Erogenic aids and optimising sport Flashcards
Overreaching
- systemic attempt to overstress the body for a short period of training
- Allowing body to adapt to stronger stimulus
- Not same as excessive training
- Caution: easy to cross into overtraining
- Short performance decrement fatigued by improved performance and function
Overtraining syndrome
- Highly individualised, subjective
- Necessary to rule out other causes
Symptoms - Decrease of strength, co-ordination, capacity
- Fatigue
- change in appetite: weight loss
- sleep and mood disturbances
- lack of motivation, vigour concentration
- Depression
Neural and endocrine factors
- overtraining stressors may act primarily hypothalamic signals
- can lead to sympathetic neural activation
- can lead to a pituitary endocrine cascade
- hormonal axes are involved
- sympathetic: adrenal medullary axis
- Hypothalamic- pituitary- adrenocortical axis
Immune responses
- circulatory cytokines
- mediate inflammatory responses to infection, injury
- increase in responses to muscle, bone, and joint trauma
- Increase physical stress + decrease of rest: systemic inflammation
- inflammation: increase cytokines via monocytes
- may act on brain and body functions, contribute to overtraining symptoms
- compromised immune factor in onset of overtraining syndrome
- overtraining suppresses immune function
- abnormally decreases lymphocytes, antibodies
- increase incidence of illness after exhaustive exercise
- exercise during illness: immune complications
how to predict
- threshold different for each athlete
- intuition (unreliable) used by most coaches and trainers
- preliminary warning symptoms
- subjective self-report measures
- increased RHR
- sleep quality
- decreased body mass
- HR during exercise
- HR variability
- likely related to intensity or volume
Psychological factors
- emotional pressure of competition: stress
- parallels with clinical depression
Physiological factors
- autonomic, endocrine and immune
- Not a clear cause and effect relation but significant parallels
Sympathetic nervous system
- increased BP
- Loss of appetite
- weight loss
- Sleep and emotional disturbances
- Increased basal metabolic rate
Parasympathetic Nervous system
- Early fatigue
- Decreased resting HR
- Decreased resting BP
- Rapid HR recovery
- More common with endurance athletes
Endocrine responses
- resting thyroxine, decrease testosterone
- Resting cortisol increase
- Testosterone- to cortisol ratio
- Indicator of anabolic recovery processes
- Altered ratio possibly indicative of protein catabolism
- Possible cause of the overtraining syndrome
- Volume- related overtraining is apparently more likely to affect hormones
- Increase in blood urea concentration
- Resting catecholamines
- Possible outside factors
- overreaching may produce same trends
- time between last training bout and resting blood sample is critical
- blood markers are helpful but not definitive diagnostic tools
Treatment and prevention
Treatment
- reduced intensity
- rest (weeks, months)
- counselling to deal with stress
Prevention
- Periodisation training
- adequate caloric intake
Detraining
Loss of training-induced adaptations
- partial or complete
- due to training reduction or cessation
- Much more substantial change than tapering
Brief period: tapering
Longer period: de-training
Immobilization
- Immediate loss of muscle mass, strength, power
Training cessation
- variation in rate of strength and power loss
- Causes
- atrophy
- reduced ability to recruit muscle fibres
- Altered rates of protein synthesis vs degradation
- Loss mitigated by low level of exercise
Detraining part 2
Muscle endurance decrease quickly
- change was seen after 2 weeks of inactivity
- Not clear whether results from muscle changes or CV changes
- Oxidative enzyme activity decreases by 40-60%
- muscle glycogen stores decrease by 40%
- Significant acid-base imbalance
- Weekly exercise tests during de-training
- blood lactate accumulation
- Bicarbonate decreases
- pH decreases
- Training: only moderate increase in speed and agility
- de-training: only moderate, decrease speed and agility
- form, skill, and flexibility are also lost
- sprint’s performance still suffers
- significant cardiorespiratory losses
- Based on bed rest studies
- significant increase in sub-max HR
- 25% decrease in Sub-max SV
- 27% decrease VO2 max
- Trained athletes, VO2 max lost faster with detraining, regained slowly
- losses occur when frequently and duration decrease by 2/3 of regular training load
- 70% VO2 max training is sufficient to maintain max aerobic capacity.