Equine wounds Flashcards

1
Q

Classification of wounds
a) Mechanical injuries
b) Thermal injuries
c) Chemical injuries

A

a)
- Most common
- Grazes, abrasions, erosions
- Bruising, hematomas, contusions
- Puncture/penetrating wounds
- Lacerations/incised wounds

b)
- Rare
- Thermal burns
- Friction rubs

c)
- Acids: cause coagulation necrosis and protein precipitation
- Alkalis: cause liquefaction necrosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Classification of wounds
a) Clean
b) Clean contaminated
c) Contaminated
d) Dirty

A

a)
- Elective/Surgical wounds created under aseptic conditions, primarily closed
- No break in technique or dirty space entered

b)
- GI, respiratory or urogenital tract (without urinary
infection) entered without significant contamination
- Or minor break in aseptic technique

c)
- Traumatic wounds = always contaminated
- elective procedure with major break in aeptic technique

d)
- > 1x10^5 bacteria
- Traumatic wound with devitalized tissues, FB, feces, chronic wound

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Stages of wound healing
a) Inflammatory/debridement
b) Proliferative repair
c) Maturation/remodelling

A

a)
- haemostasis/acute inflammation
- Immediate, lasts days to weeks
- Neutrophils (remove damaged tissue and chemo attractants) and Macrophages (release O2 radicals, cytokines, tissue growth factors)

b)
- tissue formation
- day 3-14+
- angiogenesis, fibrous and granulation tissue formation
- collagen deposition
- epithelialisation

c)
- strengthening tissue
- ends in formation of scar tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Control of haemorrhage

A
  • Horse can use ~5L without any real problems
  • Pressure bandage
  • Pressure with swab/hand
  • Topical haemostatic agent
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Preparing the wound

A
  • Clip around area
  • Clean/lavage with large volumes, moderate pressure (do not use soap solutions)
  • Can use local anaesthetic directly on wound to desensitise
  • use sterile gloves to palpate
  • debride necrotic tissue, but any tissue that may be viable should be left
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Wound closure
a) Primary closure
b) Delayed primary closure
c) Delayed secondary closure

A

a)
- clean wounds with minimal contamination
- minimal tissue loss
- best cosmetic result

b)
- up to 5 days, gives more time for contamination removal
- lavage and dress daily until healthy granulation bed present, then close

c)
- rarely performed due to lack of elasticity of skin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Second intention healing

A
  • very common
  • especially if contaminated tissue or large skin defects
  • granulation then epithelialisation, wound will be exudative
  • multiple bandage changes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Suture material used on equine skin

A
  • prolene is least reactive, most commonly used
  • can also use nylon, PDS, vicryl
  • consider absorbable sutures
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Factors affecting wound healing (5)

A
  • Degree of contamination/infection (may need to debride further, broad spectrum antibiotics, allow drainage)
  • Tension of skin edges (can cause dehiscence or necrosis at wound margin)
  • Vascularity
  • Dead space (allows fluid accumulation, use a penrose drain, mesh skin)
  • Motion (commonly over joints, must box rest)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Bandaging
a) How often to change a bandage
b) Other materials to use

A

a)
- typically daily, then reduce it after a week
- depends per case

b)
- clean, dry, sutured wound won’t need anything
- no oil-based ointments, no caustic agents
- flamazine (antibacterial)
- manuka honey (infected wounds)
- topical antibiotics (rarely used, only for chronically infected wounds)
- corticosteroids (chronic wounds, use sparingly)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Bandaging
a) Primary layer
b) Secondary layer
c) Tertiary layer

A

a)
- wicks exudate
- cushions/protects
- promotes healing environment

b)
- absorbs excess exudate
- provides pressure and support
- reduces movement of wound edges

c)
- secures other components
- provides additional support and pressure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Considerations at
a) Heel bulb laceration
b) Coronary band laceration

A

a)
- must rule out coffin joint, DDFT and navicular bursa involvement
- primary or delayed primary closure best
- hoof cast reduces movement and allows faster healing

b)
- if coronary band misaligned will result in hoof wall defects
- also use hoof cast

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Causes of chronic/non-healing wounds (5)

A
  • excess movement
  • large skin defect (may require skin graft)
  • presence of foreign body/necrotic tissue (stake wounds, inadequately debrided, bone fragment remaining, bony sequestrum formation)
  • excessive granulation (proud flesh)
  • systemic disease of patient, quite severe (nutritional status, cushings/PPID)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Chronic/non-healing wounds
Proudflesh indications and predisposing factors

A
  • When granulation tissue protrudes higher than wound edge, difficult for epithelium to grow over
  • due to inefficient acute inflammatory phase and protracted chronic inflammatory response (too much granulation tissue)
  • trim off tissue until a flat surface
  • Predisposing: chronic inflammation, motion, bandages/casts, size of horse
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Chronic/non-healing wound
Bony sequestrae cause

A
  • following when initial injury traumatises bone or blood supply to the bone
  • suspect when see a mature granulation bed but a cleft/cloaca
  • may require surgical intervention
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Skin grafting
a) When to use
b) Two types

A

a)
- speed up healing
- may be less expensive than lengthy bandaging

b)
- Pedicle graft (has vascular and nerve supply)
- Free graft (no vascular/nerve supply, must establish new vascular connections

17
Q

Skin grafts - free grafts
a) Categories
b) reasons for failure

A

a)
- Full thickness: epidermis and entire dermis
- split thickness: epidermis and only a portion of dermis
- sheet grafts/ pinch/punch/tunnel grafts

b)
- fluid accumulation
- movement
- infection (must effectively prepare recipient bed)

18
Q

Pinch/punch grafts

A
  • pinch: small discs of skin harvested using scalpel
  • punch: full-thickness plugs harvested with biopsy tool and implanted into granulation tissue using another biopsy tool
  • donor site typically taken from neck beneath mane, ventral abdomen
  • grafts inserted into holes, cover with a non-adherent dressing and bandage
  • leave on for 5 days, then change every 2-4 days
  • survival of 60-75% grafts expected
19
Q

Skin grafts
a) tunnel graft
b) split thickness graft
c) full-thickness sheet graft

A

a)
- useful for large skin defects or sites are difficult to bandage

b)
- very painful, usually requires GA
- needs specialist equipment
- accepted better than full-thickness grafts

c)
- not accepted as readilt as split-thickness
- epidermis usually sloughs

20
Q

Synovial sepsis
a) gold standard treatment
b) when to use just needle lavage
c) Common causes

A

a) Arthroscopic lavage under general anaesthesia (89% survival, 69% return to work)
b) Only on low budget cases (very poor survival rate)
c) Penetrating traumatic injury causing contamination of synovial cavity. Causes marked inflammatory response

21
Q

Synovial sepsis aetiology
a) Penetrating traumatic injury
b) Hematogenous spread of local/systemic infection to bone/synovial structures

A

a)
- common in adults, introduction of bacteria via wound
- distal limb joints and tendon sheaths commonly affected
- variable lameness: typically go lame 24-48 hours after injury
- Many bacteria can cause

b)
- occurs in foals (especially immunocompromised or systemic illness - like pneumonia, colitis, umbilical infection)
- lameness may vary, most common clinical sign is joint distension
- Most commonly caused by Streptococcus, Rhodococcus, Actinobacillus, E. coli, Salmonella

22
Q

Synovial sepsis aetiology
a) Iatrogenic infection
b) Extension of local infection

A

a)
- infection post injection or surgery
- occurs infrequently
- usually acute lameness and history of joint injection
- Osten involves Staphylococcus

b)
- Rare
- can be due to peri-synovial abcesses, cellulitis
- treat with broad spectrum antibiotics (doxycycline or penicillin/gentamicin)

23
Q

Diagnosing synovial sepsis
Synoviocentesis

A
  • collection of synovial fluid, number 1 test for diagnosis no matter the aetiology
  • fluid analysis (WBC count, % neutrophils and TP most important)
  • culture and sensitivity (often not performed as takes days to get result)
24
Q

Synovial sepsis treatement

A
  • must be rapid and aggressive (immediately refer)
  • synovial lavage to remove foreign material, organic debris, bacteria
  • systemic antibiotics (penicillin/gentamycin or oxytetracycline - but not for foals)
  • local antibiotics usually injected after lavage (aminoglycosides ie gentamicin or amikacin)
  • IV regional limb perfusion
25
Q

a) Specific synovial structures known to have worse outcomes (5)
b) Long-term possibility

A

a)
- Coffin joint
- Navicular bursa
- Calcaneal bursa
- Tarsocural joint
- Digital flexor tendon sheath

b)
- osteoarthritis, especially in prolongued treatment