Exam 1 Flashcards
LAD feeds
Apical cap
Apical Septum
Apical Lateral
Mid Anterior Lateral
Basal Anterior Lateral
Apical Inferior
Apical Anterior
Mid Anterior
Basal Anterior
Mid Anterior Septum
Basal Anterior Septum
RCA feeds
Mid Inferior Septum
Basal Inferior Septum
Mid Inferior
Basal Inferior
Mid Inferior Lateral
Basal Inferior Lateral
Circumflex feeds
Apical Lateral
Mid Anterior Lateral
Basal Anterior Lateral
Mid Inferior Lateral
Basal Inferior Lateral
Right and Left Dominance
RCA gives rise to the Posterior Descending Artery in 80%
Circumflex gives rise to the Posterior Descending Artery in 20%
Ischemia
Deficient oxygen supply to the myocardium resulting in transient or reversible injury to the myocardium
-caused by partial occlusion of a coronary artery due to atherosclerotic plaque
Ischemia on Echo
normal wall motion at rest (partial occlusion does not prevent oxygenation of the heart muscle)
abnormal wall motion with stress or exercise
Infarction
Irreversible injury to the myocardium due to prolonged ischemia
-caused by acute thrombotic total occlusion at the atherosclerotic plaque (plaque ruptures and clots)
Infarction on Echo
acute infarction demonstrates abnormal wall motion at rest; unaffected segments are hyperkinectic
Post reperfusion echo
PCI (percutaneous coronary intervention = angioplasty/stent)
CABG (Coronary Artery Bypass Graft Surgery)
Echo performed several days after to assess effectiveness
Assess systolic and diastolic function and wall segments
Echo 4 to 6 weeks post MI
-checking status of infarcted segments
-looking at actual extent of infarction
-evaluates for reduced global function and ventricular dilation
Hibernating myocardium
border myocardium that is chronically ischemic but not infarcted and could benefit from PCI or CABG
-identified with dobutamine stress test (wall motion improves with small amounts)
Development of MI complications
-MR due to infarct of segment and pap muscle
-Ventricular septal defect due to necrosis then rupture of a focal area of IVS
-Pericardial effusion seen 2-4 days post MI
-Right Ventricular Infarction associated with inferior LV infarct
-LV aneurysm due to extensive damage of thinned dyskinetic walls
-LV thrombus due to akinetic walls and stasis of blood
-Acute/End stage ischemic cardiomyopathy from severe LV dysfunction and dilation due to acute multi vessel or prox event
Delineating aneurysms
Aneurysm creates distinct outpouching of an area of the ventricle
-thin layer of myocardium
-wide neck
-ruptures early
-smooth transition from normal to thin wall
Chronic pulmonary hypertension on echo
-concentric RVH
-PSM and D shaped LV in systole and diastole
-RAE
-PV mmode: reduced a wave and mid systolic closure
-Eventual RV systolic dysfunction
-Eventual RVE
-Enlarged PA, IVC, Coronary Sinus
acute pulmonary emboli on echo
-immediate RVD
-RV systolic dysfunction
-significantly elevated pulmonary pressures and resistance
-dagger shaped TR