Exam 4 Peds Flashcards

(68 cards)

1
Q

What will be the s/s of the CSF with bacterial meniginitis?

A

cloudy
decreased glucose
increased WBC
increased protein

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2
Q

What are the precautions for bacterial meningitis?

A

droplet

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3
Q

What are S/S of growth hormone deficiency or somatotropin insufficiency?

A

Short stature: proportional height & weigh
Teeth crowding
Delayed epiphyseal closure
Delayed sexual maturation
Delayed onset of pubic, facial, axillary hair, genital growth & high-pitched voice

Labe: decreased GH
GH stimulation test (*NPO 12hr prior)
Bone scan
CT scan of head

Treatment
Somatropin (growth hormone) SQ or IM injections 6-7 days/wk
Continue therapy until x-rays show epiphyseal closure

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4
Q

What are 2 pathos of DI (diabetes insipidus). What can cause these pathologies? What is a complication of DI?

A

Central (low/absent ADH from pituitary)
Nephrogenic (lack of response to ADH)

Damage to pituitary
Meningitis
Tumors pressing on pituitary

Chronic kidney disease

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5
Q

What are S/S of DI? Treatment? Nursing interventions?

A

polydipsia
polyuria, high urine output
dehydration
hypovolemia
hypernatremia
high osmolality, low specific gravity

remove tumor
ADH replacement: desmopressin or vasopressin

Monitor I&O
med considerations/education
med bracelet

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6
Q

What are the 2 pathos of SIADH? What are S/S of SIADH? Risk?

A

High ADH
water intoxication

oliguria
hyponatremia (think diluted)
low serum osmolality
weight gain

seizures

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7
Q

What should your mind think with anything dealing with the thyroid? What are 3 types of thyroid disorders?

A

think metabolism!!
Thyroid regulates metabolism

3 Types of thyroid disorders
Congenital Hypothyroidism
Acquired Hypothyroidism (autoimmune)
Hyperthyroidism

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8
Q

What can be the causes of congenital hypothyroidism? S/S in an infant? Diagnostic tools? Treatment?

A
  • absent or nonfunctioning thyroid gland in a newborn
  • Mom was treated for hyperthyroidism
  • Mom with low iodine level or autoimmune disorder

Excessive sleepiness
Poor feeding
Large tongue
Cold, dry, & peeling extremities
Jaundice

Newborn metabolic screen
Labs: ↓ T3 & T4, ↑ TSH & lipid levels
X-ray: Delayed bone growth
Ultrasound

Synthetic thyroid hormone: levothyroxine
Vit D supplements

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9
Q

What can cause aquired hypothyroidism? S/S? Diagnostic tests? Treatment?

A

Body’s immune system makes antibodies that damage thyroid
Trauma
Surgery/Radiation
Medications

Slowed metabolism
Decreased HR, RR, BP
Cold intolerance
Goiter
Impaired growth

Labs: ↓ T4, Normal T3, ↑ TSH
Presence of antithyroid antibodies (autoimmune)
Radioactive iodine uptake test (RAIU) for carcinomas

Synthetic thyroid hormone
Levothyroxine or Synthroid

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10
Q

What are nursing considerations for hypothyroidism?

A

Monitor vital signs
Monitor respiratory status
Monitor weight
Assess for feeding difficulties
Administer medication as prescribed
Monitor for s/s

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11
Q

What is the cause of hyperthyroidism? What are S/S of hypertyroidism? Treatment?

A

genetic or autoimmune reaction causing over-secretion of thyroid hormone

high metabolic rate, weight loss
irritability
fever
rapid pulse, high BP
vomiting, diarrhea
weight loss
heat and cold intolerance
goiter

Administer supplemental vitamin D to support rapid bone growth.
Monitor thyroid levels (T3, T4 and TSH)

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12
Q

What is treatment for a hyperthyroid? What is a thyroid storm (complication of hyperthyroid)

A

1st-β-Adrenergic blocking agent (propranolol)
Anti-thyroid drug (PTU: “puts thyroid under”)
Surgery

thyroid overactivity: temp/BP/HR extremes

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13
Q

What is the primary cause of hyperparathyroidism? Secondary? Symptoms?

A

parathyroid tumor (adenoma)

kidney disease?

Hypercalcemia
Hypophosphatemia
Kidney stones
Decalcification of bones

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14
Q

What is the primary cause of hypoparathyroidism? What two minerals can cause a dysfunction in the parathyroid (Think 2 that always affect one another)? S/S? Treatment?

A

Destruction of parathyroids often the result of total thyroidectomy

too little calcium
too much phosphorus

Hypocalcemia
Paresthesia
Tetany
Chvostek sign (Twitching of facial muscles when tapping cheek)

Calcium supplementation

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15
Q

What is the hallmark of an adrenal insufficieny? What is a common adrenal insufficiency disease? S/S? Nursing considerations?

A

Under secretion of cortisol and aldosterone production leads to adrenal insufficiency

Addison’s

Extremely low BP
Ashen gray appearance
Weak pulse
Elevating temp, dehydration, hypoglycemia
hyponatremia
high potassium level
possibly seizures
sudden death

watch for S/S of hypovolemic shock
Strict intake & output
Medical alert bracelet

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16
Q

What is hypercortisolism? Primary causes? Is it common in infants? S/S? Treatment? What is the risk of an adrenalectomy?

A

Cortisol-producing tumors
benign adrenal tumors
adrenal hyperplasia
Chronic steroid use

No, rare

Moon-faced
Large bellies, thin extremities in contrast to trunk
Increased risk of infection
HTN
Hyperpigmented cheeks
Purple striae
Polyuria
Growth cessation

Signs & Symptoms
Fat accumulates on the cheeks, chin, &trunk
Moon-faced
Thin extremities in contrast to trunk
Increased risk of infection
HTN
Hyperpigmented cheeks
Purple striae
Polyuria
Growth cessation

Radiation/surgery

would go hypo and have Addison’s and now need steroids

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17
Q

What is the nature of DM 1? What causes it? What are BG levels for both hypoglycemia and hyperglycemia?

A

Autoimmune destruction & immunologic damage of beta cells causing absolute or deficiency of insulin

Idiopathic

Hypoglycemia: BG <60mg/dl
Hyperglycemia: BG >250mg/dl

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18
Q

What are S/S for DM1 in children? What are 4 labs used to confirm diagnoses?

A

SOnset abrupt and present to ED in DKA
Young children: enuresis
3 P’s: Polyuria, Polydipsia, & Polyphagia

Diagnosis & Labs
Casual BG >200mg/dl
Fasting (8 hrs) BG >126mg/dl
GTT BG> 200mg/dl
HbA1C: >7-8%

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19
Q

What are the 5 sick day rules for DM1?

A

Monitor blood glucose more frequently when sick (every 3 hours)

Test urine for ketones every 3 hours

Do NOT skip insulin or anti-diabetic meds when sick

Stay hydrated: drink 2-3 L of water (sugar-free, non-caffeinated liquids per day)

Notify doctor for blood glucose >240 mg/dl, fever >38C, urine positive for ketones, confusion, or rapid breathing (Kussmaul)

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20
Q

What are treatments for DM1?

A

Insulin administration
Regulation of nutrition (Count carbs when prepping meals)
Exercise
Stress management
Blood glucose & urine ketone monitoring

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21
Q

What is BG for hypoglycemia? S/S

A

Hypoglycemia
BG <60mg/dl

Hunger, irritability, shakiness, headache, tachycardia, decreased LOC, slurred speech, seizure/coma

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22
Q

What is BG for hyperglycemia? S/S?

A

Hyperglycemia
BG >250mg/dl

3 P’s, warm/dry skin, fruity breath, N/V, weakness, lethargy

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23
Q

What is treatment for conscious patients for hypoglycemia? What if they are unconscious?

A

Consume 10-15g simple carbohydrate (ex: 4 oz juice or soft drink, 8 oz of milk)
Recheck blood glucose frequently
Follow with a complex carbohydrate

Administer IM or subcutaneous glucagon
Repeat in 10 minutes if patient is still not conscious
Once patient is conscious (and can swallow safely), have patient consume a simple carbohydrate

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24
Q

What is some patient teaching for self-monitoring BG and administering insulin?

A

Do NOT puncture the pads of the fingers (use the skin to either side of the finger pad for better blood flow and less pain)

Perform before meals and at bedtime

Rotate subcutaneous injection sites to prevent lipohypertrophy.

Administer 4-5 injections in one site before switching to another site.

Inject at 90 degree angle (45 degree angle if thin)

Mixing insulins: Draw up clear (shorter-acting insulin) before cloudy (longer-acting insulin)

Never mix long-acting insulin (i.e. insulin glargine) with other insulins

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25
What are risk factors for DKA? S/S?
Infection Stress or illness Untreated/undiagnosed T1DM Missed insulin dose Weight loss Fruity breath odor Kussmaul breathing GI upset Dehydration Confusion
26
How is DKA confirmed? Medical treatment?
Diagnostics & Labs BG >330mg/dl Ketones in blood & urine Metabolic acidosis Hyperkalemia Treat underlying cause (infection) Administer IV fluids Administer IV insulin Check BG hourly Monitor K+ levels Administer Bicarb for metabolic acidosis
27
What are the risk factors for DM2? Medical management?
obesity inactivity HTN high triglycerides nutrition exercise anti-glycemic agent such as metformin or glyburide
28
What is PKU and it's nature? S/S? Doagnosed? Treatment?
phenylketonuria Inherited metabolic disorder: Autosomal recessive trait Newborn lacks enzyme to metabolize phenylalanine (amino acid) Builds up in bloodstream & tissues Damages brain leading to cognitive delays Early dietary interventions needed Irritability Microcephaly Vomiting Growth failure Severe cognitive impairment Seizures Newborn metabolic screen Low phenylalanine diet Avoid protein-containing foods Milk, eggs, cheese, nuts, meats, fish Infants: low phenylalanine formula
29
What is the structure of bone?
Diaphysis: shaft of the bone (primary center for ossification) Epiphysis: end of long bones Metaphysis: wide portion of long bone (meeting point of diaphysis and epiphysis) Periosteum: outer layer covering the bone
30
What sare traction types? Nursing considerations?
Skin traction (Buck and Bryant) Skeletal traction Halo traction Nursing Considerations: Don’t manipulate the weights! Maintain alignment Pain control (pharmacologic/nonpharmacologic) Neurovascular checks! Skin integrity Consider elimination needs Incentive spirometry Traction-specific considerations (Halo wrench)
31
What is compartment syndrome? Where is it commonly located? Cause? How can it be identified early on?
compression on nerves, blood vessels and muscle resulting in ischemia and tissue necrosis forearm and tibial dressing constrictions, burns, surgery, hemorrhage neurovascular checks!!
32
What are the 5 Ps for neurovascular checks?
pain paresthesia pulselessness paralysis pallor
33
What is polydactyly? Syndactyly? Treatment and nursing considerations for both?
Supernumerary digit to hand and/or foot Treatment options surgical repair/ “tying off” Nursing Considerations -parent bonding Fusion/webbing of fingers and/or toes Treatment options -surgical repair Nursing considerations -post-surgical -parent bonding -social stigma
34
What is craniosynostosis? Why is it a problem?
Fused suture lines nowhere for the brain to grow
35
What is the nature of pectus excavatum? Treatment?
“Funnel Chest” Surgical for breathing difficulties
36
What is torticollis? Risk factors? Assessment findings? Treatment?
“Twisted neck” Delivery complication Congenital or acquired Assessment findings Visible appearance Treatment and nursing care Muscle stretches Parent education PT
37
What is plagiocephaly? Prevention? Contributing factors? Treatment?
Cranial molding in infancy position changes and helmet care Contributing factor: torticollis Treatment: Physical therapy and specialized skull-molding helmets
38
What is osteogenesis imperfecta? Treatment?
Genetic abnormality of connective tissue- “brittle bone disease” Treatment Pamidronate (medication to help increase bone density)
39
What is aseptic necrosis of the femoral head? Findings? Treatment?
Legg-Calve-Perthes Disease Limp, stiffness, limited ROM, pain Varies based on severity Pain control, PT, traction, surgery
40
What is developmental dysplasia (DDH) of the hip? Cause? Findings?
Abnormal development of the hip Acetabular dysplasia: Subluxation or Dislocation When does this occur? Birth order Family history Delivery considerations Asymmetry Limited ROM Positive Ortolani test (noticeable "clunk" Positive Barlow test (palpable dislocation) Trendelenburg sign in older children
41
What are treatment options for DDH?
Dependent on age and severity Pavlick Harness (newborn-6 months) Bryant traction (adduction contracture) Hip spica cast Surgical correction (6 months-2 years)
42
What is talipes? Nature? Findings? Treatment?
clubfoot Deformity to ankle and foot Unilateral or bilateral Positional, syndromic, or congenital Deformity dependent Treatment options Splinting/Casting
43
What is scoliosis? Nature? Findings? Treatment?
Deformity of spine Lateral curvature and rotation of spine with rib asymmetry Asymmetry Routine screening (beginning at 8 years and annually) Bracing Surgical correction (spinal fusion and rod placement)
44
What is the nursing care when a lumbar puncture is performed?
before: education, topical anesthetic, consider the age of child during: C-shape, pain control, 3 tubes collected (C&S, glucose level, RBC presence) after: spinal headache, lay flat
45
What is the nursing care for a ventricular tap?
during: supine, firm hold, concern of hemorrhage after: pressur dressing, keep calm, semi-fowler's
46
What is the natures of a shunt with neurologic disorders? 2 kinds? Nursing care? Complications? What are temporary options until shunt placement?
Surgically placed catheter to shunt extra CSF away from brain Ventriculoperitoneal (VP) shunt (most common) Ventriculoarterial (VA) shunt Surgical considerations for shunt placement Shunts require revisions! Family education on shunt complications (irritability, lethargy, vomiting) Infection prevention (infected shunt can lead to meningitis) Reservoirs External drains Ventricular tap
47
What drug classes are commonly used to treat neurological disorders and their indications?
Osmotic diuretics (mannitol) Indication: decrease cerebral swelling Corticosteroids (dexamethasone) Indication: decrease cerebral swelling Antiepileptics (gabapentin, phenytoin, clonazepam) Indication: decrease incidence and severity of seizures Antispasmodics and skeletal muscle relaxants (Botulin toxin, baclofen) Indication: decrease muscle spasms and severe spasticity
48
What are causes of increased ICP? Findings? In infants? In children? What are 2 types of posturing?
↑CSF, blood in CSF, cerebral edema, head trauma, infection, tumors, hydrocephalus, Guillain-Barre Syndrome Can occur with multiple neurologic disorders Headache/irritability/restlessness VS change (↑ Temp/BP, ↓HR/RR) Pupillary changes (diplopia, dilation, Doll’s eye reflex) Infant: bulging fontanel, increased OFC, irritability, restlessness, high-pitched/shrill cry, poor feeding, sunset eyes Children: nausea, headache, vomiting, blurred vision, decline in school performance, seizures Posturing Decorticate (arms adducted/flexed on chest, legs extended/internally rotated) *cerebral cortex dysfunction Decerebrate (rigid extension/adduction of arms, wrists pronated, legs extended) *midbrain dysfunction
49
What is hydrocephalus? Cause? Findings? Testing?
Abnormal accumulation of CSF in the ventricles or subarachnoid space typically due to an obstruction or interference with absorption Congenital (at birth) or acquired (incident) In-utero detection Bulging fontanel, widened suture lines Bossing forehead, sunset eyes OFC measurement ICP assessment (↑ Temp/BP, ↓HR/RR) Ultrasound, CT, MRI Transillumination (highlights fluid)
50
What is treatment for hydrocephalus? Complications? Nursing care?
Treatment based on cause Diuretics (if caused by overproduction of CSF) Shunt placement (if obstruction is the cause) Complications Increased ICP leading to brain tissue damage motor/mental deterioration Education Pre/post-op considerations for shunt placement Shunt education and care Psychosocial considerations
51
What is the nature of cerebral palsey? 4 types?
A group of nonprogressive disorders of upper motor neuron impairment that result in motor dysfunction. Caused by abnormal brain development or damage to the developing brain that leads to cell destruction of the motor tracts. Four main types: Spastic/Pyramidal (most common- stiff/tight muscles) Dyskinetic/Extrapyramidal (non-spastic, involuntary muscle movement) Ataxic (non-spastic, shaky movements- affects balance) Mixed (combination damage-severe impairment)
52
What is assessed with cerebral palsey?
Full health history and evaluation of prenatal/perinatal/postnatal risk factors (anything contributing to anoxia to brain) Sensory alterations (strabismus, refractive disorders, visual perception/field defects, speech impairment) Vision, speech, or hearing impairments Neurologic findings (toe walking, arching, posturing, hyperreflexia, seizures) Attention deficit disorder, Autism Seizure activity Cerebral asymmetry
53
What is the nature of spina bifida? Cause? What is the benign spina bifida? How is it identified? What are the more serious form of spina bifida and the 3 types?
Neural tube defect characterized by lack of fusion of posterior surface of the embryo Due to maternal malnutrition, medications, insufficient folic acid, and/or family Hx Spina bifida occulta Internal defect common to lumbosacral area (no spinal cord involvement) Dimpling and/or hair tuft to sacral area (follow-up necessary) Spina bifida cystica (protruding sac) Meningocele Spinal fluid and meninge involvement Myelomeningocele (most common) Spinal fluid, meninge, and nerve involvement Encephalocele (cranial meningocele
54
What are assessment findings with spina bifida? What are complications of surgical closure of the sac? Nursing considerations?
Dependent upon lesion involvement Muscle flaccidity Lack of bowel control Rectal prolapse Scoliosis/kyphosis Skin integrity Increased ICP Bowel/bladder control Orthopedic considerations (protect the sac and keep moist with NS dressing, prone positioning, OFC measurements Post-op considerations (ICP monitoring, CSF leakage prone positioning infection prevention ROM exercises Intermittent bladder catheterization (inpatient and possibility of home)
55
What is the nature of meningitis? Presentation in infants? Children? What will be found in CSF with a confirmed diagnosis? Medications Tx? Precaution measures?
Often result of a URI Intense HA (sharp pain upon head movement) Infant: poor suck, weak cry Older child: arched back, neck hyperextension CSF results: ↑WBC/protein, glucose comparison to blood glucose (bacterial meningitis will have cloudy CSF) Medications/Tx Antibiotic therapy (primary therapy) Intrathecal (CSF) injections Corticosteroids (dexamethasone)- for bacterial meningitis Diuretics Analgesics Isolation precautions: droplet
56
What can cause encephalitis? S/S? Considerations?
Occurring from protozoan, bacterial, fungal, or viral invasions (enterovirus, mosquito-borne illness, measles, mumps, varicella) Headache, fever, ataxia (muscle loss), lethargy, comatose, + Brudzinksi or Kernig sign Childhood vaccination necessity Long-term considerations from neurologic damage
57
What is GBS? Naute? Cause? Symptoms? Tx?
Group Beta Strep (GBS) Infection Major cause of meningitis in newborns Cause: in-utero/delivery contact Symptoms: respiratory distress/pneumonia  meningitis (lethargy, fever, bulging fontanel) Treatment: antibiotics
58
What is the most common cause of botulism? S/S? Tx?
Cause: contaminated food (commonly honey) Symptoms are progressive: hypotonia  weak gag  paralysis Treatment: Botulinum immune globulin therapy, supportive
59
What are types of seizures based on age and causes? Newborn? Infant/toddler? 3+ years?
Newborn seizures (head twitching, lip smacking, cyanosis and/or apnea) Cause: trauma, anoxia at birth, hypoglycemia, infection Infantile spasms Generalized seizure typically from trauma, metabolic disease, or infection Poisoning/Drugs Febrile (SUDDEN spike in temp; 102F to 104) Tonic-Clonic Absence
60
What is the nature of a toni-clonic seizure? Clonic? Absence?
Tonic-clonic (grand mal) No warning Most prevalent Tonic phase (eyes roll upward, contraction of whole body, increase salivation, apnea) Clonic phase (violent jerking, mouth foaming, incontinence) Postictal state (semiconscious, vomiting/headache, lethargic, no recollection of seizure) Absence (petite mal) Loss of consciousness, blank stare, minimal behavior change, momentary confusion, automatisms (lip smacking, eyelid twitching)
61
What are VS changes in ICP?
temperature and blood pressure increase, and pulse and respiratory rate decrease.
62
What constitutes a neurologic assessment?
mental status motor function cranial nerves reflexes sensory function
63
What will analysis of CSF indicate when there is menigitis and why?
increased white blood cell and protein levels increased ICP glucose level less than 60% of blood glucose (because bacteria have fed on the glucose)
64
What is an example of traction used for femur in infant less than 2 years?
Bryant traction, used for fractured femurs in children younger than 2 years, is an example of skin traction
65
What is an example of traction used for lower extremities in older children?
Buck extension is an example of skin traction used for immobilizing lower extremity fractures in older children.
66
What is treatment for juvenile idiopathic arthritis?
Because JA is a long-term illness, therapy includes a balanced program of exercise, rest, and medication administration to relieve pain, restore function, and maintain joint mobility
67
What is the Gower sign?
rise from the floor only by rolling onto their stomachs and then pushing themselves to their knees. To stand, they press their hands against their ankles, knees, and thighs (they “walk up their front”), called a Gower sign.
68
What meds are used for hyperthyroidism? What are Considerations
Propranolol PTU PTU can cause immunosuppression so look for leukopenia and thrombocytopenia