Exam 2: Immob, pain, LLAF Flashcards
What do you attend to during observation?
- alignment
- atrophy
- edema/effusion
- color
- callus patterns
- shape
- signs of CRPS/RSD
- abnormal skin lesions/moles
What joint does “pronation/supination twist” occur at?
-tarsometatarsal
3 mechanisms that maintain arches in foot.
1) wedging of the interlocking tarsal and metatarsal bones
2) tightening of the ligaments on the plantar aspect of the foot
3) the intrinsic and extrinsic muscles of the foot and their tendons.
Ottawa Rules with Buffalo Modifications
1) tenderness over lateral malleolus to 6cm proximally
2) tenderness over medial malleolus to 6cm proximally
3) tenderness over navicular
4) tenderness over base of fifth metatarsal
Effects of immobilization on bone
- bone loss
- resorption > formation
- seen w/in 2 weeks
- drops 55-60% by week 12. after 12wk = permanent changes
-dec elastic resistance (more brittle)
Effects of immobilization on articular cartilage
- softening
- dec thickness
- adhesions
- dec proteoglycan synthesis
- pressure necrosis
- > 30 day immobilization = OA
Effects of immobilization on periarticular connective tissue
- dec GAG and water content
- dec lubrication
- abnormal cross linkage
- excessive fatty fiber deposition -> scar tissue
- clinical: ankylosis, jt stiffness, or joint contracture
Effects of immobilization on ligaments
- makes weaker
- dec fiber size and density = dec CSA
- dec GAG level
- haphazard arrangement of collagen fibers
- dec collagen synthesis
- inc osteoclastic activity at bone-ligament junction
Effects of immobilization on muscle
- dec size and mass
- dec strength
- inc or dec length depending on position of immobilization
- dec endurance
- dec recruitment
- slower contractions
- slow twitch > fast twitch
Effects of immobilization on nervous system
Dysponetic signaling = the misuse of energy in functional disorders. Reversible.
First oder pain stuff
- A delta, C fibers, (A beta) dorsal horn (SG)
2nd order pain stuff
- T (transmission) cells. Spinal cord to thalamus
lateral ST = a delta
anterospinothalamic = C fibers
periaquaductal gray = opiate
3rd order pain stuff
from thalamus to CNS higher centers
How does inflammation affect nerve endings?
- activates nociceptive receptors at nerve endings.
- increases threshold of firing, inc firing rate
= primary hyperalgesia - silent nociceptors activated (respond to inflammation in area) -> inc firing rate
- second messenger system activated = greater permeability and excitability
- catecholamines released as stress response = inc firing rate.
Peripheral nerve sensitization
- pain along nerve or distribution
- pain w/ palpation
- trigger points
- burning
- pain linked with stress
- off-loading nerve = relief
Central nervous sensitization
- not in neat boundaries
- spread
- multiple areas linked
- similar pain on contralateral side
- “chasing pain”
- temporal summation
- significant BPS factors
Sympathetic nervous system is regulated by what?
- brain and hypothalamus
- arousal, fear, and readiness. Part of upregulation in central sensitization
Cortical “smudging”
- decreased clarity of motor/sensory representation
- thought to explain why pain spreads.
Pain neuromatrix
Inputs: cognitive, sensory, emotion
Output: pain perception, action, stress-regulation
“neurotag”
- central sensitization
- pain “program”/memory/matrix
- recursive process between parts of brain related to a pain experience
A typical pain neurotag: 9 steps
1) pre-motor/motor
2) cingulate cortex
3) pre-frontal cortex
4) amygdala
5) sensory Cortex
6) hypothalamus/thalamus
7) Cerebellum
8) hippocampus
9) Spinal cord
Premotor/motor (neuro tag fx)
- organize and prepare movement
Cingulate cortex (neuro tag fx)
- concentration, focusing
Prefrontal Cortex (neuro tag fx)
- problem solving, memory
Amygdala (neuro tag fx)
- fear, fear conditioning, addiction