Exam 2 Flashcards

test/tools, intro, ear disorders, hearing impairment, rhinosinusitis

1
Q

Audiometry

A
  • tests for low/high pitch, loudness, and distinguishing voice and white noise
  • performed in office or by audiologist
  • Risk: none
  • Gold standard: pure tone audiology
  • technique: place headphones on ear, series of noises are played into headphones and pt signals when they hear nose
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2
Q

Tympanometry

A
  • detects problems with fluid/wax buildup, perforated eardrum, ossicle bone damage, tumor in middle ear
  • can dx otitis medis, hearing disorders
  • can be done in primary care or audiologist
  • procedure: insert tympanometer into ear, present a probe tone, want ear canal and middle ear air pressures to be equal
  • Looking for compliance: TM moves with small changes in pressure
  • Noncompliance: TM is stiff/does not move easily - reduced mobility
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3
Q

Newborn hearing test

A
  • 2 common tests
  • passing test does not indicate clear hearing in adult life
  • Otoacoustic emissions (OAE): test outer hair cells response to sound, not adequate for neural hearing loss or any middle ear pathology
  • Automated auditory brainstem response (AABR): auditory nerve and brainstem respond to sound, wears small earphones and electrodes, pass/fail results
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4
Q

Nasopharyngoscopy

A
  • allows visualization of structures from the nasal cavity to vocal cords
  • indicated for suspected tumors, polyps, obstructions, hypertrophied tonsils, vocal cord nodules
  • Fiberoptic gold standard
  • pt is sitting and tube is inserted through nare and down pharynx
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5
Q

Tympanocentesis

A

-small puncture through tympanic membrane to draw fluid or administer medication directly to middle ear

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6
Q

Pneumatic Otoscopy

A
  • examination that determines mobility of pt tympanic membrane in response to pressure changes
  • indicates presence of effusion
  • gold standard for TM
  • want to see movement
  • dx: otitis media with effusion, perforation, tympanosclerosis
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7
Q

Tympanostomy tubes

A
  • small plastic or metal tubes surgically placed in TM to allow drainage of fluid
  • indications: fluid in both ears for 3 months and hearing loss or problems with pain, balance, repeated infections
  • benefits: decrease ear infections, improved hearing
  • Risks: infection
  • tubes fall out in 1-2 years
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8
Q

Allergy Testing

A
  • skin test

- ELISA and RAST

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9
Q

Strep testing

A
  • test for antibodies of group A step from throat swab

- do culture if negative - gold standard

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10
Q

Bordetella

A
  • “whooping cough” requires pertussis test
  • collect nasopharyngeal secretions or blood
  • culture is gold standard
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11
Q

Rhinoplasty

A
  • nose job
  • changes nose shape or improve function
  • injury, breathing problems, devited septum, birth defect, cosmetic
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12
Q

Uvuloplasty

A
  • surgical remedy to snoring and some sleep apneas
  • removing part or entire uvula with laser
  • not covered by insurace
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13
Q

Perichondritis

A
  • infection of ear cartilage
  • serious
  • usually secondary to trauma or psedumonas
  • hard to cure due to poor vascularity
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14
Q

Cellulitis v. Perichondritis

A
  • cellulitis: soft tissue inflamed. Lobe is inflamed and it may spread upward
  • perichondritis: cartilage inflamed. lobe is not involved
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15
Q

Relapsing polychondritis

A
  • same, but bilateral

- probably auto-immune related

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16
Q

Otitis Externa

A
  • inflammatory disorders of the ear canal not middle ear
  • Culprits: psedumonas, fungas, staph aureus
  • Swimmer’s ear: excess water in ear canal
  • Malignant external otitis is often associated with DM
  • Sx: otalgia, pruritis, discharge
  • Exam: erythema, edema, pain, can’t really see TM
  • tx: otic drops (can use wick), abx if bacterial, isopropyl alcohol for drying, half-strength vinegar for fungal or psedumonal
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17
Q

Ceruminosis

A
  • accumulate cerumen
  • interferes with hearing
  • gently irrigate ear
  • Cerumenex soften wax and then irrigate
  • loops and scoops for stubborn wax
  • typically not painful, but if contacting TM very painful
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18
Q

Acute otitis media

A

-bacterial infection of middle ear
-S. pneumo, M. cat, H. flu, viruses very important as well
-precipitated by a URI
-more common in children
-

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19
Q

Acute Otitis Media Diagnosis

A
  • mobility of the TM is the gold standard for determining infection
  • single best predictor of AOM is TM immobility
  • combo of TM immobility and “cloudiness” is best
  • dx: moderate to severe TM bulge, mild TM bulge and ear pain/intense TM erythema
  • no effusion then not AOM
  • bulging TM, decreased/distorted/absent light reflex, redness
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20
Q

Acute otitis media risk factors

A
  • Age: 6mo-18mo
  • Gender: male>females
  • Day care centers
  • Pacifier use: slight increase
  • Breast feeding: decrease
  • Smoking
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21
Q

Acute otitis media history

A
  • PMHx: known/suspected medical problem, prior surgeries, current medications, allergies, hospitalizations, immunizations status
  • CC
  • HPI: symptoms, description of each, onset, course, sacred 7
  • ROS: fever, appeitite, URI sx, n/v/d, abd pain, lethargic, cough, runny nose, decreased hearing
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22
Q

Normal TM

A
  • translucent/transparent
  • gray or pink color
  • neutral position
  • fully mobile with pneumatic otoscope
  • no effusion
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23
Q

Acute Otitis Media TM characteristics

A
  • opaque
  • red, yellow, white color
  • Bulging or full position
  • reduced mobility but may respond to positive pressure on pneumatic otoscopy
  • effusion present
  • white/cloudy stuff is pus
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24
Q

Acute Otitis Media tx

A
  • decrease fever and pain
  • prevent developmental delays
  • 80% heal spontaneously
  • 94% w/ abx
  • tx pain: tylenol/ibuprofen, warm compresses
  • observation and waiting is a good tx plan if pt is not really sick
  • Amoxicillin: first line
  • Augmentin: second line, H. flu
  • S.pneumonia needs larger dose of amoxicillin not augment
  • should see improvement in 72 hours using abx
  • effusion can presist for 12 weeks
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25
Q

Acute Otitis Media Prevention

A
  • vaccines: pneumococcal vaccine and influenza

- ear tubes

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26
Q

Otitis Media w/ Effusion (OME)

A
  • not really otitis as there is no infection
  • serious middle ear effusion
  • can last months
  • Worry about hearing: can affect language development
  • decreased mobility of TM
  • TM often retracted, but not always
  • occurs spontaneously b/c of poor estachian tube function or as inflammatory response following AOM
  • Referral to ENT
  • Place tubes
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27
Q

Otitis Media w/ Effusion (OME) TM characteristics

A
  • translucent or opaque
  • gray or pink color
  • neutral or retracted position
  • reduced mobility, responds to negative pressure on pneumatic otoscopy
  • effusion present
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28
Q

OME concerns

A
  • hearing loss
  • effects on speech, language, and learning
  • quality of life
  • need accurate diagnosis to distinguish AOM from OME
  • distinguishing OME in health child from child at risk for developmental delays
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29
Q

OME guidelines

A
  • use pneumatic otoscopy as primary diagnostic method to distinguish OME from AOM
  • tympanometry is an option
  • distinguish child with OME who is at risk for speech, language, or learning problems and promptly evaluate and tx
  • children not at risk watch for 3 months
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30
Q

Hearing loss with OME

A
  • HLs < 20 dB (normal hearing): repeat hearing test should be performed in 3-6 months if OME persists
  • HLs 21-39 dB (mild hearing loss): audiologic evaluation, repeat hearing test in 3-6 months if OME persists
  • HLs > 40 dB (moderate hearing loss): audiologic evaluation, surgery recommended, can impact speech, language, academic performance
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31
Q

Benign Paroxysmal Positional Vertigo (BPPV)

A
  • benign, but scary
  • ever time someone moves they get very dizzy and throw up
  • get a club of cells that don’t role to one side. This sends different signal to the brain from side to side and that gives you vertigo
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32
Q

Vertigo

A
  • produced by: Meniere disease, BBPV, labyrinthitis, acoustic tumors
  • n/v are frequently associated with vertigo and dizziness
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33
Q

Maneuvers

A
  • Dix-Hallpike: localize labyrinthine dysfunction - positive test with affected ear down, nystagmus toward affected ear
  • Epley: reposition otoliths - referral to audiology, ENT, or PT
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34
Q

Meniere disease

A
  • inner ear disorder
  • AKA: idiopathic endolymphatic hydrops
  • Onset: 40-60 yr
  • Prevalence: 50-200 per 100,000
  • too much fluid that causes a triad (tinnitus, hearing loss, vertigo)
  • caused by buildup of endolymph pressure
  • typically unilateral (nystagmus, possible ear fullness)
  • intermittent attacks (last minutes to hours, n/v, sweating, pallor, falling down)
  • Not positional: can’t reproduce with changing head position like BBVP
  • Risk: white, genetic, stress, allergy
  • Differential: teriary syphilis, MS, vestibular schwannoma, BPPV
  • Lab: RPR (syphilis), CT
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35
Q

Meniere disease tx

A
  • meclizine
  • scopolamine patch behind ear
  • diuretics to reduce fluid, low Na+ diet
  • endolymphatic shunt (surgical)
36
Q

Vestibular neuronitis

A
  • sudden onset of vertigo related to neural afferents w/o inflammation
  • vertigo due to imbalance in vestibular inputs
  • possible involvement of latent herpes simplex virus type 1
37
Q

Labyrinthitis

A
  • inflammatory disorder of inner ear or labyrinth
  • hearing loss, vertigo, w/wo tinnitus, systemic signs
  • hearing loss always present
  • tends NOT to recur
38
Q

Vestibular neuronitis vs. labyrinthitis

A
  • vestibular: no inflammation, does not having hearing loss

- labyrinthitis: inflammatory, hearing loss

39
Q

ABRS

A
  • bacterial infection of maxillary, frontal, or ethmoid sinuses
  • not viral, not inflamed, not allergic
  • acute onset
  • must distinguish between ABRS and viral illness
40
Q

Parthenogenesis of ABRS

A
  • Rhinitis to sinusitis
  • cold/URI to inflammation to bacterial infection
  • Mucociliary clearance is important in keeping sinuses clear of fluid
41
Q

Microbial etiology for sinus infection

A
  • Viruses: most common by far (rhinovirus, parainfluenza, etc.)
  • Bacterial: community acquired or nosocominal
  • Fungal: uncommon
42
Q

Bacterial sinusitis

A
  • community acquired: S.pneumoniae, H. influenza, M.catarrhalis and group A strep, Staph aureus
  • Nosocomial: nasogastric tubes or NG tube (staph, pseduomonas, and G-)
43
Q

ABRS Guidelines Diagnosis

A
  • persistent symptoms or sign for more than 10 days
  • onset with severe symptoms (high fever, purulent nasal discharge, facial pain)
  • “double-sickening”: sick for a few days, got better and then got sick again (just as bad or worse)
44
Q

ABRS Guidelines Therapy

A
  • empiric antimicrobial therapy be initiated as soon as clinical diagnosis of ABRS is established
  • Amoxicillin-clavulanate not amoxicillin alone
  • alternative management if symptoms worsen (in 2-3 days) or fail to improve (3-5 days)
45
Q

Further studies for ABRS if therapy is not working

A
  • cultures be obtained by direct sinus aspiration (gold standard)
  • cultures of the middle meatus may be considered as alternative in adults
  • Nasopharyngeal cultures are unreliable
  • CT for pt with suspected suppurative complications (don’t do MRI or sinus film typically)
  • seriously ill and immunocompromised, continue to deteriorate clinically despite extended courses of antimicrobial therapy, or have recurrent bouts of acute rhinosinusitis refer to ENT
46
Q

ABRS signs and symptoms

A
  • nasal secretions of any color
  • congestion and facial pressure / HA are common to both viral and bacterial
  • recent onset (<7-10 days) with no fevers likely to be viral not ABRS
  • failure to resolve in 7-10 days
  • higher fever / “severe” symptoms even if <7 days
  • “re-sickening”
  • foul odor for mouth
  • dental pain (maxillary - upper teeth)
  • Anosmia (can’t smell)
47
Q

ABRS red flags

A
  • abnormal vision
  • change in mental status
  • periorbital edema
  • high fevers
48
Q

ABRS physical exam

A
  • vital signs: may be febrile or afebrile
  • eye: possible clear discharge, otherwise normal
  • nose: turbinates swollen, possible purulent discharge visible
  • throat: likely inflamed, absence of tonsillar exudates, possible posterior drainage, possible posterior pharyngeal cobblestoning
  • face: tenderness to palpation/percussion of maxillary and/or frontal sinuses (especially unilateral)
  • neck: possible anterior cervical lymphadenopathy
  • chest: should be normal, but cough possible
49
Q

Cobblestoning

A
  • clumps of hypertropied lymphoid tissue at the posterior pharynx due to CHRONIC postnasal drainage and irritation of the tissue
  • not specific for ABRS
50
Q

ABRS tests

A
  • transillumination
  • sinus puncture and aspiration (gold standard)
  • radiology: CT (very sensitive) only order for pt with recurret ABRS or suspect structural problem
51
Q

ABRS treatment

A
  • Antibiotics
  • pain/fever: antiinflammatories
  • congestion: decongestant -tx 5 days only “Afrin rule”
  • expectorants: guaifenisen (Mucinex/Robitussin)
  • Avoid 1st generation antihistamines (thickens secretions)
  • guidelines support saline irrigation and intranasal corticosteroids
52
Q

Complications for ABRS

A
  • periorbital tissues
  • osteomyelitis
  • CNS: meningitis, brain abscess
  • chronic sinusitis
  • paranasal sinus cancer should always be a differential of sinusitis
53
Q

AVRS

A
  • AKA: URI
  • do not ABRS specific signs
  • almost never a fever (possibly a low grade at beginning)
  • shorter duration: resolves on own in 5-7 days
  • similar c/o w/ ABRS (congestion, nasal d/c, ha, pressure, possible tenderness, fluid in sinuses, possible associated hx of allergies)
54
Q

AVRS treatment

A
  • treat symptoms
  • avoid antibiotics
  • be alert for ABRS
  • must decided if viral or bacterial
  • viral: block inflammation events (antihistamines, NSAID’s, cough suppressant, decongestants, mucolytics)
55
Q

Hadley’s take on sinus infection tx

A
  • if bacterial tx with augmentin with doxy second
  • Afrin 2 spays each nostril BID x4 day THEN STOP
  • anti-inflammatory meds (ibuprofen/naproxen) for comfort
  • Guaifenesin (mucinex/robitussin) also drink a lot of fluids
  • maybe saline sinus rinse if copious discharge
  • if allergic trigger nasal steroid spray (not Flonase). Start concurrently with Afrin and continue long term, consider chronic antihistamine
  • pt education (why they are not getting antibiotic)
56
Q

Chronic sinusitis sx

A
  • chronic (>12 weeks)
  • anterior or posterior mucopurulent drainage
  • nasal obstruction
  • facial pain/fullness/tenderness
  • purulent mucus or edema in middle meatus or ethmoid region OR polyps in nasal cavity or middle meatus OR imaging showing inflammation
57
Q

Chronic sinusitis etiology

A
  • persistent infection
  • allergy and other immunologic disorder
  • intrinsic factors of upper airway
  • superantigens
  • colonizing fungi that induce/sustain eosinophilic inflammation
  • aspirin sensitivity
  • all affecting mucociliary clearance
58
Q

Chronic sinusitis history

A
  • major symptoms: purulent anterior / posterior nasal drainage, nasal obstruction or blockage, facial congestion/fullness, facial pain/pressure, hyposmia, anosmia
  • minor symptoms: HA, ear pain/fullness, halitosis, dental pain, cough, fever, fatigue
  • duration
  • hx of previous sinus surgery
  • hx of asthma, allergies, immunocompromising disease
  • active or passive tobacco use
59
Q

Chronic sinusitis physical exam

A
  • nasal exam: purulent drianage, polyps , septal deviation, turbinate hypertrophy/edema, consider endoscopic exam
  • sinus palpation/percussion for tenderness - consider sinus CT
  • Ears: TM fluid
  • Neck: LAD
  • throat: postnasal drip
  • Eye: oculomotor involvement
  • lung: lower infection / asthma
60
Q

Allergic rhinosinusitis

A
  • non-infectious
  • look for allergic history
  • chronicity: possible recurrent ABRS or AVRS
  • clear rhinorrhea with associated allergic sx (sneezing, itching eyes/ears, itchy throat)
  • benign PE, possible swollen/boggy turbinates, cobblestoning, clear eye discharge, clear fluids behind TMs
61
Q

Allergic triggers

A
  • seasonal allergic rhnitis (pollens)
  • Perennial allergic rhinitis: symptoms all year round (dust mites, mold, pet dander)
  • allergy and asthma are closely tied; suspect one when you see the other
62
Q

Allergic treatment

A
  • avoidance therapy
  • drug therapy: inhibit release and action of mediators, reversal of inflammatory response
  • Immunotherapy: repeated long term injection (for one specific allergy - not multiples)
63
Q

Drug Allergic treatment

A
  • antihistamines
  • anticholergic agents: stop mucus secretion
  • corticosteroid nasal spray: anti-inflammatory
  • mast cell stabilizer: cromolyn sodium and sodium nedocromil
  • Leukotriene antagonists: give symptomatic relief by inhibiting vascular permeability and eosinophilic inflammation
  • nasal saline lavage: wash away mucus
64
Q

Vasomotor rhinitis

A
  • one form of nonallergic rhinitis
  • triggers are cold air, strong odors, stress, or inhaled irritants
  • sx are rhinorrhea, sneezing, and congestion
  • due to parasympathetic overactivity of nasopharynx
65
Q

Vasomotor rhinitis treatment

A
  • avoidance of triggers
  • Ipatropium nasal spray is anticholinergic
  • Intranasal steroid of unclear benefit
  • oral antihistamines (1st gen) may help due to anticholinergic effects
  • intranasal antihistamines: Astelazine -anti-inflammatory
  • Intranasal and oral sympathomimetics: oxymetazolien, pseduoephedrime (decongestants) - can help promote vasoconstriction
66
Q

Vasomotor rhinitis relatives

A
  • other nonallergic rhinitis
  • occupational rhinitis
  • hormonal rhinitis: estrogen increase (puberty most common cause)
  • drug induced rhinitis: over use of topical decongestants
  • gustatory rhinitis: response to spicy foods
  • NARES (nonallergic rhinitis with eosinophilia syndrome) abnormal prostaglandin metabolism, can precede NSAID sensitivity
67
Q

Nasal polyps

A
  • associated with asthma, chronic sinus infections, cystric fibrosis, allergic rhinitis, hypoosmia
  • outgrowth of nasal mucosa
  • can be removed, but most return
68
Q

Nasal polyps triad

A
  • ASA sensitivity is found in 1/3 of pt with polyps, rhinosinusitis, and asthma
  • chronic rhinitis followed by astham and asa sensitivity with subsequent development of nasal polyposis
  • type 1 hypersensitivity
  • sx: watery eyes, rhinorrhea, flushing of head, neck, and chest, bronchoconstriction, wheezing, cyanosis, n/v/d, cramps
  • very responsive to bronchodilators
69
Q

Symptoms associated with hearing loss

A
  • tinnitus
  • hyperacusis
  • vertigo/dizziness
  • aural fullness/pain
  • otorrhea
  • delayed speech / language development in children
70
Q

Type A tympanogram

A
  • normal
  • TM intact
  • like looks like pointy mountain
  • pressure peak is at 0 (equal pressure)
  • Type As: “s” is shallow or stiff, suggest middle ear system (ossicular fixation, thickened/scarred TM)
  • type Ad: “d” is deep, can suggest ossicular discontinuity, also seen with monomeric, healed TMs, post PE tube or perforation
71
Q

Type B tympanogram

A
  • opposite of type A
  • “flat”
  • consistent with middle ear fluid
  • flat tymp PLUS high volume indicates TM perforation or pt has PE tube
72
Q

Type C tympanogram

A
  • negative middle ear pressure / TM retraction
  • consistent with eustachian tube dysfunction
  • 0 +/- 100daPa range is normal
  • peak off center
73
Q

Sound treated booth

A
  • only proper way to perform a hearing evaluation

- hearing test not done in booth is a hearing screening

74
Q

Audiometric symbols

A
  • right is red
  • blue is left
  • O—O right ear, air conduction
  • X—X left ear, air conduction
  • (blue) unmasked bone conduction
75
Q

Types of hearing loss

A
  • sensorineural (SNHL)
  • conductive
  • mixed
  • central
  • seduohypacusis
76
Q

Causes of sensorineural hearing loss

A
  • congenital/genetic
  • presbycusis
  • bacterial/viral infection
  • medications / ototoxicity
  • acoustic nerve tumors
  • meniere’s disease
  • sudden: autoimmune ear disease, vascular, perilymphatic, fistula, unknown
  • acoustic trauma / noise exposure
77
Q

Noise induced hearing loss

A
  • type of SNHL

- most common occupational illness or injury

78
Q

SNHL treatment

A
  • prevention / education/ hearing conservation measures
  • amplification (hearing aids)
  • medications (sudden loss, Meniere’s, autoimmune ear disease)
  • surgery (cochlear/brainstem implant)
79
Q

Conductive hearing loss

A
  • inner ear is fine, but sound is not conducted to the inner ear
  • air conduction issue
80
Q

Causes of conductive loss

A
  • cerumen impaction
  • foreign body
  • external ear infection
  • middle ear fluid / infection
  • TM perforation
  • otosclerosis
  • tumors /growths in external or middle ear
  • congenital anomalies
81
Q

Conductive hearing loss treatment

A
  • cerumen management
  • surgery (for the 10% that can be corrected)
  • medication
  • amplification
82
Q

Conductive hearing loss treatment

A
  • cerumen management
  • surgery (for the 10% that can be corrected)
  • medication
  • amplification (hearing aids)
83
Q

Mixed hearing loss

A

-combination of SNHL and conductive hearing loss

84
Q

Central hearing loss

A
  • stroke or head injury is effecting hearing

- anatomy of ear and nerve is working fine

85
Q

Pseudohypacusis hearing loss

A
  • faked hearing loss

- can be purposeful or psych related

86
Q

Hearing loss prevention

A
  • hearing protection
  • education
  • screening and early intervention
  • healthy lifestyle and exercise
87
Q

Chronic sinusitis treatment

A
  • bugs: staph aureus, coagulase negative staph, anaerobes, G-, fungus
  • Abx tx for 3-4 weeks
  • intranasal steroids, saline, oral steroids, decongestants, mucolytics
  • smoking cessation
  • surgery if anatomical cause found