Exam 3 Flashcards
what are the 4 main body fluids HIV is commonly spread?
- blood
- semen
- breast milk
- vaginal secretions
HIV is most often transmitted through what? (3)
- sexual contact
- parenteral
- perinatal
what type of sexual contact is highest risk for HIV transmission?
anal sex
re: parenteral HIV transmission, what years were the higher risk years for transmission via blood products?
1978-1985 (before screening of blood products)
what are interventions for parenteral HIV transmission?
HARM REDUCTION
cleaning needles, needle exchange, PreP
how long should babies be treated with HIV drugs after delivery (from positive pregnant person)?
4-6 weeks post delivery
increased viral load means what?
increased chance of transmission
what is best thing health care workers can do to prevent HIV transmission?
standard precautions + use of safety needles
+ post exposure prophylaxis within 24 hours if exposed ◡̈
what does the 4th generation HIV testing detect? + in how many days?
HIV antibodies: 21 days
HIV p24 antigen: 14 days
if 4th gen HIV testing is positive, what is protocol? if negative, what is protocol?
positive –> test for HIV strain
negative –> test with another test (NAAT)
re: home testing kits for HIV, what should be done?
results verified w/further testing
what is viral load measuring? + what can it track?
amt of HIV viral RNA in blood
tracks: infectivity + therapy effectiveness
ideally, how often should a patient with positive HIV diagnosis be monitoring their viral load?
per Messer, q 2-3 months after diagnosis
what would we see re: these labs with AIDS?
lymphocytes:
CD4:
viral load:
lymphocytes: LOW w/fully progressed AIDS
CD4: decreases over time
viral load: increases over time (ART meds can mitigate this)
re: HIV/AIDS, CBC + CMP can help to track development of what?
opportunistic infections
what are the 3 stages of HIV/AIDS?
- acute (flu-like; VERY contagious)
- chronic (asymptomatic; can transmit to others)
- AIDS
what is timeframe to see acute HIV stage after transmission?
2-4 weeks after transmission
what are the 2 AIDS-defining parameters?
- CD4 <200
or - development of opportunistic infections
what is normal CD4? what are the CD4 counts associated with each HIV stage?
normal 500-1500
- acute: >500
- chronic: 200-499
- AIDS: <200
- unknown: not enough info about CD4 or opportunistic infections
who should be screened for HIV?
13-65 yrs old (at least one test)
higher risk/repeated high risk exposures (IVDU or sex workers) –> annually/frequently
what is the PRIORITY assessment of positive HIV/AIDS patient?
KNOW
early detection + management of infections
patient education
what is PRIORITY nursing care for patient with HIV/AIDS?
handwashing!! (infection risk - keeping patient safe)
what is most common opportunistic infection with AIDS?
KNOW
Pneumocystis pneumonia (PCP)
how does pneumocystis pneumonia present? (name s+s)
KNOW
PNA:
cough, dyspnea, SHOB, crackles in lungs, fever, tachypnea
what is tx for pneumocystis pneumonia?
KNOW
antibiotics (bactrim) + support (O2 + positive pressure)
these txs can increase risk of infection even more :(
disseminated MAC has ONLY been seen with which 2 scenarios with AIDS patients?
KNOW
- CD4 <50
or - patients not on ART drugs
if patient receives PPD test and is HIV positive, what is positive result?
> 5mm
may not be best screening test for immunocompromised - best with NAAT, CXR or sputum
what do we need to KNOW about Kaposi’s sarcoma r/t AIDS?
that it improves with ART drugs
per Messer
what interventions r/t oxygenation should we implement for HIV/AIDS patients?
elevate HOB + O2 therapy
what interventions r/t nutrition should we implement for HIV/AIDS patients?
high calorie, high protein, low fat
small, frequent meals
how can we educate patients with HIV/AIDS to prevent infection? (7)
- monitor vitals + temp
- no fresh plants, flowers or sick visitors
- avoid raw foods
- handwashing
- don’t handle pet litter
- bathe daily
- don’t share personal items
what are the characteristics of all autoimmune disease?
- periods of exacerbation + remission
- chronic + progressive
- systemic effects: fever, fatigue, weakness, anorexia, weight loss
- inflammation of connective tissues
what is the pathophysiology of RA?
forms autoantibodies + rheumatoid factors that affect healthy tissues (especially likes synovial joints)
what can happen with RA over time if left untreated?
major deformities + bone fusions
when does RA often present?
winter months
what are the s+s of RA in early stage?
red, warm, tender, swollen + stiff joints - symmetrical
what joints are most commonly affected with RA?
upper extremity joints (synovial)
what are the s+s of RA in late stage? (5)
- inflammation in more joints
- gel phenomenon
- joint effusions
- spindle like fingers
- muscle atrophy –> dec ROM
TMJ with RA signifies what?
SEVERE disease
RA affecting cervical joints puts a person at risk for what?
paralysis
aside from the “usual” systemic effects of RA, what other things can you see? (3)
- subcutaneous nodules (forearms)
- vasculitis
- paresthesia –> foot drop
“Very Painful Shit”
re: RA, what is vasculitis? what does it present as? what is person at risk for?
what: inflammation of vessels
presentation: periungual lesions on fingers
@ risk for: organ ischemia (kidneys + lungs most worrisome)
what are Baker’s cysts and which condition are they associated with?
enlarged popliteal bursae; associated with RA
tx of underlying disease is best for these
what is sjorgen’s syndrome?
attacking glands that lubricate; often with another autoimmune disease
eyes, mouth, vagina
re: RA, what should we be examining with these patients in regard to quality of life?
how are their ADLs impacted? are their medications working and helping them to achieve their ADLs? <3
diagnosis for RA
no definitive; only supportive; very difficult
if an arthrocentesis was performed on a person with suspected RA, what findings would you expect to see?
fluid cloudy w/WBCs
what is our priority intervention for person wtih RA?
rest, balance + setting priorities
if patient with RA was too tired to take a bath and wanted to skip it, what would be your best response?
teaching them why hygiene is so important, especially with immunosuppressive medications + offering a bed bath with CHG wipes
describe the differences between RA + Osteoarthritis
RA: autoimmune, symmetrical, worse after resting
OA: wear and tear/degenerative, generalized, worse after activity
pathophysiology of SLE
production of antinuclear antibodies + immune complexes
re: SLE, the immune complexes have an affinity for which organ?
kidneys
can impact the organs directly or the vessels that perfuse them
SLE is most common in which population
females of color
what is the unique sign of SLE?
“butterfly rash”
function of which organ is VERY IMPORTANT with SLE?
kidneys - because the immune complexes are nephrophilic + cause systemic inflammation
this condition is triggered by stress or cold and NOT exclusive to Lupus
Reynaud’s Phenomenon
extreme pallor or red/blue of fingers –> can lead to loss of digits
what is our priority nursing education for patient with SLE?
avoid direct sunlight + wear sunscreen (skin protection)
+ derm referral is also important
what symptom do we educate patients with SLE to monitor for to recognize impending exacerbations?
low grade fever
what is systemic sclerosis?
autoimmune disorder causing hardening of skin + eventually inflammation of connective tissue
what is leading cause of death with systemic sclerosis?
kidney sclerosis (systemic inflammation)
limited cutaneous systemic sclerosis is associated with which syndrome? describe it.
CREST
Calcium deposits in skin Raynaud's phenomenon Esophageal Dysfunction Sclerodactyly (tightened skin on fingers/toes) Telangiectasis (spider capillaries)
s+s of systemic sclerosis (6)
- painful + stiff joints
- pitting edema
- shiny skin (b/c of hardening)
- taut skin
- joint contractures
- loss of ROM
re: systemic sclerosis, describe the organ involvement (GI, lungs, CV, kidney)
digestive tract –> dysphagia
CV –> raynaud’s
lungs –> pulmonary HTN
kidneys –> DEATH
what is #1 cause of death with systemic sclerosis?
kidney failure
based on googling, this is not accurate, but according to messer, kidneys are the cause
re: systemic sclerosis and involvement of digestive tract, what would be your priority intervention?
ability to swallow safely (r/t dysphagia)
what are 3 interventions for patient with scleroderma?
- bed cradle (avoid skin breakdown + pain)
- foot board (prevent contractures)
- keep room warm (prevent raynaud’s)
what is gout?
systemic arthritis r/t uric acid build up
NOT autoimmune
what is a very common cause for secondary gout?
kidney disease (hyperuricemia b/c of impaired excretion)
with acute gout attack, where is the pain often manifested?
big toe (excruciating!!)
what happens with chronic/tophaceous gout?
urate crystals deposit in major organs
re: gout, urate crystals have an affinity to which organs?
kidneys (nephrophilic)
s+s of gout
joint inflammation + severe pain
interventions for gout (2) + what things should be avoided?
- fluids
- low purine diet
- avoid ASA, diuretics, stress
what foods should people avoid with gout? give examples
KNOW
purine-containing foods
avoid organ meat, shellfish, oily fish w/bones
(egg would be good protein choice)
main interventions for patients with sensory problems
know how to optimize communication + keep patient safe (per Messer) **
what is our priority assessment and intervention for patients with sensory deficits?
communication + anxiety (per Messer) + keeping them safe
what is cataracts?
lens opacity + cloudiness
what are the 2 major risk factors for cataracts?
KNOW
aging (>70) + sun exposure (not wearing sunglasses)
what would you see re: vision with person with cataracts?
blurred vision + decreased color perception
–> leads to double vision