Exam 3- knee Flashcards
how much ROM for knee during the swing phase
60 deg
what compensates for knee if full ext is not acquired
ankle becomes hypermobile
why does the hip not compensate for loss of knee ext
hip is IR
why is tibial ER needed
complete screw home mechanism
maximal congruency
cover larger articular cartilage surface area
what is the ROM goal for TKA
120 flx
what is ROM for stair descent
90-120 flx
what is ROM for sit to stand
105 flx
what is the ROM and arthro for kneeling and deep squat
90-150 flx
femoral ER and post glide
ABD
lat glide
what are characteristics of ligament and capsule
dense connective tissue
type 1 collagen
low elastin
fibrocytes
multidirectional
what sprain, extraarticular or intraarticular needs sx
intraarticular within joint space
what is the healing phase for sprains
initial tensile strength - 3-5 weeks
dense connective tissue- 12 weeks
normal strength- 10-12 months
what should be the purpose needing to achieve following a sprain
stabilization
tissue proliferation
how can we treat sprains
POLICED
MET
how does the ACL run
superior
posterior
lateral
how are the cruciate ligaments named
where they are attached on the tibia
what does the ACL resist
anterior tibial glide
IR
for a noncontact ACL injury what are the nonmodifable RF
gender - F
2 weeks following menstruation
bony morphology
congenital hypermobility
for a noncontact ACL injury what are the modifable RF
high shoe surface friction (wrong shoe for surface)
high BMI
how does m strength play into ACL tears as a RF
lower overall
ham to quad ratio- predicts LE control
why are hams so important to the ACL
decrease ant. tibial translation produced by quads
what visual alignment can show low impaired LE control and be a risk for ACL tears
knee valgus
hip ADD
trunk lean
trunk RT
why does decreased knee flx with increase GRF be a problem
pt creates more stability for protection
why does greater visual motor strategy for balance be a RF for ACL tears
they use more of their eyes for visual stability rather than sensory control to stabilize more= impaired LE
what are the RF for 2nd ACL injury and what m needs to be addressed
same RF
ER of hip
what are symptoms of ACL tear
effusion
pop
giving way
WB activities limited
what can be found in a SCAN for ACL tears
ROM- limited and painful, hyperext and IR
RST- weak
ST- + distraction
what sp test can be positive for an ACL tear
Anterior drawer
lachmans
pivot shift
why might ACL sp test have false negatives
swelling- distending capsule
hamstring guarding
mensical tear
what can create the arthrogenic m inhibition to the quads after an ACL tear
pain
effusion
jt laxity
m weakness/incoordination
how can we test arthrogenic changes in the knee after an ACL tear
observation
palpation
m testing
what are the 3 primary early goals for ACL Rx
full to nearly full ROM, EXT- no later 4 weeks
minimal to no swelling
quad activation/coordination/endurance
what does full ROM for an ACL at 4 weeks depend on
if thats the only thing torn
why is full ROM important
overall healthier joint
what should be achieved for quad activation
SLR without extension lag
how can a quad set that is >90 of uninvolved side be misleading
uninvolved side is having side effects from pain, jt laxity, and swelling from involved side so both are inhibited
what is the PT Rx for ACL recovery
early WB
POLICED
functional bracing
MT
MET
how might NMES benefit an ACL recovery
significant increase in quad strength
isometric varying angles
what is our end goal for MET for an ACL recovery
intense resistive training without inducing pain
what MET should be emphasized for recovery of an ACL recovery
concentric and eccentric training
why is non WB activity have more load than WB early on
all loading is through the quads where as WB activities involves more groups of m
greater moment arm
for early ACL recovery, what range should OC and CC activities be performed in to not put as much load on the ACL
OC- 90-45
CC- 0-45
why does walking have the same amount of load as non WB activities
terminal knee ext
when are OC and CC activities okay to be performed
walking with correct form, correct trunk and LE control
why should HS be strengthen and coordinated with ACL recovery
decrease ant tibial glide produced by quads
besides knee proprioception, what else needs to be addressed for ACL recovery
trunk proprioception to minimize twist and lean
with neuromuscular training, what are we trying to minimized/decrease with ACL recovery
minimize frontal and transverse plane motion
decrease GRF
with neuromuscular training, what are we trying to promote/increase with ACL recovery
promote sagittal plane and trunk flx
progress speed and difficulty
emphasis on jump and balance
what is the MET prescription for an ACL recovery
2-3x for 6-10 months
how does BFR benefit ACL recovery
similar strength and hypertrophy as high intensity training
increased growth hormone
decreased myostatin
how does a vertical drop jump have a similar loading to non WB ext
increased loading with rate of deceleration
how does ACL recovery change if pt had partial meniscetomy
no change
how does ACL recovery change if pt has a meniscal repair
slower progression due to greater protection needed for meniscal healing
how does ACL recovery change if pt has a bone bruise
delays that leads to more difficulty reaching full ext and proper quad function
how does an MCL tear change ACL recovery
most often not surgically repaired
what are precautions for MCL recovery
only sagittal plane activity for 4-6 wks
limite tibial ER and valgus stress
how does articular cartilage debridement change ACL rehab
WBAT 3-5 days
no ACL delays
how does the articular cartilage sx compare
debridement
OATS and ACI
microfx
what are the grafts for ACL sx
BPTB auto and allograft
SGT
what pre op RF can limit prognosis
weakness= poor outcomes
what is the healing phase for BPTB autograft
initial weakening - first 4 wks
graft into bone- 6-8 wks
dense fibrous tissue- 8-12 wks
why is the first 4 weeks of the BPTB autograft weak
blood vessels are weakened until new blood vessels can form
why do BPTB autograft pt have anterior knee pain
patellar tendinitis/osis
more demand on less supply
what are the advantages of BPTB allograft
symptoms improve faster
pre pubescent population
avoids ant knee pain
what is the healing phase for BPTB allograft
graft to bone- 8-12 wks
dense fibrous tissue- 8-12 wks
what are the advantages of SGT graft
prepubescent to avoid epiphyses disruption
avoid ant knee pain
as a result of STG graft, when should pure HS strengthening start
6-8 wks
delay heavy strengthening for 12 wks