Ep General Flashcards

0
Q

Recordings egm

A

Distal to proximal if the catheter

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
1
Q

EP test indications

A
Syncope in structural heart disease
Wide complex tachycardia 
Assessment of Brady cardia
Sudden death 
For mapping
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Hv interval

A

35-55

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

EAD

A

Phase 3
Bradycardia
Torsades
Idiopathic VT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Dad

A

Phase 4
Digitoxicity
Ca, catecholamines excess
Cpvt

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Increased automaticity

A

Sinus tach
AT focal
AIVR
Idiopathic VT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Reentry

A

Afl
Avnrt
Avrt
MMVT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Qrs

A

Narrow if you use normal conduction

Wide if it uses Accessary pathway

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Atrial ectopy

At, avrt,avnrt

A

Ventricular ectopy

AVRT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Short RP
AvNRT
Orthodontic avrt

A
Long RP
Atrial tachycardia 
Atypical avnrt
Orthodontic avrt with slow pathway
Sinus tachycardia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Atrial tachycardia

A

Does not use normal conduction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Avnrt

A

Terminate with adenosine when you block the AV node

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Vt

A

R in v1 >40 ms
>60 msec from qrs onset to S nadir
Notched down stoke S wave in V1V2
Any Q in V6

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Brugada criteria

A

Absence or RS complex VT
R to S interval >100 msec one lead VT
AV dissociation VT
VT morphology in V1-2 or V6 VT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Intra cardiac tracing

A

More V’s than A’s VT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Cardiac action potential

A
Phase o sodium 
Phase 1 transient outward current
Phase 2-3 calcium in potassium out
Phase 4 ca k reequilibriate 
Phase 4 If pacer
16
Q

Vaughn Williams

A

Class 1 sodium channel
Inhibit depolarization slow conduction
Class 3 increase refractoriness and prolong repolarization

17
Q

Sodium blockers

A
Type1A quinidine 
Intermediate kinetics
Prolong APD/QT torsades 
1b Lido rapid kinetics
1c slow kinetics qrs prolonged
18
Q

Class 3 potassium channel blockers

A

Block potassium channels mediating I kr, Iks
Prolonged refractory/ APD
Ibitelide also prolongs sodium channel opening

19
Q

Prevention of VT

A

Sotolol sword study increase death
Dronedarone andromeda increase death
Amio gesica, emiat, camiat mixed results

20
Q
SCD-Heft 
Lvef <35% class 2,3
A

Icd therapy better than amio

21
Q

Vt VF

A

Lido and procan and bretylium removed from CPR

Amio improves survival ARREST

22
Q

To prevent icd shocks

A

Amio>sotalol >betablockers

Consider cathter ablation for mmvt

23
Q

Indication for use svt

A

Class 1 vagal, adenosine, calan

Class 2 beta blockers amio dig

24
Q

Chronic medal therapy svt without structural heart disease

A

BB, ca blockers
Flecanide rythmol
Sotalol
Amio

25
Q

Svt LVH

A

Dronedarone amio

26
Q

Cad

A

Defetolide sotalol
Amio
Dronedarone

27
Q

Heart failure

A

Amio

Defetolide

28
Q

Maintain NSR

A

Amio>sotalol> rythmol >Dronedarone

29
Q

Dronedarone

A

Don’t use in CHF and persistent afib

30
Q

Renal

A

Sotolol
Dig
Difetilide

31
Q

Hepatic

A

Lido
Amio
Rythmol

32
Q

CYP2D6 deficiency

A

Enhanced bb activity with rythmol

33
Q

Increase DFT

A

Mexiletine
Flec/ rythmol
Amio

34
Q

Decrease DFT

A

Sotolol

Dofetilide