Exam 1 Flashcards

1
Q

LAD feeds

A

Apical cap
Apical Septum
Apical Lateral
Mid Anterior Lateral
Basal Anterior Lateral
Apical Inferior
Apical Anterior
Mid Anterior
Basal Anterior
Mid Anterior Septum
Basal Anterior Septum

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2
Q

RCA feeds

A

Mid Inferior Septum
Basal Inferior Septum
Mid Inferior
Basal Inferior
Mid Inferior Lateral
Basal Inferior Lateral

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3
Q

Circumflex feeds

A

Apical Lateral
Mid Anterior Lateral
Basal Anterior Lateral
Mid Inferior Lateral
Basal Inferior Lateral

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4
Q

Right and Left Dominance

A

RCA gives rise to the Posterior Descending Artery in 80%
Circumflex gives rise to the Posterior Descending Artery in 20%

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5
Q

Ischemia

A

Deficient oxygen supply to the myocardium resulting in transient or reversible injury to the myocardium
-caused by partial occlusion of a coronary artery due to atherosclerotic plaque

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6
Q

Ischemia on Echo

A

normal wall motion at rest (partial occlusion does not prevent oxygenation of the heart muscle)
abnormal wall motion with stress or exercise

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7
Q

Infarction

A

Irreversible injury to the myocardium due to prolonged ischemia
-caused by acute thrombotic total occlusion at the atherosclerotic plaque (plaque ruptures and clots)

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8
Q

Infarction on Echo

A

acute infarction demonstrates abnormal wall motion at rest; unaffected segments are hyperkinectic

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9
Q

Post reperfusion echo

A

PCI (percutaneous coronary intervention = angioplasty/stent)
CABG (Coronary Artery Bypass Graft Surgery)
Echo performed several days after to assess effectiveness
Assess systolic and diastolic function and wall segments

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10
Q

Echo 4 to 6 weeks post MI

A

-checking status of infarcted segments
-looking at actual extent of infarction
-evaluates for reduced global function and ventricular dilation

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11
Q

Hibernating myocardium

A

border myocardium that is chronically ischemic but not infarcted and could benefit from PCI or CABG
-identified with dobutamine stress test (wall motion improves with small amounts)

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12
Q

Development of MI complications

A

-MR due to infarct of segment and pap muscle
-Ventricular septal defect due to necrosis then rupture of a focal area of IVS
-Pericardial effusion seen 2-4 days post MI
-Right Ventricular Infarction associated with inferior LV infarct
-LV aneurysm due to extensive damage of thinned dyskinetic walls
-LV thrombus due to akinetic walls and stasis of blood
-Acute/End stage ischemic cardiomyopathy from severe LV dysfunction and dilation due to acute multi vessel or prox event

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13
Q

Delineating aneurysms

A

Aneurysm creates distinct outpouching of an area of the ventricle
-thin layer of myocardium
-wide neck
-ruptures early
-smooth transition from normal to thin wall

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14
Q

Chronic pulmonary hypertension on echo

A

-concentric RVH
-PSM and D shaped LV in systole and diastole
-RAE
-PV mmode: reduced a wave and mid systolic closure
-Eventual RV systolic dysfunction
-Eventual RVE
-Enlarged PA, IVC, Coronary Sinus

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15
Q

acute pulmonary emboli on echo

A

-immediate RVD
-RV systolic dysfunction
-significantly elevated pulmonary pressures and resistance
-dagger shaped TR

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16
Q

Symptoms of PE

A

SUDDEN ONSET OF:
-SOB
-sharp chest pain
-cough with possible hemoptysis
-rapid HR
-sweating
-signs of cyanosis