Exercise Prescription for CR & PR (Part 2) Flashcards

1
Q

What is HIIT training?

A
  • Alternating period of intensive exercise (>90% VO2 max) with periods of passive or mod/mild intensity (<40% VO2 max)
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2
Q

What are the benefits of HIIT training in cardiac populations?

A
  • Maintain high intensity for longer
  • Greater training stimulus
  • Increases peak O2 uptake
  • Changes in ventricular & endothelial function
  • Improves QOL
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3
Q

What did Guiraud et al 2012 find regarding HIIT training in CR?

A
  • Stable CAD: Safe, well tolerated, increases exercise length
  • HF: Safe, well tolerated, improved adherence & motivation
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4
Q

What did Currie et al 2013 find regarding HIIT training in CR following recent CAD event?

A
  • Compared HIIT to MICE
  • Both improved brachial artery flow-mediated dilation & peak VO2
  • No difference between interventions
  • HIIT required less time (common barrier)
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5
Q

What did Rognmo et al 2012 find regarding HIIT training in CR?

A
  • Risk of CV event low after HIIT and MICE in CR setting

- HIIT should be considered more in patients with CHD

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6
Q

What did Moholdt et al 2014 find regarding HIIT training in CR?

A

Exercise intensity is an important determinant of improving VO2 peak in patients with CHD (> intensity = > benefits)

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7
Q

What did Aamot et al 2012 find regarding HIIT training in CR?

A
  • Exercise mode is not essential for improving exercise capacity
  • Home-based may be an option for low-mod risk patients with CAD
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8
Q

What did Koufaki et al 2012 find regarding HIIT training in CR?

A

HIIT is feasible & well-tolerated but not more effective than MICE

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9
Q

What are the key features of HIIT established by the research?

A
  • Max exercise test performed pre-program
  • Optimal medical treatment &/or undergo re-vascularisation (stable)
  • Supervised program with staff trained to handle medical emergencies
  • Improves patient adherence & motivation
  • Intensity matter
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10
Q

What factors contribute to ventilatory limitation of exercise in COPD patients?

A
  • Dynamic hyperinflation & airway obstruction
  • Max expiratory flow rate reached during exercise
  • Causes further gas trapping
  • Increased FRC
  • Inspiratory capacity reaches TLC
  • Inability to increase TV
  • Increased RR & dead space ventilation
  • Alveolar ventilation & impaired gas exchange
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11
Q

What are the causes of dyspnoea during exercise in COPD patients?

A
  • Dynamic hyperinflation causes restriction of TV
  • Altered respiratory mechanics: Increased loading & length-tension properties of respiratory muscles causes fatigue
  • Impaired gas exchange (increased PaCO2, decreased PaO2)
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12
Q

What happens when COPD patients reduce their activity levels due to dyspnoea?

A
  • Peripheral muscle deconditioning
  • Decreased oxidative capacity
  • Early onset lactic acidosis during exercise
  • Increased CO2 production & ventilator drive
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13
Q

What are the causes of CV limitations to exercise in COPD patients?

A
  • Increased pulmonary vascular resistance
  • Decreased right ventricular stroke volume
  • Can lead to right-sided heart failure
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14
Q

What are the causes of skeletal muscle limitations to exercise in COPD patients?

A
  • Decreased oxidative capacity
  • Decreased CSA & strength
  • Altered muscle composition
  • Myopathy due to hypoxia, inflammatory mediators
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15
Q

What other mechanisms can limit exercise capacity in COPD patients?

A
  • Nutritional impairments

- Psychological factors

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