Exam Flashcards

1
Q

Define gingivitis

A

Inflammation of gingiva
Is the earliest stage of disease in the oral cavity which can lead to periodontitis

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2
Q

Define periodontitis

A

Inflammation of the underlying tissue and periodontum

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3
Q

Gingiva is the :

A

First line of defence protecting underlying bone and supporting tissue

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4
Q

What does gingiva provide?

A

The main mechanical barrier to infection

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5
Q

Where is ginigival sulcus secreted?

A

Through the sulcular walls to flush out debris from the sulcus

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6
Q

What kind of properties does the gingival sulcus have?

A

Anti-microbial

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7
Q

What does saliva contain? Why?

A

Calcium and fluoride to help maintain the integrity of the enamel

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8
Q

What is saliva? What does it do?

A

Bactericidal, aids in the healing of oral mucosa

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9
Q

What is the enamel bulge?

A

The area where the tooth bulges out at the gingival margin

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10
Q

What does the shape of enamel bulge and how it meets in the gingival, do?

A

Work to keep the sulcus free of debris

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11
Q

What does normal occlusion do?

A

Helps remove plaque before it hardens to calculus

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12
Q

Post dental prophylaxis plaque formation : (bacteria)

A

No bacteria present

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13
Q

Deposit of acellular film called ? made up of saliva ?

A

Acquired pellicle
Glycoproteins

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14
Q

Plaque a few minutes after post dental prophylaxis : (bacteria)

A

Gram positive cocci and rods (aerobic)

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15
Q

What happens to the acquired pellicle when bacteria attaches? (few mins after dental prophylaxis)

A

Plaque starts to form

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16
Q

Plaque formation 6hrs after dental prophylaxis : (bacteria)

A

Gram negative cocci and rods (aerobic)

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17
Q

Plaque formation 24-48hrs after dental prophylaxis : (bacteria)

A

Anaerobic motile rods and spirochetes

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18
Q

Organic substances pertaining to 24hrs post dental prophylaxis

A

Exfoliated epithelial cells
Leukocytes
Macrophages
Protein
Lipid

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19
Q

Inorganic substances pertaining to 24hrs post dental prophylaxis (c & p)

A

Calcium and phosphorus, due to saliva pH of 7.4

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20
Q

7 factors affecting the occurrence of periodontal disease

A

Food debris
Varies
Missing/maloccluded teeth
Mouth breathing/rock chewing
Systemic diseases
Nutrition
Breed

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21
Q

Food debris with periodontal disease

A

creates a great environment for plaque (hard food rather than soft food)

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22
Q

Caries, with periodontal disease

A

Where the enamel has been destroyed or worn, so provides a great place for bacteria and plaque

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23
Q

Missing or maloccluded teeth with periodontal disease can…

A

increase the amount of retained food

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24
Q

Mouth breathing/rock chewing with periodontal disease

A

Dehydrated oral cavity renders plaque together tougher and stickier

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25
Q

Systemic diseases with periodontal disease

A

Can decrease an animals defences
Increasing invasion of bacteria

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26
Q

Nurtrition with periodontal disease

A

Malnutrition can weaken defences
Types of food can affect occurrence

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27
Q

Breeds pertaining to periodontal disease

A

Certain breeds are more prone to occlusion problems due to the shape of their mouth

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28
Q

How does plaque lead to periodontal disease?

A

Once the plaque is mature and extended into the gingival sulcus, the environment easily grows anaerobic bacteria

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29
Q

How is plaque leading to periodontal disease, prevented?

A

By preventing plaque from maturing and growing into the sulcus

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30
Q

What is periodontitis?

A

Inflammation or disease that affects the deeper structures

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31
Q

What is periodontal index?

A

A measure of the amount of periodontal attachment loss

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32
Q

List the 6 stages of periodontal disease (gingivitis)

A

Healthy (GI0)
Marginal gingivitis (GI1)
Moderate gingivitis (GI2)
Severe gingivitis or early periodontitis (GI3)
Moderate periodontitis
Severe periodontitis

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33
Q

Healthy gingivitis

A

GI0
Firm and resilient gingiva
Minimal sulcus depth

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34
Q

Marginal gingivitis

A

GI1
Gingival inflammation
No increase in sulcus depth
Mild odour
Increased crevicular fluid
Vasculitis of vessels at junctional epithelium
Leukocytes into epithelium on sulcus
Becomes more permeable

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35
Q

Moderate gingivitis

A

GI2
Increase inflammation with edema
Gingiva bleeds when probed
Gingiva becomes friable and encroaches on the crown
Increased sulcus depth due to increased gingival size

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36
Q

Severe gingivitis or early periodontitis

A

GI3
Severe inflammation
Periodontal ligament inflamed
Deeper pocket formation
Some epithelial loss
Bleeding
Periodontitis present

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37
Q

Moderate periodontitis

A

Formation of deep pockets
Bone and epithelial attachment loss
+/- gingival hyperplasia or gingival recession
Increased vasculitis with destruction of periodontal ligament

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38
Q

Severe periodontitis

A

Advanced breakdown of periodontal structures
Significant gingival recession
Tooth mobility
Severe pocket
More than 50% bone loss

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39
Q

What do you record when charting?

A

Pocket depth
Gingival recession
Attachment loss

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40
Q

Where do you measure attachment loss?

A

From the cemento-enamel junction to the bottom of the pocket

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41
Q

How to measure gingival recession

A

An estimate from the CEJ to the current free gingival
Then add 1-2mm to account for normal gum line

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42
Q

How to measure pocket depth

A

Measured from the current free gingival margin to the bottom of the pocket

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43
Q

Numbers when grading periodontitis

A

PD0
PD1
PD2
PD3
PD4

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44
Q

PD0

A

Healthy gingiva and deeper periodontal structure

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45
Q

PD1

A

Gingivitis only, with no attachment loss

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46
Q

PD2

A

Less than 25% attachment loss

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47
Q

PD3

A

25-50% attachment loss

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48
Q

PD4

A

Greater than 50% attachment loss

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49
Q

Numbers when grading mobility

A

M0
M1
M2
M3

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50
Q

M0

A

No tooth mobility

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51
Q

M1

A

Slight tooth mobility

52
Q

M2

A

Moderate tooth mobility

53
Q

M3

A

Severe tooth mobility

54
Q

Numbers when grading furcation

55
Q

F1

A

Furcation exposed

56
Q

F2

A

Furcation undermined

57
Q

F3

A

Furcation open through to the other side

58
Q

Things to address pre-prophylaxis

A

Age and general health
Head exam
Inside the oral cavity

59
Q

Why do we assess patients age and general heath before a prophylaxis?

A

Most patients are older, so may present with other medical problems
Pre-screen to assess organ function

60
Q

Assessing for abnormalities on a head exam pre prophylaxis

A

Midline of face
Position
Eye appearance
Sinus area
Facial bones
Biting plane
Temporimandibular joint
Lips

61
Q

Assessing inside the oral cavity pre prophylaxis

A

Buccal/labial mucosa
Soft and hard palate
Tongue and sublingual
Saliva
Breath
Throat and tonsils
Foreign bodies
Plaque and calculus

62
Q

What do you give pre-prophylaxis in order to help combat the floor of bacteria into the blood stream?

A

Antibiotics

63
Q

What to treat for when gingivitis is present pre prophylaxis (bacteria)

A

Aerobic staph/strep

64
Q

What to treat for when periodontitis is present pre prophylaxis (bacteria)

A

Anaerobic and aerobic

65
Q

When to treat mild gingivitis pre prophylaxis

A

1hr before prophylaxis

66
Q

When to treat severe gingivitis pre prophylaxis

A

1 day before

67
Q

When to treat severe periodontitis pre prophylaxis

A

7-10 days before

68
Q

Patient safety when performing a prophylaxis

A

Cuffed ET tube
Gauze packing
Adjustable table for drainage
Eye covers for patient

69
Q

Technician safety when performing a prophylaxis

A

Chlorhex rinse
Goggles, face mask, and gloves
Proper and safe use of machines and equipment

70
Q

Equipment required when performing a prophylaxis

A

Scaling tools
Anesthetic

71
Q

Steps to performing a dental prophylaxis

A

Charting of plaque and calculus
Chlorhex rinse to decrease contaminants
Plaque and calculus supra-gingival
scaling
Sub-gingival scaling with a curet
Explore with explorer and chart abnormalities
Polish (sub and supra-gingival areas)
Sub-gingival irrigation (Chlorhex)
Fluoride treatment
Charting

72
Q

Why do we utilize radiography in dentistry?

A

Most serious pathology in the oral cavity stems from the gingiva which cannot be seen without a radiograph

73
Q

Pertaining to clients, why is it good to take radiographs in dentistry?

A

For legal reasons, good to show below the gum line as it will aid in explaining why a tooth needs to be extracted, etc

74
Q

What will be determined when taking dental radiographs on a young animal?

A

The presence of adult tooth buds

75
Q

Pertaining to periodontal disease, why is it good to take dental radiographs?

A

Can show the extent of bone loss
Can also indicate periodontal disease

76
Q

Why do we take dental radiographs in patients with missing teeth?

A

To see if they are coming in or they are permanently missing

77
Q

Dental radiographs prior to a tooth extraction will determine

A

If it needs to be extracted

78
Q

Dental radiographs while doing a tooth extraction

A

To indicate the type of root you are working with

79
Q

Dental radiographs after a tooth extraction

A

To check for anything retained

80
Q

Why do we take dental radiographs of the mandible and maxilla?

A

To assess for fractures
Checking for body density

81
Q

Why do we take dental radiographs of chipped or broke teeth?

A

To help determine the extent of the fracture

82
Q

What do we do post-prophylaxis?

A

Monitor the oral cavity
Monitor the animal
Give post-op meds

83
Q

Why do we do oral post prophylaxis checks?

A

To make sure there are no complications and that healing is happening if there were extractions

84
Q

Why do we monitor the animal post prophylaxis?

A

To make sure there are no complications and to make sure the patient is eating properly

85
Q

What are some post prophylaxis medications that are given?

A

Antibiotics if there was extensive periodontal disease
Chlorhex rinse for 2-3 to help healing

86
Q

What is the most common species for resorptive lesions?

87
Q

Where do resorptive lesions occurs?

A

Most begin at the neck of the tooth at the CEJ

88
Q

What teeth are most commonly seen with resorptive lesions?

A

Premolars and molars, but can be seen on canines and incisors

89
Q

What surface of the tooth are resorptive lesions most commonly found?

A

Buccal and labial surface

90
Q

What are resorptive lesions?

A

Bacterial destruction within the tooth

91
Q

Resorptive lesions start at the CEJ where the cementum and dentin are softer, and what?

A

They are constantly in contact with the subgingival plaque bacteria

92
Q

What are resorptive lesions filled with?

A

Odontoclasts, which absorbs dentin and enamel

93
Q

Name the 6 factors thought to cause resorptive lesions

A

Periodontal disease
Diet
Viral cause
Regurgitation of hairballs
Genetics
Gingivitis/stomatitis

94
Q

What is the number 1 indicator of resorptive lesions?

A

Periodontal disease

95
Q

What do people think diet can do with resorptive lesions?

A

Some think it can increase acidifiers

96
Q

What do some think phosphate in diet has to do with the cause of resorptive lesions?

A

May have to do with the decrease in phosphate in diets, which decreases the remineralization of the teeth

97
Q

Viral causes pertaining to resorptive lesions

A

Can decrease the immunological response

98
Q

Many cats with resorptive lessons are found to be what positive?

A

FIV and FeLV

99
Q

Why can hairballs cause resorptive lesions?

A

Can increase the amount of stomach acids in the mouth

100
Q

What purebred cat breeds are more prone resorptive lesions?

A

Persians
Siamese
Russian blue

101
Q

What could over breeding cats do in terms of resorptive lesions?

A

Can decrease immune systems

102
Q

What would resorptive lesions associated with gingivitis/stomatitis be due to?

A

Inflammatory resorption

103
Q

What is LPS?

A

Lymphocytic plasmacytic stomatitis

104
Q

Signs/history of LPS in cats

A

Halitosis
Increased saliva
Dysphagia
Inappetence
Weight loss

105
Q

Tests to run with LPS

106
Q

Name the classes of lesions

A

Class 1
Class 2
Class 3
Class 4
Class 5

107
Q

Class 1 lesions

A

Early lesion, only unblocking enamel or cementum, will feel like a roughened area

108
Q

How are class 1 lesions found?

A

Usually found by subgingival exploration

109
Q

Class 2 lesions

A

Significant lesions, deeper with increased tooth destruction

110
Q

Where do class 2 lesions reach?

A

Dentin, but not pulp chamber

111
Q

Class 3 lesions

A

Deep in the pulp chamber, but not much crown loss

112
Q

Where do class 3 lesions reach?

A

Through the dentin, into the pulp chamber, and will bleed on probing

113
Q

What may class 3 lesions be covered by?

A

A pulpal polyp

114
Q

Class 4 lesion

A

Extensive lesions with considerable loss of tooth structure
Could be missing a large portion of the crown
Could be discoloured and very easily shattered

115
Q

Class 5 lesion

A

Passive destruction with separation of root and crown

116
Q

What kind of crown might you see with a class 5 lesion?

A

A floating crown with almost no root OR just the root remaining under very inflamed gingiva

117
Q

Treatment of class 1 lesion

A

Smoothing lesion and applying a fluoride treatment
Must have all edges smooth before adding fluoride

118
Q

How long does a fluoride treatment for class 1 lesions last?

A

6-12 months plus home care

119
Q

What is the treatment for class 2 lesions?

A

Restoration with glass ionomers or extraction of the tooth
Must be dry and polished to a smooth surface

120
Q

Treatment for class 3 and 4 lesions

A

Extraction of the tooth, removing the whole root

121
Q

Treatment of class 5 lesions

A

Floating crowns are extracted and roots are extracted if gingival irritation is present

122
Q

How can class 5 treatment of lesions be a problem?

A

Can become a problem if a cyst forms around the root

123
Q

3 ways to prevent lesions

A

Home care when young
Regular dental exams and prophylaxis
Feed appropriate diets

124
Q

Home care lesion prevention

A

Brushing teeth with a fluoride toothpaste

125
Q

What will regular dental exams/prophylaxis prevent for lesions?

A

Increase the chance of finding the lesions in the early stages of

126
Q

Which kind of food stays on the teeth more than the other?

A

Moist food stays on the teeth more than dry

127
Q

What will happen when you decrease the amount of retained food stuck in the teeth?

A

Will decrease the plaque accumulation