Exam 3 Flashcards

1
Q

What is the main function of the gallbladder?

A

store excess bile from the liver

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2
Q

What does the gallbladder do while we eat?

A

push bile into small intestine to aid in digestion

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3
Q

When bile leaves the liver, where does it travel?

A

liver –> common bile duct –> cystic duct –> gallbladder

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4
Q

What are the components of bile?

A
  1. bilirubin
  2. bile salts
  3. cholesterol
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5
Q

What is bilirubin a product of

A

the breakdown of RBCs in the liver

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6
Q

What is the function of bile salt s

A

help fat breakdown in the small intestine

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7
Q

Why is our stool brown colored

A

bilirubin colors it

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8
Q

What is cholecystitis commonly caused by

A

triggered by a high-fat diet because this overworks the gall bladder

remember that bile salts aid in the digestion of fat

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9
Q

What is cholecystitis?

A

inflammation of the gallbladder

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10
Q

What is cholelithiasis

A

gall stone

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11
Q

What is choledocholithiasis

A

stones in the common bile duct

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12
Q

What is cholangitis

A

bile duct inflammation

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13
Q

What is the main difference between acute and chronic cholecystitis

A

acute has a rapid onset and more severe symptoms

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14
Q

White gallstones are usually caused by

A

excess cholesterol (hypercholesterolemia)

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15
Q

black gallstones are usually caused by

A

bilirubin

(liver failure pts due to cirrhosis)

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16
Q

What are risk factors for cholecystitis?

A
  1. 4 F’s
  2. high cholesterol/high fat diet
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17
Q

What are the four F’s?

A

female, fat, forty, fertile (premenopausal)

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18
Q

Is a female postmenopausal female on hormone replacement therapy at risk of cholecystitis

A

YES

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19
Q

Where is pain usually located with cholecystitis

A

right upper quadrant

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20
Q

Where may pain radiate to with cholecystitis

A

right shoulder

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21
Q

What is biliary colic?

A

extreme pain caused by obstruction of cystic duct due to stone

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22
Q

Biliary colic usually increases when

A

the stone is moving

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23
Q

What are other assessment findings of cholecystitis?

A

-flatulence
-dyspepsia
-eructation
-N/V

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24
Q

What are chronic manifestations of cholecystitis?

A

-jaundice
-clay colored stools
-icterus
-steatorrhea

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25
Q

What do patients with chronic cholecystitis develop jaundice

A

-build up of bilirubin in the blood (blockage of common bile duct is prohibiting the movement of bilirubin from moving to the small intestine)

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26
Q

Why do patients with chronic cholecystitis develop clay colored stools

A

lack of bilirubin the small intestine

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27
Q

What is icterus

A

yellowing of the eyes

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28
Q

Why do patients with cholecystitis have steatorrhea

A

bile salts are not breaking down fats

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29
Q

WBCs will be elevated, decreased, or normal in cholecystitis

A

elevated (mostly due to inflammation)

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30
Q

Liver enzymes will be elevated, decreased, or normal in cholecystitis?

A

MAY elevate if there is an obstruction and they cannot leave the liver

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31
Q

What were the liver enzymes she mentioned in class

A

-alkaline phosphatase
-aspartate
-amintotransferase (AST)
-lactate dehydrogenase (LDH)

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32
Q

Bilirubin will increase, decrease, or be normal with cholecystitis?

A

may elevate with obstruction

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33
Q

Pancreatic enzymes may increase, decrease, or be normal with cholecystitis?

A

may elevate if there is a blockage

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34
Q

What are the pancreatic enzymes

A

amylase and lipase

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35
Q

What diagnostic imaging is done to diagnosis cholecystitis

A

-ultrasound
-CT for followup
-HIDA scan

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36
Q

What is a hepatobiliary imilodiacetic acid (HIDA) scan

A

a nuclear medicine test to watch the function of the gallbladder

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37
Q

is the HIDA scan better for chronic or acute cholecystitis

A

chronic

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38
Q

Should we assess for allergies to shellfish, iodine, or contrast dye with HIDA scan? why or why not

A

NO because contrast dye is not used

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39
Q

What is inserted into the patient during a HIDA scan

A

radioactive tracer

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40
Q

Should our client be NPO prior to a HIDA scan>

A

yes

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41
Q

what is the treatment for acute cholecystitis in most cases

A

surgery

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42
Q

Why is surgery the most common treatment for cholecystitis

A

prevent peritonitis

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43
Q

What are non-surgical interventions for cholecystitis

A
  1. pain management
  2. dissolving stones
  3. lithotripsy
  4. percutaneous transhepatic biliary catheter
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44
Q

What medications are used to dissolve or stabilize gallstones?

A

-ursodiol and chenodiol

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45
Q

What is the most important thing to remember if a patient is receiving medication to dissolve their gall stones

A

they need and ultrasound q 6 months

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46
Q

How does lithotripsy help in treating gallstones?

A

breaks them down into smaller pieces so they can pass through ducts

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47
Q

What is a percutaneous transhepatic biliary catheter

A

-relieves pressure and promotes the flow of bile into the small intestine or into an external drainage bag

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48
Q

Nursing considerations for bile drainage bag following transhepatic biliary catheter

A

-empty regularly and track the output
-investigate if there is decreased output
-check surrounding skin and make sure no drainage is leaking out
\

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49
Q

What is a cholecystectomy

A

removal of the gallbladder

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50
Q

What are the two ways a cholecystectomy can be performed

A

laparoscopic or open

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51
Q

what are the benefits to a laparoscopic cholecystectomy

A

decreased risk of complications, recover faster

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52
Q

What are complications to monitor for after cholecystectomy

A

-bleeding, infection, etc

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53
Q

Nursing interventions after cholecystectomy

A

pain management, monitoring for complications

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54
Q

What key education points should we provide for a patient who has had a cholecystectomy

A

importance of maintaining low fat diet

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55
Q

What are the two types of pancreatic functions

A

endocrine and exocrien

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56
Q

Endocrine functions of the pancreas includes

A

glycemic control - release of insulin and glucagon

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57
Q

Exocrine functions of the pancreas include

A

release of pancreatic enzymes to break down carbs, proteins, fats

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58
Q

Acute vs chronic pancreatitis

A

acute has rapid onset and more severe symptoms

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59
Q

What is the most common cause of pancreatitis

A

alcohol use (chronic or short term binge drinking)

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60
Q

Is pancreatitis more common in men or women

A

men

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61
Q

When is acute pancreatitis seen more often in the ER

A

during holiday season when drinking is involved

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62
Q

Can gallstones cause the development of pancreatitis

A

yes

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63
Q

How do gallstones lead to pancreatitis

A

amylase and lipase get stuck in pancreatic duct and begin to autodigest the pancreas

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64
Q

What diagnostic procedure increases the risk of pancreatitis?

A

ERCP

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65
Q

what does ERCP stand for

A

endoscopic retrograd e cholangiopancreatography

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66
Q

What is an ERCP

A

extension of EGD that is used to assess for problems in the pancreatic and biliary ducts

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67
Q

How does an ERCP sometimes lead to the development of pancreatitis

A

this increases pancreatic inflammation

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68
Q

Pancreatitis presents with pain where

A

epigastric / LUQ

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69
Q

Pain from pancreatitis may radiate to the ?

A

back, flank, or shoulder

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70
Q

What position may relieve pain from pancreas

A

fetal position

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71
Q

Other symptoms of pancreatitis include?

A

1 . N/V
2. fever
3. jaundice
4. cullens sign
5. turners sign

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72
Q

What is Cullens sign?

A

dark blue discoloration around the naval

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73
Q

What is turners sign?

A

dark blue discoloration in the flank area

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74
Q

What is the cause of cullens sign and turners sign

A

hemorrhaging

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75
Q

Will pancreatic enzymes increase during pancreatitis?

A

Yes
-amylase will be 3x normal

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76
Q

Will liver enzymes (ALT and AST)increase, decrease, or remain normal in pancreatitis

A

elevate if blockage is occurring

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77
Q

Will bilirubin increase, decrease, or remain normal in pancreatitis

A

increase if there is a blockage

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78
Q

Why does glucose increase during pancreatitis

A
  1. increased stress on the bdoy
  2. inability to secrete insulin
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79
Q

Pancreatitis can lead to the development of what disease?

A

Type 1 DM

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80
Q

Will CRP be increased, decreased, or normal during pancreatitis

A

increased

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81
Q

What electrolyte imbalances are expected during pancreatitis

A

hypocalcemia, hypomagnesemia

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82
Q

Why are patients NPO with NG tube placement with pancreatitis

A

to decompress, prevent N/V

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83
Q

What IV meds are given during pancreatitis

A

-IV fluids
-electrolyte replacements

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84
Q

Why is a PPI given for a patient with pancreatitis?

A

decease gastric acid secretion

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85
Q

What pain medication may we expect a patient with pancreatitis be placed on?

A

PCA morphine or hydromorphone (dilaudid)

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86
Q

Patients with pancreatitis may be placed on prophylactic ________

A

antibiotics

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87
Q

Complications of pancreatitis include (long list sorry)

A

infection, abscess, pseudocysts, diabetes, MODS, ARDS, DIC, paralytic ileus

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88
Q

Is pain as severe with chronic pancreatitis?

A

no

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89
Q

Is the presence of ascites more likely to develop in acute or chronic pancreatitis

A

chronic

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90
Q

Is steatorrhea more likely to develop in acute or chronic pancreatitis

A

chronic

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91
Q

Are patients with chronic pancreatitis at risk of developing jaundice

A

yes

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92
Q

Patients with chronic pancreatitis may develop dark urine due to?

A

bilirubin

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93
Q

What diagnostic scan is used to identify pancreatitis

A

CT

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94
Q

What laboratory values are used to diagnose pancreatitis

A

amylase/lipase
bilirubin
alkaline phosphatase
glucose

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95
Q

What nutrition requirements are needed for a client with chronic pancreatitis

A

high protein-high calorie

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96
Q

clients with chronic pancreatitis are at risk of

A

significant loss of weight and muscle mass, DM

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97
Q

Is pain management a priority when treating clients with chronic pancreatitis

A

yes

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98
Q

To decrease gastric acid, clients with chronic pancreatitis are placed on

A

long term PPI

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99
Q

What is pancreatic enzyme replacement therapy

A

synthetic forms of amylase and lipase

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100
Q

When must PERT synthetic enzymes be taken

A

every time the patient eats to help aid in digestion

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101
Q

Does pancreatic cancer have a poor prognosis

A

yes

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102
Q

s/s of pancreatic cancer include

A
  1. painless jaundice
  2. pale stool
  3. itching
  4. dark urine
  5. glucose intolerance
  6. LUQ mass
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103
Q

Why do patients developing itching with pancreatic cancer

A

build up of bile salts and bilirubin

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104
Q

Is treatment for pancreatic cancer ever curative

A

no - goal is to extend life and quality of life

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105
Q

What are the treatments for pancreatic cancer

A
  1. chemo and radiation
  2. pain management
  3. surgery
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106
Q

what are surgical options for treating pancreatic cancer

A
  1. Whipple procedure
  2. partial pancreatectomy
  3. radical pancreatectomy
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107
Q

What is removed during a Whipple procedure

A

head of pancreas, duodenum, portion of jejunum, part or all of stomach, gallbladder

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108
Q

Clients post-op for pancreatic cancer procedure will be sent to the

A

ICU

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109
Q

Interventions post-op for pancreatic cancer include

A
  1. NPO with NG tube
  2. IV fluids
  3. biliary drains
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110
Q

Patients post-op from pancreatic cancer procedures should be placed in what position and why

A

semi-fowlers to reduce pressure on the abdomen

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111
Q

Nurses should monitor for what complications after a Whipple procedure

A

hemorrhage
infections
electrolyte imbalances

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112
Q

What is the function of the kidneys?

A

acid-base balance
filter and excrete waste
maintain BP
fluid balance
hormone secretion (EPO)

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113
Q

How does the kidneys help maintain normal hemoglobin and hematocrit

A

-releases erythropoietin which is a hormone that stimulates the production of red blood cells

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114
Q

How do the kidneys help with absorption of calcium

A

kidneys activate vitamin D –> vitamin D helps intestines absorb calcium

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115
Q

Characteristics of AKI

A

-sudden onset
-may not progress
-good prognosis
-high mortality if RRT is required or illness is prolonged

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116
Q

Characteristics of CKD

A

-gradual onset
-progressive to permanent
-prognosis depends
-ESKD fatal without RRT

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117
Q

Without RRT, ESKD will likely lead to

A

cardiac failure

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118
Q

why is it so important to manage CKD properly

A

it can slow the progression of the disease

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119
Q

What is meant by pre-renal AKI

A

caused by decreased perfusion!

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120
Q

Causes of pre-renal AKI include

A

-hemorrhage
-heart failure
-MI
-hypovolemia
-sepsis
-shock

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121
Q

What is meant by Intra-renal AKi

A

happens within the kidneys

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122
Q

Causes of intra-renal AKI include

A

-glomerulonephritis
-nephrotoxic drugs
-pyelonephritis

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123
Q

another name for intra-renal AKI

A

intrinsic AKI

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124
Q

Examples of nephrotoxic drugs that can cause intra-renal AKI

A
  1. NSAIDs
  2. antibiotics
  3. contrast dye
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125
Q

Special consideration for type 2 diabetics and contrast dye

A

metformin must be held to prevent AKI

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126
Q

How to assess kidney function after receiving contrast dye

A

-monitor creatinine levels before and after administration

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127
Q

What is meant by post renal AKI

A

occurs after the kidneys

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128
Q

Causes of post renal AKI

A

-stones
-BPH
-cystitis
-Hydronephrosis
-prostate, cervical, bladder, and colon cancers

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129
Q

Assessment findings of AKI include

A
  1. oliguria
  2. hypertension
  3. FVO
  4. SOB
  5. azotemia
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130
Q

What is azotemia

A

build-up of waste products in the blood

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131
Q

in AKI, serum BUN and creatinine will?

132
Q

What is creatinine

A

a byproduct of muscle breakdown

133
Q

The breakdown of creatinine should usually occur at

A

a constant rate

134
Q

IN AKI, GFR and creatinine clearance will?

135
Q

What electrolytes will be elevated in AKI

A

Potassium, sodium, and phosphorus

136
Q

in AKI, which electrolyte will decrease

137
Q

What ABG will be seen in patients with AKI

A

metabolic acidosis

138
Q

in AKI, H/H will eventually

139
Q

What is the number one intervention for treatment of AKI

A

treat underlying cause!! (ex. hemorrhage = blood products ; kidney stone = lithotripsy)

140
Q

If AKI is caused by hypovolemic shock, would we give fluids or diuretics?

141
Q

If AKI is caused by hypovolemic shock, would we give vasopressors or antihypertensives?

A

vasopressors

142
Q

If AKI is caused by heart failure, would we give fluids or diuretics

143
Q

T or F: maintaining fluid balance, BP, and electrolyte balance is crucial for managing patients with AKI

A

TRUE! remember types of interventions are based on underlying cause!!!

144
Q

What type of dialysis are patients with AKI typically placed on?

A

continuous renal replacement therapy

145
Q

Nutrition therapy is a very crucial aspect of managing AKI and is typically done by

A

the registered dietician!

146
Q

Why is nutrition hard to manage in AKI

A

there is an increased rate of protein breakdown in the muscles

147
Q

What is a common finding in stage 1 CKD

148
Q

What is a common finding in stage 2 CKD

A

microalbuminuria

149
Q

When should discussion of RRT begin in the CKD stages

A

stage 4 to being preparing patient

150
Q

In stage 5 CKD, GFR is less than

151
Q

In stage 5 CKD, interventions include

A

RRT and kidney transplant

152
Q

For a diabetic patient with CKD, they require a _____ q _____ months

A

urinalysis ; 3

153
Q

What are we monitoring for when we have diabetic patients submit for a UA

A

microalbuminuria (monitors their progression of kidney disease!)

154
Q

What are the two main causes of CKD

A
  1. hypertension
  2. diabetes
155
Q

Other causes of CKD include

A

-lupus
-rheuatoid arthritis
-HIV
-NSAIDs
-smoking / tobacco

156
Q

Over time, patients with CKD will experience less

A

urine production

157
Q

is FVO common in CKD

158
Q

What blood dyscrasia should we monitor for in our patients with CKD

159
Q

Electrolyte imbalances for clients with CKD are

A

the same as AKI

160
Q

The buildup of waste products in the blood (uremia) can lead to this complication

A

pericarditis

161
Q

Why is heart failure a complication of CKD?

A

the heart is having to work so much harder and will eventually weakne

162
Q

Fluid volume overload is patients with CKD will lead to this complicaiton

A

hypertension

163
Q

the kidneys reduced ability to filter blood components also contributes to this complicationl

A

hyperlipidemia

164
Q

How does anemia develop in patients with CKD

A

reduced production of EPO = less production of RBCs

165
Q

What is asterixis?

166
Q

How does CKD cause asterixis?

A

waste product build up

167
Q

What is renal osteodystrophy

A

complication of CKD due to loss of vitamin D

-vitamid D loss = loss of calcium absorption = brittle bones

168
Q

Patients with renal osteodystrophy require ?

A

calcium and vitamin D supplementation

169
Q

Uremic symptoms that develop in patients with CKD include?

A

-uremic frost
-halitosis
-mouth inflammation

170
Q

What is halitosis?

A

foul smelling breath

171
Q

The biggest intervention for managing CKD includes

A

-lifestyle medications

-controlling hyperlipidemia
-controlling blood glucose
-controlling hypertension

172
Q

Patient teaching for CKD includes

A

-maintain a healthy weight
-get plenty of exercise
-eat healthier
-manage diabetes and hypertension

173
Q

In broad terms, hemodialysis functions as an

A

external kidney

174
Q

the external kidney in the hemodialysis machine is known as the

175
Q

What is used as the filter in peritoneal dialysis

A

the peritoneal sac

176
Q

IN peritoneal dialysis, waste products drain from the peritoneal cavity and into

A

a drainage bag

177
Q

Long term dialysis access is typically an

A

AV fistula

178
Q

What is an AV fistula

A

where an artery and vein are connected

179
Q

What is important to assess when our patient has an AV fistula

A

-a thrill is palpated
-a bruit is heard
-surrounding skin for infection

180
Q

Important considerations when our patient has an AV fistula

A

they are a limb restriction in that arm!
-no IVs, no BPs

181
Q

A temporary dialysis catheter is known as a ?

A

hemodialysis catheter

182
Q

Although hemodialysis catheters are very similar to central lines, they have a larger lumen. what does this increase the risk of ?

A

clots forming inside

183
Q

Will clients with a short term hemodialysis catheter have peripheral IV lines

184
Q

Should medications be given through hemodialysis catheter

185
Q

What special circumstances may allow for hemodialysis catheters to be used as central line

A
  1. no peripheral IV can be obtained
  2. during a codeW
186
Q

What MUST be obtained before giving meds through a hemodialysis catheter

A

Provider permission

187
Q

What is done to prevent clots in a hemodialysis catheter line

A

they are heparinized (heparin left to sit in catheter)

188
Q

Before giving meds through hemodialysis catheter, the nurse should

A

-remove 10 ML of blood to ensure there is no heparin left in line

189
Q

Complications of AV fistula include

A

-thrombosis of AV access
-stenosis of AV access
-ischemia to site distal of AV access

190
Q

Complications of dialysis include

A
  1. infection
  2. heart failure
  3. aneurysms
191
Q

Why is heart failure a complication of dialysis

A

hemodynamic overload from AV access

192
Q

Why may aneurysms form as a complication of dialysis

A

-from repeated needle punctures and pressure exerted during dialysis

193
Q

Considerations for peritoneal dialysis

A

-they must be able to do it at home

194
Q

Is multiple sclerosis a chronic disease?

195
Q

What are the causes of multiple sclerosis?

A

auto-imune, genetic, infection

196
Q

What is the pathophysiology of MS

A

there is a breakdown of myelin sheath leading to decreased nerve impulses/ messages

197
Q

What is the leading cause of disability in young and middle age adults

A

multiple sclerosis

198
Q

Most adults are diagnosed with MS before the age of

199
Q

What is the function of myelin?

A

transmission of electrical impulses

200
Q

What are the types of MS?

A
  1. relapsing-remitting
  2. secondary progressive
  3. primary progressive
  4. progressive-relapsing
201
Q

what is relapsing - remitting MS

A

symptoms vague in beginning –> then symptoms go COMPLETELY away –> then come back a little worse

202
Q

what type of MS does relapsing-remitting turn into

A

secondary progressive

203
Q

What is secondary progressive MS?

A

progression without remission periods

204
Q

What is primary progressive MS?

A

they never have any period of no sx or less severe sx

205
Q

What is progressive-relapsing MS?

A

symptoms never completely go away but there are periods where they are more vague

206
Q

What type of climate is MS more common in

A

colder climates

207
Q

What increases the risk of developing MS

A

-Northern European ancestry
-1st degree relative
-onset 20-50 years old
-being a woman

208
Q

Why is MS so hard to diagnose

A

vague symptoms in the beginning stages are often missed as MS

209
Q

What are early symptoms of MS

A

-muscle weakness
-fatigue
-brain fog

210
Q

What are assessment findings of those with MS

A

-tremors
-mobility issues
-dysphagia
-difficulty speaking
-vision problems
-bowel and bladder dysfunction
-sexual dysfunction

211
Q

Psychosocial and cognitive findings of MS

A

-depression and anxiety
-self-esteem / body image issues
-alterations in ADLs
-short-term memory loss
-inability to concentrate
-impaired judgment

212
Q

Is there a definitive diagnostic test for MS?

213
Q

What is the gold standard diagnostics used for MS

A
  1. must have presence of symptoms
  2. must have 2 areas of white looking plaque on MRI
214
Q

how does MS appear on an MRI

A

-white spots on MRI that are plaques/lesions

215
Q

A lumbar puncture for CSF will contain these if the patient has MS

A

-increased proteins
-white blood cells

216
Q

What is evoked potential testing

A

where electrodes are placed on patient to assess the transmission of impulses on the optic nerve

217
Q

A dysfunction of impulses during an evoked potential test may indicate

218
Q

What medications are given for MS patients

A

-immunomodulators
-anti-inflammatories

219
Q

Why are immunomodulators given for patients with MS

A

slow disease progression and suppress immune system

220
Q

Medical marijuana is approved for patients with MS. What symptoms does this aim to aid in?

A

pain, tremors, spasms, muscle stiffness

221
Q

Nursing consideration for use of medical marijauan

A

-alters judgment and cognition (which may make these symptoms of MS worse)

222
Q

What can be done for clients with MS to improve their mobility?

A

-assistive devices
-PTOT
-rehab
-exercise

223
Q

What interventions may be needed to help patients with MS manage their cognition

A

-therapy
-caledars
-reminders for medications
-med boxes
-safe home
-items accessible to them

224
Q

What vision change is often seen in MS

A

diplopia (double vision)

225
Q

How to manage diplopia

A

-patching one eye and alternating sides q few hours

226
Q

What should patients with MS avoid to prevent exacerbation

A

-overexertion
-stress
-extreme temp
-extreme humidity
-people with infections

227
Q

What is a complete spinal cord injury

A

-across entire width of spinal cord
-full deficits

228
Q

What is an incomplete spinal cord injury

A

-through only some of the spinal cord
-partial deficits

229
Q

1/3 of spinal cord injuries occur from

230
Q

Who is most likely to receive a spinal cord injury

A

young, caucasian man

231
Q

What is the most common cervical cord injury

232
Q

SCI between c2-c5 result in

A

loss of breathing ; will require mechanical ventilation

233
Q

SCI between c5-c6 result in

A

paraplegia and arm weakness

234
Q

Most common thoracic injury

235
Q

Most common lumbar injury

236
Q

Injuries to T12 and L1 often result in

A

paraplegia

237
Q

What happens during a SCI caused by hyperflexion

A

a sudden and forceful movement of head forward

238
Q

what happens during SCI caused by hyperextension

A

sudden movement of head backwards

239
Q

What happens during a SCI caused by excessive rotation

A

very quick and sudden rotation beyond point of normal

240
Q

What happens during a SCI caused by penetrating trauma

A

spinal cord severed by knife, gunshot, etc

241
Q

What happens during spinal cord injury caused by axial loading

A

the spinal cord is compressed

242
Q

Head rests are meant to prevent which type of SCI

A

hyperextension

243
Q

Critical teaching about car safety to prevent SCI

A

-make sure there are headrests
-make sure headrest is at appropriate height !

244
Q

what is important to know before treating a SCI

A

-how the injury occurred
-baselines motor and sensory function before injury

245
Q

Order of assessment for SCI includes

A

-ABC
-neuro status
-spina cord perfusion
-GCS

246
Q

How to assess mobility and sensory function in clients with SCI

A

-go down the body and evaluate where patient can move and feel
-mark the place where this stops on the body

247
Q

Overtime, control of _____ and _____ function can be lost after SCI

A

bowel and bladder

248
Q

GI/GU changes after SCI include

A

-loss of control
-loss of sensation to go
-loss of knowing when to go

249
Q

Psychosocial issues after SCI include

A

-depression and anxiety
-potenital loss of job/income
-unable to live at home
-etc

250
Q

Initial management of SCI includes

A

-maintaining airway
-IV fluids
-pressors
-monitoring for complications

251
Q

Immobilization of the spinal cord should be done with a

A

c - collar

252
Q

Patients with SCI are at risk of developing this GI complication

A

stress ulcers

253
Q

What medications are given to prevent the development of stress ulcers in patients with SCI

A

PPI / histamine blockers

254
Q

Nursing considerations for c - collar include

A
  1. always assume there is a cervical spine injury until they are cleared
  2. do not take it off
  3. correct size
255
Q

How to position patients in C - collars

A

make sure we are log rolling

256
Q

Complications to monitor for when our patient is in a C-collar

A

skin breakdown
airway clearance/ compression

257
Q

what is a halo device

A

-a type of traction used to relieve pressure on cervical spine by holding head up

258
Q

A major complication of the halo device is

259
Q

Nursing considerations for halo device

A
  1. infection prevention - pin care
  2. monitoring neurostatus
  3. skin breakdown
260
Q

To prevent the halo from moving, it is important to monitor

A

that it is always in the right spot

261
Q

Can the fleece part of the halo device be changed?

262
Q

Can patients go home with a halo device?

263
Q

Client teaching for halo device

A
  1. do not remove
  2. do not drive
264
Q

For a patient in a halo device, what must be kept with them at ALL times

A

-a device to remove the halo if needed

265
Q

How do patients in halo devices sleep

A

sitting up

266
Q

How do patients in halo devices drink

A

using a straw

267
Q

Patient teaching for getting dressed in halo device

A

-use button up shirt that is 2-3 times too big

268
Q

3 major complications of SCI include

A
  1. neurogenic shock
  2. spinal shock
  3. autonomic dysreflexia
269
Q

What type of shock is neurogenic shock

A

distributive

270
Q

What causes neurogenic shock?

A

-loss of sympathetic tone and autonomic control
-loss of epi and norepi

271
Q

Where in the spinal cord is more likely to cause neurogenic shock

A

T6 and above

272
Q

Symptoms of neurogenic shcok

A

bradycardia, hypotension

273
Q

Neurogenic shock is treated iwth

A

atropine, fluids

274
Q

What is spinal shock?

A

temporary loss of sympathetic activity below level of injury

275
Q

In what time frame can spinal shock occur

A

within 30-60 minutes of injury to 6 weeks after injury

276
Q

Will spinal shock reverse?

A

yes ; is only temporary

277
Q

is spinal shock considered a localized shock?

A

yes ; directly in spinal cord

278
Q

Manifestations of spinal shock include

A

-flaccid paralysis
-loss of reflexes
-loss of bowel and bladder

279
Q

What is autonomic dysreflexia?

A

exaggerated sympathetic response

280
Q

What triggers autonomic dysreflexia?

A

stimulation of bowel or bladder

281
Q

Examples of bowl or bladder stimulation include?

A

severe constipation, urinary catheters, UTI, bladder distention, scrotal compression, UTI, hemorrhoids

282
Q

Symptoms of autonomic dysreflexia include

A

-severe hypertension
-headache
-bradycardia

283
Q

Other symptoms of autonomic dysreflexia include

A

-nausea
-blurred vision
-sweating

284
Q

Complications of autonomic dysreflexia include

A

-seizures
-coma
-death

285
Q

What position should patients with autonomic dysreflexia be placed in

A

sitting / high followers

286
Q

Why are patients with autonomic dysreflexia placed in high fowlers positioin

A

lower blood pressure and intracranial pressure

287
Q

Treatment for autonomic dysreflexia depends on

288
Q

What will be given to manage the HTN in autonomic dysreflexia

A

nifedipine
nitrates
make sure to continuously monitor BP

289
Q

Long term complications of SCI include

A

-immobility
-skin breakdown
-infection (PNA)
-VTE
-depression
home environment changes

290
Q

Musculoskeletal changes for SCI include

A

-osteopenia
-muscular atrophy
-heterotopic ossification

291
Q

what is heterotypic ossification

A

when bone tissue forms outside of the skeletal system

292
Q

SCI can cause longterm changes to

A

sexual functions, bladder and bowel functions

293
Q

What percentage of adults experience back pain

294
Q

What is the leading cause of work disability

295
Q

Examples of lower back pain

A

sciatic nerve - burning, stabbing, pain down leg or footE

296
Q

Examples of cervical back pain

A

burning, stabbing pain down arm, pain in neck, upper back and shoulder pains, headache

297
Q

Does back pain include numbness and tingling

298
Q

Will managing weight and getting regular exercise prevent back pain

299
Q

Avoiding this substance will help reduce back pain

300
Q

Adequate intake of ______ and _____ __ will help prevent back pain

A

calcium; vitamin D

301
Q

Do high heels increase risk of back pain

302
Q

Occupational education to prevent back pain

A
  1. lift with legs
  2. use working height
  3. ask for help
303
Q

What is most commonly used to diagnose back pain

A

-symptoms
Pain, limited mobility, paresthesias

304
Q

What imaging is typically done first in back pain complaints

305
Q

What is usually done before insurance will approve CT/MRI for back pain

A

-physical therapy
-medication management

306
Q

What is a bone scan?

A

-used to assess perfusions to bone using a radioactive tracer

307
Q

Does a bone scan require contrast

A

NO (it is nuclear)

308
Q

Increased vascularity to a bone seen on a bone scan indicates

A

presence of a tumor

309
Q

Can massage and heat be used as back pain interventions

310
Q

Is spinal manipulation okay to use for addressing back pain

311
Q

Patient education if they are seeking chiropractor/ spinal manipulation

A
  1. make sure they are licensed
  2. make sure they do x-rays before
312
Q

What medications can be given for back pain

A
  1. NSAIDs
  2. creams and sprays
313
Q

A tens unit does what?

A

-gives little electrical impulses into muscles and may help relieve back pain

314
Q

What is ziconotide?

A

a central acting analgesic

315
Q

How does ziconotide help with back pain?

A

-it is an implantable pain pump
-gives a little bit of medication at a time

316
Q

Is a ziconotide pain pump considered a last resort

317
Q

Ziconotide has a BBW for

A

-mental health issues (psychosis)

318
Q

Patient teaching for ziconotide

A
  1. do not take if you are struggling with mental health
  2. report changes ot mental health
  3. you must come back in for medication to be refilled
319
Q

What are key concerns to address immediately post op any spinal surgery

A
  1. ABC’s
  2. Bleeding
  3. infection
320
Q

Can CSF leak occur following spinal surgery?

321
Q

Clients experiencing a CSF leak will experience

A

severe headache

322
Q

what is halo’s sign?

A

-CSF leak: will appear red with yellow around the edges

323
Q

GI/GU complications following spinal surgery include

A

-urinary retention
-paralytic ileus

324
Q

Why are fat embolisms common after spinal surgery?

A

many surgeries manipulate bone where fat globules are stored

325
Q

Key sign of fat embolism (difference between PE and FE)

A

petechiae on the chest

326
Q

Nerve root pain is common after spinal surgery and is known as

A

persistent or progressive lumbar radiculopathy

327
Q

What is failed back surgery syndrome?

A

when symptoms persist even after surgery