Exam 1 Flashcards

1
Q

What is the rational for perio treatment?

A
  • Control etiology
  • Control inflammation
  • Control pain & discomfort
  • Restore periodontal health
  • Maintain long-term function of dentition
  • Regeneration of lost bone and soft tissue
  • Maintain/restore to esthetic level
  • Control local inflammatory response = control of systemic inflammation = good general health
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Why do we treat perio?

A
  • Disease is an infection
  • Disease is chronic
  • Cannot remove all plaque and calculus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are the legal requirements we are obligated to fulfill?

A

We must:

  • Diagnose disease
  • Inform pt of disease
  • Offer appropriate treatment
  • Treat to standard of care
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the variables of probing?

A
  • Inflammation
  • Probe diameter
  • Tapered vs parallel
  • Force (.15 - .75 N)
  • Band width (.7 - 1mm)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How is probing done?

A
  • 6 measurements/tooth
  • Measure oral sulcular epithelium pocket
  • Its a measure of free gingiva
  • Attached gingiva = margin to MGJ - depth of gingival sulcus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is a healthy probing depth?

A

0-3 mm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the characteristics of severe chronic perio?

A
  • Localized < 30% teeth
  • Generalized > 30% teeth
  • Slight/Moderate/Advanced
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the characteristics of an Initial Lesion?

A
  • 2-4 days
  • Inflammatory infiltrate - PMN
  • Vasculitis
  • Loss of perivascular CT = collagen
  • Increased GCF
  • No CAL or bone loss
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the characteristics of an Early Lesion?

A
  • Bleeding on probing (1st sign)
  • 4-7 days
  • Acute inflammation - PMNs
  • Chronic inflammation begins - lymphocytes/macrophages
  • 70% loss of collagen in lamina propria
  • Fibroblasts show damage
  • Pseudopocket formation begins
  • Edema and erythema of marginal gingiva
  • Increased GCF
  • Loss of gingical stippling
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What are the characteristics of an Established Lesion?

A
  • 2-3 weeks
  • Edema & erythema
  • Bleeding upon probing,
  • Gingival changes (color, contour, consistency
  • No bone loss
  • Soft tissue retraction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the characteristics of Advanced Lesion?

A
  • AKA “Periodontitis”
  • Activation of osteoclasts - bone loss
  • Bleeding on probing
  • Apical margination of JE
  • Mobility
  • Cytokines, MMPs, Prostaglandins, Leukotrines
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

How do you calculate Clinical Attachment Loss?

A
  • Probing depth - Gingival margin
  • If gingival margin is coronal to CEJ, PD - GM
  • If gingival margin is apical to CEJ, PD + GM
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the biological width and how wide is it usually?

A
  • Dimension that exists naturally around all teeth
  • About 2mm
  • Junctional epithelium + Connective tissue attachment
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How do you measure mobility and how is it classified?

A
  • Measured with 2 instruments
  • Class 1 - .2 > x < 1 mm
  • Class 2 - > 1 mm (buccal to lingual)
  • Class 3 - > 1 mm + axial displacement (apical margination)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What does bleeding upon probing indicate?

A

Active disease, presence of micobial biofilm/plaque, ulcerative sulcus/pocket epithelium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What does probing depth show with BOP, along with recorded data over time?

A
  • Probing depth (BOP) - active disease
  • Recorded data - disease progression or stability
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What is the normal distance between the CEJ and alveolar bone?

A

1.5 - 2 mm (accomodates biological width)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What is the average PDL width?

19
Q

What probe do you use to measure furcations and what does it measure?

A

Nabors probe - horizontal bone loss

20
Q

What is the difference between gingivitis and periodontitis?

A
  • Clinical presentations
  • Plaque is necessary for both
  • May not have signs of inflammation with perio
  • No bone loss or PDL loss with gingivitis
  • Apical margination of JE with periodontitis
  • Gingivitis may never develop into periodontitis (pt immunology plays role in pathogenesis)
21
Q

What are the characteristics of healthy gingiva?

A
  • Color: coral pink, melanin is variable
  • Contour: anterior - papillae pyramidal, posterior - papillae are slightly pyramidal
  • Consistency: firm & resilient
  • Texture: stippling
  • Some PMN, macrophages present, no collagen destruction, some GCF, intact epithelial barrier
22
Q

What data do we collect to diagnose perio?

A
  • Probing depths
  • Bleeding upon probing
  • Clinical attachment levels
  • Width of attached gingiva
  • Gingival recession
  • Furcation involvement
  • Mobility
  • Radiographic evidence of bone loss
  • Plaque/calculus
23
Q

What are the characteristics of chronic periodontitis?

A
  • Most prevalent in adults
  • Loss of furcation bone, gingival recession, gingival appearance can be consistent with acute or chronic inflammation
  • BOP, exudate from pockets, not amenable to treatment with systemic antibiotics
  • Tooth mobility, Suprabony & Intrabony pocket formation (≥ probing depth), loss of alveolar bone, periodontal abscess
  • Painless except abscess
24
Q

The average rate of CAL in patients with untreated chronic periodontitis ranges from ______ per year for facial and lingual surfaces, and _____ per year for interproximal areas.

A
  • 0.1 to 0.3 mm
  • 0.3 mm
25
During the same time period, untreated periodontal patients will lose ______ more teeth than those patients who receive treatment, or in a 10 year period, untreated periodontal patients will lose ________ teeth while those patients who receive treatment will lose ___ tooth.
* 3.5 times * 3.5 – 4.0 * 1
26
What is the Continuous Model of periodontal progression?
* All sites show continuous progession of clinical attachment loss
27
What is the Random Burst Model of periodontal progression?
* All sites exhibit loss over time, but with random bursts of activity
28
What is the Asynchronous Burst Model of periodontal progression?
* Several sites have one or more bursts of activity * Prolonged period of inactivity * Cumulative extent of destruction varies over time * Some sites don't have attachment loss
29
How do we diagnose slight/moderate/advanced?
* Slight - 1-2 mm CAL * Moderate - 3-4 mm CAL * Advanced - \>5 mm CAL
30
How are intrabony pockets classified?
By the number of remaining walls: 1,2 or 3 Circumferential or Interdental Craters
31
What are the percentages of mandibular vs maxillary occurences of interdental craters?
* 63% - mandibular intrabony defects * 35% - maxillary intrabony defects
32
How is a well-controlled diabetic treated?
Just like a non-diabetic patient
33
What are some signs of diabetes and periodontal disease?
* Rapid alveolar bone loss * Poor response to treatment * Oral signs/symptoms * Xerostomia, burning mouth, periodontal abscesses, multiple abscesses, caries, candidiasis * Periodontal considerations: * Impaired wound healing, increased plaque & bone resportion, altered PMN chemotaxis, abscesses
34
With type 2 diabetes, obesity has been associated with increased what?
Susceptibility to periodontal disease
35
What is a common perio problem for pregnant women and why?
Pyogenic granulomas - P. intermedia growth is facilitated by progesterone (menadione = napthoquinone) Hormones - decreased estrogen, increased progesterone
36
What are some of the adverse outcomes of perio disease in pregnant women?
* Pre-term birth \< 37 weeks gestation * Low birth weigh \< 2500g * Preeclampsia - hypertension b/w week 20-41 * Fetal growth restriction and development, CNS involving hippocampus (emotions/memory)
37
When can you treat pregnant women?
* 1st trimester - emergencies only, no radiology * 2nd trimeseter - good * 3rd trimester - selective treatment
38
What medications are safe/not safe during pregnancy?
* Avoid (D) – Clarithromycin & Tetracyclin, Metronidazole (B) * Caution (C) – Ciprofloxacin, Gentamicin, Vanocomycin * OK (B) – Penicillins, Erythomycin, Clindamycin, Cephalosporins * Avoid aspirin during breastfeeding
39
What are medications that affect gingivitis?
* Pheytoin (Dilantin) - anti-seizure * Cyclosporine (Sandimmune) - immunosuppressant * Ca Channel Blockers (Nifedipine) - hypertension
40
How long must you wait to treat a patient after he or she has had an MI?
6 months
41
What are the Red Complex bugs?
* Prevotella gingivalis * Tannerella forsythia * Treponema denticola
42
What is A.A. associated with?
Aggressive chronic perio
43
What is Prevotella intermedia associated with?
Pregnancy
44
What's the difference between suprabony and intrabony bone loss?
* Suprabony - horizontal bone loss * Intrabony - vertical bone loss