equine derm 2 Flashcards
what infectious dz is this
signalment, dx, tx
Papilloma
may be congenital
mainly in younger horses (1 to 4-years old)
equine papilloma virus (‘everywhere’)
multiple wart like lesions, mainly head,
incidentally elsewhere
diagnosis on clinical presentation
treatment usually not warranted
prognosis is good
cryosurgery, surgery, creams
common areas for sarcoids
ears,groin, axila, face, and eyelid
it is a fibro epithelial tumor
clinical appearance of occult sarcoid
it manifest as a ring of alopecia with slight scaling or skin thinning
they are commonest in the medial thigh region
dicsuss lesions of verrucose saicoid
the lesions are cauliflower(warty) in appearance.
they can be focal or diffuse
single or multiple
the lesions usually produce large amounds of keratin and so have a flaky grey or scabby appearance
discuss the types of nodular sarcoids
type A and B
in the type A there is no epithelial component and the skin and the tumor can be moved independently of each other.
in type B there is significant epithelial component which results in the binding of the tumor mass under the skin so that independent movement is not possible.
this type of sarcoid has a fleshy appearance very like granulation tissue but
fibroblasticsarcoid
affected wounds may be extrememly difficult to tx
type 1 fibroblast has has a narrow pedunculated stalk attachment to the skin and therefore there may be obvious involvement of the subcutis in the region of the attachment while type 2 the tumor has a wide base usually wider than the underlying exuberant mass
what kind of sarcoid tumor is this one
mixed sarcoid tumor
this tumor is simply a mixture of the other types in varying proportions
in reality many of the common lesions fall under this category
therapy is difficult/
the most common skin tumors in horses
sarcoids
dx for sarcords
bx
differentiate types of sarcoids
tx for sarcoids
cryosx and bcg immunotherapy are possible
px for sarcoids
guarded
they dnt metastasis but therapy is nt always successful
what causes this dz
how is it transmitted and cured
ehv3
contagious veneral disease
caused by EHV-3
transmission by coitus (via insects,
fomites and inhalation)
incubation about 7 days
systemic corticosteroids may
reactivate the diseases
depigmentation may persist after
lesions have healed
rest the animal and dont breed till the dz has healed
which dz is this, clinical appearance ,pathogenesisas well as characteristics
Dermatophilus congolensis
bacteria invade epidermis and root shafts
in rainy weather in horses at pasture
dorsum and lateral parts of the body
painful but no pruritus
crusts under which there is a pink-red open lesion
underside of crust is moist and yellow-greenish
pus may be present
in laboratory samples mention ‘dermatophilosis’
tx for drmatophylosis
improvement of the management
removing crusts (painful!)
clip as far as possible
repeated bathing and drying
systemic antibiotics (costs!)
disease is not highly contagious but it is
sensible to isolate affected horses
which dz is this,etiology, common areas, characterisitics, tx
Folliculitis and furunculosis
several bacteria
most often on tack-areas
papels – pustules
often quite painful, rarely pruritic
pustules may open and exude pus
treatment is hygiene !
sometimes systemic antibiotics indicated
which bacteri cuses this
common areas
abscess
Corynebacterium pseudotuberculosis
mostly pectoral muscle
ventral abdomen
groin
cs of corynebcterium pseudotubeculosis infection
lame or reluctant to walk
depression
weight loss
small to very large abscesses
complications like purpura
haemorrhagica or ulcerative
lymphangitis
bacteria is difficult to culture
discuss tx for corynebacterium pseudotuberosis
external abscesses
- mature abscesses (hot pack)
- abscess drainage and flushing
- NSAID’s (bute)
internal abscesses
- long course proc-penicillin / TMPS
limb infection (ulcerative lymphangitis
px for corynebacterium psydotubercolosis
external abscesses good
internal abscesses poor
ulcerative lymphangitis guarded
what causes cellulitis
several bacteria, often Staphylococci
often primary ‘entrance’ not detected
acute onset of severe swelling and pain of
one limb, often a hind limb
often extremely lame
often tachycardia and febrile
complications: cutaneous necrosis and
sloughing, laminitis, bacteremia
tx for cellulitis
immediate systemic antibiotics
hydrotherapy
support bandages other limbs
NSAID’s
if within 24 hours no improvement
corticosteroids
name the dz
characterists
Tail pyoderma
folliculitis and furunculosis of the tail
may cause severe pruritus and
automutilation
therapy
- clipping infected area and bathing
- systemic treatment with antibiotics
prognosis is guarded
name this condition
etiology
lesions,tx
Botryomycosis = bacterial pseudomycetoma
mostly coagulase positive staphylococci
mostly solitary nonpruritic nodular growths
incidentally multiple pustules, nodules and
draining tracts over large areas of the body
affected horses are otherwise healthy
surgical excision for solitary lesions - good
TMPS systemically for multiple lesions - poor
which lesion is dz
dermatophytosis
types of dermatophysis
superficial skin dz
trichophyton spp and microspora spp
pathogenesis for dermatophytosis
they primarily invade the hair shaftsto weaken it and cause breakage
some produce substances that can act as an irritatant or allergens and therefore produce pruritus
describe the lesions of dermatophytosis
it often starts with small papules with erected hairs. then the classical ring shaped lesions with alopecia and some scalling may develop or may be obvious as some lesions may coalesce or appear as paint brush type areas.
dx for drmatophytosis
it is done thru clinical appearance but confirmation can be done through microscopic examination of hairs and crusts for hyphae and conidia.
while cx is the only definitive way.
transmission of fungal infections
they are contracted through direct contact and theough indirect contact by saddle girth, brushes etc
tx for dermatophytosis
it is self limiting
tx is recommended to stop transmission or reduction in performance
use antifungal topicals
characteristics of fungal infections, incubation period, signalemnt, early signs
incubation period 6 days – 6 weeks
young and debilitated horses predisposed
early signs may be urticaria
variable degree of associated crust and
scale which is often ‘silvery’
hair can be lost in annular patches =
‘ring worm’
lesions of dermatophyte infection overlap
with bacterial infection
dermatophytosis is a zoonosis,
tx for dermatophytosis
reduce contagion to the environment
maximise horse’s ability to respond
(correct predisposing factors)
hasten resolution of the infection
topical therapy
systemic therapy? No!
vaccination
vaccination protocol for dermatophytosisi
Insol® Dermatophyton where to vaccinate?
deep intra-muscular
muscle is preferably ‘relaxed’
thigh is preferred
pectoral muscle is also suitable
preferably not in the neck
alternate sides between
vaccinations
When to vaccinate with
Insol®Dermatophyton
horses with clinical signs of dermatophytosis
possible infected horses
horses due to travel
(second injection has to be administered
at least 14 days prior to departure)
all horses on premises with historically
problems
hygiene is essential !!!
n.b horse has dermatophytosis,
but does not show clinical signs (yet),
these symptoms will show soon
after vaccination
When not to vaccinate with Insol® D ?
reduced immunity
- foals younger than 5 months
- sick or stressed horses
pregnant mares ??
close before important events!
hygiene is essential !!!
where do u normally find malazezia
mostly in intertrigenous areas (axilla,
groin, udder, prepuce, incidentally
coronitis)
what are the lesions of malaszia
very rare in horses
greasy, waxy, often malodorous dermatitis
example of a deep fungal infection
sporothrichosis
it is a chronic progressive sporadic skin and sq/ lymphatic infection caused by sporothrix schenckii
infection is usually introduced thru small skin wounds
cs of sporottricosis
firm,well dermacated, painless sq nodulesassociated with cored hardened lymphaticscommnly on limb regions (fetlock most common)
it is potentially zoonotic