Equine Arrhythmias Flashcards

1
Q

Arrythmias are the same in the horse except?

A
  • Very large heart, with large myocardium
  • Huge excess capacity at rest, therefore very high vagal tone
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2
Q

The vagus nerve controls which parts of the heart? What is the effect of this?

A

SAN and AVN
Increased vagal tone affects these nodes

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3
Q

The cardiac action potential conduction system pacemaker and threshold potentials can be influenced by?

A

Autonomic Nervous system
K+, Na+ and Ca2+ concentrations
Drugs

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4
Q

What is the function of the AV node?

A

Slow down depolarisation between the atria and ventricles so they don’t contract at the same time

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5
Q

What is the resting potential?

A

The difference between positive and negative potential difference in the myocytes
Resting potential is negative

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6
Q

What is the refractory period?

A

The time when a cardiac myocyte cant carry another wave of depolarisation

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7
Q

How is the pacemaker action potential different to the cardiomyocyte action potential?

A

Pacemaker action potential has a creeping resting potential that slowly increases until threshold is reached and it has a dramatic increase
Regularly sends out waves of depolarisation

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8
Q

Describe how to carry out an equine ECG

A

Base apex lead:
- Left arm (+): heart apex
- Right arm (-): mid right jugular furrow
- Neutral: remote from heart

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9
Q

The P wave represents?

A

Atrial depolarisation starting at the SA node

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10
Q

The P-R segment on an ECG represents?

A

Delay at the AV node
Prevents synchronous atrial / ventricular contraction

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11
Q

The QRS complex represents?

A

Depolarisation that starts at the AV node

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12
Q

The T wave on an ECG represents?

A

Ventricular repolarisation
- can change morphology / polarity at different heart rates = normal

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13
Q

Describe the heart sounds in the horse

A

Arial depolarisation and contraction = S4 ‘B’
Ventricular depolarisation and contraction = S1 ‘LUB’
Ventricular repolarisation and relaxation = S2 ‘DUP’

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14
Q

What are the 2 consequences of depolarisation starting at an ectopic focus (spreads over the atria in a different direction)?

A

Different wave of depolarisation
Different shaped ECG (p-wave)

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15
Q

Describe how an ectopic ventricular pacemaker alters the ECG

A
  • Normally starts at the AV node and spreads down the conduction fibres.
  • If the ectopic focus starts at another location i.e. the ventricular wall, it spreads in a different direction and takes longer. So the ECG is wider and appears bizarre
  • Ectopic ventricular pacemaker close to conduction system: run of ventricular tachycardia
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16
Q

Name the 2 common physiological arrythmias in horses

A

1st and 2nd degree AV block
Sinus arrythmias/bradycardia/tachycardia

17
Q

Name the 4 common pathological arrythmias in horses

A

Atrial Premature complexes (APC)
Atrial fibrillation
Ventricular Premature complexes
Ventricular tachycardia

18
Q

Name the 4 less common pathological arrythmias in horses

A
  • Atrial tachycardia
  • Junctional escape complexes/premature contractions
  • Ventricular fibrillation
  • 3rd degree AV block
19
Q

Describe the features of second degree AV block in horses

A
  • Common physiological arrhythmia at rest
  • Regular SA node depol.
  • Conduction pathway normal
  • AV node stops spread of depolarisation to ventricles every 3 - 4 beats
20
Q

Describe the ECG of a horse with 2nd degree AV block

A
  • Regular R-R intervals
  • Pause is approx double R-R interval
  • Normal QRS morphology
  • Normal P waves (and therefore S4)
  • Dropped beat
21
Q

What change is seen on the ECG in a horse with 1st degree AV block?

A

Prolonged P-R interval

22
Q

What change is seen on the ECG in a horse with sinus arrythmia?

A

Periodic waxing and waning of the R-R interval

23
Q

What change is seen on the ECG in a horse with sinus block or arrest?

A
  • Occasional pauses of 2+ R-R intervals
  • No P or QRS T on ECG
24
Q

Describe how an ECG would appear if a horse has atrial fibrilation

A
  • Irregular R-R intervals
  • Absence of P waves
  • ‘F’ (fibrillation) waves
  • QRS normal morphology
25
How is the heart rhythm affected by atrial fibrillation?
Becomes irregularly irregular Long pauses Premature beats No 4th heart sound Prominent S3
26
If atrial fibrillation is suspected, how is it diagnosed?
Echocardiography Check for underlying Pathology: - Serum electrolytes / FE - Complete Blood count - Cardiac troponin
27
How may a horse present if it has atrial fibrillation?
Poor performance Fading during race Epistaxis Often Incidental finding in sedentary / low level athletes
28
How can atrial fibrillation be differentiated from 2nd degree AV block?
Careful auscultation
29
AF in the horse without any underlying cardiac pathology called ...?
Lone atrial fibrillation
30
Is atrial fibrillation a cause of heart failure?
No AF can be an effect of other cardiac disease = ‘Secondary AF’
31
What are the two main options for treating atrial fibrillation?
1. Pharmacologic conversion –Quinidine sulphate 2. Transvenous electrical cardioversion
32
Describe how quinidine sulfate is used to treat AF
Oral Main effect = increases refractory period for atrial cells STOP if horse does not convert or shows severe side effects
33
Is a horse with atrial fibrillation suitable to ride?
- Low but still increased risk of collapse - Perform an exercising ECG up to/beyond level they will be ridden - Ectopic ventricular beats = not suitable - Only suitable for light work if in sustained AF
34
Regular atrial premature contractions can lead to..?
Atrial fibrillation
35
How many VPCs / APCs are significant?
>2 isolated premature at peak exercise >5 pairs or paroxysms post exercise