Exam 2 Flashcards
What is a AAA
- permanent, localized, outpouching or dilation of wall of aorta
- AAA more common: 3/4 of aortic aneurysms
- occur in men more, increases with age
- most occur below renal arteries
- larger aneurysm = greater risk of rupture
Clinical manifestations of AAA
often asymptomatic
- can have mimicking abdominal or back disorder symptoms
- deep constant pain in abdomen
- low BP
- pulsation in belly button
- “blue toe syndrome”
Causes and risk factors of AAA
Causes:
- degenerative
- congenital
- mechanical: penetrating or blunt trauma
- inflammatory
- infectious
Risk factors:
- age, male gender
- HTN, CAD
- family history
- tobacco use
- high cholesterol
- lower extremity PAD
- carotid artery disease
- previous stroke
- excess weight or obesity
Treatment of AAA: small vs larger
Small aneurysm: size < 5.4 cm
- conservative therapy
- risk factor modification (lower BP, tobacco cessation, optimize lipid profile, gradual increase in activity)
- sizing 4-5.4cm: monitor every 6-12 months
- size < 4cm: ultrasound every 3 years
Larger than 5.5 cm:
- surgical repair
- can happen earlier in those with genetic disorder, rapidly expanding aneurysm, symptomatic patients, high rupture risk
Nursing care and education for AAA
- watch for signs of cardiac, pulmonary, cerebral and lower extremity vascular problems
- establish baseline data to compare postop
- signs of aneurysm rupture
- quality and character of peripheral pulses; mark pedal pulses and any lesions before surgery
- monitor renal and neuro status
Goals:
- normal tissue perfusion
- intact motor and sensory function
- no complications related to surgical repair (thrombosis, infection, rupture)
PAD vs PVD
Clinical manifestations of PAD
symptoms occur when arteries are 60-75% blocked
- classic symptom: intermittent claudication
– ischemic muscle pain caused by constant level of exercise
– resolves with rest
– reproducible
- paresthesia
- cold legs
- thin shiny taut skin, no hair
- diminished pulses
- pallor w/ leg elevation, pain w/ elevation, hang legs on bed
6 Ps: pain (unrelieved and at rest), pallor, pulselessness, paresthesia, paralysis, poikilothermia
Causes and Risk factors of PAD
Risk factors:
- tobacco use
- atherosclerosis
- diabetes
- HTN
- high cholesterol
- age > 60
PAD results from arteriosclerosis that usually occurs in the arteries of the LE and is characterized by inadequate blood flow
Nursing process for PAD
- assessment (sub, obj)
- nursing diagnoses: ineffective tissue perfusion, activity intolerance
- planning; overall goals w/ PAD:
– adequate tissue perfusion, relief of pain, increased exercise tolerance, intact healthy skin, increase knowledge - implementation (health promotion, acute, ambulatory)
- evaluation of the goals
complications, drugs/tx for PAD
Complications:
- nonhealing arterial ulcers and gangrene
- amputation
- wound infection, delayed healing
- tissue necrosis
- arterial ulcers over bony prominences
Drugs:
- ACE inhibitors reduce symptoms
- drugs for intermittent claudication
Treatments - want to reduce CVD risk factors
- PTA, revascularization, decrease CVD risks (diet, statins, antiplatelets, ACE inhibitors, beta-blockers), angiogenesis
- IR and catheterization, atherectomy, etc
- surgical procedures
- control BP, tobacco cessation, high A1C control, change diets
Clinical manifestations of PVD
- pain with dangling feet and needing to elevate legs
- exudate drainage
- irregular sores around ankles
- lower leg swelling
- dependent edema
V: voluptuous pulses - warm legs
E: edema
I: irregularly shaped
N: no sharp pain
Y: yellow and brown ankles
Causes and Risk factors of PVD
Can be caused by varicose veins, chronic venous insufficiency, or venous embolism
- narrowing of veins interfering with return of blood to heart
Risk factors:
- smoking
- atherosclerosis
- DM, HTN
- high cholesterol
Nursing care, drug/tx for PVD
Nursing care: help with blood flow back to heart
- keep hydrated
- movement every 1-2 hours, prevent sitting
- elevate legs for 20 mins ev 4-5hrs
- compression stockings
- assess HR, BP, pulses
Drugs/tx:
- exercise
- dietary changes
- do not cross legs
- compression stockings
- anticoags if needed
VTE - what is it, what does it include?
venous thromboembolism; includes DVT and pulmonary embolism
- clot formation occurs when localized platelet aggregation and fibrin entrap RBCs, WBCs, and more platelets
- deep veins of arms or legs, pelvis, vena cava, and pulmonary system
Clinical manifestations of VTE
superficial vein thrombosis:
- most common
- palpable, firm, cordlike vein
- itchy, painful, red and warm
- mild fever, leukocytosis
VTE including DVT and PE:
- unilateral leg edema
- pain
- erythema
- chronic venous insufficiency
- embolism of thrombotic fragments
- warmth, redness
- coughing up blood, chest pain
Causes and risk factors of VTE
- venous stasis: dysfunctional valves, inactive extremity muscles, at risk population
- endothelial damage
- hypercoagulability of blood
risk factors:
- sedentary lifestyle
- obesity
- DM
- atherosclerosis
Nursing care, drugs/tx for VTE
Nursing care:
- pain relief
- decrease edema
- no skin ulcerations, bleeding complication
- no PE
Three VTE measures:
- early and progressive mobilization
- graduated compression stockings
- intermittent pneumatic compression devices to increase venous return
Drug therapy: anticoagulants
- vitamin K antagonists
- thrombin inhibitors
- factor Xa inhibitors
Raynaud’s Phenomenon
episodic, vasospastic disorder of small cutaneous arteries
- fingers and toes most commonly affected
- more common in women (15-40 y/o)
Clinical manifestations of Raynaud’s Phenomenon
in stress or cold: fingers and toes turn white-blue-red episodes
- primary: disease occur in any stress or cold
- secondary: phenomenon underlying connective tissue diagnosis (lupus, scleroderma) that damages the arteries
Causes, diagnosis, and treatment for Raynaud’s Phenomenon
Pathogenesis: abnormalities in vascular, intravascular, and neuronal mechanisms that cause vasodilation
Dx: ANA titer, clinical symptoms
Tx: vasodilators or sympathectomy in extreme cases
Teaching for Raynaud’s Phenomenon
avoid the cold, dress warmly, decrease caffeine intake, manage stress levels, stop smoking
Nervous System Assessment
Acute head injury: concussion - clinical manifestations
- headache
- confusion
- dizziness
- sometimes loss of consciousness
Acute head injury: concussion - causes
- mild TBI caused by jolt to head
- immediate medical eval is crucial