🤰🏾- Exam 3 Flashcards
Risk factors of ectopic pregnancy
- hx of previous ectopic pregnancy
- hx of std’s
- multiple partners
- tubal sterilization
- infertility
- IUD
- multiple induced abortions
- assisted reproductive techniques
- infection/inflammation
Signs of ectopic pregnancy
Missed menstrual period
Positive pregnancy test
Abdominal pain
Vaginal “spotting”
Linear salpingostomy vs salpingectomy
Linear salpingostomy- incision along the length of a Fallopian tube to remove an ectopic pregnancy and preserve the tube
Salpingectomy- removal of the tube
What are the signs and symptoms that suggest tubal rupture or bleeding
Pelvic, shoulder or neck pain ; dizziness or faintness ; increased vaginal bleeding
Gestational trophoblastic disease (hydatidiform mole)
Trophoblasts (peripheral cells that attach the fertilized ovum to the uterine wall) develop abnormally
Complete: no fetus present
Partial: fetal tissue or membranes present
**avoid uterine stimulus = embolus
-removed by vacuum aspiration then curettage
Clinical manifestations of hydatidiform mole
Vaginal bleeding
High hCG levels
Large uterus
Excessive nausea and vomiting
Early development of preeclampsia
wait 1yr to get pregnant again, continuous follow-up to detect malignancies
Nursing interventions for early pregnancy hemorrhagic conditions
- Provide test and procedure information
- teach infection prevention: first 72hrs greasy risk, no tampons, consult provider about sex
- provide dietary information: high in iron to increase HnH (liver, red meat, spinach, egg yolk, carrots, raisins) high in vitamin c (citrus fruit, broccoli, strawberries, cantaloupe, cabbage, green peppers) adequate fluid intake (2500mL per day) folic acid
- educate on reporting signs of infection: fever >100.4, vaginal odor, pelvic tenderness, malaise
Placenta previa
Implantation of the placenta in the lower uterus closer to the cervical os
Three classifications of placenta previa
Marginal- lower border is more than 3cm away from the os
Partial- partial covering of os (within 3cm)
Total- placenta completely covers cervical os
What is the classic sign of placenta previa
Sudden onset of painless uterine bleeding in the last half of pregnancy
What should NOT be done with placenta previa
No manual vaginal exams , use ultrasound to locate position of placenta
No pitocin to prevent uterine stimulation
Abruptio placentae
Premature separation of the placenta
Occurs when a clot forms on the maternal side of the placenta
Risk factors for abruptio placentae
- cocaine use causes vasoconstriction (narrowing of blood vessels) leading cause
- maternal hypertension, cigarette smoking, multigravida status, short umbilical cord, abdominal trauma, premature ROM, hx of premature separation of placenta
Clinical manifestations of abruptio placentae
1 bleeding
2 uterine tenderness that may be localized at the site of the abruption
3 uterine irritability with frequent low-intensity contractions and poor relaxation between contractions
4 abdominal or low back pain that maybe described as aching or dull
5 high uterine testing tone
– back pain, nonreassuring FHR patterns, signs of hypovolemic shock , fetal death, board like uterus
Uterine resting tone
Degree of uterine muscle tension when the woman is not in labor or during the interval between labor contractions
Signs of hypovolemic shock
Increased ❤️ rate
Increased RR
Decreased BP
Pale color, Cold and clammy skin
Decreased urine output
Weak, diminished, or “threads” pulses
Decreased O2 saturation
Nursing interventions for late pregnancy hemorrhagic conditions
- monitor for signs of hypovolemic shock: tachycardia, weak peripheral pulses, decreased BP, tachypnea, decreased O2 sat, cool/pale skin
- Monitor the fetus: tachycardia, decreased baseline variability, presence of late decelerations
- promote tissue oxygenation: position, O2, fluids
- fluid replacement
- prepare for surgery
- emotional support
Gestational HTN
Also known as PIH (pregnancy induced hypertension)
Systolic > 140 or diastolic > 90
After 20 weeks
Protein is NOT present
Can lead to chronic HTN if persists after birth
Mild preeclampsia
Unknown cause
Systolic > 140 or diastolic > 90
Develops after 20 weeks
Protein IS present in urine= proteinuria
Risk factors for pregnancy related HTN
First pregnancy
1st pregnancy for man who has previously fathered a preeclamptic pregnancy
Age > 35yrs
Anemia
Family or hx of PIH
chronic HTN or predicting vascular or kidney disease
Obesity
DM
Antiphospholipid syndrome
Multifetal pregnancy
Severe preeclampsia
Systolic > 160 or diastolic > 110
^ proteinuria
Generalized edema , weight gain , oliguria
Epigastric pain
Magnesium sulfate
Calcium antagonist and CNS depressant that acts as an anticonvulsant
Therapeutic level: 4-8 mg/dL
Antidote: Calcium Gluconate (1g at 1mL/min)
Nursing assessment for severe preeclampsia and magnesium toxicity
- rest: on left side
- diet: decreased Na
- weight: daily provides estimate of fluid retention
- VS: mag sulfate decreases RR
- EFM
- Breath sounds: excess moisture associated with pulmonary edema
- DTRs: noreflex= mag toxicity/ hyperreflex= ^ seizure risk
- urinary output: at least THIRTY mL/hr
- LOC: drowsiness indicates therapeutic mag
- labs: ^ creatinine, liver enzymes, or decreased platelets
Signs of magnesium toxicity
- RR rate of less than 12
- maternal O2 sat of lower than 95%
- absence of DTRs
- sweating, flushing
- altered sensorium (confused, lethargic, slurred speech, drowsy, disoriented)
- hypotension
HELLP syndrome
Hemolysis, elevated liver enzymes and low platelets
Liver enzymes ^ when hepatic blood flow is obstructed by fibrin deposits / low platelets occur cuz of vascular damage from vasospasm
Deliver vaginally
R upper quadrant, epigastric or lower r chest pain
Treatment: mag sulfate and hydralazine
Disseminated intravascular coagulation
Life threatening defect in coagulation
Labs: decreased fibrinogen, platelets, increased fibrin degradation and d-dimer is present
Hyperemesis gravidarum
Persistent uncontrollable vomiting that can continue throughout pregnancy leading to serious complications
Therapeutic management: meds, fluid replacement
eat foods high in K+ and Mg+
Characteristics of Hyperemesis gravidarium
- loss of 5% or more of pregnancy weight
- dehydration
- acidosis from starvation
- elevated levels of blood and urine ketones
- alkalosis from loss of hydrochloric acid in gastric fluids (vomiting)
- hypokalemia
Home visits
low risk infants vs high risk infants
Low risk- recognize jaundice, family adaptation, adequacy of maternal support
High risk- specialized equipment, ensure proper electricity/heat/telephone. Medically fragile: vents, oxygen therapy, apnea monitors, trach care, tube feedings, suctioning and cAre of IV sites
Red flags of telephone triage
- emergency situation (respiratory difficulty, bleeding) call back in 5 mins to make sure parents seek help
- illness (fever, dehydration, change in feeding behavior, unusual rashes)
- present problem or remedies aren’t working
- parents affect seems inappropriate for situation
Car seat test
Infants less than 37 weeks or LBW are placed in car seat and monitored for VS and oxygen level for 90mins
What are the 5S’s to consoling colic
Swaddle
Side lying
Shushing sound
Swinging
Suck on pacifier
Miliaria
Prickly heat
Develops in infants who are too warmly dressed in any weather / may also occur in infants with fever
Appears as red base with papules or clear vesicles in the center
Treatment: remove excess clothing or lukewarm bath
**avoid ointments
Ways to correct regurgitation
“Spit up”
Caused by overfeeding
Treat: burp, smaller frequent feed, enlarge nipple hole, upright after feeding
Growth in first 3 months
Gains 1oz/day (2lb per month)
Grows 1.4inches per month
Infant milestones
Focus best within a range of 8-12 inches
By 3 months follow objects horizontally
Social smile at 1-3months
Start “cooing” at 1-4 months
Laugh at 3-6 months
Well baby checkups occur at what age
1 2 4 6 9 12 months old
Common signs of illness in infants
Temp > 100.4
Vomiting
Watery stools or significant increase in stools
Blisters, sores or rashes
Listlessness of sleeping more than usual , irritability or crying more than usual
Coughing, frequent sneezing, runny nose
Pulling or rubbing at ear , drainage from the ear
Seek help immediately if infant displays signs of
Dyspnea
Rr > 60
Retractions
Cyanosis or extreme pallor
Difficult to arouse/keep awake
Plagiocephaly
Flattening of the head which was rounded at birth , may result from prolonged lying in the supine position
SIDS
Abrupt unexplained death of an infant less than 1 year old
3rd leading cause of death in infAnts from birth -1yr
Most common cause of death in infants from 1 month-1 yr
Common predisposing factors for PPH
- overdistention of the uterus (multiple gestation, large infant, hydraminos)
- precipitate labor or delivery
- prolonged labor
- use of forceps or vacuum extractor
- cesarean birth
- placenta previa, accreta or low implantation
- surgery, oxytocin, tocolytics, mag sulfate
Current definitions of postpartum hemorrhage
Blood loss of more than 500mL after vaginal birth, 1000mL after cesarean, a decrease in hematocrit of 10% or more since admission or the need for a blood transfusion
Management and predisposing factors for uterine atony
Management- Fundal massage, bimanual compression, medicine, fluid, blood replacement, hysterectomy
Factors- exhaustion, overexpansion, multi parity, prolonged oxytocin, hyper and hypo uterine contraction, retained placenta
Causes of early postpartum hemorrhage vs late postpartum hemorrhage
Early- uterine atony, trauma to the birth canal, hematoma, retention of placenta fragments,
Late- subinvolution and placental fragments that remain attached
Pharmacological measures to treat PPH and things to note
- oxytocin
- methylergonovine maleate (methergine) is given IM. Contraindicated in hypertension
- carboprost tromethamine (hemabate, prostin/15M) is given IM. Contraindicated in asthma and causes diarrhea. Keep refrigerated
- cytotec
Predisposing factors of retained placenta
Attempts to deliver placenta before it separates from the uterine wall, manual removal of the placenta, placenta accreta, previous cesarean and uterine leiomyomas
Therapeutic management of retained placenta
Oxytocin , methylergonovine , dilation and curettage , antibiotics
Subinvolution of the uterus
Causes, management, nursing considerations
Delayed return of uterus to non-pregnant state
Causes- retained placental fragments and pelvic infection
Management- methergine PO and antibiotics
Nursing considerations- education about warning signs, excessive bleeding and possible infection. Pelvic or fundal pain, backache and pelvic pressure or fullness
Nursing considerations/management of hypovolemic shock
Control bleeding , check VS q3-5 mins , give 8-10L of oxygen and draw labs for H&H, clotting studies and type and cross
Factors that increase the risk of thrombosis
Inactivity Prolonged bed rest Obesity Cesarean birth Sepsis Smoking Hx of thrombosis DM Trauma Prolonged labor Age > 35 Dehydration
What are the major causes of thromboembolic disorders in pregnancy or birth
Endothelial injury
Causes: increased clotting factor, venous stasis, vessel injury= prolonged in stirrups/constriction/flexion