Exam 2 Flashcards

1
Q

Define trauma from occlusion.

A

Damage to the periodontium caused by stress on the teeth produced directly or indirectly by teeth in opposing jaw

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2
Q

Trauma from occlusion happens when? And is considered to be?

A

Considered to be pathologic.

Forces of occlusion that exceed the adaptive capacity of the periodontium.

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3
Q

What are the variables to the relationship of occlusal trauma to periodontal disease?

A
  • Direction of force
  • Magnitude of force
  • Duration of force
  • Frequency of occurance
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4
Q

What parts of the periodontium are affected by occlusal forces?

A
  • Cementum
  • PDL
  • Alveolar bone proper
  • NOT - the gingiva and junctional epithelium
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5
Q

What is fremitus?

A

A tremulous vibratory movement of a tooth when teeth come into functional contact - generally detected by a finger palpation

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6
Q

What happens during trauma from occlusion on the compression side?

A
  • Compression of PDL fibers - initial decrease in width of PDL space
  • Loss of fiber orientation
  • Rupture of capillaries and hemorrhage into PDL spaces
  • Resorption of alveolar bone proper followed by widening ot PDL space
  • Root resorption if severe
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7
Q

What happens during trauma from occlusion on the tension side?

A
  • Tension (stretching) of fibers with initial increase in PDL space
  • Rupture of PDL fiber bundles
  • Compression of PDL capillaries and hemorrhage into PDL perivascular spaces
  • Apposition of new alveolar bone proper followed by decrease in PDL space
  • Cemental tearing
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8
Q

When does Primary Occlusal Trauma occur?

A

When occlusal forces are excessive (increased) and the amount of alveolar bone support is normal.

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9
Q

When does Secondary Occlusal Trauma occur?

A

When occlusal forces are normal or excessive and the amount of alveolar bone support is reduced.

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10
Q

What is occlusal hyperfunction, and what is it considered to be?

A

Slight increase in occlusal force, considered to be a physiologic adaptation and not a pathologic entity.

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11
Q

What are the clinical characteristics of Occlusal Hyperfunction?

A
  • Increase in # and diameter of collagen fiber bundles in PDL
  • Increased width of PDL
  • Increased density and thickness of alveolar bone proper (lamina dura)
  • Radiographic evidence of osteosclerosis of alveolar bone with PDL insertions
  • Slight or undetectable tooth mobility
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12
Q

What are the characteristics of Occlusal Hypofunction?

A
  • Mild weakening of the tooth supporting structures due to a lack of physiologic stimulation
  • Considered to be a physiologic adaptation and not a pathologic entity.
  • Can only be diagnosed by histology
  • Decrease in # of PDL fiber bundles but normal orientation
  • Decrease physiologic turnover and remodeling of alveolar bone
  • Narrowing of PDL space, no change in tooth mobility
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13
Q

What is the total removal of occlusal forces resulting in lack of the level of physiologic stimulation required to maintain normal form and function called?

A

Disuse Atrophy

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14
Q

What is disuse atrophy considered to be?

A

A physiologic adaptation rather than a pathologic feature of disease

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15
Q

What are the clinical characteristics of disuse atrophy?

A
  • Radiographic evidence of decreased width of PDL space
  • Increased tooth mobility - always
  • Absence of occlusal antagonist
  • Loss of orientation of principle fiber bundles of PDL
  • Significant decrease in # of bone trabeculae (localized osteoporosis)
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16
Q

Trauma from occlusion, in the absence of inflammation does not cause:

A
  • Gingivitis
  • Periodontitis
  • Pocket formation

But it makes these worse

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17
Q

What is the key concept of the animal study dealing with occlusal trauma and periodontal disease?

A
  • Periodontitis with superimposed occlusal trauma produces more bone loss than periodontitis alone
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18
Q

What is the key concept in the human study dealing with occlusal trauma and periodontal disease?

A
  • Strong association between initial occlusal discrepancies and periodontal prognosis and increased periodontal probing depth of posterior teeth
  • Strong association between untreated occlusal discrepancies and the progression of periodontal disease.
  • Occlusal treatment significantly reduces the progression of periodontal disease over time.
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19
Q

What is the creation of additional problems or complications as a result of treatment called?

A

Iatrogenic Disease

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20
Q

Gingival margin overhangs are associated with what?

A
  • Gingival inflammation
  • Bone loss
  • Microbial plaque and calculus accumulation
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21
Q

For a furcation involvement - how much bone loss must there be?

A

33%

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22
Q

What are some characteristics of interproximal caries?

A
  • Occur below the contact point
  • Follows the direction of enamel rods
  • Cone-shaped radiolucency
  • Spreads along the DEJ
23
Q

What is the microbial agent for interproximal caries?

A

Streptococcus Mutans

24
Q

What are some characteristics of Root Caries?

A
  • Involves cementum and dentin
  • Initiated in areas of food impaction
  • Common in periodontal patients following surgical exposure of root surfaces
  • Saucer-shaped morphology
  • Enamel is not involved
  • Microbial agent:
    • Actinomyces viscosus
    • Candida albicans?
25
What does caries detection by radiographic exam require?
* Very thorough clinical exam * Flawless radiographic technique * Experience in radiographic interpretation
26
How sensitive are X-rays in the diagnosis of proximal caries?
60%
27
What are the modifying factors of furcation involvement?
* Anatomic * Supervised neglect * Difficult cleansibility * Restorations
28
For a maxillary molar - what are the facial, mesial, and distal furcation depths?
* Facial - 4 mm * Mesial - 3 mm * Distal - 5 mm
29
For a maxillary bicuspic (canine) what are the mesial and distal furcation depths?
* Mesial - 7 mm * Distal - 7 mm
30
For a mandibular molar - what are the facial and lingual furcation depths?
* Facial - 3 mm * Lingual - 4 mm
31
For root trunk furcations - what is considered short, average, and long?
* Short - 1-2 mm * Average - 3 mm (facial mandibular 1st molar) * Long - \>4 mm
32
Why do premolars with furcation involvement have a very poor prognosis?
Because of the root trunk length
33
On a mandibular molar - how often are there root concavaties on the mesial and distal roots?
* Mesial - 100% (0.7 mm) * Distal - 99% (0.5 mm)
34
On a maxillary molar - how often are there root concavaties on the Mesial Buccal, Distal Buccal and Palatal roots?
* MB - 94% (0.3 mm) * DB - 31% * P - 17%
35
Bifurcation ridges are present in what percentage of mandibular molars?
73%
36
What angle do you need to come from with a Nabors probe to measure furcations on the mesial and distal?
* Mesial - Palate * Distal - Buccal
37
What is Glickman's Class I furcation involvement?
* Incipient bone loss * Radiographically not evident
38
What is Glickman's Class II furcation involvement?
* Partial bone loss (cul-del-sac) * Depth with vary: early or advanced * Radiograph may or may not appear
39
What is Glickman's Class III furcation involvement?
* Through and through * Inter-radicular bone is completely absent * Radiographic evidence is a usual finding
40
What is Glickman's Class IV furcation involvement?
* Through and through with furcation exposure due to gingival recession * Almost always shows on radiographs
41
What's the difference between the Glickman classification system and the Hamp sytem?
* Glickman Classification * Class I - IV * Hamp System * Class I - III (III and IV are combined) * Class I \< 2 mm, Class II \> 2 mm, Class III through and through
42
What's a problem with measuring furcations in first molars?
The furcation entrance is more narrow than standard curette in most first molars - 81% \< 1.0 mm Curette width = 1.25 mm
43
What are the different grades of Cervical Enamel Projections?
* Grade 1 - distinct change in the CEJ that projects toward the furca * Grade 2 - CEP approaching the furcation * Grade 3 - CEP at the roof of or into the furcation
44
What are the percentages of Cervical Enamel Projections on mandibular molars, maxillary molars?
* Mandibular - 28.6% * Maxillary - 17%
45
What is the association between the presence of a cervical enamel projection and furcation involvement?
50%
46
What are the percentages of accessory canals on maxillary and mandibular 1st and 2nd molars?
* Max 1st - 36% * Mand 1st - 32% * Max 2nd - 24% * Mand 2nd - 12%
47
What is the order of frequency of individual tooth loss?
1. Max 2nd molar 2. Max 1st molar 3. Mand 2nd molar 4. Mand 1st molar The least likely are: 1. Max Cuspid 2. Mand Cuspid \*\*\*
48
What are the percentages of patients with gingival overgrowth by drug regimen? Carbamazepine? Phenytoin Sodium? Phenytoin Sodium + Sodium Valporate? Phenytoin Sodium + Carbamazepine? Phenytoin Sodium + Carbamazepine + Phenobarbital?
* 0% - Carbamazepine * 52% - Phenytoin Sodium * 56% - Phenytoin Sodium + Sodium Valporate * 71% - Phenytoin Sodium + Carbamazepine * 83% - Phenytoin Sodium + Carbamazepine + Phenobarbital
49
What does Wennstrom (1996) say about how much keratinized and attached gingiva is enough?
At least 2 mm attached - keratinized mucosa is necessary for maintaining health
50
What are the 2 types of grafts and what do they involve?
1. Free Gingival Graft 1. Increased keratinized tissue 2. Connective Tissue Graft 1. Increased keratinized tissue and **root coverage**
51
What are the treatment options for increasing the width of attached gingiva?
* Apically positioned flap (APF) * Free autogenous gingival graft (FGG) * Subepithelial connective tissue graft (CTG)
52
What are the treatment options for obtaining root coverage?
* Subepithelial connective tissue graft (CTG) * Semi-lunar incision + coronal positioning (Tarnow procedure) * Lateral pedicle flap (LPF)
53