Adaptations to Aerobic Training Flashcards

1
Q

What is cardiorespiratory endurance?

A
  • Ability to sustain prolonged, dynamic exercise.
  • Improvement through multisystem adaptations.
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2
Q

What is endurance training

A
  • Maximal endurance capacity= increased VO2 max
  • increase sub-max endurance capacity:
    • lower HR at the same submaximal exercise intensity
    • More related to competitive endurance performance
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3
Q

What are the 3 aspects of Muscular and what role do they play?

A
  1. Fibre type
    - Increase the size and number of type 1 fibres
    - Type 2x may perform more like type 2a
    - Modest increase in the number of type 1 fibres
  2. Capillary supply
    - increase the number of capillaries supplying each fibre
    - May be key factor in VO2 max
  3. Myoglobin
    - an increase of myoglobin content by 75% to 80%
    - supporting increase of oxidative capacity in muscle
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4
Q

What are the 2 types of Muscular function?

A
  1. Mitochondrial function
    - increase the size and number
    - Magnitude of change dependent on training volume
  2. Oxidative enzymes
    - increase activity with training
    - Continued increase even after VO2 max plateaus
    - Enhanced glycogen sparing
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5
Q

What is lactate threshold under Metabolic?

A
  • To a higher percentage of VO2 max
  • Decrease of lactate production, increase in lactate clearance
  • Allowing higher intensity without lactate accumulation
    Respiratory exchange ratio (RER)
  • Decrease at both absolute and relative sub-max intensities
  • Increase dependent on fat and decrease dependent on glucose
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6
Q

What is the long term improvement in metabolic?

A
  • Highest possible VO2 max is achieved after 12-18 months
  • Performance continues to increase after VO2 max plateaus because lactate threshold continues to increase with training
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7
Q

What are the factors dictating individual Responses?

A
  1. Training status and pre-training VO2 max: - Relative improvement dependent on fitness
    - More sedentary individuals: greater increase
    - More fit individual: smaller increase
  2. Heredity:
    - Finite VO2 max range determined by genetics (VO2 max altered within that range by training)
    - VO2 max is more similar for identical than fraternal twins, 25% - 50% of VO2 max variance due to heredity
  3. Sex
    - Untrained female VO2 max < untrained male VO2 max
    - Trained female VO2 max closer to trained male VO2 max
    - Differences in older men vs older women
  4. High vs low responders
    - Genetically determined variation in VO2 max for same training stimulants and compliance.
    - Physiological mechanisms
    - Reasons for tremendous variation in training outcomes for given training conditions.
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8
Q

Adaptations to HIIT

A
  • Stimulates mitochondrial biogenesis and changes in the capacity for carbohydrate and fat transport and oxidation
  • Increase VO2 max and mitochondrial content
  • Mitochondrial biogenesis depend largely on exercise intensity
  • Potentially > with HIIT than MICT when work matched
  • SIT= MICT despite the reduced volume
  • VO2 max
    • HIIT > MICT when work matched
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9
Q

Heart size

A
  • With Aerobic training, increase heart mass and LV chamber size
  • Increase plasma volume, increase LV volume, increase EDV, increase SV Volume loading effect
  • Increased wall thickness, allowing a more forceful contraction of the left ventricle
  • Increased internal dimensions due to an increase in ventricular filling (preload).
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10
Q

Stroke volume

A

SV increase after training
- Resting, sub-max, max
- Plasma volume increase with training, increases EDV, increases preload
- Resting and sub-max HR and decrease with training, increase filling time, increase EDV
- Increase LV mass with training, increase force of contraction
- Alternated increase TPR with training, decrease in afterload.
- SV adaptations to training decrease with age

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11
Q

Heart Rate

A
  1. Resting HR
    - Decreases markedly
    - Increases parasympathetic, decreases sympathetic activity in the heat
  2. Sub-max HR
    - decreases HR for the same given absolute intensity
    - More noticeable at higher sub-max intensities
  3. Max HR
    - no significant change with training (decreases with age)
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12
Q

Cardiac output

A

Little or no change at rest or during sub-max exercise with training
Maximal Q increases considerable due to increase of SV

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13
Q

Cardiovascular

A
  • Increase of blood flow to active muscle
  • Increase capillarization, capillary recruitment
    - Increase capillary-to-fibre ratio
    - Increase total cross-sectional area for capillary exchange
  • decrease blood flow to inactive regions
  • Increase total blood volume
    Preventing decrease in venous return as a result of more blood in capillaries
    Blood volume: total volume increase rapidly
  • increase blood plasma via increase plasma protein, increase water and NA+ retention
  • Increase red blood cell volume
  • Decrease plasma viscosity
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14
Q

Respiratory

A
  • Pulmonary ventilation
    - decrease at given sub-max intensity
    - increase at maximal intensity due to increasing tidal volume and respiratory frequency
  • Pulmonary diffusion
    Unchanged during rest and at sub-max intensity
  • increase at max intensity due to increase lung perfusion
  • Arterial- venous O2 difference
    - Increase due to increase of O2 extraction active muscle blood flow
  • Increase O2 extraction due to increase oxidative capacity
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