Exam 4 - Peds Flashcards
what is the blood shift with increased pulmonary blood flow?
blood shifts L to R
from high to low pressure
with increased pulmonary blood flow, what would you see manifest in the lungs?
pulmonary edema
what are the 3 types of increased pulmonary blood flow?
- ventricular septal defect
- atrial septal defect
- patent ductus arteriosis
type of heart defect:
hole between R + L atria
atrial septal defect
type of heart defect:
hole between R + L ventricle
ventricular septal defect
ventricular septal defect murmur is best heart where?
L sternal border
which heart defects have a loud + harsh murmur?
atrial septal defect + ventricular septal defect
type of heart defect:
duct between pulmonary artery + aorta doesn’t close @ birth
patent ductus arteriosis
which heart defect murmur sounds like a machine hum?
patent ductus arteriosis
what medication can be given to treat patent ductus arteriosis?
indomethacin (constricts the duct)
this class of defect is defined as blood exiting heart meets narrowed area
obstruction to blood flow
with obstruction to blood flow defects, pressure is increased or decreased BEFORE the obstruction?
increased
with obstruction to blood flow defects, pressure is increased or decreased AFTER the obstruction?
decreased
obstruction to blood flow defects lead to what? (r/t cardiac fxn)
decreased CO
usual tx for obstruction to blood flow defects
cardiac cath - balloon angioplasty
type of heart defect:
narrowing of aortic valve (harder to get blood out to systemic circulation)
aortic stenosis
type of heart defect:
narrowing of pulmonary valve/artery
pulmonary stenosis
s+s of aortic stenosis (6)
think of patho…
- HF
- faint pulses
- hypotension
- tachycardia
- poor feeding (infants)
- exercise intolerance + dizziness (kiddos)
all from not getting enough blood/O2 to system
pulmonary stenosis causes an obstruction of blood flow from the R ventricle into the pulmonary system, which can lead to what happening with the R ventricle?
cardiomegaly - b/c of increased work of heart
pulmonary stenosis can cause which respiratory S+S?
cyanosis w/activity
type of heart defect:
narrowing of lumen of aorta
Coarctation of the aorta
s+s of coarctation of the aorta (5)
- nosebleed
- HF
- elevated BP/pulse in UE
- decreased/cool skin in LE
- leg cramps w/activity (not getting enough blood)
what is the blood shift with decreased pulmonary blood flow?
R to L shift
deoxygenated blood into systemic circulation b/c not reaching lungs
what are the 3 types of obstruction to blood flow defects?
- aortic stenosis
- pulmonary stenosis
- coarctation of the aorta
what are the 2 types of defects that fall under decreased pulmonary blood flow?
- tetralogy of fallot
- tricuspid atresia
2 T’s
tet spells are associated with which class of heart defects?
decreased pulmonary blood flow
tetralogy of fallot has 4 defects. what are they?
- Ventricular septal defect
- Overriding aorta
- Pulmonary stenosis
- Ventricular hypertrophy (from the pulmonary stenosis)
with tetralogy of fallot, tet spells often occur when for infants?
crying + feeding
with tetralogy of fallot, tet spells often occur when for children?
playing
main manifestation of tetralogy of fallot
cyanosis / tet spells
what position can we put an infant in to help with tetralogy of fallot? or a position you might see a kid put themselves in to get better oxygenation?
knees to chest
type of defect:
closure of tricuspid valve + septal defect
tricuspid atreisa
s+s of tricuspid atresia in infants (3)
- cyanosis
- dyspnea
- tachycardia (infants)
s+s of tricuspid atresia in kids (2)
think more long term/chronic
- hypoxemia
2. clubbing
most common complication of patients with congenital heart disease
HF
s+s of R sided HF (6)
- edema
- bulging fontanel (infant)
- swollen eyelids
- weight gain
- swelling in hands + feet
- HTN
- decreased urinary output
s+s of L sided HF (3)
- dyspnea
- orthopnea
- adventitious lung sounds
MOA of digoxin
helps with contractility of heart
therapeutic digoxin range
0.8-1.2
1st s+s of dig toxicity
vomiting
what lab value should be closely monitored with digoxin therapy?
potassium
low levels risk dig toxicity
hold dig for:
child HR: ?
infant HR: ?
child HR: 70 or less
infant HR: 90 or less
after cardiac cath for child, what should happen with dressing?
remove and replace with bandaid after 1 day.
keep dry + clean
no strenuous activity
acute systemic vasculitis (inflammation of vessels) → weaken vessel walls
kawasaki disease
s+s in acute phase of kawasaki disease (9 - move head to toe / all over body)
- high fever (no response to antipyretic)
- enlarged lymph nodes
- red eyes w/o drainage
- bright red + chapped lips + strawberry tongue
- throat inflammation
- heart issues
- swelling + redness in hands + feet
- blistering rash
- joint pain
what is unique about fever in acute phase of kawasaki disease?
doesn’t respond to antipyretics
s+s of subacute phase of kawasaki disease? (3)
- peeling of hands and feet
- fever resolution
- other s+s start to resolve
how long does kawasaki disease take to resolve (lab findings) in the convalescence phase?
6-8 weeks