Exam 1 Outline Flashcards

Examination Prep

1
Q

What are Erikson’s stages of Development?

A
  1. Trust vs Mistrust
  2. Autonomy vs Shame & Doubt
  3. Initiative vs Guilt
  4. Industry vs Inferiority
  5. Identity vs Role Confusion
  6. Intimacy vs Isolation
  7. Generatively vs Stagnation
  8. Ego Integrity vs Despair
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2
Q

Trusting Aunties Invite Important Insights Into Growing Elderly

A
  1. Trust vs Mistrust
  2. Autonomy vs Shame & Doubt
  3. Initiative vs Guilt
  4. Industry vs Inferiority
  5. Identity vs Role Confusion
  6. Intimacy vs Isolation
  7. Generatively vs Stagnation
  8. Ego Integrity vs Despair
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3
Q

Trust vs Mistrust

A

Infancy (1to 2) yrs
Trust: belief world is safe
Ex. mother and loving caregiver
Mistrust: world is unpredictable and dangerous
EX. don’t receive consistent care

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4
Q

Autonomy vs Shame & Doubt

A

Early childhood 2- 4 yrs old
Both parents are involved
Sense of independence and self control
building confidence or shame

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5
Q

Initiative vs Guilt

A

Preschool age 4 to 5 years
Try new things, learn basic principles
Learning from entire family
Learn to make decision through play and social interaction

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6
Q

Industry vs Inferiority

A

School Age 5 to 12 yrs
Discover difference from others
productive and elevate own work
Neighbors and School have most influence

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7
Q

Identity vs Role Confusion

A

Adolescence (13 to 19)
Different Social Roles
Identity Crisis
Peers and Role Models have most influence

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8
Q

Intimacy vs Isolation

A

Early Adulthood 20 to 40 years
let go of prior relationships
ability to love
friends and partners center to development

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9
Q

Generatively vs stagnation

A

adulthood 40 to 65 years
leisure time used in creativity
leading next generation
not able to deal with situations causes stagnation
work and home are main influences

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10
Q

Ego Integrity vs Despair

A

maturity 65 to death
look back at life
comparison with mankind

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11
Q

What are the stages of Piaget’s Theory of Cognitive Development

A
  1. Sensorimotor
  2. Pre-operational
  3. Concrete Operational
  4. Formal Operational
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12
Q

Some People Can Fly

A
  1. Sensorimotor
  2. Pre-operational
  3. Concrete Operational
  4. Formal Operational
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13
Q

Sensorimotor

A

Birth to 2 yrs old
learn through senses and movement
Develop Object permanence

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14
Q

What is Object Permanence

A

something continues to exist when it is out of sight

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15
Q

Pre-operational

A

2 to 7 years old
symbolic thoughts, magical thinking (wishes cause events to occur), animism

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16
Q

What is animism

A

treating inanimate object as alive

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17
Q

Concrete Operational

A

7 to 11 years
logical thought
understanding of cause and effect
learn conservatism

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18
Q

what is conservatism

A

matter does not change when its form is altered
ex. small cup of water vs large

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19
Q

Formal Operational

A

11 years to adulthood
engage in abstract though
engage in deductive reasoning, logic based problem solving

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20
Q

Infant Physical Growth

A
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21
Q

Infant Physical Growth : Weight

A

6 months = 2x weight
12 months = 3x weight

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22
Q

Infant physical growth: length

A

6 months = rapid length
12 months = increased by 50%

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23
Q

Infant Physical Growth: Head circumference

A

6 wks (2 months)= posterior frontal close
18 months (2 yrs)= anterior fontanel close

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24
Q

Toys for 2 months

A

black & white, high contrast
soft rattles
soft textured toys
soft hanging toys

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25
Q

Toys for 30 months yr old

A

Building block
shape sorter
pretend play toys (dolls, kitchen set)
push and pull toys (toy car with pull string)
picture books
Musical instruments

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26
Q

When can a infant start to point to body parts

A

around 18 to 24 months

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27
Q

When can a child draw a circle?

A

around 3 yrs old

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28
Q

when can a child start to point?

A

around 9 to 12 months

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29
Q

When does a child start to have a pincer grasp?

A

9 to 12 months

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30
Q

What is Meningitis?

A

Viral illness
contagious
Patho: infectious organism enter the CNS through bloodstream
Prevention meningococcal vaccination
S/S: fever, headache, nuchal rigidity, photophobia, positive Kernig and Brudzinski sign
Infant: poor feeding, bulging fontanels, high pitched crying

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31
Q

B.B.B.

A

Brudzinke
Brain Bending (Flexion)
Bending Knee

Indicated positive Brudzinki for meningitis

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32
Q

Kernig

A

Positive sign

K: knee
E: Extension
Painful

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33
Q

Indication of Meningitis via Labs

A

bacterial: cloudy low glucose, high protein, high WBC, gram positive
Viral: clear, normal glucose and protein, slightly elevated WBC, negative gram stain

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34
Q

Physical Assessment: Newborn

A

No illness
Poor muscle tone
weak cry
poor suck
refuses feeding/vomiting/diarrhea
possible fever: hypothermia
NO NUCHAL RIGIDITY

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35
Q

Physical Assessment: 3 months to 2 years

A

bulging fontanels
seizures
high pitched cry
pupuric/petechial rash
fever, irritability
poor feeding/vomtting
POSSIBLE NUCHAL RIGIDITY

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36
Q

Physical Assessment 2 years to adolescence

A

seizures
fever/chills
severe headache
vomiting
petechiall/purpuric rash
photophobia & Irritability = professes to drowsiness, delirium and coma
Involvement of joints
Chronic Draining Ear
NUCHAL RIGIDITY
KERNIG SIGN
BRUDZINSKI SIGN

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37
Q

Petechiae and Purpura Rash

A
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38
Q

Physical Exam Signs: Brudzinski Neck sign

A

Helps diagnose meningeal irritation: meningitis
Physical Exam:
1. pt lies flat on back (supine position)
2. flexion of neck (toward chest)
Positive: involuntarily flexes knees and hips when neck flexed
Negative: no involuntary flexion of the knees or hips when neck flexed

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39
Q

Physical Exam Signs: Nuchal Rigidity

A

refers to stiffness of the neck/limitation of flexion due to pain or resistance
associated with meningitis and inflammation
Positive: cannot flex the neck forward due to pain/stiffness present

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40
Q

Physical Exam: Kernig Sign

A

Assess for meningeal irritation/pain and resistance to leg extension when the hip is flexed
Physical Exam:
1. pt lies flat on back
2. examiner flex pt hip and knee to 90 degree
3. attempt to extend pt knee
Positive Sign: pain or resistance during extendtion of knee when hip is flexed.

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41
Q

Techniques to help patient feel better of meningitis

A

Create quiet, calm environment
Dim light
Limit Noise
Close Blinds
Positioning and comfort
Pain management
Hydration and nutrition

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42
Q

What are seziures?

A

abnormal , involuntary excessive electrical discharge of neurons within the brain

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43
Q

What are partial (focal) seizures?

A

involve one area of the brain

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44
Q

Types of Partial Seizures

A

Simple Partial Seizures with Motor Manifestation
Simple Partial Seizures with Sensory Manifestation
Complex Partial Seizures
Unclassified

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45
Q

Simple Partial with Motor Characteristics

A

Aversive Seizure
Rolandic

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46
Q

Simple Partial Sensory Characteristics

A

one part brain affected
No LOC
involuntary movement, change or in emotions

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47
Q

Unclassified Seizures

A

West Syndrome
Lennox-Gastaut Syndrome
Febrile Seizures

48
Q

West Syndrome (infantile Spasms)

A

Peak: 3 to 7 months
Sudden, brief, symmetric muscle contractions
Flexed head, extended arms drawn up
Possible Nystagmus or eye deviation
Possible loss of consciousness
TREATMENT: ADRENOCOTICOTOP IC HORMONE

49
Q

Infantile Spasms VERSUS Infantile Seizures

A

INFANTILE SPASMS
sudden, brief muscle jerks or spasms of head, neck, arm, trunk
occur in cluster
no loss of consciousness
onset 3 to 12 months
INFANTILE SEIZURES
localized jerking with altered consciousness

50
Q

Lennox-Gastaut Syndrome

A

Mixture with cognitive defects
Hyperactive and aggressive behavior
poor prognosis
difficult to treat

51
Q

Febrile Seziures

A

Associated with a sudden spike in temperature 38.9 to 40 (102 to 104)
15 to 20 second
TREATMENT:
Acetaminophen
ibuprofen
dress in light clothing
administer tepid sponge baths

52
Q

What are generalized seziures?

A

involved the entire brain

53
Q

Risk Factors for Seizures

A

cerebral Edema
Febrile episode
intracranial infection/hemorrhage
cyst/tumor in brain
lead poisoning
metabolic conditions
tetanus, shigella, salmonella

54
Q

Risk factors for Epilepsy

A

Congenital Defect
Anoxia
Trauma
Hemorrhage
Infection
Hypoglycemic injury
migraine
uremia
cardiovascular dysfunction

55
Q

Seizure Causes Mnemonic: VITAMIN

A

V– Vascular: stroke, embolic stroke
I–Infection: meningitis, encephalitis, cerebral malaria
T–Trauma: head injuries
A— AV malformations: cavernous alformation
M– Metabolic: hypoglycemia, hyponatremia, hypoxia
I– Idiopathic
N–Neoplasms: primary or secondaries

Others: sleep deprivation, drug overdose, fever, eclampsia, hyrocephalus, multiple scerlosis

56
Q

What are the generalized motor Seizures

A

Atonic
Clonic
Tonic
Mycoclonic
Tonic-clonic

57
Q

What is Atonic Seizures

A

DROP seizures consisting of sudden loss of muscle tone
(falls unexpectedly)

58
Q

Atonic Seizures Characteristics

A

Drop Attacks
2 to 5 years of age
muscle tone is lost for few second (falls)
period of confusion follows

59
Q

What is a clonic Seizure?

A

repetitive jerking or twitching

60
Q

What is a Tonic Seizure ?

A

sudden stiffness and rigidity of muscles

61
Q

What is a Myoclonic Seizure

A

short, jerking movement of muscles resembling muscle spasms

62
Q

Myoclonic Seizures Characteristics

A

Variety of Seizures episodes
brief contraction of muscle or groups of muscle
symmetric or asymmetric involvement
No Postictal State
might not lose consciousness
involve only face and trunk or more extremities

63
Q

What is a Tonic-Clonic Seizure

A

sudden stiffness (tonic) and repetitive jerking (clonic)

64
Q

Tonic-Clonic Seizures Characteristics

A

10 to 20 second duration
loss of consciousness
eye roll upward
tonic contraction of entire body: arms flexed/leg and head extended
piercing cry
thoracic and abdominal muscles contraction
mouth snaps shut and tongue can be bitten
flushing
loss of swallowing reflux
increased salivation
apnea leading to cyanosis
ONSET WITHOUT WARNING

65
Q

Tonic-clonic Seizures examples: Clonic

A

30 to 50 seconds
violent jerky movement of the body
foaming in the mouth
rhythmic contraction/relaxation of trunk and extremities
incontinent of urine or feces
gradual slowing of movement until cessation

66
Q

Tonic-clonic Seizures examples: Postictal State

A

30 minutes
remain semiconscious: arouses with difficulty
impairment of fine motor movement
headache, vomiting, visual, speech difficulties
confused for several hours
lack of coordination
sleeps for several hours, feels tired, complain of sore muscles
no recollection of seizures

67
Q

What are the generalized non-motor seizures

A

Typical Absence seizures
Atypical Absence seizures
Eyelid myoclonia

68
Q

What is typical absence seizures

A

affected person stops what they are doing and stare intto space
eyelid fluttering
short duration 10 seconds

69
Q

Absence Seizures characteristics

A

Onset: 4 to 12 and ceases by teenage years
LOC (level of consciousness) 5 to 10 seconds
blank stare, motionless
resembles day dreaming
can drop items
automatisms
momentarily confused
can immediately resume previous activities

70
Q

what is an atypical absence seizures

A

longer duration
staring into space, lipsmacking, repetitive hand movement such as wringing

71
Q

what is eyelid myoclonia seziure

A

brief twitches of eyelid

72
Q

Nursing Care for During Seizures

A

Protection from injury
position: maintain airway
watch the clock
note onset, time, characteristics
Do not restrain a child
loosen restriction clothing
DO NOT attempt to put anything in child mouth
prepare for oxygenation
remove glasses
remain calm and stay with the child

73
Q

Nursing Care for Post-Seizures

A

Side-lying
check for breathing
position of head and tongue
neuro checks
allow for rest
reorient due to confusion or agitation
remain with the client
don’t offer food or beverage until fully awake
encourage child to describe the period

74
Q

Client Education for Seizures

A

Periodic lab testing
DONT ABRUPTLY stop medication
adhere to medication regimen
medication interaction
helmets during sports
water safety
avoid triggers
ketogenic diet

75
Q

Ketogenic Diet

A

used for children younger than 8 years of age with myoclonic or absence seizures
high fat, low carbs, low protein diet causing ketosis as body uses fat for metabolism
ketosis slow electrical impulses that cause seizures

76
Q

What are types of CROUP (Upper Airway)

A

acute laryngotracheobronchitis
acute spasmodic laryngitis

77
Q

Generalized Croup Characteristics

A

hoarseness
cough: barking or brassy
degree of inspiratory stridor
degrees of respiratory distress
obstruction in the larynx (swelling)
affecting trachea, larynx, bronchi

78
Q

Croup: Acute Laryngotracheobronchitis

A

most common
6 months to 5 years
causative organism are viral agent
preceded by uri
gradual onset with low fever
barky cough, stridor, wheezing, retraction
inflammation of mucosa lining the larynx and trachea causing narrowing of the airway
more common in boys than girls

79
Q

Croup: acute spasmodic laryngitis

A

spasmodic/midnight/twilight
occurs at night
recurrent paroxysmal attacks of laryngeal obstruction
1 to 3 years old
allergies and hypersensitivity

80
Q

Management of Croup at Home

A

warm mist from hot running water in a closed bathroom
humidification: cool mist, outdoors, open freezer door

81
Q

Management of Croup at Hospital

A

PO or IV fluids
high humidity with cool mists
nebulizer treatments
corticosteroids

82
Q

Nursing care Measures for Children with Croup

A

Assess respiratory status
Positioning
Humidifie air
administration of medications
fluid encouragement
monitor worsening symptoms

83
Q

Patient Education of Croup

A

recognizing early signs
encouragement of cool mist and humidifier
if difficulty breathing occurs immediate medical attention needed
Calming child

84
Q

Upper Airway: Acute Epiglottis

A

onset abruption
supraglottic obstruction
MEDICAL EMERGENCY
rapidly progress to severe respiratory distress
child goes to bed asymptomatic and awaken with sore throat and painful swallowing

85
Q

Acute Epiglottis symptoms

A

fever
appears sicker than clinical finding
drooling tripod position
irritable
anxious
restless
retractions
thickened or muffled voice
throat is red and inflamed
epiglottis large, cherry red, edematous

86
Q

Emergency Of EPIGLOTTIS

A

due to inflammation of epiglottis and rapid progression for complete airway obstruction causing respiratory failure
1. potential for rapid airway obstruction
2. rapid deterioration of breathing
3. risk of sudden and complete airway obstruction
4. high risk of respiratory failure

87
Q

What is Asthma

A

chronic inflammatory disorder of the airways characterized by bronchial hyper responsiveness, recurring symptoms and airway obstruction

88
Q

Characteristics of Asthma

A

chronic inflammatory disorder of airways and obstruction of the bronchial tree
inflammatory process are mast cells, eosinophils, T-lymphocytes

89
Q

Pathophysiology of Asthma

A

Chronic inflammatory process
hypersensitivity of bronchioles to irritants
bronchospasm and mucus production leading to air trapping
respiratory failure

90
Q

What are the hypersensitivity of bronchioles to irritants?

A

INTRINSIC
strong emotions, exercise, conditions (GERD, fistula), endocrine factors (thyroid disease, menses, pregnancy)
EXTRINSIC
food allergy (dairy, nuts), plants (weed, trees, pollens) dust, mites, animals (cat, dogs), exposure to chemicals
virus or infection

91
Q

What are types of bronchospasm and mucus production leading to air trapping

A

hypoxemia
hyper ventilation
obstruction

92
Q

What is the Course of Asthma Attack

A

Environmental Factors
to
Provoking airway inflammation
to
Resulting in coughing, wheezing, chest tightness, breathlessness
to
progress to airway hyper- responsiveness
to
obstruction to partial to total

93
Q

Symptoms of Asthma

A

Dyspnea
Cough
Inspiratory wheezing
SOB
prolonged Expiration
Accessory Muscle Use
Tripod Positioning
Hyper resonance or Percussion
Cyanosis to nail beds
Tripod positioning

94
Q

Inhaler Instructions for Asthma

A

Metered-dose: pressurized canister to release a fixed dose of medication
Dry powder inhaler: deliver medication in form of power and take deep breaths
Nebulizer: delivery of medication in a mist form

95
Q

What is Cystic Fibrosis

96
Q

Diagnosing Cystic Fibrosis

A

Family History
Positive Sweat Chloride test
absence of pancreatic enzymes
fecal fat test
LFTs elevated
chest X-ray

97
Q

Diagnosis of Early Infancy

A

sweat Cl- test
DNA identification of Mutant Genes
Abnormal Nasal Potential Difference Measurements

98
Q

Screening of Newborn Cystic Fibrosis

A

Detect an abnormal chloride secretion in sweat
CFTR Gene Mutation
IRT (Immunoreactive Tryspinogen)
DNA Analysis of F508 Gene Mutation

99
Q

Carrier Screening of Cystic Fibrosis

A

F508 GENE
70% ni Caucasian population
30% in African American population

100
Q

Enzyme Administration for Children with Cystic Fibrosis

A

prescribed pancreatic enzyme replacement therapy to aid in digestion of food and ensure they receive adequate nutrition

given due to
pancreatic insufficiency developing which leads to malnutrition, poor growth, fatigue
&
ensure they can break down food properly absorb the nutrients needed for growth, maintain healthy weight

Aid in digestion, nutrient absorption, promotion of health growth, maintain energy levels

101
Q

Concussion: Nursing Care Measures

A
  1. Monitor for worsening symptoms
  2. perform regular neurological assessments (glasgow coma scale)
  3. promotion of mental and physical rest in the first 24 to 48 hrs
  4. gradual return to activity after being asymptomatic 2 to 48 hr
  5. education on symptom management
102
Q

Concussion: Patient education for prevention

A

educate on importance of avoiding second impact syndrome
not to return to sports or physical activities too soon

103
Q

Reye Syndrome Labs findings

A

Elevated Liver Enzymes: indicate liver dysfunction
Hyperammonemia: elevation of ammonia level
Hypoglycemia: low blood sugar
Prolonged PT: indicating clotting problems

104
Q

Reye Syndrome: Mannitol Administration

A

Mannitol given to reduce intracranial pressure where brain swelling is a concern
works by drawing fluid out of the brain into the bloodstream for excretion by the kidneys

105
Q

SID Prevention (Sudden Infant Death Syndrome)

A

place infant on their backs to sleep
avoid soft bedding, pillow, or stuffed toys in crib
sleeping in crib in the same room not same bed
avoid overheating
offer a pacifier during sleep to reduce risk

106
Q

Toddler Burn Prevention

A

Keep hot items out of reach
Supervise toddler near hot appliances
cover electrical outlet
installations of safety gates like kitchen

107
Q

Poisoning/indigestion Preventions

A

Store household cleaning products and medications in childproof containers
Keep hazardous substances locked
educate parents of safe disposal of old meds
use child-resistant caps on medicine bottles

108
Q

Post-op Tonsillectomy Care: Nursing Care Measures

A

Pain management (acetaminophen/ibuprofen)
Hydration
Monitor bleeding (swallowing, restlessness, vomiting blood)
Positioning: semi-upright to prevent choking
Education

109
Q

Pneumonia Signs & Symptoms

A

Cough: green or yellow
Fever
SOB: increased respiratory rate
chest pain/tightness
fatigue/loss of appetite

110
Q

Pneumonia: Progression of Infection

A

worsening cough/difficult breathing
increased work of breathing
signs of sepsis (persistnet fever, tachycardia, low blood pressure)

111
Q

Pneumonia: Hospitalization reasons

A

Severe respiratory distress : ventilation/oxygenation
Inability to maintain hydration or persistent vomiting
high fever not responding to treatment

112
Q

Respiratory Distress Signs and Symptoms

A

increased respiratory rate or effort
nasal flaring, retractions of the chest wall
grunting or wheezing
cyanosis (blueish lips/face/extremities)
decreased breath sounds or stridor

113
Q

Respiratory syncytial virus: risk factors for complications

A

prematurity (infant born before 37 weeks)
chronic lung disease
heart disease
Immune system compromise
<6 month infants & elderly at more risk

114
Q

Respiratory syncytial virus: Reason for hospitalization

A

severe respiratory distress: supplement oxygen, intubation
Dehydration: inability to maintain oral intake/vomting/diarrhea
Increased work of breathing: nasal flaring, grunting, retraction

115
Q

Respiratory syncytial virus: Transmission

A

Direct Contact: respiratory secretion (droplets from coughing or sneezing)
Contaminated Surfaces: surviving on surfaces for several hours
Spread through hands: virus transfer when touching infected surfaces and touching the face