-respiratory behavior
99
Q

what are the sings of disengagement in infant feeding?

A
  • pushing nipple out
  • no active rooting/sucking,
  • unable to re-alert
  • use of weak suck
100
Q

what is involved in a child/infant evaluation for dysphagia?

A

Swaddling – important to regulate state, improves coordination; midline flexion – arms/legs to chest
Treatment strategies are used during evaluation to compensate for deficits if noted

Caregiver focus
If dysfunction or risk of aspiration present, pursue MBS or FEES