EXAM 3: GENITAL/URINARY Flashcards

1
Q

What population of children suffers the most from UTIs

A

uncircumcised boys younger than 3 months

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2
Q

What Sx is s major indicator of pyelonephritis

A

fever

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3
Q

What is Cystitis

A

inflammation of the bladder

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4
Q

What is Urosepsis

A

bacterial illness; urinary pathogens in blood

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5
Q

What pathogen is responsible for 80% of UTIs

A

Escherichia coli

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6
Q

The single most important contributing factor to UTI formation is

A

stasis

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7
Q

Describe Vesicoureteral Reflux

A

Retrograde flow of urine from bladder to upper urinary tract

Primary
Congenital abnormal insertion of ureters into the bladder

Secondary
Result of an acquired condition

Reflux increases chance for febrile UTI and can lead to scarring

Reflux with infection—Most common cause for pyelonephritis

Surgical management
Prevention with antibiotics

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8
Q

Any child who exhibits the following should be evaluated for a UTI:

A

Incontinence in a toilet-trained child

Strong or foul-smelling urine

Frequency or urgency

Dysuria

Gross hematuria

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9
Q

MANIFESTATIONS OF UTI in a neonate/infant

A

Poor feeding, vomiting
FTT
Excessive thirst
Frequent urination
Foul-smelling urine
Pallor, fever
Persistent diaper rash
Increased RR

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10
Q

What causes obstructive uropathy

A

kidney stones, urethral stricture, prostate/uterus issues

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11
Q

What is is called when the foreskin doesn’t move down penis and stays

A

PHIMOSIS

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12
Q

What is is called when theres a bunch of fluid in the scrotum that makes the testicles blow up like a baloon

A

Hydrocele

if it doesnt go away after a year, surgery

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13
Q

What is it called when the testes dont descend

A

Cryptorchidism

theres Abdominal, canalicular, ectopic

Anorchism is absence of testis

Will need surgert

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14
Q

What is it called when the male’s pee hole isn’t where its supposed to e

A

Hypo/epispadias

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15
Q

What are 6 symptoms of nephrotic syndrome

A

edema in face, pale skin fissure, hyperlipidemia, proteinuria, vomiting, tachycardia

HIGH PROTIEURIA
HYPOALBUMINEMIA

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16
Q

What’s the difference between primary and secondary nephrotic syndrome (and congenital)

A

Primary disease
Also known as
“Idiopathic nephrosis”
“Childhood nephrosis”
“Minimal change nephrotic syndrome” (MCNS)

Secondary nephrotic syndrome
In association with glomerular damage

Congenital nephrotic syndrome
Autosomal recessive disorder

17
Q

Describe the PATHOPHYSIOLOGY OF NEPHROTIC SYNDROME

A

Glomerular membrane
Normally impermeable to large proteins
Becomes permeable to proteins, especially albumin
Albumin lost in urine (hyperalbuminuria)
Serum albumin decreased (hypoalbuminemia)
Fluid shifts from plasma to interstitial spaces

18
Q

Nursing care for nephrotic syndrome

A

Low-to-moderate protein intake
Sodium restriction when a lot of edema is present

Medications
Steroids, immunosuppressants, diuretics

19
Q

What are signs that nephrotic syndrome has been controlled

A

Protein-free urine
Acute infections prevented
Edema absent or minimal
Nutrition maintained
Metabolic abnormalities controlled

20
Q

What is the difference between chronic and ACUTE GLOMERULONEPHRITIS

A

Acute: Sx suddenly, resolve w/ Tx

Chronic: if acute isnt treated and/or when disease develops slowly that leads to IRREVERSIBLE INJURY TO KIDNEYS

21
Q

What is the difference between [primary and secondary GLOMERULONEPHRITIS

A

Primary is where the infection starts in the glomeruli

Secondary is where glomeruli are infected by a systemic infection (Strep, Lupus, Sickle cell disease)

22
Q

What are th Sx of ACUTE GLOMERULONEPHRITIS

A

Oliguria, edema (periorbital), hypertension

Hematuria

Bleeding in upper urinary tract causes urine to appear smoky

Proteinuria
Increased amount of protein reflects increased severity of renal disease

23
Q

Diagnostic evaluation of acute glomerulonephritis

A

History of strep infection, mild cold, or nothing
Urinalysis—Proteinuria and hematuria
Azotemia—Elevated BUN and creatinine levels
Serum antibodies
X-rays—Cardiac enlargement, pleural effusion
Renal biopsy

24
Q
A
25
Q

Nursing considerations for glomunephritis

A

Manage edema
Daily weight measurements
Accurate input and output
Daily abdominal girth measurements

Nutrition
Low sodium
Fluid restriction

Susceptibility to infections

26
Q

Describe Hemolytic-uremic syndrome

A

Uncommon acute renal disease occurring in infants and small children

Etiology—Thought to be related to bacterial toxins

Symptoms
Hemolytic anemia, thrombocytopenia, renal injury, CNS symptoms
Hemolytic-uremic syndrome ()

Pathophysiology
Endothelial lining of glomerular arterioles become swollen and occluded, damaging RBCs as they pass
Damaged RBCs are removed by the spleen

Diagnostic evaluation
Anemia, thrombocytopenia, renal failure

Therapeutic management
Supportive care
Dialysis if needed
Hemolytic-Uremic Syndrome ()

Prognosis
Recovery rate of 95%

27
Q

Describe RENAL FAILURE

A

Inability of kidney to
Excrete waste
Concentrate urine
Conserve electrolyte
s
Can occur suddenly or slowly

Azotemia
Accumulation of nitrogenous waste in blood

Uremia
Retention of the nitrogenous wastes has resulted in toxic symptoms

28
Q

Difference between acute and chronic renal failure

A

Acute renal failure (ARF)
Inadequate perfusion
Kidney disease
Urinary tract obstruction

Chronic renal failure (CRF)
More than 50% of the functional renal capacity is destroyed
Few normal nephrons remain
Kidneys no longer able to maintain fluid and electrolyte balance
Clinical syndrome called “uremia”

29
Q

Describe DIALYSIS

A

The process of separating colloid and crystalline substances through a semipermeable membrane

Methods
Peritoneal dialysis-preferred method in children

Hemodialysis

Hemofiltration

30
Q

Difference between primary and secondary amenorrhea

A

Primary Amenorrhea
-No secondary sex characteristics and no menarche by 14-15 yo
-OR-
-Secondary sex characteristics present but no menarche by 16 yo
-No uterine bleeding after attaining sexual maturity

Secondary Amenorrhea
-Absence of menses for >6 mos or > 3 cycles
-Most common cause Pregnancy!
-Other causes: eating disorders, stress, severe wt loss