exam 1: clinical mgmt, soft tissue injury, post op Flashcards
5 things you need to know to clinically manage your patient
- nature of dysfunction ie decreased AROM
- stage of condition ie acute vs subacute
- tissue reactivity ie high vs low
- subject reactivity ie high vs low
- functional goals ie what does patient want to get back to?
3 stages of tissue healing
- inflammatory (acute): 0-6 days
- proliferative (subacute)
- repair and healing 4-21 days
- repair and regeneration 48hrs-8weeks - maturation and remodeling (chronic) 14+ days
muscle pain/spasm cycle
trauma/pain/inflammation–>
- restricted movement
- circulatory stasis (retention of metabolites, tissue ischemia)
- pain
- muscle spasm
- back to restricted movement
stages of healing and ROM
acute: pain before reaching tissue resistance
early subacute: pain at beginning of tissue resistance
late subacute (settled): pain at end of resistance
goals in acute stage
decrease pain, effusion/edema, maintain function/mobility, pt. ed, facilitate movement into subacute stage
ther ex during acute stage
- rest and protection
- classical PROM
- submax muscle setting (w/o pain)
contraindications: AROM, stretching/resistance exercises
MEAT vs RICE
MOVEMENT
EXERCISE
ANALGESICS
TREATMENT
effects of PROM
prevents: contractures, fluid stasis, thrombus formation
Decreases pain, nourishes cartilage, distributes synovial fluid, stimulates healing along lines of stress
specific interventions/dosages for acute (protection) phase
- PROM
- mild jt mobs
- muscle setting
- massage
*workout associated/nearby areas to maintain strength/function
impairments during subacute stage
contractures, weakness, decreased functional use of associated areas
exercises during subacute stage
multiple angle submax isometrics AROM muscular endurance protected weight bearing exercises no heavy eccentrics early on
progression of stretching during subacute stage
- warming up tissue ie bike ride
- inhibition techniques (prevent muscle guarding)
- jt mobs
- stretching
- massage
- use new ROM
pathophysiological characteristics during acute stage
inflammatory stage:vascular changes, exudate,clot, phagocytosis, early fibroblastic activity
treatment:decreases pain, prevent progressive inflammation, maintain mobility/strength of other areas
pathophysiological characteristics during subacute stage
repair and healing:removal of noxious stimuli,capillary beds, granulation tissue, collagen formation, fibrous healing aligns to stress, TISSUE IS FRAGILE
treatment: ROM, jt mobs, scar mobs, light resistance, bone loading
pathophysiological characteristics during chronic stage (maturation and remodeling)
maturation and remodeling: contracture, alignment of stress, maturation to collagen type I
treatment: provide stress (stretch, active contraction, estim), bone loading
chronic inflammation
- fail to eliminate injuring agent
- weakens tissue
- continued pain, swelling
- muscle guarding lasting hours post activity, stiffness and loss of ROM 24 hrs post exercise