equine derm 2 Flashcards

1
Q

what infectious dz is this

signalment, dx, tx

A

Papilloma
may be congenital
mainly in younger horses (1 to 4-years old)
equine papilloma virus (‘everywhere’)
multiple wart like lesions, mainly head,
incidentally elsewhere
diagnosis on clinical presentation
treatment usually not warranted
prognosis is good
cryosurgery, surgery, creams

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2
Q

common areas for sarcoids

A

ears,groin, axila, face, and eyelid

it is a fibro epithelial tumor

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3
Q

clinical appearance of occult sarcoid

A

it manifest as a ring of alopecia with slight scaling or skin thinning

they are commonest in the medial thigh region

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4
Q

dicsuss lesions of verrucose saicoid

A

the lesions are cauliflower(warty) in appearance.

they can be focal or diffuse

single or multiple

the lesions usually produce large amounds of keratin and so have a flaky grey or scabby appearance

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5
Q

discuss the types of nodular sarcoids

A

type A and B

in the type A there is no epithelial component and the skin and the tumor can be moved independently of each other.

in type B there is significant epithelial component which results in the binding of the tumor mass under the skin so that independent movement is not possible.

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6
Q

this type of sarcoid has a fleshy appearance very like granulation tissue but

A

fibroblasticsarcoid

affected wounds may be extrememly difficult to tx

type 1 fibroblast has has a narrow pedunculated stalk attachment to the skin and therefore there may be obvious involvement of the subcutis in the region of the attachment while type 2 the tumor has a wide base usually wider than the underlying exuberant mass

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7
Q

what kind of sarcoid tumor is this one

A

mixed sarcoid tumor

this tumor is simply a mixture of the other types in varying proportions

in reality many of the common lesions fall under this category

therapy is difficult/

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8
Q

the most common skin tumors in horses

A

sarcoids

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9
Q

dx for sarcords

A

bx

differentiate types of sarcoids

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10
Q

tx for sarcoids

A

cryosx and bcg immunotherapy are possible

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11
Q

px for sarcoids

A

guarded

they dnt metastasis but therapy is nt always successful

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12
Q

what causes this dz

how is it transmitted and cured

A

ehv3

contagious veneral disease
caused by EHV-3
transmission by coitus (via insects,
fomites and inhalation)
incubation about 7 days
systemic corticosteroids may
reactivate the diseases
depigmentation may persist after
lesions have healed

rest the animal and dont breed till the dz has healed

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13
Q

which dz is this, clinical appearance ,pathogenesisas well as characteristics

A

Dermatophilus congolensis
bacteria invade epidermis and root shafts
in rainy weather in horses at pasture
dorsum and lateral parts of the body
painful but no pruritus
crusts under which there is a pink-red open lesion
underside of crust is moist and yellow-greenish
pus may be present
in laboratory samples mention ‘dermatophilosis’

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14
Q

tx for drmatophylosis

A

improvement of the management
removing crusts (painful!)
clip as far as possible
repeated bathing and drying
systemic antibiotics (costs!)
disease is not highly contagious but it is
sensible to isolate affected horses

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15
Q

which dz is this,etiology, common areas, characterisitics, tx

A

Folliculitis and furunculosis
several bacteria
most often on tack-areas
papels – pustules
often quite painful, rarely pruritic
pustules may open and exude pus
treatment is hygiene !
sometimes systemic antibiotics indicated

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16
Q

which bacteri cuses this

common areas

A

abscess

Corynebacterium pseudotuberculosis

mostly pectoral muscle
ventral abdomen
groin

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17
Q

cs of corynebcterium pseudotubeculosis infection

A

lame or reluctant to walk
depression
weight loss
small to very large abscesses
complications like purpura
haemorrhagica or ulcerative
lymphangitis
bacteria is difficult to culture

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18
Q

discuss tx for corynebacterium pseudotuberosis

A

external abscesses
- mature abscesses (hot pack)
- abscess drainage and flushing
- NSAID’s (bute)
internal abscesses
- long course proc-penicillin / TMPS
limb infection (ulcerative lymphangitis

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19
Q

px for corynebacterium psydotubercolosis

A

external abscesses good
internal abscesses poor
ulcerative lymphangitis guarded

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20
Q

what causes cellulitis

A

several bacteria, often Staphylococci
often primary ‘entrance’ not detected
acute onset of severe swelling and pain of
one limb, often a hind limb
often extremely lame
often tachycardia and febrile
complications: cutaneous necrosis and
sloughing, laminitis, bacteremia

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21
Q

tx for cellulitis

A

immediate systemic antibiotics
hydrotherapy
support bandages other limbs
NSAID’s
if within 24 hours no improvement
corticosteroids

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22
Q

name the dz

characterists

A

Tail pyoderma
folliculitis and furunculosis of the tail
may cause severe pruritus and
automutilation
therapy
- clipping infected area and bathing
- systemic treatment with antibiotics
prognosis is guarded

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23
Q

name this condition

etiology

lesions,tx

A

Botryomycosis = bacterial pseudomycetoma
mostly coagulase positive staphylococci
mostly solitary nonpruritic nodular growths
incidentally multiple pustules, nodules and
draining tracts over large areas of the body
affected horses are otherwise healthy
surgical excision for solitary lesions - good
TMPS systemically for multiple lesions - poor

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24
Q

which lesion is dz

A

dermatophytosis

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25
Q

types of dermatophysis

A

superficial skin dz

trichophyton spp and microspora spp

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26
Q

pathogenesis for dermatophytosis

A

they primarily invade the hair shaftsto weaken it and cause breakage

some produce substances that can act as an irritatant or allergens and therefore produce pruritus

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27
Q

describe the lesions of dermatophytosis

A

it often starts with small papules with erected hairs. then the classical ring shaped lesions with alopecia and some scalling may develop or may be obvious as some lesions may coalesce or appear as paint brush type areas.

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28
Q

dx for drmatophytosis

A

it is done thru clinical appearance but confirmation can be done through microscopic examination of hairs and crusts for hyphae and conidia.

while cx is the only definitive way.

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29
Q

transmission of fungal infections

A

they are contracted through direct contact and theough indirect contact by saddle girth, brushes etc

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30
Q

tx for dermatophytosis

A

it is self limiting

tx is recommended to stop transmission or reduction in performance

use antifungal topicals

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31
Q

characteristics of fungal infections, incubation period, signalemnt, early signs

A

incubation period 6 days – 6 weeks
young and debilitated horses predisposed
early signs may be urticaria
variable degree of associated crust and
scale which is often ‘silvery’
hair can be lost in annular patches =
‘ring worm’
lesions of dermatophyte infection overlap
with bacterial infection
dermatophytosis is a zoonosis,

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32
Q

tx for dermatophytosis

A

reduce contagion to the environment
maximise horse’s ability to respond
(correct predisposing factors)
hasten resolution of the infection
topical therapy
systemic therapy? No!
vaccination

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33
Q

vaccination protocol for dermatophytosisi

A

Insol® Dermatophyton where to vaccinate?
deep intra-muscular
muscle is preferably ‘relaxed’
thigh is preferred
pectoral muscle is also suitable
preferably not in the neck
alternate sides between
vaccinations

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34
Q

When to vaccinate with
Insol®Dermatophyton

A

horses with clinical signs of dermatophytosis
possible infected horses
horses due to travel
(second injection has to be administered
at least 14 days prior to departure)
all horses on premises with historically
problems
hygiene is essential !!!

n.b horse has dermatophytosis,
but does not show clinical signs (yet),
these symptoms will show soon
after vaccination

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35
Q

When not to vaccinate with Insol® D ?

A

reduced immunity
- foals younger than 5 months
- sick or stressed horses
pregnant mares ??
close before important events!
hygiene is essential !!!

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36
Q

where do u normally find malazezia

A

mostly in intertrigenous areas (axilla,
groin, udder, prepuce, incidentally
coronitis)

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37
Q

what are the lesions of malaszia

A

very rare in horses
greasy, waxy, often malodorous dermatitis

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38
Q

example of a deep fungal infection

A

sporothrichosis

it is a chronic progressive sporadic skin and sq/ lymphatic infection caused by sporothrix schenckii

infection is usually introduced thru small skin wounds

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39
Q

cs of sporottricosis

A

firm,well dermacated, painless sq nodulesassociated with cored hardened lymphaticscommnly on limb regions (fetlock most common)

it is potentially zoonotic

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40
Q

tx for sporotrichosis

A

sodium iodide

sx removal of nodules is also possible

41
Q

what infection is this

A

3-year old Quarter horse mare - phaeohyphomycosis

42
Q

common transmission of mange

A

direct or indirect through grooming materials

43
Q

this mange live in skin surface and feed on skin debris

A

choriotic mange

it is caused by chorioptic equi

no interspecies transmission

and the mite doesnt affect pple

survival off host 70 days

44
Q

season for choriotic mange and area they are mostly found in the body

A

is often identified in winter in horses with feathered fedlocks.

transmission is thru direct contact.

the dz may be transmitted from season to season by horses that show no cs

45
Q

cs of chorioptic mange

A

cs are often limited to distal limbs and consists of scalling, fine papular eruptions but in heavy infectations the whole body can be affecte leading to general signs of pruritus, irritability and even wt loss

horses may stump their feet because of irritation

they may rub posts and bite at the limbs

46
Q

contributing factors for mange and season

A

mostly late winter – early spring
contributing factors
- crowding
- prolonged stabling
- suboptimal nutrition
- environmental temperature
limited host specificity

47
Q

characteristics of sarcoptic mange

A

0.25-0.6 mm in diameter
tunnel through epidermis
feed on tissue fluids + epidermal cells
life cycle on host ± 2-3 weeks
off-host survival time only a few days
‘endangered species’

N.B it is a digging mite while choriotic is a non burrowing mite

48
Q

location of sarcoptic mange, and clinical appearance

A

starts on the head
very severe pruritus
anaemia may occur
secondary infections may occur
mites very difficult to find
often ‘diagnostic therapy’

49
Q

tx for sarcopts mange

A

treatment with appropriate spray or dip
at least two times - 10-14 days between
few or even no products licensed
in the Netherlands we use foxim – Sebacil®
ivermectine should be useful
reportable disease in many countries

50
Q

characteristics of this mange

A

Psoroptes equi

0.4-0.8 mm long
nonburrowing
feed on tissue fluids
life cycle on host ± 10 days
14-18 days off-host survival time
but incidentally up to 84 days?

51
Q

clinical appearance of psoroptes mange

A

pruritic dermatitis of trunc and/or
ear disease with head shaking
mane and tail seborrhoea
mites rarely found

52
Q

treatment for psoroptes mange

A

ivermectine is very effective in the
treatment of psoroptes
otoacariasis and/or dermatitis
eggs are not killed – second dose
after 14 days necessary
efficacy of ivermectine may be
explanation for ‘extinction’
often unnecessary treatment in the
ears??

53
Q

characterists of this ectoparasite

A

Thrombiculidiasis
thrombiculid larvae ± 0.2-0.4 mm long
thrombiculid adults and nymphes are freeliving
larvae normally feed on tissue fluids of small
rodents, but incidentally horses
generally in late summer and fall
entire life-cycle 50-70 days (7-10 days
feeding as larvae on host)

54
Q

lesions of Thrombiculidiasis

dx and tx

A

infestation primarily in pastured horses
papules and wheals
muzzle, face, distal limbs
to make the diagnosis: chigger larva
seen in the centre of the lesion
variable pruritus
often self-limiting / topical treatment

55
Q

characteristics of this ectoparasite

A

Demodex
± 0.2 mm long (D.caballi + D. equi )
normal residents of the skin in the hair
follicles and sebaceous glands
feed on normal debris
life cycle takes ± 20-35 days on the host
survival off host few hours to days
probably not a contagious disease

56
Q

when does demodex become clinical

A

only clinical after long-term
glucocorticoid treatment
most times asymptomatic
alopecia and scaling, over the face, neck,
shoulders and forelimbs
papules and pustules may be seen
deep skin scrapings necessary
treatment not attempted

57
Q

this parasite is contacted by horse which live close to chickens

A

Dermanyssus gallinae

± 0.6-1.0 mm long
incidentally on horses
pruritic papules and crusts in contact areas
horse can be sprayed or dipped
eradication of the mite from the premises
(and the poultry)

58
Q

which tick is this plus cs, season

A

Dermacentor reticulatus

same as ixodes

spring n summer

incidentally in spring and summer
may attack any part of the body surface
paples, pustules, wheals
later crusts, erosions, ulcers, alopecia
pain and pruritus are variable
anaemia

59
Q

which ectoparasite is this

discuss its cs, season and lesions

A

Werneckiella equi
3-6 mm in lenght (nits 1-2 mm)
‘moving dust’
more in winter months
feeds on cutaneous debris
prefers dorsal trunk
pruritus mild to moderate
varying degrees of scaling and alopecia

60
Q

which parasite caused this lesions

A

Werneckiella equi

61
Q

name this parasite, its favourable areas in the body

A

Haematopinus asini
3-6 mm in lenght (nits 1-2 mm)
very rare in our area
feeds on blood and tissue fluid
favor main, tail and fetlocks
may cause anaemia

62
Q

this parasite is active in summer and
adult females glue eggs to hairs on legs
and shoulders

A

Gastrophiliasis – bots
Gasterophilus intestinalis = G. equi
G. nasalis, G. haemorrhoidalis, G. pecorum,
G. inermis
active in summer
adult females glue eggs to hairs on legs
and shoulders
cutaneous gastrophiliasis – 1-2 mm wide
greyish-white crooked streaks

63
Q
A

Gastrophiliasis – bots

64
Q

which parasite is this

A

Hypoderma bovis and H. lineatum
close contact to cattle
egg – larvae – skin penetration –
subcutaneous tissue
H. bovis – spinal canal and epidural fat
H. lineatum – submucosal connective
tissue of the oesophagus
second stage larvae to dorsum
swellings of the back with breathing pore

65
Q

Hypoderma – therapy

A

confirm diagnosis !!
enlarging breathing pore and extracting
the larva
surgical excision of the whole nodule
wait and allow larvae to drop out by
themselves
prevention: deworming with ivermectin
or moxidectin

66
Q

common places for this parasite

A

Hippobosca equina – louse fly
adult louse suck blood
tender to cluster in the perineal
and inguinal regions
only in import horses
horses may be ‘irritated’
therapy: insecticide

67
Q

this fly adult blowflies lay clusters of
light-yellow eggs in wounds

A

Lucilia sericata – myiasis
adult blowflies lay clusters of
light-yellow eggs in wounds
larvae hatch within 8-72 hours
larvae reach full size in 2-19 days
treatment
- cleansing and debriding
- topical insecticide
- symptomatic therapy

68
Q

where do u find oxyubiris

A

Oxyuris equi , Probstmayria vivipara
lives in coecum and colon
adult females lay eggs around anus
variable pruritus
incidentally restless and irritable
acetate tape – diagnosis
therapy: routine worming

also knon as pin worms

69
Q

clinical dz for habronemiasis

discuss season

A

summer sores
swamp cancer
granular dermatitis
begins in spring and summer
regresses partially or completely in winter

70
Q

discuss the lyf cycle of hebronemiasis

A

H. muscae, H. majus + Draschia megastoma
adults inhabit the stomach
eggs and larvae passed with faeces
Musca domestica + Stomoxys calcitrans
infectious larvae deposited on the horse in
moist areas or open wounds
larvae near mounth are swallowed –
complete parasitic life-cycle

71
Q

treatment for habronemiasis

A

perhaps partly a hypersensitivity disorder
- sporadic (one animal in a herd)
- seasonal
- recurs in same horse every summer
- systemic corticosteroids may be curative
no optimal therapeutic protocol
(surgery, local and systemic therapy)

72
Q

which parasite did this

A

Parafilaria multipapillosa
parafilariasis
haemorrhagic filariasis
summer bleeding
only import horses in the Netherlands
adult worms (3-7 cm) live in the
connective tissues coiled within nodules
nodules open at the surface and
discharge a blood exsudate (eggs, larvae)
therapy: time + avermectines?

73
Q

which parasite is this

where does it live in the body

its intermidiate host

A

Onchoceriasis
O. cervicalis, O. reticulata, O. gutturosa
adult worms live in lichamentum nuchae
or in connective tissues of tendons
microfilaria numerous in ventral midline
Culicoides spp. are intermediate host
in the Netherlands not diagnosed for
over 25 years

74
Q

name the parasite and its characteristics

A

Halicephalobiasis
Halicephalobus gingivalis =
Halicephalobus deletrix = Micronema
deletrix = Rhabditis gingivalis
details of life cycle not known
multiple organ systems can be infected
cutaneous lesions uncommon
diagnosis confirmed by biopsy
treatment ??

75
Q

proper way of cooling the horse

A

30 s. whole body cooling with cold water
30 s. walking in a circle
temperature should fall 1oC / 10 min.
allow horse to drink

76
Q

when should you stop cooling the horse

A

if body temperature < 38.0-39.0 oC
if skin over the quarters is cool after
walking
if respiratory rate < 30 breaths/min
if horse shivers

77
Q

what condition is this

A

Arabian fading syndromeo

78
Q

what condition is this

A

melanoma

79
Q

what lesion is this

A

sarcoid

80
Q

what dz is this

A

melanoma

81
Q

what condition is this

A

aloppecia areata

82
Q

which paraste causes this

A

choriotic mange

83
Q

which dz is this

A

Leucosis Eosinophilic granuloma

84
Q

what condition is this

A

saicoid

wound that grows

85
Q

what condition is this

A

habronemiasis

wound that grows

86
Q

what parasite is this one

A

sucking lice

87
Q

what parasites causes this

A

Chorioptes equi

Chorioptes equi , often lives with many!

88
Q

how is this dz dx

A

diagnosis by ‘diagnostic treatment’.

89
Q

what is going on here

A

Thick bite – Ixodes ricinus

90
Q

which tick spp is this

A

Hyalomma
marginatum

91
Q

which tick spp i this

A

Dermacentor reticulatus
http://upload.wikimedia.org/wikipedia/commons/7/77/Dermacentor_reticulatus_M_070825.jpg

92
Q

which condition is this

A

Dermatophilus congolensis – specific crusts

Painful and pink lesions

93
Q
A
94
Q
A
95
Q
A
96
Q
A
97
Q
A
98
Q
A
99
Q
A