EQUINE Flashcards
What is string halt?
Stringhalt is a myoclonic disease affecting one or both pelvic limbs. It causes spasmodic hyperflexion of the leg. The etiology is unknown but sweet pea poisoning is thought to be associated with the condition. Diagnosis is based on clinical signs, but electromyography can be used to confirm the diagnosis. Treatment involves tenectomy of the lateral digital extensor tendon; however, not all cases respond to the treatment.
Bog spavin, also known as tarsal hydrarthrosis, is often due to poor conformation and can result in increased synovial fluid formation. Both hindlimbs are usually affected. The horse is usually not lame from this condition. Distension may spontaneously appear and disappear in young horses.
What is the problem with treating with piperazine?
The correct answer is large die-off of ascarids resulting in impactions. Treatment with piperazine will result in rapid death of ascarids. If a horse has a large burden of worms, this may be contraindicated as they may die rapidly and result in obstruction, as most likely occurred in this case. There is no reported specific adverse reaction of piperazine in Hanoverian horses. Small bowel bacterial overgrowth is not an issue. This would not happen after treatment with piperazine, especially a day after treatment. Infarction of the caudal mesenteric artery is not seen with use of piperazine.
Which of these is the most appropriate treatment for a horse with equine protozoal myeloencephalitis?
The correct answer is trimethoprim-sulfa and pyrimethamine. Equine protozoal myeloencephalitis is caused by Sarcocystis. This combination of drugs blocks folate metabolism by the protozoan at multiple steps and is generally agreed to be the most effective treatment for this condition. Recently, ponazuril has been approved and labeled for the treatment of EPM in horses and works by the same general mechanism.
What disease would be a top DDX for equine urinary incontinence?
Cauda equina neuritis and herpes virus myelocephalopathy
What are the CS of potomac horse fever?
Colic, fever, laminitis; looks like salmonella
What is Fistulous Withers?
Brucella abortus
What are the signs of Horner’s in horses?
The correct answer is sweating. Horner’s syndrome is caused by a disruption of the vagosympathetic trunk. Clinical signs include sweating (on the ipsilateral head and neck), enophthalmos (sinking of the eye ball), ptosis (drooping of the eye), and miosis (constricted pupil). Additionally, you will likely see a prolapsed third eyelid due to the enophthalmos allowing the third eyelid to just prolapse.
Two mares are presented for crusting dermal lesions on their dorsum and pasterns. The lesions are “paintbrush” like lesions and have purulent material exudating from them. A direct smear of the lesion shows branching, filamentous, gram positive bacteria. What is your most likely diagnosis?
The answer is dermatophilosis. Dermatophilus congolensis is a gram positive, non-acid fast, facultative anaerobic actinomycete. It causes a crusting dermatitis in large animals when there is high moisture on the skin as well as mechanical irritation. Horses with long, wet haircoats are often affected. Lesions are commonly on the dorsum and pasterns of horses and are classically described as paintbrush lesions. It can be diagnosed with a direct smear preparation. Treatment includes keeping the haircoat clean and dry and penicillins if the lesions are severe. Lesions typically heal rapidly.
A 4 year old horse presents to you with a mucopurulent nasal discharge, lethargy, and depression. On physical exam, the horse has a temperature of 103.7F (39.8 C) and has markedly enlarged mandibular and retropharyngeal lymph nodes. You suspect that the horse has strangles. What would you expect to see if you aspirated one of the enlarged lymph nodes?
The correct answer is purulent inflammation and gram positive cocci with large capsules. The causative agent of strangles is streptococcus equi ssp. equi which is a gram positive cocci with a large capsule. It causes suppurative abscessation and enlargement of the mandibular and retropharyngeal lymph nodes.
A 2-year old Quarter Horse presents for intermittent muscle fasciculation followed by weakness. What condition should you suspect?
The correct answer is hyperkalemic periodic paralysis (HYPP). HYPP is seen in Quarter Horses due to a point mutation in a key part of a skeletal muscle sodium channel subunit. This results in elevation of the resting membrane potential to increase the likelihood of depolarizing. Excess concentrations of potassium can result in failure of the sodium channels to inactivate. Therefore, treatment is directed at decreasing dietary potassium.
Treatment of stringhalt in a horse involves which of the following?
The correct answer is tenectomy of the lateral digital extensor. Stringhalt is a myoclonic disease affecting one or both pelvic limbs. It causes spasmodic hyperflexion of the leg. The etiology is unknown but sweet pea poisoning is thought to be associated with the condition. Diagnosis is based on clinical signs, but electromyography can be used to confirm the diagnosis. Treatment involves tenectomy of the lateral digital extensor; however, not all cases respond to the treatment.
A horse presenting for discharge around the ear should be assessed for this condition.
The correct answer is dentigerous cyst. This occurs as a result of having tooth germ tissue in the ear. Usually, a swelling at the base of the ear and observe a seromucoid discharge can be identified. Treatment is surgical removal of the tissue.
How many permanent teeth do horses have?
Horses have between 40-42 permanent teeth.
Which of these would you expect to see in a horse that has had significant blood loss?
The correct answer is hypoproteinemia. The expected findings in a horse with blood loss are anemia (normocytic, normochromic), hypoproteinemia, and responsive bone marrow.
You are presented with a 6-month old colt with a 5-day history of lethargy, intermittent diarrhea, weight loss, and ventral edema. A quick check of the PCV and TP reveal a PCV of 30% (28-42%) and TP of 3.2 g/dl (6.8-8.2 g/dl). Abdominal ultrasound (see image) demonstrates thickening of the small intestinal wall. What is the most likely diagnosis based on signalment, history and clinical findings?
Although not as commonly as in pigs, L. intracellularis can infect horses (and other species). Similar to pigs, the infection usually involves weanling age horses and causes thickening of the small intestine and hypoproteinemia. The low protein is commonly observed as ventral edema clinically. Both Clostridium and Salmonella can cause diarrhea at any age but are not as commonly associated with ventral edema. Intussusceptions do occur but also typically result in colic.
Which of the following is not believed to be a cause of physitis in young horses?
The correct answer is osteochondrosis. Physitis in young horses involves swelling around growth plates in long bones. Physitis is sometimes thought to be a component of osteochondrosis, but the other answer choices have been suggested to be causes of physitis.
Approximately how long does it take for spermatogonia to mature to sperm in the stallion?
The correct answer is 60 days. For this reason, a stallion with poor quality semen should be re-evaluated in 2 months before making a final judgment.
Which cells does caprine arthritis encephalitis virus infect?
monocytes.
Where do nephrosplenic entrapments occur in the horse?
The correct answer is between the spleen and the left body wall. The nephrosplenic ligament runs from the left kidney to the spleen, and a nephrosplenic entrapment occurs when a portion of large intestine is looped over the nephrosplenic ligament, becoming trapped between the spleen and the left body wall.
What is the most common infectious cause of abortion in horses?
The correct answer is equine herpesvirus. This is the most common cause of late-term abortions and accounts for about 25% of all diagnosed abortions in the horse.
A 5-year old brood mare presents with a history of intermittent hind limb lameness. The onset of the lameness has been gradual. The mare is an active cutting horse. With your initial hands-off examination, you compare the structural integrity of both hocks (see image). Without using any further diagnostics, what disease process and corresponding anatomical structures do you suspect is causing the lameness?
The correct answer is bone spavin: osteoarthritis of the distal intertarsal joint and/or tarsometatarsal joint. Bog spavin is synovial distention of the tarsocrural joint, which you cannot see or appreciate with the given image. The only true answer with the information given is osteoarthritis of the distal tarsal and tarsometatarsal joint. All you have to do is memorize which spavin correlates with the appropriate anatomy. This question is designed to help you use the information given and not to get caught up in over analyzing the question.
You are examining a 3-year old Thoroughbred gelding for lameness of the right forelimb. The lameness persists with a palmar digital and abaxial sesamoidian nerve block but is improved by 90% after a low 4-point (low palmar) nerve block. The source of the lameness is likely in which region of the limb?
The low 4-point (low palmar) nerve block typically provides local anesthesia to the metacarpophalangeal (fetlock) joint and below. An injury to this area is most likely. The proximal suspensory ligament and carpal-metacarpal joint would require local anesthesia more proximally.An 8-year old Quarterhorse brood mare presents with an acute onset of colic. On presentation she has a heart rate of 58, respiratory rate of 28, and is seen rolling on the ground. CRT is approximately 3.0 seconds and mucous membranes are slightly red. She has rectal temperature is 101.6F (38.7 C). Gastrointestinal sounds are completely absent and gastric reflux yielded 15L of brown- to yellow- colored fluid. Peritoneal fluid analysis yielded a cloudy yellow fluid with a protein of 2.8 gm/dl and white blood cell count of 11,354/ul. Which of the following is not a likely diagnosis?