everything Flashcards
condition where gastric content and enzymes backflow into the esophagus
GERD
primary treatment of GERD
diet and lifestyle changes
medication treatment of GERD
antacids
H2 receptor antagonists
proton pump inhibitors
untreated GERD leads to
inflammation
breakdown
long term complications (barretts esophagus)
adenocarcinoma of esophagus
condition where lining of esophagus gets damaged by acid reflux
barretts esophagus
GERD prevention includes
BMI below 30
smoking cessation
limit alcohol/tobacco use
low fat diet
no eating 2 hrs before bed
no tight fitting clothes
elevate head of bed 6-8 inches
risk factors of GERD
obesity
old age
sleep apnea
nasogastric tube
what type of foods relax LES and cause GERD
fatty fried foods
chocolate
caffenated beverages
peppermint
spicy foods
tomatoes
citrus fruits
alcohol
burning sensation in the esophagus is called
pyrosis
pain when swallowing is called
odynophagia
what color will esophageal lining be in GERD pt
red
allows visualization of esophogus and can reveal barretts epithelium or esophagitis
EGD
diagnostic procedure done under moderate sedation to observe for tissue damage
EGD
what should you verify has returned before giving a pt oral fluids or food after a EGD
gag reflex
manifestations of esophageal perforation
fever
pain
dyspnea
bleeding
most accurate method of diagnosing GERD
esophageal pH monitoring
diagnostic procedure where small catheter is placed through nose into esophagus to get pH readings for 24-48 hrs
esophageal pH monitoring
nursing action for esophageal pH monitoring
have pt log food/beverages consumed, manifestations and activity during 24 hr test period
records lower esophageal sphinter pressure & peristaltic activity of esophagus
esophageal manometry
identifies hiatal hernia, strictures or structural abnormalities that contribute to GERD
barium swallow
not eliminating barium places pt at risk for what?
fecal impaction
proton pump inhibitors
pantaprazole
omeprazole
esomeprazole
rabeprazole
lansoprazole
what do proton pump inhibitors do?
stop stomach cells from pumping acid into the stomach
what to watch for in diabetic pts taking PPIs
electrolyte imbalances
hypoglycemia
signs of C.diff diarrhea secondary to PPI use
abdominal cramping
fever
diarrhea
long term PPI use can increase the risk for what especially in older pts
fractures
types of antacids
aluminum hydroxide
magnesium hydroxide
calcium carbonate
sodium bicarbonate
what do antacids do?
neutralize acid in stomach to make it less abrasive
what med cant be taken w antacids
levothyroxine
what function should you check for pts taking magnesium hydroxide (antacid)
kidney function
when is acid secretion highest
1-3 hrs after eating
bedtime
when should acacia be taken
when acid secretion is high
how long should you wait to take other meds before/after antacids
1 hr
h2 receptor antagonists
ranitidine
famotidine
cimetidine
nizatidine
what do h2 receptor antagonists do
reduce stomach acid secretion
difference between antacids & h2 receptor
h2 takes longer to kick in but lasts longer
use h2 receptor antagonists carefully in pts with
kidney disease
pt education for h2 receptor antagonists
take w meals and at bedtime
take 1 hr apart from antacids
med that increases motility of esophagus and stomach
metoclopramide
what should you monitor in pt taking metoclopramide
extrapyramidal adverse effects (involuntary movement)
procedure that uses radio frequency energy from an endoscope to decrease vagus nerve activity
stretta
post op stretta pt education
clear liquids for first 24 hrs
no NSAIDS for 10 days after
report CP, abd pain, bleeding, difficulty swallowing, dyspnea, nausea/vomiting
what is used to treat bleeding esophageal varices
vassopressin
what does peppermint do to indigestion
increases indigestion (bad for hiatal hernias)
most common area for peptic ulcer
duodenum
what infection causes peptic ulcers
H. pylori
how do you get H pylori
food, water, or exposure to body fluids
curlings ulcer is found in patients
with burns
cushings ulcers are found in patients with
head/brain trauma
what is used prophylactically to prevent stress ulcers
PPIs
gastric ulcer pain usually occurs
30-60 mins after a meal
gastric ulcer pain is exacerbated by
ingestion of food
does gastric ulcer pain usually occur at night
no
what kind of ulcer pain is found in malnourished patients
gastric ulcer
what ulcer pain has hematemesis as a symptom
gastric ulcer
patient with what kind of ulcer would be throwing up blood
gastric ulcer
what ulcer pain has melena as a symptom
duodenal (peptic) ulcer pain
patient with dark stool would have what kind of ulcer
peptic (duodenal) ulcer
when would pt with duodenal (peptic) ulcer feel pain
1.5-3hrs after a meal
which ulcer will wake patient up at night
peptic (duodenal) ulcer
which ulcer would occur in a well nourished patient
peptic (duodenal) ulcer
which ulcer is relieved by food or an antacid
peptic (duodenal) ulcer
physical signs of peptic ulcer
epigastric pain or tenderness
vomiting blood
poo-ing blood
losing weight
how is H pylori tested for
endoscopy
urea breath test
stool sample test
hemoglobin and hematocrit
stool sample
how does a urea breath test find H.pylori
pt drinks carbon rich urea solution
blows into collection container
positive urea breath test
carbon dioxide will be released if h pylori is present
foods to avoid w/ ulcer
coffee
tea
carbonated beverages
meds for ulcers
antibiotics
H2 receptor antagonists
PPIs
antacids
mucosal protectants
what to do before EGD procedure
start 2 large bore IV catheters
what to do after EGD procedure
monitor vitals
NPO until gag reflex returns
surgerys if ulcers dont heal
gastrectomy
vagotomy
pyloroplasty
procedure that part or whole stomach is removed
gastrectomy
procedure that vagus nerve is cut to decrease gastric acid production
vagotomy
how is vagotomy done to prevent post op complications
laparoscopically
procedure where opening between stomach opening and small intestine is enlarged to increase gastric emptying rate
pyloroplasty
what happens to pts HR w peptic ulcer
tachycardia
abdominal signs of peptic ulcer
rigid abdomen
rebound tenderness
what happens to peptic ulcer pt BP
hypotension because of GI bleed
what is dumping syndrome
high sugar food moves from stomach to small intestine too quick
gastritis caused by h pylori infection
nonerosive
gastritis caused by NSAIDS, alcohol use, recent radiation
erosive gastritis
gastritis w sudden/short onset and results in gastric bleeding if severe
acute gastritis
chronic gastritis can be caused by
autoimmune disease
bacterial infection
lab tests for gastritis
CBC (anemia)
blood/stool antibody test (h. pylori)
urea breath test (h pylori measurement)
pernicious anemia is treated with
monthly vitamin b12 injections
what does famotidine do
antacid (stops acid production)
ulcerative colitis is characterized by
frequent stools
cramping/abdominal pain
exacerbations/remissions
edema & inflammation in the rectum and rectosigmoid colon is called
ulcerative colitis
Inflammation and ulceration of GI tract at distal ileum is called
crohns disease
cultures w high risk get ulcerative colitis
caucasian
jewish
culture w high risk for crohns disease
jewish
crohns disease requires what monthly
vitamin b12 injections
part of abdomen that ulcerative colitis is felt
LLQ
part of abdomen crohns disease is felt
RLQ
hematocrit & hemoglobin lvls with ulcerative colitis
decreased
ESR lvls with ulcerative colitis
increased
WBC with ulcerative colitis
increased
CRP w/ ulcerative colitis
increased
albumin w/ ulcerative colitis
decreased
electrolytes w/ ulcerative colitis
decreased
hematocrit & hemoglobin in crohn’s disease
decreased
ESR in crohns disease
increased
WBC in crohns disease
increased
CRP in crohns disease
increased
albumin in crohns disease
decreased
folic acid and b12 in crohns disease
decreased
urynalisis in crohns disease will show
WBC
electrolytes in crohns disease will be
decreased
diagnostic procedures for ulcerative colitis
colonoscopy
sigmoidoscopy
barium enema
CT scan/MRI
stool exam
crohns disease diagnostic procedure
endoscopy
proctosigmoidoscopy
colonoscopy
abd US
barium enema
life threatening inflammation of peritoneum and abdominal cavity lining
peritonitis
cause of peritonitis
bacteria in peritoneal cavity
nursing actions for peritonitis
place pt in fowlers (drain fluid)
monitor for hypovolemia
adminitser hypertonic IVF & antibiotics
crohns disease diet
high protein diet
ulcerative colitis diet
low fiber diet
inflammation of gallbladder wall
cholecystitis
cholelithiasis is
gallstones
where is bile stored
the gallbladder
cholecystitis risk factors
females
oral contraceptives
obesity
older pts
type 2 DM (high triglycerides)
crohns disease
rapid weight loss
native/mexican american
cholecystitis would be felt where
RUQ radiating to right shoulder
physical assessment test for cholecystits
murphys sign
positive murphys sign
pain w/ deep inspiration during palpation of R subcostal
food that causes pain in pt w cholecystitis
high fat food caused by biliary colic
physical findings in cholecystitis pts
jaundice
icterus
clay colored stool
steatorrhea
dark urine
cholecystitis WBC labs will be
increased (inflammation)
cholecystitis blood bilirubin will be
increased
cholecystitis amylase and lipase will be
increased
AST, LDH & ALP can indicate what in cholecystitis
common bile duct is obstructed
cholecystitis diagnosis exams
US
abd CT
hepatobiliary scan (HIDA)
endoscopic retrograde cholangiopancreatography
magnetic resonance cholangiopancreatography
bile duct obstruction can cause
ischemia
gangrene
rupture of gallbladder wall
a ruptured gallbladder can cause
bile peritonitis
when can a pt eat a normal diet of choice after a cholecystectomy
upon discharge
what pain would a pt s/p laparascopic cholecystectomy experience
shoulder pain (from free air introduced into the abdomen during surgery)
what does chenodiol do?
dissolves gallstones over 2 years
what does the pancreas secrete
insulin
glucagon
autodigestion of pancreas by enzymes that activate early before reaching stomach
pancreatitits
pancreatitis is felt where in the abdomen
LUQ, midepigastric and radiates to the back
pancreatitis pain is worse when
lying down
pancreatitis pain is relieved by what position
fetal position
sitting upright
bending forward
what does insulin do to glucose
decreases it
what does glucagon do to glucose
increases it
lipase breaks down
lipids
amylase breaks down
carbohydrates
what worsend cholecystitis pain
movement and eating
referred pain is
pain felt in different area than problem area
nerves that elevate the diapraghm
frenic nerves
pancreatitis risk factors
gallbladder stones
alcoholism
diet (high fat meal)
pancreatitis meds
antibiotics
h2 receptor antagonists
PPIs
pancreatic enzymes
hypercalcemia will cause pt to be
fatigued
bradycardia
hyperactive muscles
what will pancreatitis do to blood glucose
increase
pancrelipase should be given with what
every meal or snack
what should pt do after taking pacrelipase
drink full glass of water
what can you sprinkle pancrelipase on
non protein food
severe, boring epigastric pain that radiates to back, left flank, or left shoulder is a sign of
pancreatitis
gray-blue discoloration in periumbillical area is a manifestation of
pancreatitis
inflammation of liver cells
hepatitis
permanent scarring of liver that is caused by chronic inflammation
cirrhosis
most common type of hepatitis
viral hepatitis
can hepatitis be asymptomatic
yes
hepatitis A is transmitted through
food/water contaminated w feces
hepatitis B is transmitted through
blood
you get hepatitis B from
unprotected sex w infected person
contact w infected blood
substance use disorder
hepatitis C is transmitted through
blood
hepatitis D is transmitted through
co-infection with hepatitis B
hepatitis E is transmitted through
contaminated food/water
physical manifestations of hepatitis
fever
vomiting
dark color urine
clay colored stool
jaundice
HBV lab tests
ALT (elevated)
AST (elevated)
ALP (elevated)
total bilirubin (elevated)
HBV antibodies (will be present)
normal ALT level
4-36 units/L
normal AST
0-35 units/L
normal ALP
30-120 units/L
total bilirubin
0.3-1.0 mg/dL
HCV labs
AST
ALP
ALT
total bilirubin
HCV antibodies
EIA
HCV PCR
HDV labs
intrahepatic delta antigen identification
HDV antibodies
hepatitis diagnostic procedures
liver biopsy
what to do during liver biopsy
put pt in supine position w RUQ exposed
exhale and hold for 10 secs while needle inserted
what to do during liver biopsy
put pt in supine position w RUQ exposed
exhale and hold for 10 secs while needle inserted
post op liver biopsy
put pt in right side lying position
assess for pneumothorax
medication for chronic HCV
antiviral medication
HCV treatment
combination of peginterferon alfa-2a & ribavirin (antivirals)
are there meds for HBV & HEV?
no only supportive care
chronic hepatitis results from
HBV
HCV
HDV
chronic hepatitis increases risk for
liver cancer
post necrotic cirrhosis is caused by
viral hepatitis
laennecs cirrhosis is caused by
chronic alcohol use
biliary cirrhosis is caused by
chronic biliary obstruction or autoimmune disease
s/s of cirrhosis
cognitive changes
GI bleeding
splenomegaly
ascites
jaundice
petechiae
red palms
spider angionomas
cirrhosis labs
ALT (elevate)
AST (elevated)
ALP (elevated)
blood bilirubin (elevated)
blood protein (decreased)
blood albumin (decreased)
PT/INR (prolonged)
ammonia (increase)
creatine levels (increase)
cirrhosis diagnostic procedures
US
CT
MRI
liver biopsy
EGD
ESRC
monitor what for cirrhosis
resp status
skin integrity
fluid balance
vital signs
neurologic status
GI status
pain status
meds for cirrhosis
diuretics
beta blocking agent
lactulose
nonabsorbable antibiotic
procedures for cirrhosis
paracentesis
endoscopic sclerotherapy
transjugular intrahepatic portosystemic shunt
surgical bypass shunting procedures
liver transplant
pre op for paracentesis procedure
assis pt to bathroom to void
during paracentesis procedure
put pt supine with elevated bed head
apply dressing over puncture
post op paracentesis procedure
measure fluids and document amount/color
send to lab
asses puncture site for drainage
weigh pt
diet for pt with cirrhosis
high calorie mod fat
low sodium (ascites)
lowprotein (encephalopathy)
small frequent well balanced meals
cirrhosis complications
hepatic encephalopathy
esophageal varices
acute graft rejection post liver transplantation
medication for hepatic encepholopathy
lactulose (reduces ammonia)
signs hepatic encepholopathy is worsening
asterixis (hand flapping)
fetor hepaticus (bad breath)
HBV diet
high calorie high carbs
inappropriate amounts of T3 and T4 indicate
hypo or hyperthyroidism
what gland secretes TSH
anterior pituitary gland
T3 normal range
70-205 in adults
40-180 in old ppl
T4 normal range
4-12 mcg/dl up to 60 yrs old
5-11 mcg/dL over 60 y/o
increased TSH indicates
hypothyroidism
decreased TSH indicates
hyperthyroidism
TSH normal range
0.3-0.5 mU/L
hbA1c measures blood glucose for the past
120 days
lab values of pt with SIADH
low sodium
increased urine osmolarity
high urine sodium
increased urine specific gravity
excess growth hormone in adults w increased body part size but not height
acromegaly
untreated acromegaly can cause
HTN
DM
cardiac issues
acromegaly risk factors
age
benign tumors
s/s of acromegaly
headaches
visual disturbance
joint pain
hyperglycemia
barrel chest
types of diabetes insipidus
primary neurogenic
secondary neurogenic
nephrogenic
defects in pituitary gland cause lack of ADH
primary neurogenic
lack of ADH caused by infetions or tumors near pituitary gland
secondary neurogenic
renal tubules that dont react to ADH
nephrogenic
diabetes insipipidus urine labs will all be
decreased
diabetes insipidus blood labs will be
increased
meds for diabetes insipidus
desmopressin
chkorpramide
thiazide
hyperthyroidism risk factors
graves disease
thyroiditis
toxic adenoma
toxic goiter
synthroid hormone replacement
levothyroxine
levothyroxine increases the effects of what drugs
warfarin
levothyroxine is increased how often
every 2-3 weeks
levothyroxine should be taken on
an empty stomach before breakfast
how long is levothyroxine treatment
lifelong
abnormal menstrual periods (mennorrhia)
dry skin
hoarseness are manifestations of
hypothyroidism
methimazole treats
graves disease
disease caused by long term gluccocorticoid use
cushings syndrome
result of tumor in pituiatary gland resulting in release of ACTH hormone
cushing disease
cushings disease risk factor
females 20-40
cushings disease labs
cortisol levels (elevated)
urine (elevated free cortisol)
ACTH levels (elevated)
salivary cortisol (elevated)
blood potassium and calcium lvls (decreased)
blood glucose level (increased)
blood sodium level (increased)
lymphocytes (decreased)
pt with cushings disease increased risk for
infection
gastric ulcer
bone fractures
pt s/p transsphenoidal hyposectomy nasal drainage should be testes for
glucose
primary addisons disease causes
autoimmune dysfunction
TB
histopasmosis
adrenalectomy
cancer w metastasis
abd radiation therapy
secondary addissons disease causes
steroid withdrawal
hypophysectomy
pituitary neoplasm
high dose radiation of pituitary gland
s/s of addissons disease
weight loss
salt craving
hyperpigmentation
weakness/fatigue
nausea/vomiting
abdominal pain
constipation/diarrhea
addisons disease labs
blood electrolytes (increased)
BUN/Creatine (increased)
blood glucose (decreased)
blood/salivary cortisol (decreased)
unable to produce insulin
type 1 DM
diabetes screening `
BP greater than 140/90
HgA1c over 5.7%
HDL less than 35
triglycerides greater than 250 mg/dL
rapid acting insulin is given
before meals
rapid acting insulin onset
10-30 mins
give rapid acting insulin with
intermediate or long acting insulin
regular insulin (short acting) should be given
30-60 mins before meals
NPH insulin (intermediate acting insulin) should be given
given between meals
long acting insulin should be given
anytime but at the same time daily