final Flashcards
components of peripheral vascular assessment
Colour, temperature, capillary refill, edema, pulses
components of a peripheral neurological assessment
Pain, sensation, motor function
Purpose of neurovascular assessment
Early detection of impaired blood flow or damaged nerves
Essential in preventing permanent deficits, loss of a limb & even death
What are the 6 p’s of compartment syndrome?
Pain
Pallour
Paralysis
Pulselessness
Paresthesia
Pressure
What is osteoporosis
A chronic, metabolic bone disease wherein there low bone mass, and structural deterioration of bone tissue occur
Predisposes patients to increased risk of fracture at hip, wrist and spine
What may cause osteoporosis?
Hereditary
Nutrition
Exercise
Hormones
How does Osteoporosis manifest?
Back pain
Loss of height
Spinal deformation
Pathophysiology osteoporosis
Bone resorption exceeds bone deposition
Which gender is at greater risk for developing osteoporosis & why?
Women
- (1) women tend to have lower calcium intake;
- (2) women have less bone mass because of their generally smaller frame;
- (3) bone resorption begins at an earlier age in women and is accelerated at menopause;
- (4) pregnancy and breastfeeding deplete a woman’s skeletal reserve unless calcium intake is adequate; and
- (5) longevity increases the likelihood of osteoporosis, and women live longer than men
Diagnostics osteoporosis
History & physical exam, serum calcium, phosphorous, and alkaline phosphate levels, bone mineral density, dual-energy X-ray absorptiometry (DEXA), quantitative ultrasound, radiology only detect when 25-40% lost, FRAX = 10yr risk hip fracture & 10yr risk for major osteoporosis fracture
Medical Management osteoporosis
Estrogen replacement therapy, bisphosphonates, selective estrogen receptor modulator, teriparatide, salmon calcitonin
Nursing Managementosteoporosis
Proper nutrition (high diet with calcium), calcium supplementation, vitamin D supplements, exercise, prevention of fractures, medications, education about smoking
What is a fracture?
A fracture is a disruption of normal bone continuity occurring following an elevated level of stress being placed on bone tissue beyond that which it may otherwise manage
What are some examples of bone fractures?
Closed fracture
Open fracture
Transverse fracture
Spiral fracture
Greenstick fracture
Comminuted fracture
Oblique fracture
Pathologic fracture
Stress fracture
What is an open fracture?
broken bone that penetrates the skin
Complete or incomplete fracture
Complete = break completely through bone
Incomplete = fracture occurs partly across bone shaft but bone still in 1 piece
Displaced or non-displaced fracture
displaced: ends of the bone have come out of alignment
non-displaced: bone typically stays aligned in an acceptable position for healing
What are the clinical manifestations of bone fractures?
Deformity
Edema, swelling
Muscle spasm
Pain, tenderness
Ecchymosis
Loss of normal function
Inability to bear weight on or use affected part, guards & protects extremity against movement
What is fracture reduction?
restoration of the fracture fragments to anatomic alignment and positioning
What is a closed reduction?
-manual realignment of the bones
-no surgery
-cast/splint
What is an open reduction
Fracture realigned with surgery with various internal devices
Medical management: Immobilization
External (cast, splints) or internal fixations
Casting or splinting, external fixation, internal fixation, prophylactic antibiotic therapy, surgical debridement & irrigation, tetanus & diphtheria immunization
Nursing Management fractures
Neurovascular status, elevate limb, ice therapy, increase vitamin D, education
Neurovascular assessment
Drug therapy
Nutritional therapy
Preoperative and postoperative
Cast care
Ambulation and assistive devices
What is a cast?
temporary immobilization device
What is external fixation?
Devices consist of pins that are placed through the skin into the fractured bone and connected to external frame
What is internal fixation?
Devices, pins and screws surgically inserted at time of realignment
Delayed union:
healing progresses more slowly than expected but eventually occurs
Nonunion:
fails to heal properly despite treatment
Malunion:
heals in expected time but in unsatisfactory position, possibly resulting in deformity or dysfunction
Angulation:
heals in abnormal position in relation to midline of structure (type of malunion)
Pseudoarthosis:
type of nonunion occurring at fracture site in which a false joint is formed with abnormal movement
Refracture
new fracture occurs at original site
Myositis ossificans:
deposition of calcium in muscle tissue at site of significant blunt muscle trauma or repeated muscle injury
Care of patients in a cast
Neurovascular assessment (bit of a running theme)
Pain assessment
Cast care teaching
Assessment for complications (compartment syndrome, infection)
What is an intracapsular fracture in the hip?
A fracture within the joint capsule
What is an extracapsular fracture in the hip?
A fracture not within the joint capsule
Clinical manifestations of hip fractures
- External rotation
- Muscle spasm
- Shortening of affected leg
- Severe pain and tenderness
Medical management of hip fractures
- medical (traction or casting); not ideal
- surgical (open reduction (move bones back into place & internal fixation (screws, pins), pre-op); common
Preoperative management of fractures
Preoperative check list
Consent
Pain management (muscle relaxation, analgesia)
Patient education
(Exercise for unaffected leg and both arms, weight bearing status)
Postoperative management of fractures
Vital signs
Pain management
Neurovascular assessment (it’s a pattern)
Position / mobility
Maybe put in fall protections (hip protectors)
What is Osteoarthritis?
A chronic, progressive process where new tissue is formed in response to cartilage destruction
Noninflammatory disorder of the synovial joints
Osteoarthritis clinical manifestations
Worsening joint pain
Limitation of movement
Crepitus (bone rattling)
Stiffness
Deformity
causes osteoarthritis
Known event or condition that directly damages cartilage or causes joint instability
Decrease estrogen, obesity, inflammation
patho osteoarthritis
Cartilage damage that triggers a metabolic response at the level of the chondrocytes, causing the articular cartilage to become dull, yellow, and granular causing it to become less functional
diagnostics for osteoarthritis
History & physical exam, radiological studies of involved joints (CT scans, MRI, bone scan), synovial fluid analysis
Medical Management osteoarthritis
Acetaminophen, nonsteroidal anti-inflammatory drugs, antibiotics, intra-articular hyaluronic acid, opioids, reconstructive joint surgery, rest, joint protection
Nursing Management osteoarthritis
Nutrition & weight management, rest & use of assistive devices, therapeutic exercise, heat & cold application, herb therapy (glucosamine), yoga
Arthroplasty
reconstruction or replacement of a joint
What is Bipolar I disorder?
Characterized by one or more manic episodes generally with a major depressive occurrence.
Manic behaviour must last at least 1 week.
The mood disturbance is severe enough to cause marked impairment
Not otherwise explained by different drugs / conditions
What is Bipolar II disorder?
At least 1 episode of hypomania
One or more depressive episodes
Never had a manic episode
Usually begins with a depressive episode
What is hypomania?
Symptoms are the same as mania but last only 4 days
No impairment in social or occupational functioning
Hypomanic episode is a distinctive change in functioning not otherwise seen when asymptomatic
If psychosis is present, it’s mania
Bipolar impact on health
Medications used to treat disorder have some important side effects
Bipolar behaviour may include dangerous activities such as reckless driving, unsafe sex
bipolar impact on economic decisions & well being
High risk behaviour during mania may cause reckless spending, gambling
Friends and family may feel guilt, grief or worry
Family roles change during periods of illness
schizoaffective
Meets diagnostic criteria for schizophrenia (need 2 of these) = Delusions, Hallucinations, Disorganized speech, Grossly disorganized or catatonic behaviour, Negative symptoms
mood component = bipolar type (mania & depression), depressive type
Delusions or hallucinations for 2 or more weeks in the absence of a major mood disturbance
Long interrupted time of illness
What is mania?
a mood disorder marked by a hyperactive, wildly optimistic state with 3 of:
- Inflated self-esteem - grandiosity
- Decreased need for sleep
- Talkativeness
- Flight of ideas
- Distractibility
- purposeless non goal-directed activity
MSE focus with mania:
Appearance / Behaviour
Mood affect
Speech
Thought form / content
Perceptual Disturbances
What types of medications are used for mania?
Mood stabilizers
Antipsychotics
Benzodiazepines
Lithium toxicity: Early
Polyuria
Ataxia
Blurred vision
Diarrhea
extreme thirst
Lithium toxicity: Severe
Confusion/disorientation, memory impairment, seizures, nystagmus
Priorities of care for bipolar med adherence
May initially require IMs
Monitor for side effects
Concerns:
Sedation, CNS depression, substance use, EPS, lithium toxicity, anticholinergic effects, blood work, weight gain (important), dependence, tolerance, benzo & other drugs, cardiovascular effects (prolonged QT waves), agranulocytosis, endocrine effects (hyperprolactinemia = producing breast milk) thyroid problems, dermatological effects = acne, steven johnson syndrome = rash, changes in urination, diabetics, osteoporosis
priorities of care safety bipolar
(not just about suicide, homicide; need to safe from others, financial, sexual, vulnerability, social media, risky behaviour)
Redirection / distraction
Limit setting
Timeouts
Quiet/low stimulus environment
Private room
Seclusion room (last resort)
complicaitons after fracture infection
Assess = Early detection, warmth, redness, pus, swelling, increase in pain
Treat = Nutrition, rest, antibiotics, wound care, pain management
Prevent = Sterile techniques, prophylactic antibiotics, standard precautions
complicaitons after fracture compartment syndrome
Prevent = Early detection, assessing, neurovascular checks
Assess = Neurovascular, 7 Ps
Treat = Fasiostomy, open cast, loosing dressing, bivalving
complicaitons after fracture fat embolism syndrome (FES)
manifest as acute respiratory distress
Prevent = Careful mobilization
Assess = ARDS (acute respiratory distress syndrome)
Treat = Supportive treatment for presenting symptoms
complications of fractures venous thromboembolism
Prevent = Compression stockings/devices, enoxaparin/heparin, mobilize, range of motion exercises
Assess = Redness, swelling, pain, parasthesias, DDIMER, WBC
Treat = Aimed at preventing further blood clots
Cast care teaching
Don’t = Get wet, Remove padding or insert objects, Bear weight x 48hrs, Cover cast with plastic for prolonged periods
Do = Report signs of possible problem, Apply ice over fracture site to reduce edema, Relieve itchiness: blowdryer on cool setting, elevate it, tapping, Move joints above & below regularly, Elevate higher than heart for 24-48hr
What is Electroconvulsive therapy (ECT)
a treatment that involves inducing a brief seizure by delivering an electrical shock to the brain
A safe and effective treatment for a variety of psych conditions
ECT indications for use
Acute suicidality
Psychotic features
History of poor response to medications
History of good response to ECT
Risk of standard antidepressants treatment outweighing risk of ECT
Mania
Schizophrenia
ECT contraindications
Unstable/severe cardiovascular conditions
Aneurysm or vascular malformation
Increased intracranial pressure
Recent cerebral infarction
Pulmonary conditions (COPD, asthma, pneumonia)
Pace maker
Psychotropic medications during ECT
Usually you continue them. Special attention must be paid to mood stabilizers and Benzos due to the anti-seizure effects of these medications
Frequency of treatment ECT
2-3 ECT treatments/week (Mon, Wed, Fri)
6-12 treatments
above is used early in treatment course when a rapid response is important (i.e. mania, catatonia, high suicide risk)
continuation phase: 1st 6 months
especially vulnerable time for re-emergence of symptoms
day before ECT
Assess pt’s physical and mental status
Begin ECT/pre-op checklist
Encourage & assist patient with personal hygiene if needed
Encourage expression of concerns & anxiety related to ECT
Maintain NPO from midnight
Weight them for anaesthia dosing
Patient Preparation
Be NPO,
Remove jewelry, hair accessories, contact lens, glasses, hearing aids, glasses, dentures
Pre-ECT medications (if ordered) and most routine medications 1 hour prior with sips of water
Void
Pre ECT Vital Signs
Pre ECT
Assess the education required
Implement education
Document education provided
Ensure chart forms are on pt’s chart including (Consent form/form 5, Checklists, Record of anesthesia, Record of ECT)
ECT morning
Complete ECT pre checklist
Confirm NPO has been maintained
Encourage pt to void immediately before transport
Assess anxiety
Provide reassurance & support
Post ECT
Assess pt’s physical & mental status (DOCUMENT)
VS within 5 mins of their return to unit
Assess frequency of observation required based on assessments and LOC
Assess safety of environment and pt’s ability to ambulate & swallow to take held morning medications & breakfast
Assess & document above & any side effects
Side effects include = Nausea, headache, muscle pain (acetaminophen), Acute confusion (resolves on own & orientate)
What is anaesthesia?
A state wherein there was a loss of sensation by depressing the CNS or PNS
Anaesthetic agents
Can decrease the level of consciousness, stimulate muscle relaxation and or cause a loss of response to stimuli
Types of Anaesthesia
Local and general anaesthesia
Local anaesthesia
occurs when sensation is lost to a limited part of the body without loss of consciousness
Loss of pain to specific area, no effect on respiratory function
General Anaesthesia
Causes loss of sensation to the entire body, usually resulting in loss of consciousness
Paralysis of respiratory function a worry
Local anaesthesia (LA) route types
Peripheral:
Skin (topical)
Nerve block
Infiltration (direct injection such as dental freeze)
Central:
Spinal or intraspinal anaesthesia- injected into the area near the spinal cord within the vertebral column; intrathecal and epidural
LA - Mechanism of action
Interfere with nerve transmission in specific areas of the body by blocking both the generation & conduction of nerve impulses by blocking the movement of sodium, potassium, calcium ions
Known as membrane stabilizing as decreases nerve cell permeability
LA s/e
Adverse effects are uncommon, and allergy is rare (may occur preservatives)
Resulting symptoms may be restlessness, anxiety, hypotension or dysrhythmias
Signs of CNS toxicity are excitement leading to irritability & confusion
examples of LA
Lidocaine most commonly used
Side effects =
Inadvertent IV injection occurs
Excessive dose or rate of injection is given
Slow metabolic breakdown
Inject into highly vascular tissue
general anesthesia (GA)
The goal of a GA is to provide a rapid and complete loss of sensation.
Signs of a GA include:
*Total analgesia
*Loss of consciousness
*Loss of memory
*Loss of body movement (muscle relaxation and reflex reduction)
Procedural Sedation
Is general anaesthesia achieved by a single drug?
Not frequently, no.
Multiple are usually used to rapidly induce unconsciousness, relax muscles and maintain anaesthesia
Balanced anaesthesia is safer as less anaesthetic is used
Does ECT require a balanced approach?
YES
How is GA usually administered
IV or by inhalation
IV meds such as Propofol are given as they act within a few seconds
Inhalation meds such as halothane and Nitrous oxide are used to sustain anaestheisa
General Anaesthetic mechanism of action
Exact mechanisms unknown
Varies according to agent used
Lipid solubility of anaesthetic agents determines its potency
Nerve cell membranes & blood brain barrier have a high lipid content, allowing for anaesthetic agents to concentrate in cell membranes
What is thiopental sodium?
barb = It induces anaesthesia in like 30 seconds and lasts 10-30 minutes
Can be used alone for procedures less than 15mins, ECT meets this specification often involving anaesthesia for about 10 mins
What are the most commonly used GA agents?
Propofol is the most common though Etomidate and Methohexital are also used
What other agents may be used in adjunct to major GA agents? (Not common)
Benzos (Though they are NOT used for ECT due to anti-seizure effects)
Opioids (fentanyl)
Ketamine (The choice of little green gremlins everywhere. Also more often used as an animal tranquilizer)
What is succinylcholine?
depolarizing neuromusclar junction blocker
What does succinylcholine do?
Depolazing: occupiers acetylcholine receptors creating muscle paralysis
Often used to reduce the amount of anaesthetic used
What are some succinylcholine indications?
During ECT to prevent damage to musculo-skeletal system
The jaw is the only muscle not totally relaxed, thus indicating the need for bite-blockers
Mechanical ventilation will ALSO need to be used.
Side effects of SUCCylcholine
Respiratory depression
*Hypotension
*Tachycardia
*Urinary retention
*Dysrhythmias
*Delirium - hallucinations, confusion, excitability.
Malignant hyperthermia (rare)
What is malignant hyperthermia?
*Occurs during or after succinylcholine
*Sudden elevation in body temperature (40°C)
*Muscle rigidity or unexplained aches
*Dark brown urine
*Bleeding
*This is a life threatening emergency
Additional medications used with ECT
Atropine
Ranitidine
Metoclopramide
Tylenol
What is Atropine?
*An anticholinergic
*Indicated during ECT for clients who have excessive secretions.
*Inhibits the action of acetylcholine at postganglionic sites located in the secretory glands
*Low doses decrease sweating, salivation and respiratory secretion.
Why do we use Atropine?
It’s an antidote for NMBA (Neuromuscular blocking agent) that we use in SMALL amountsto prevent bradycardia, hypotension, and secretions
What is Ranitidine?
*An antihistamine -histamine H2 antagonist
*Indicated during ECT for clients at risk for heartburn, acid reflux (gastric hypersecretory states).
*Inhibits the action of histamine at the H2 receptor site in the gastric parietal cells, leading to decreased gastric secretions.
What is metoclopramide?
*An antiemetic
*Indicated during ECT for clients who are at risk for or experience postoperative nausea and vomiting.
*Stimulates motility of the upper GI tract and accelerates gastric emptying, by blocking dopamine receptors.
metoclopramide side effects
Drowsiness, extrapyramidal reactions, restlessness, neuroleptic malignant syndrome
nursing process anathesia
Assess past history of surgeries & response to anaesthesia
Assess allergies to medications
Pre-op (Medical history, full physical exam, lab tests, substance use)
Post-op (Type of anaesthesia used, monitor LOC, airway, respiratory depression, cardiovascular depression)
Implement safety measures during recovery
Reorient client to surroundings
Provide preoperative teaching as necessary
Osteoarthritis
Cartilage begins to get destroyed between the joints
Chronic progressive process where new tissue is formed in response to cartilage destruction
Non inflammatory disorder of synovial joints (as progresses may be inflammation due to the bone on bone not the actual pathophysiology)
Diagnostics osteoarthritis
Bone scan, Xray, MRI, CT
Goal: confirm disease and stage progression of joint damage and slow it down
clinical manifestations osteoarthritis
Worsening joint pain
Limitation of movement
Crepitus
Stiffness
Deformity
medical management osteoarthritis
Rest and joint protection
Heat and cold therapy
Nutritional therapy & exercise
Complementary & alternative therapies
Drug therapy (pain management = nonopioids (acetaminophen)
osteoarthritis risk
Genetic links
Menopause
Increased weight
Strenuous exercise
Occupational
surgical management osteoarthritis
management
Hip = total hip arthroplasty
Knee = total knee arthroplasty
Goal = restore joint motion by replacing arthritic bone
What is anxiety?
A normal response to stress
Consists of three parts:
Psychological arousal (fight, flight response)
Cognitive Process
Coping strategies
Anxiety becomes abnormal when it impacts life negatively / causes interference in life
Causes of anxiety disorders
Genetics
Increases by 5 if someone on your family has
Temperament
Persons nature (low stress tolerance)
Neurotransmitters
Low serotonin levels = anxiety???
Norepinephrine = physical symptoms of anxiety (GI upset, tachycardia)
GABA
Life experiences
Trauma, financal, relationship, physical health concerns
Anxiety levels
mild, moderate, severe, panic
What is generalized anxiety disorder?
Characterized by persistent and excessive anxiety and worry about occupational/social/interpersonal situations
Affects daily functioning
Has physical symptoms
GAD can be debilitating disorder with a serious negative impact on quality of life
Often associated with other disorders