Fall 2020 Final Exam Flashcards
Chapman: Chapman’s reflexes are a classic modality of diagnosis and treatment in osteopathic medicine. They are palpable as “gangliform” contractions, and thought to be due to hypercongestion of local lymphatics.
Each specific palpatory change relates to a specific disease or condition (anatomic “map”). All are bilateral except for which of the following?
a. stomach (acidity)
b. liver
c. spleen
d. pancreas
All of the above
- Stomach
- -acidity (L)
- -peristalsis (L) - Liver and gallbladder (R)
- Spllen (L)
- Pancreas (R)
Chapman: Chapman’s reflexes are a classic modality of diagnosis and treatment in osteopathic medicine. They are palpable as “gangliform” contractions, and thought to be due to hypercongestion of local lymphatics.
Each specific palpatory change relates to a specific disease or condition (anatomic “map”). All are bilateral except for which of the following?
a. stomach (acidity)
b. liver
c. spleen
d. pancreas
All of the above
- Stomach
- -acidity (L)
- -peristalsis (L) - Liver and gallbladder (R)
- Spleen (L)
- Pancreas (R)
- Appendix (R)
Chapman: True/False - Chapman’s points were developed in the 1900’s by Frank Chapman during a time when lab testing and imaging was not reliable, and non-toxic medications were not readily avaialble. A book based on these points (Endocrine Interpretation of Chapman’s Reflexes) was published in the 1930’s after his death, and was the first to describe the inter-related neuroendocrine immunity systems.
True
Chapman: Chapman’s reflexes are distinct, hyper-congested areas of the lymphoid tissues in the fascia. Each point is associated with a specific organ, each has an anterior and posterior reflex, and they all can can present with soreness/tenderness in acute stages.
What are theoretical causes of Chapman’s reflexes?
a. Lymphatic system
b. ANS
c. Myofascial
d. Endocrine system
All of the above
- Lymphatics **
- ANS: segmentation
- Myofascial: histopathology studies of biopsies – no tissue changes
- Endocrine: less studied
Chapman: The etiology of Chapman’s reflexes is based on irritation, disease or stress of an organ. This leads to increased ________, which then leads to lymphatic stasis and boggy, ropy, shotty and thickened myofascial nodules
inc. sympathetic tone
* lymph vessels innervated by SNS fibers (facilitation from irritation leads to constricted lymph vessels/stasis and accumulation of pro-inflammatory substances)
Chapman: True/False - Palpatory tissue changes are most likely from ANS input and lymphatic congestion. Changes occur most often or are palpable in the intercostal and spinal areas, but may occur in the extremities.
True
Chapman: Chapman’s reflexes may be acute or chronic (based on duration of the condition). This aspect of chronicity can be appreciated by its tactile properties.
A patient presents with tender, non-radiating, tapioca (or BB) type points. They are smooth and circumscribed, but firm (dense/not hard). The are discretely palpable, and fixed in one place (may move slightly). The points are located in the deep aponeurosis or fascia.
This describes acute or chronic?
Answer: Acute
Chronic:
- less tender
- less discrete, somewhat confluent
- Generalized inc. tension
- -rubbery nodule, stringy, ropy, firm
Chapman: True/False - In chronic or severe cases, coalescent mats of “string of pearls” may be felt (especially with points on the lower extremities)
True
Chapman: How do Chapman’s points differ from other myofascial points?
a. Tenderness is present, but not the sole criteria
b. location, lymphatic congestion and myofascial tissue changes are important criteria
c. they are not counterstrain or trigger points
d. they are counterstrain and trigger points
A-C
Chapman: A positive Chapman’s reflex occurs when both the anterior and posterior points are present. They may be in the deep fascia, or periosteum (not only subcu tissue).
Anterior reflexes follow intercostal sympathetic nerves. They are located in the intercostal spaces near the sternum. These reflexes are more discrete than the posterior ones. What are their uses/characteristics?
- Used for Dx
- 1st in Tx sequence
- After Tx posterior, use anterior to assess efficacy
(if no change, it becomes a diagnostic point – pathology is too great, or another factor influencing it)
Chapman: True/False - Posterior reflexes are located between the spinous processes and transverse processes of adjacent vertebrae. They are less discrete than the anterior and tend to present with a more rubbery feel. They often resolve with treatment of the anterior reflex.
True
Chapman: Classic uses for Chapman’s reflexes include:
- Diagnosis of organ issues or conditions
- Treatment to influence lymphatics
- Treatment to influence visceral function (via nervous system)
What are the modern uses of Chapman’s reflexes?
Constructing differential diagnoses
NOTE: Never make a Dx based solely on a non-tender Chapman’s reflex (Wilson Rule)
NOTE 2: Never ignore or trivialize a tender Chapman’s reflex (Wilson Rule)
Chapman: True/False - Literature has demonstrated that hospitalized patients with pneumonia have a high predictability of presenting with Chapman reflex points classified for lungs. Furthermore, Chapman reflex point examination was proposed to be useful in evaluating patients with a potential diagnosis of pneumonia. This relationship was statistically significant.
True
Chapman: Treatment for Chapman reflexes should begin with anterior reflexes. Light massaging in the rotary direction (clockwise or counterclockwise) should be applied for 20-60 seconds until the lymphatic conegestion diminishes or a change in myofascial tissue is detected.
What should be treated next?
posterior reflexes
*then re-check anterior side and retreat if still tender and congested
NOTE: do not use excessive pressure – be gentle
Chapman: The appendix is the exception to ID’ing Chapman points. Where is the Appendix located?
Anterior: Tip of 12th rib on the R
Posterior: T10-11 spinous process/transverese process (R)
Chapman: To perform an ENT screen, the middle ear, sinuses, tonsils and larynx reflex points should be assessed. Where is the point for the sinuses?
Superior aspect of the 2nd rib
posterior: C2 transverse process
Chapman: To perform a pulmonary screen, the larynx, bronchus, upper lung and lower lungs should be assessed. What is the point for the lower lung?
anterior: 4th Intercostal space
posterior: T4 - midway between SP and TP
Chapman: To perform a cardiovascular screen, the myocardium, bronchus, esophagus, and thyroid should be assessed. What is the point for the myocardium?
Anterior: Between ribs 2 and 3 at sternocostal jx (R)
Posterior: T2/T3
Chapman: To perform an upper GI screen on the left side, the pylorus, stomach (acidity and peristalsis), spleen and SI should be assessed.
On the right, the pancreas and appendix should also be palpated. What are the points for the pancreas and appendix?
Pancreas:
- -anterior: Ribs 7 and 8 at costochondral jxn (R)
- -posterior: T7-T8 (R)
Chapman: For a lower GI screen, the Ascending colon, Transverse colon, and Descending colon should be assessed. What are the CHapman’s points of each?
- Ascending: Proximal Right IT band
- Transverse colon: Distal Right and Left IT band
- Descending colon: Proximal left IT band
Posterior: Triangle including L2, L3, L4 across to tip of the crest of the innominate (posterior points)
Chapman: What is the Chapman point for the rectum?
anterior: lesser trochanter
posteiror: lower edge of iliosacral joint
Chapman: A urinary screen involves the following:
- Adrenals (R and L)
- Kidney (R and L)
- Urethra (midline)
- Urinary bladder (midline)
- Ureter (R and L)
A genital screen involves:
- Ovary (R and L)
- Uterus (R and L)
- Broad ligament (F); Prostate (M) - R and L
What are the points for the Adrenals, Kidneys, and Ovaries?
- Adrenal:
- -anterior: 1” over, 2” above umbilicus
- -posterior: T11, T12 - Kidney:
- -anterior: 1” over, 1” up
- -posterior: T12, L1 - Ovary
- -Anterior pubic bone
- -posterior: T9-T11
Chapman: Chapman’s reflexes can aid in differential diagnosis in conjunction with other findings (correlate with established diagnosis). Treatment of these points may ease the course of the disease process as well as reduce related pain.
True/False - It is important to document as viscerosomatic reflex.
True
Lower resp: A patient presents with breathlessness, wheezing, and coughing. He complains of increased sputum production and decreased tolerance to exercise. He has been a smoker for over 40 years. You suspect
COPD
- mucus accumulation/dec. cilia fxn
- destruction of alveoli; hyperinflation
- accessory muscle use
- inflammation (proteinases/ox stress)
Lower Resp: Major systemic co-morbidities of COPD include:
- Exercise intolerance
- Remodeling of the musculoskeletal system
- Anxiety from Dyspnea
- Systemic inflammation
_____ is associated with de-conditioning, isolation, depression, and decreased muscle mass from lack of exercise
Exercise intolerance
Lower Resp: Major systemic co-morbidities of COPD include:
- Exercise intolerance
- Remodeling of the musculoskeletal system
- Anxiety from Dyspnea
- Systemic inflammation
Osteoporosis may be seen in COPD pateients due to inc. steroid use and ____
remodeling of the musculoskeletal system
Lower Resp: Major systemic co-morbidities of COPD include:
- Exercise intolerance
- Remodeling of the musculoskeletal system
- Anxiety from Dyspnea
- Systemic inflammation
True/False - Systemic inflammation results from a “spill over” of multiple mediators, cytokines, inflammatory cells (from lung), and is associated with inc. oxidative stress. It is also associated with inc. susceptibility to infections, cardiovascular disease, and diabetes.
True
*loss of lean body mass – inc. metabolism (cachexia - work hard)
Lower Resp: Musculoskeletal features of COPD patients include expansion and rigidity of the rib cage. The diaphragm flattens with less mobility, reducing efficiency of breathing due to loss of passive recoil.
Patients tend to assume the “tripod” position which features increased kyphosis, forward head posture, and overuse of accessory muscles. What are common TART findings?
Varied
*C3-5 (phrenic nerve) effects on diaphragm function
NOTE: Tx diaphragm and neck
Lower resp: OMM may be utilized to improve respiratory symptoms in COPD patients. The following may be treated to improve respiration in COPD patients. List examples of each
a. Parasympathetics (CN X)
b. Sympathetics (T1-6)
c. Musculoskeletal
d. Lymphatics
- Parasympathetics (CN X)
- -duboccipital decompression - Sympathetics (T1-6)
- -soft tissue (paraspinals)
- -rib raising - Musculoskeletal
- -doming diaphragm
- -pectoral traction - Lymphatics
- -thoracic inlet and lymphatic pump
Lower resp: Neurologic considerations for Tx of COPD focus on viscerosomatic reflexes to help balance the SNS and PNS. These reflexes intersect with biomechanical considerations.
Omm can help to normalize the autonomics. List the Reflexes associated with the lungs
T1-6 (SNS)
OA, AA, C2 (Vagus)
Chapman’s (Upper lung: 3rd ICS, T3; Lower lung: 4th ICS, T4)
Lower resp: True/False - High vagal tone (PNS) can lead to bronchoconstriction and inc. watery secretions into the airway. Facilitation of the vagus may occur through OA, AA and C2.
Conversely, Sympathetic firing my increase production of thick, tenacious secretions and lead to bronchodilation (T1-6).
True
Lower Resp: True/False - There is a significant association between pneumonia and the presence of chapman’s reflex points for the lung.
True
Lower resp: Biomechanical considerations of COPD should include assessing the limitations of the musculoskeletal system as well as its effects on the neurological system. For example, stimulation of intercostal muscle spindles out of phase with breathing may induce breathlessness.
Interventions that improve _____ and ___ mobility, and muscle legnth in the upper quadrant may improve pulmonary function
neck and thoracic mobility
- rib cage
- diaphragm (dec. work of breathing/inc. intrathoracic pressure)
- accessory muscles (scalenes, pec minor)
- thoracic kyphosis (dec. thoracic spine mobility)
Lower respiratory: Venous and Lymphatic flow or lack thereof may have an impact on COPD via
- Inc. inflammation
- Inc. immune response
- Edema and congestion
Treatment of COPD should include considerations on how to improve flow as well as increase ease of respiratory excursion (biomechanical). This can be done by OMM (adjunctive) which may aid in decreasing inflammation and infections while also improving the efficiency of breathing.
True
Other Tx: --smoking cessation --medications (steroids - fluticasone; LABA - relax SM/formoteral; Anticholinergic muscarinic antagonist -bronchodilation tiotropium) --provide O2 as needed
Lower respiratory: Metabolic/Energy Considerations -Patients with COPD are at increased risk for developing diabetes and other inflammatory conditions. They may also suffer from fatigue (multi-factorial) and malnutrition. It is important to increase protein intake in COPD patients, as they tend to lose SK muscle mass from chronic pulmonary disease.
True/False - Tx with OMM and pulmonary rehab may improve biomechanical efficiency, resp. mechanics, muscle stregnth, exercise tolerance and posture.
True
Lower respiratory - Behavioral - COPD patients tend to have increased risk of developing depression and/or anxiety.
Pulmonary rehabilitation, O2 treatment and self-management programs may help, along with exercise regiments to improve motivation. What are other considerations regarding behavior management?
- Smoking cessation
- Environmental (allergens/pollutants)
- Medication compliance/inhaler education
- Hydration
Lower respiratory: The sensation of breathlessness is closely related to the sensation of respiratory effort.
It increases when the load on respiratory muscles ______, or when the capacity of respiratory muscles _____, or both.
- increase
2. decrease
Myofascial: Which of the following is a function of fascia?
a. binds, protects, envelopes and separates tissues
b. connects structures that permits transmission of forces
c. possesses qualities of stregnth, elasticity and energy storage
d. provides protective enzymes and Igs
A-C
Myofascial: List the following in order from dense irregular to dense regular
- aponeurosis
- proper fascia
- ligaments
- tendons
- proper
- aponeurosis
- ligaments
- tendons
Myofascial: One of the functions of myofascial tissue is to bind, protect, envelope and separate tissues. Which of the following types of fascia play a role in this?
a. superficial fascia
b. intramuscular fascia
c. aponeurosis
d. tendons
A-C
*deep fascia, aponeurosis, intramuscular, superficial
Myofascial: Fascia plays arole in connecting structures permitting the transmission of forces. What types of fascia are involved in this?
a. ligaments
b. tendons
c. aponeurosis
d. deep fascia
A and B
Myofascial: Fascia also plays a role in providing stregnth, elasticity and energy storage. Which types of fascia play a role in this?
a. ligaments
b. tendons
c. superficial fascia
d. aponeurosis
all of the above
–all types of fascia (superficial, intramuscular, deep, apo, ligaments, tendons)
Myofascial: Fascia is regulated by multiple receptors including:
- Golgi tendon organs
- Pacini mechanoreceptors
- Ruffini mechanireceptors
- Interstitial (Type 3 and 4) mechnoreceptors
What are the functions of the above receptors or what do they detect?
- Golgi tendon organs
- -active streching movements - Pacini mechanoreceptors
- -changes in pressure and vibrations - Ruffini mechanireceptors
- -changes in pressure (rapid/sustained) - Interstitial (Type 3 and 4) mechnoreceptors
- -sensory
Myofascial: Fascia forms a continuous tensional network throughout the human body, covering and connecting every single organ, every muscle, and every nerve and muscle fiber. There are several properties/studies of fascia including:
- Viscoelasticity
- Creep
- Wolff’s law
- Hysteresis
The viscoelastic nature of fascia describes it as being both elastic and shapable. ___1___ enables it to maintain deformed shapes (permanent change in intermolecular bonds), while ___2__ enables fascia to return to its original shape after being loaded (recoil).
- Viscosity
- Elasticity
*fascia exhibits combined properties
Myofascial: Fascia forms a continuous tensional network throughout the human body, covering and connecting every single organ, every muscle, and every nerve and muscle fiber. There are several properties/studies of fascia including:
- Viscoelasticity
- Creep
- Wolff’s law
- Hysteresis
____ describes continuous deformation under constant load. The tissue lengthens when it is subjected to load over a prolonged period of time. Continued deformation can lead to dec. ability to resist furture loads.
Creep
- The duration of the load is more important than the amount of force.
- contributing factor in repetitive strain injuries
Myofascial: Fascia forms a continuous tensional network throughout the human body, covering and connecting every single organ, every muscle, and every nerve and muscle fiber. There are several properties/studies of fascia including:
- Viscoelasticity
- Creep
- Wolff’s law
- Hysteresis
______ describes mechanical stressor affecting tissue differentiation and growth characteristics. Examples of dysfunction include increased stress or load that contribute to remodelling (e.g. heel spur – tight plantar fascia). Therapy can involve external forces that reduce stress and restore structure, or improving structure to restore normal functional motion.
Wolffs law
Myofascial: Fascia forms a continuous tensional network throughout the human body, covering and connecting every single organ, every muscle, and every nerve and muscle fiber. There are several properties/studies of fascia including:
- Viscoelasticity
- Creep
- Wolff’s law
- Hysteresis
_______ is the capacity of the fascia to change length (stretch) in a therapeutic way when loaded and unloaded. Energy (heat) is normally released in the process and it does not return via the same pathway when unloading.
Hysteresis
**myofascial release (utilize this property of fascia – tissue feels warmer after Tx)
Myofascial: Myofascial release (MFR) is a system of diagnosis and Tx which engages continual palpatory feedback to achieve release of myofascial tissues.
- ______ involves loading the dysfunctional myofascial tissues and engaging them into the restrictive barrier with constant force.
- ____ involves loading and guiding the dysfunctional tissue into the position of ease
- direct
- indrect MFR
*light - moderate pressure with complimentary positioning
Myofascial: Fascia is capable of changing length and altering muscle tone and neural facilitation. Thus, application of external forces via MFR can help restore the normal structure and function as well as promote self-healing.
What are the steps of MFR
- Gentle compression – engage tissue/underlying fascia
- Monitor motion in all planes
- -inferior vs. superiro
- -medial vs. lat.
- -clockwise vs. counter - determine ease-bind (direct or indirect)
- hold until release is palpated
Myofascial: Relative contraindications of MRF include exercise caution in the case of
a. fractures
b. open wounds
c. acute thermal injury
d. aortic aneurysm
all of the above
- bony/soft tissue infections
- DVT
- anticoagulation
- disseminated/focal neoplasm
- recent post-operative sites
Compensatory pattern: Dr. Gordon Zink suggested that there’s a recurring compensatory pattern that occurs in patient’s with somatic dysfunction. There are two main types:
- Common compensatory
- Uncommon compensatory
How do these differ?
- COmmon compensatory
- -80% people
- -L/R/L/R - Uncommon
- -20% of people
- -R/L/R/L
Compensatory pattern: True/False - Uncompensated patterns are non-alternating, disparate patterns that are seen in patients who are acutely ill, who fail to respond to ordinary treatment approaches, or who have a history of trauma or chornic illnesses.
True
Compensatory pattern: The presence of transition zones (anatomic weak areas) in the body are believed to be influential in the development of compensated patterns. These zones are:
- Occipitoatlantal (OA)
- Cervicothoracic (CT)
- Thoracolumbar (TL)
- Lumbosacral (LS)
What are the junctions and diaphragms associated with these transition zones?
- Occipitoatlantal (OA)
- -craniocervical jxn
- -tentorium cerebelli - Cervicothoracic (CT)
- -cervicothoracic jxn
- -thoracic inlet/outlet - Thoracolumbar (TL)
- -thoracolumbar jxn
- -rep. diaphragm - Lumbosacral (LS)
- -lumbosacral jxn
- -pelvic diaphragm
The diaphragm descends on inhalation, and ascend on exhalation.
Compensatory: The respiratory circulatory model focuses on improvement of O2 and waste removal. Treatment goals include removing restriction to lymphatic flow, enhancing the mechanism for respiratory/circulatory homeostasis, and mobilizing lymphatic fluid.
The following is the sequence for treating a patient in respiratory distress with compensatory pattern. Give examples of treatments.
- Thoracic inlet
- Maximize diaphragmatic motions
- Inc. pressure differential/fluid flow
- Mobilize congested tissue
- Thoracic inlet
- -direct MFR thoracic inlet - Maximize diaphragmatic motions
- -Direct MFR diaphragm, OAD, L/S decompression - Inc. pressure differential/fluid flow
- -Rib raising - Mobilize congested tissue
- -thoracic/lymphatic pump
lymph > venous > arterial (most resistant)
lower respiratory: True/False - OMT has been shown to improve well being, performance of daily activities and 6MWT in COPD patients.
However, the only thing that has been shown to reduce decline in FEV1 is smoking cessation
True
*majority of patients believed they could breathe better after receiving osteopathic manipulation
Lower resp: A study was conducted examining the effects of OMT combined with pulmonary rehab vs. pulmonary rehab alone. What was found?
both improved breathing
Lower respiratory: Use of OMT in a patient with a lung infection is meant to encourage immune system function (respiratory/circulatory model).
Examples of OMT include thoracic inlet release, rib raising, thoracic lymphatic pump, pectoral traction, and diaphragmatic myofascial release.
What are techniques that are appropriate for improving the neurological and biomechanical models?
- Neurologic
- -OA decompression, Cervical ME (target phrenic nerve), rib raising - Biomechanical
- -Scalene MFR, pectoral traction, rib raising, rib walking, diaphragm MFR
Lower respiratory: A 75 y/o female with a 2 year history of Cryptogenic organizing pneumonia presents to the OMM clinic for “sciatica”. She had 6 weeks of R buttock pain that radiated down the back of her thigh. She still has shortness of breath but does not need supplemental O2.
Exam showed no neurologic changes or suspicion for a herniated disc. She had R. piriformis and psoas TPs. These did not resolve even with various technique approaches. What should you try next?
- -double check Dx
- -Check the other side for dysfunction/Tx it
- -Check for something keeping the system hung up
- -systemic/metabolic problems? e.g. uncontrolled diabetes
Lower respiratory: CABG requires cutting open the sternum and spreading the rib cage apart, which causes stress on the rib cage and associated soft tissue.
True/False - Do not touch the surgical site 2nd/3rd day following surgery. You can target the biomechanical and respiratory/circulatory system to assist with healing after surgery. It may also be helpful for residual musculoskeletal chest pain months after surgery.
True
*OMT thoracic inlet MFR, rib raising w/ paraspinal muscle stretch to L2, soft tissue cervica, OA
GI: An 18 year old male presents to the OMM clinic for a follow up regarding low back pain. He complains of midi back pain in addition to his low back pain. He reports heartburn over the past few months. He states his diet consists of coffee and tacos mainly. Patient denies weight loss or bloody/dark stools.
You suspect?
GERD
symptoms: worse lying down or after acidic foods
* dec. peristalsis
* can cause chronic cough/hoarseness
* atypical chest pain, SOB
GI: GERD is associated with poor tone and development of the lower esophageal sphincter. It is due to transient relaxation of the LES and can be affected by:
- Foods (coffee, chocolate, etOH, mints)
- Drugs (B-agonist, Ca2+ blocker, Nitrates, anti-Ach, nicotine)
- Hormones (progesterone, glucagon, VIP)
There may also be an increased pressure gradient between the stomach and esophagus from obesity, pregnancy or gastroparesis.
- inc. acid production (vagal and stomach distension)
* dec. mucosal defense from NSAIDS
GI: The standard medical approach to Tx GERD includes:
- Lifestyle modifications (diet, weight loss, raise head)
- Meds (antacids, H2 blockers or PPIs)
- Screening procedures (EGD’s, Barrett’s)
- Refractory – consider surgery (fundoplication)
What are OMT approaches?
- ANS
a. SNS: T5-T9
b. PNS: Vagus (AO, AA, C2) - Diaphragm and attachments to lower thoracics, upper lumbars, and lower 6 ribs (**T12)
- Tx viscerosomatic/Chapmans
acidity: T5
peristalsis: T6 - Linea alba
- -celiac ganglion
*especially useful when other approaches haven’t helped
GI: What are the risks of Tx with PPI’s?
a. inc. risk fractures (post-menopausal women)
b. Inc. C diff infections (dec. acidity in infants – alters gut flora)
c. inc. vitamin/mineral deficiencies (Mg, B12)
d. Inc. comunity acquired pneumonia (short term)
all of the above
*drug interaction with clopidogrel
GI: GERD is a disease of the foregut. The foregut is composed of the esophagus, stomach, duodenum (to ampulla of vater), liver, pancreas, gallbladder and biliary tract).
What is the vacular supply of the foregut? Innervation?
- Vascular
- -Celiac artery - SNS
- -T5-9
- -celiac ganglion - PNS
- -vagus nerve
NOTE: superior mesenteric T10-11; inferior mesenetric T12-L2)
GI: What are SNS effects on the GI system?
a. decreased gut motility, constipation and distension
b. contraction of rectal sphincter
c. vasodilation
d. dec. mucosal defenses of the gut
A, B, D
*vasoconstriction
GI: What are PNS effects on the GI system?
a. relaxation of sphincters
b. Inc. gut motility and peristalsis
c. vasodilation
d. inc. secretions
all of the above
GI: What are the effects of the ANS on the lower esophageal sphincter?
- SNS
- -celiac ganglion, T5-T9
- -contraction of LES - PNS
- -Vagus
- -relax LES