Exam 2 Flashcards

test/tools, intro, ear disorders, hearing impairment, rhinosinusitis (87 cards)

1
Q

Audiometry

A
  • tests for low/high pitch, loudness, and distinguishing voice and white noise
  • performed in office or by audiologist
  • Risk: none
  • Gold standard: pure tone audiology
  • technique: place headphones on ear, series of noises are played into headphones and pt signals when they hear nose
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2
Q

Tympanometry

A
  • detects problems with fluid/wax buildup, perforated eardrum, ossicle bone damage, tumor in middle ear
  • can dx otitis medis, hearing disorders
  • can be done in primary care or audiologist
  • procedure: insert tympanometer into ear, present a probe tone, want ear canal and middle ear air pressures to be equal
  • Looking for compliance: TM moves with small changes in pressure
  • Noncompliance: TM is stiff/does not move easily - reduced mobility
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3
Q

Newborn hearing test

A
  • 2 common tests
  • passing test does not indicate clear hearing in adult life
  • Otoacoustic emissions (OAE): test outer hair cells response to sound, not adequate for neural hearing loss or any middle ear pathology
  • Automated auditory brainstem response (AABR): auditory nerve and brainstem respond to sound, wears small earphones and electrodes, pass/fail results
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4
Q

Nasopharyngoscopy

A
  • allows visualization of structures from the nasal cavity to vocal cords
  • indicated for suspected tumors, polyps, obstructions, hypertrophied tonsils, vocal cord nodules
  • Fiberoptic gold standard
  • pt is sitting and tube is inserted through nare and down pharynx
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5
Q

Tympanocentesis

A

-small puncture through tympanic membrane to draw fluid or administer medication directly to middle ear

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6
Q

Pneumatic Otoscopy

A
  • examination that determines mobility of pt tympanic membrane in response to pressure changes
  • indicates presence of effusion
  • gold standard for TM
  • want to see movement
  • dx: otitis media with effusion, perforation, tympanosclerosis
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7
Q

Tympanostomy tubes

A
  • small plastic or metal tubes surgically placed in TM to allow drainage of fluid
  • indications: fluid in both ears for 3 months and hearing loss or problems with pain, balance, repeated infections
  • benefits: decrease ear infections, improved hearing
  • Risks: infection
  • tubes fall out in 1-2 years
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8
Q

Allergy Testing

A
  • skin test

- ELISA and RAST

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9
Q

Strep testing

A
  • test for antibodies of group A step from throat swab

- do culture if negative - gold standard

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10
Q

Bordetella

A
  • “whooping cough” requires pertussis test
  • collect nasopharyngeal secretions or blood
  • culture is gold standard
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11
Q

Rhinoplasty

A
  • nose job
  • changes nose shape or improve function
  • injury, breathing problems, devited septum, birth defect, cosmetic
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12
Q

Uvuloplasty

A
  • surgical remedy to snoring and some sleep apneas
  • removing part or entire uvula with laser
  • not covered by insurace
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13
Q

Perichondritis

A
  • infection of ear cartilage
  • serious
  • usually secondary to trauma or psedumonas
  • hard to cure due to poor vascularity
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14
Q

Cellulitis v. Perichondritis

A
  • cellulitis: soft tissue inflamed. Lobe is inflamed and it may spread upward
  • perichondritis: cartilage inflamed. lobe is not involved
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15
Q

Relapsing polychondritis

A
  • same, but bilateral

- probably auto-immune related

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16
Q

Otitis Externa

A
  • inflammatory disorders of the ear canal not middle ear
  • Culprits: psedumonas, fungas, staph aureus
  • Swimmer’s ear: excess water in ear canal
  • Malignant external otitis is often associated with DM
  • Sx: otalgia, pruritis, discharge
  • Exam: erythema, edema, pain, can’t really see TM
  • tx: otic drops (can use wick), abx if bacterial, isopropyl alcohol for drying, half-strength vinegar for fungal or psedumonal
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17
Q

Ceruminosis

A
  • accumulate cerumen
  • interferes with hearing
  • gently irrigate ear
  • Cerumenex soften wax and then irrigate
  • loops and scoops for stubborn wax
  • typically not painful, but if contacting TM very painful
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18
Q

Acute otitis media

A

-bacterial infection of middle ear
-S. pneumo, M. cat, H. flu, viruses very important as well
-precipitated by a URI
-more common in children
-

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19
Q

Acute Otitis Media Diagnosis

A
  • mobility of the TM is the gold standard for determining infection
  • single best predictor of AOM is TM immobility
  • combo of TM immobility and “cloudiness” is best
  • dx: moderate to severe TM bulge, mild TM bulge and ear pain/intense TM erythema
  • no effusion then not AOM
  • bulging TM, decreased/distorted/absent light reflex, redness
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20
Q

Acute otitis media risk factors

A
  • Age: 6mo-18mo
  • Gender: male>females
  • Day care centers
  • Pacifier use: slight increase
  • Breast feeding: decrease
  • Smoking
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21
Q

Acute otitis media history

A
  • PMHx: known/suspected medical problem, prior surgeries, current medications, allergies, hospitalizations, immunizations status
  • CC
  • HPI: symptoms, description of each, onset, course, sacred 7
  • ROS: fever, appeitite, URI sx, n/v/d, abd pain, lethargic, cough, runny nose, decreased hearing
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22
Q

Normal TM

A
  • translucent/transparent
  • gray or pink color
  • neutral position
  • fully mobile with pneumatic otoscope
  • no effusion
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23
Q

Acute Otitis Media TM characteristics

A
  • opaque
  • red, yellow, white color
  • Bulging or full position
  • reduced mobility but may respond to positive pressure on pneumatic otoscopy
  • effusion present
  • white/cloudy stuff is pus
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24
Q

Acute Otitis Media tx

A
  • decrease fever and pain
  • prevent developmental delays
  • 80% heal spontaneously
  • 94% w/ abx
  • tx pain: tylenol/ibuprofen, warm compresses
  • observation and waiting is a good tx plan if pt is not really sick
  • Amoxicillin: first line
  • Augmentin: second line, H. flu
  • S.pneumonia needs larger dose of amoxicillin not augment
  • should see improvement in 72 hours using abx
  • effusion can presist for 12 weeks
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25
Acute Otitis Media Prevention
- vaccines: pneumococcal vaccine and influenza | - ear tubes
26
Otitis Media w/ Effusion (OME)
- not really otitis as there is no infection - serious middle ear effusion - can last months - Worry about hearing: can affect language development - decreased mobility of TM - TM often retracted, but not always - occurs spontaneously b/c of poor estachian tube function or as inflammatory response following AOM - Referral to ENT - Place tubes
27
Otitis Media w/ Effusion (OME) TM characteristics
- translucent or opaque - gray or pink color - neutral or retracted position - reduced mobility, responds to negative pressure on pneumatic otoscopy - effusion present
28
OME concerns
- hearing loss - effects on speech, language, and learning - quality of life - need accurate diagnosis to distinguish AOM from OME - distinguishing OME in health child from child at risk for developmental delays
29
OME guidelines
- use pneumatic otoscopy as primary diagnostic method to distinguish OME from AOM - tympanometry is an option - distinguish child with OME who is at risk for speech, language, or learning problems and promptly evaluate and tx - children not at risk watch for 3 months
30
Hearing loss with OME
- HLs < 20 dB (normal hearing): repeat hearing test should be performed in 3-6 months if OME persists - HLs 21-39 dB (mild hearing loss): audiologic evaluation, repeat hearing test in 3-6 months if OME persists - HLs > 40 dB (moderate hearing loss): audiologic evaluation, surgery recommended, can impact speech, language, academic performance
31
Benign Paroxysmal Positional Vertigo (BPPV)
- benign, but scary - ever time someone moves they get very dizzy and throw up - get a club of cells that don't role to one side. This sends different signal to the brain from side to side and that gives you vertigo
32
Vertigo
- produced by: Meniere disease, BBPV, labyrinthitis, acoustic tumors - n/v are frequently associated with vertigo and dizziness
33
Maneuvers
- Dix-Hallpike: localize labyrinthine dysfunction - positive test with affected ear down, nystagmus toward affected ear - Epley: reposition otoliths - referral to audiology, ENT, or PT
34
Meniere disease
- inner ear disorder - AKA: idiopathic endolymphatic hydrops - Onset: 40-60 yr - Prevalence: 50-200 per 100,000 - too much fluid that causes a triad (tinnitus, hearing loss, vertigo) - caused by buildup of endolymph pressure - typically unilateral (nystagmus, possible ear fullness) - intermittent attacks (last minutes to hours, n/v, sweating, pallor, falling down) - Not positional: can't reproduce with changing head position like BBVP - Risk: white, genetic, stress, allergy - Differential: teriary syphilis, MS, vestibular schwannoma, BPPV - Lab: RPR (syphilis), CT
35
Meniere disease tx
- meclizine - scopolamine patch behind ear - diuretics to reduce fluid, low Na+ diet - endolymphatic shunt (surgical)
36
Vestibular neuronitis
- sudden onset of vertigo related to neural afferents w/o inflammation - vertigo due to imbalance in vestibular inputs - possible involvement of latent herpes simplex virus type 1
37
Labyrinthitis
- inflammatory disorder of inner ear or labyrinth - hearing loss, vertigo, w/wo tinnitus, systemic signs - hearing loss always present - tends NOT to recur
38
Vestibular neuronitis vs. labyrinthitis
- vestibular: no inflammation, does not having hearing loss | - labyrinthitis: inflammatory, hearing loss
39
ABRS
- bacterial infection of maxillary, frontal, or ethmoid sinuses - not viral, not inflamed, not allergic - acute onset - must distinguish between ABRS and viral illness
40
Parthenogenesis of ABRS
- Rhinitis to sinusitis - cold/URI to inflammation to bacterial infection - Mucociliary clearance is important in keeping sinuses clear of fluid
41
Microbial etiology for sinus infection
- Viruses: most common by far (rhinovirus, parainfluenza, etc.) - Bacterial: community acquired or nosocominal - Fungal: uncommon
42
Bacterial sinusitis
- community acquired: S.pneumoniae, H. influenza, M.catarrhalis and group A strep, Staph aureus - Nosocomial: nasogastric tubes or NG tube (staph, pseduomonas, and G-)
43
ABRS Guidelines Diagnosis
- persistent symptoms or sign for more than 10 days - onset with severe symptoms (high fever, purulent nasal discharge, facial pain) - "double-sickening": sick for a few days, got better and then got sick again (just as bad or worse)
44
ABRS Guidelines Therapy
- empiric antimicrobial therapy be initiated as soon as clinical diagnosis of ABRS is established - Amoxicillin-clavulanate not amoxicillin alone - alternative management if symptoms worsen (in 2-3 days) or fail to improve (3-5 days)
45
Further studies for ABRS if therapy is not working
- cultures be obtained by direct sinus aspiration (gold standard) - cultures of the middle meatus may be considered as alternative in adults - Nasopharyngeal cultures are unreliable - CT for pt with suspected suppurative complications (don't do MRI or sinus film typically) - seriously ill and immunocompromised, continue to deteriorate clinically despite extended courses of antimicrobial therapy, or have recurrent bouts of acute rhinosinusitis refer to ENT
46
ABRS signs and symptoms
- nasal secretions of any color - congestion and facial pressure / HA are common to both viral and bacterial - recent onset (<7-10 days) with no fevers likely to be viral not ABRS - failure to resolve in 7-10 days - higher fever / "severe" symptoms even if <7 days - "re-sickening" - foul odor for mouth - dental pain (maxillary - upper teeth) - Anosmia (can't smell)
47
ABRS red flags
- abnormal vision - change in mental status - periorbital edema - high fevers
48
ABRS physical exam
- vital signs: may be febrile or afebrile - eye: possible clear discharge, otherwise normal - nose: turbinates swollen, possible purulent discharge visible - throat: likely inflamed, absence of tonsillar exudates, possible posterior drainage, possible posterior pharyngeal cobblestoning - face: tenderness to palpation/percussion of maxillary and/or frontal sinuses (especially unilateral) - neck: possible anterior cervical lymphadenopathy - chest: should be normal, but cough possible
49
Cobblestoning
- clumps of hypertropied lymphoid tissue at the posterior pharynx due to CHRONIC postnasal drainage and irritation of the tissue - not specific for ABRS
50
ABRS tests
- transillumination - sinus puncture and aspiration (gold standard) - radiology: CT (very sensitive) only order for pt with recurret ABRS or suspect structural problem
51
ABRS treatment
- Antibiotics - pain/fever: antiinflammatories - congestion: decongestant -tx 5 days only "Afrin rule" - expectorants: guaifenisen (Mucinex/Robitussin) - Avoid 1st generation antihistamines (thickens secretions) - guidelines support saline irrigation and intranasal corticosteroids
52
Complications for ABRS
- periorbital tissues - osteomyelitis - CNS: meningitis, brain abscess - chronic sinusitis - paranasal sinus cancer should always be a differential of sinusitis
53
AVRS
- AKA: URI - do not ABRS specific signs - almost never a fever (possibly a low grade at beginning) - shorter duration: resolves on own in 5-7 days - similar c/o w/ ABRS (congestion, nasal d/c, ha, pressure, possible tenderness, fluid in sinuses, possible associated hx of allergies)
54
AVRS treatment
- treat symptoms - avoid antibiotics - be alert for ABRS - must decided if viral or bacterial - viral: block inflammation events (antihistamines, NSAID's, cough suppressant, decongestants, mucolytics)
55
Hadley's take on sinus infection tx
- if bacterial tx with augmentin with doxy second - Afrin 2 spays each nostril BID x4 day THEN STOP - anti-inflammatory meds (ibuprofen/naproxen) for comfort - Guaifenesin (mucinex/robitussin) also drink a lot of fluids - maybe saline sinus rinse if copious discharge - if allergic trigger nasal steroid spray (not Flonase). Start concurrently with Afrin and continue long term, consider chronic antihistamine - pt education (why they are not getting antibiotic)
56
Chronic sinusitis sx
- chronic (>12 weeks) - anterior or posterior mucopurulent drainage - nasal obstruction - facial pain/fullness/tenderness - purulent mucus or edema in middle meatus or ethmoid region OR polyps in nasal cavity or middle meatus OR imaging showing inflammation
57
Chronic sinusitis etiology
- persistent infection - allergy and other immunologic disorder - intrinsic factors of upper airway - superantigens - colonizing fungi that induce/sustain eosinophilic inflammation - aspirin sensitivity - all affecting mucociliary clearance
58
Chronic sinusitis history
- major symptoms: purulent anterior / posterior nasal drainage, nasal obstruction or blockage, facial congestion/fullness, facial pain/pressure, hyposmia, anosmia - minor symptoms: HA, ear pain/fullness, halitosis, dental pain, cough, fever, fatigue - duration - hx of previous sinus surgery - hx of asthma, allergies, immunocompromising disease - active or passive tobacco use
59
Chronic sinusitis physical exam
- nasal exam: purulent drianage, polyps , septal deviation, turbinate hypertrophy/edema, consider endoscopic exam - sinus palpation/percussion for tenderness - consider sinus CT - Ears: TM fluid - Neck: LAD - throat: postnasal drip - Eye: oculomotor involvement - lung: lower infection / asthma
60
Allergic rhinosinusitis
- non-infectious - look for allergic history - chronicity: possible recurrent ABRS or AVRS - clear rhinorrhea with associated allergic sx (sneezing, itching eyes/ears, itchy throat) - benign PE, possible swollen/boggy turbinates, cobblestoning, clear eye discharge, clear fluids behind TMs
61
Allergic triggers
- seasonal allergic rhnitis (pollens) - Perennial allergic rhinitis: symptoms all year round (dust mites, mold, pet dander) - allergy and asthma are closely tied; suspect one when you see the other
62
Allergic treatment
- avoidance therapy - drug therapy: inhibit release and action of mediators, reversal of inflammatory response - Immunotherapy: repeated long term injection (for one specific allergy - not multiples)
63
Drug Allergic treatment
- antihistamines - anticholergic agents: stop mucus secretion - corticosteroid nasal spray: anti-inflammatory - mast cell stabilizer: cromolyn sodium and sodium nedocromil - Leukotriene antagonists: give symptomatic relief by inhibiting vascular permeability and eosinophilic inflammation - nasal saline lavage: wash away mucus
64
Vasomotor rhinitis
- one form of nonallergic rhinitis - triggers are cold air, strong odors, stress, or inhaled irritants - sx are rhinorrhea, sneezing, and congestion - due to parasympathetic overactivity of nasopharynx
65
Vasomotor rhinitis treatment
- avoidance of triggers - Ipatropium nasal spray is anticholinergic - Intranasal steroid of unclear benefit - oral antihistamines (1st gen) may help due to anticholinergic effects - intranasal antihistamines: Astelazine -anti-inflammatory - Intranasal and oral sympathomimetics: oxymetazolien, pseduoephedrime (decongestants) - can help promote vasoconstriction
66
Vasomotor rhinitis relatives
- other nonallergic rhinitis - occupational rhinitis - hormonal rhinitis: estrogen increase (puberty most common cause) - drug induced rhinitis: over use of topical decongestants - gustatory rhinitis: response to spicy foods - NARES (nonallergic rhinitis with eosinophilia syndrome) abnormal prostaglandin metabolism, can precede NSAID sensitivity
67
Nasal polyps
- associated with asthma, chronic sinus infections, cystric fibrosis, allergic rhinitis, hypoosmia - outgrowth of nasal mucosa - can be removed, but most return
68
Nasal polyps triad
- ASA sensitivity is found in 1/3 of pt with polyps, rhinosinusitis, and asthma - chronic rhinitis followed by astham and asa sensitivity with subsequent development of nasal polyposis - type 1 hypersensitivity - sx: watery eyes, rhinorrhea, flushing of head, neck, and chest, bronchoconstriction, wheezing, cyanosis, n/v/d, cramps - very responsive to bronchodilators
69
Symptoms associated with hearing loss
- tinnitus - hyperacusis - vertigo/dizziness - aural fullness/pain - otorrhea - delayed speech / language development in children
70
Type A tympanogram
- normal - TM intact - like looks like pointy mountain - pressure peak is at 0 (equal pressure) - Type As: "s" is shallow or stiff, suggest middle ear system (ossicular fixation, thickened/scarred TM) - type Ad: "d" is deep, can suggest ossicular discontinuity, also seen with monomeric, healed TMs, post PE tube or perforation
71
Type B tympanogram
- opposite of type A - "flat" - consistent with middle ear fluid - flat tymp PLUS high volume indicates TM perforation or pt has PE tube
72
Type C tympanogram
- negative middle ear pressure / TM retraction - consistent with eustachian tube dysfunction - 0 +/- 100daPa range is normal - peak off center
73
Sound treated booth
- only proper way to perform a hearing evaluation | - hearing test not done in booth is a hearing screening
74
Audiometric symbols
- right is red - blue is left - O---O right ear, air conduction - X---X left ear, air conduction - (blue) unmasked bone conduction
75
Types of hearing loss
- sensorineural (SNHL) - conductive - mixed - central - seduohypacusis
76
Causes of sensorineural hearing loss
- congenital/genetic - presbycusis - bacterial/viral infection - medications / ototoxicity - acoustic nerve tumors - meniere's disease - sudden: autoimmune ear disease, vascular, perilymphatic, fistula, unknown - acoustic trauma / noise exposure
77
Noise induced hearing loss
- type of SNHL | - most common occupational illness or injury
78
SNHL treatment
- prevention / education/ hearing conservation measures - amplification (hearing aids) - medications (sudden loss, Meniere's, autoimmune ear disease) - surgery (cochlear/brainstem implant)
79
Conductive hearing loss
- inner ear is fine, but sound is not conducted to the inner ear - air conduction issue
80
Causes of conductive loss
- cerumen impaction - foreign body - external ear infection - middle ear fluid / infection - TM perforation - otosclerosis - tumors /growths in external or middle ear - congenital anomalies
81
Conductive hearing loss treatment
- cerumen management - surgery (for the 10% that can be corrected) - medication - amplification
82
Conductive hearing loss treatment
- cerumen management - surgery (for the 10% that can be corrected) - medication - amplification (hearing aids)
83
Mixed hearing loss
-combination of SNHL and conductive hearing loss
84
Central hearing loss
- stroke or head injury is effecting hearing | - anatomy of ear and nerve is working fine
85
Pseudohypacusis hearing loss
- faked hearing loss | - can be purposeful or psych related
86
Hearing loss prevention
- hearing protection - education - screening and early intervention - healthy lifestyle and exercise
87
Chronic sinusitis treatment
- bugs: staph aureus, coagulase negative staph, anaerobes, G-, fungus - Abx tx for 3-4 weeks - intranasal steroids, saline, oral steroids, decongestants, mucolytics - smoking cessation - surgery if anatomical cause found