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PHTY2101 > EOS > Flashcards

EOS Flashcards

1
Q

NWB Gait with walking Aid

A

Start with tripod position

  • Step to gait (crutches move together and feet move together)
  • Step through gait (crutches forward together, limbs forward together past crutches)
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2
Q

PWB Gait with walking aid

A

Start with tripod position

  • Step to (crutches and affected limb together, unaffected follows)
  • Step through (crutches forward with affected unaffected follows and goes beyond)
  • 4 point reciprocal - RC, LL, LC, RL
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3
Q

Transfers

A

Transfers ( both crutches in one hand, standing using arm rest or bed, transfer crutches)

Stairs - always lead with good leg and crutches stay with bad

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4
Q

Shrinker sock measurement

A

Above knee - thickest part and 5cm above distal stump

Below knee - patella and 5cm above distal stump

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5
Q

K level classification

A 
0- unable 
1 - in house 
2- some in community 
3- fully community
4- above expected level
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6
Q

Hypertrophic scar and its risk factors

A

Redness (increased blood supply), raised and thick (build of granulation tissue), less pliability (minimal regen of elastin fibres), reduced skin stretch (from changes in ground substances and constant contraction through myofibroblast activity). Altered sensation (painful, itchy)

Main risk factor is the time of healing (longer = increased risk)
Site of scar (across joints), depth of wound, skin grafting
Skin grafting increases risk, full thickness has highest increase

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7
Q

Positions for acute stage management

A 
Neck - slight extension (30)
Shoulder - 90 abd
Elbows - full ext
Hips - neutral rotation
Knee - full ext 
Feet - plantar grade
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8
Q

Factors that contribute to contracture development

A

Depth of wound, TBSA, duration of immobilisation, muscle weakness (unable to oppose force), race and skin type, growth spurts, other pathology leading to more immobilisation (heterotopic ossification or factures)

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9
Q

3 phases of wound healing

A

Inflammation (1-5 days)

Proliferation (3-5, 3 weeks). Fibroblasts synthesising collagen and ground substance. Fibroblasts differentiate into myofibroblasts (contraction). Wound is closed by the end of this phase (scar tissue)

Remodelling (3 weeks – 12//18 months). Collagen forms cross links to increase tensile strength of scar tissue. Orientation of collagen becomes less random and more parallel.

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10
Q

Surgical burn management

A

Moving down with increased burn depth (also means increased difficulty for healing)

  • Autograft (gold standard)
    • Surrounding joints immobilised to allow graft to take
    • Spilt thickness (donor site will heal on its own) or full thickness (site requires split thickness to cover)
    • Meshed grafts allow better and earlier taking as well as exudate being able to be evacuated from under. Non meshed are better aesthetically but are more fragile in early stages and nurse will need to prick to evacuate.
  • Cultured autologous keratinocytes
    • Large TBSA burns can be covered without donor sites, nil rejection. It is fragile due to limited dermal attachments
  • Homograft - Cadaver allograft
    • Acts almost like a bandaid as tissue rejection will occur in 7-14 days
    • Can be used over autograph to increase rigidity
  • Skin substitutes
    • Materials are removed and often require grating after
  • Flaps
    • When skin graft is not enough to cover a wound, cover exposed bone/tendon ect
    • Either skin or muscle flaps
    • Flaps are transferred with their own blood supply
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11
Q

Escharotomy and fasciotmy description as well as their considerations for burns patients

A

Escharotomy
Incision into burnt skin to restore distal circulation

Fasciotomy
Relieve swelling and pressure via incision

When mobilising make sure that only light ROM for Fasciotomy. Can mobilise with escharotomy need to observe for bleeding.

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12
Q

Physio in grafting phase

A

Stop to allow graft to take

Should have maintained full ROM prior to grafting

Immobilise joint above and below graft

Maintain ROM in other areas

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13
Q

Physio managment in acute stage burn injury (pre graft)

A
  • Assessment (sub/obj)
  • Respiratory
  • Oedema
    • Elevation
    • Breathing exercises (prox lymphatics)
    • Active exercise for muscle pump (while elevated)
    • Compression bandage in hands or lower limb (DONT if peripheral vascular disease)
  • Exercise
    Minimise risk of contracture (movement in opposite direction)
    ROM -> resisted
  • Early mobility
    Mobilised day 1 post burn if able in FWB
    • Escharotomies down legs
    • Exposed Achilles
    • Burns to soles of feet (need adequate protection)
  • Positioning
    • Need to be in anti deformity positions (+/- splints)
  • Pain management
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14
Q

Physio management in scar maturation

A

Scar assessment
-Location, skin mobility/contracture, strength and function

Contracture prevention:

  • Exercise (ROM, hold/contract relax, strength, CV)
  • Splinting to avoid positions

Other scar management

  • Compression garments
  • Contact media
  • Scar massage
  • Surgical
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15
Q

Long term outcomes and considerations for burns

A

Not just about skin

With >20% TBSA = hyper metabolism. Need to start early gym and CV to prevent wasting

Exercise also decreases risk of heterotopic ossification (deep joint pain + decreased ROM)

Shorter time to fatigue (worse with increased TBSA)

Often have itch, need to avoid (moisturise, massage, medication)

Skin care:

  • loss of lubrication so moisturising required
  • Sun protection more important
  • Heat intolerance
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16
Q

Key components of objective assessment after ortho

A
  • Respiratory
    • Deep breaths
    • Cough
  • Circulatory
    • DVT (specific to patient, Achilles repair may have these)
      • Swelling of calf
      • Redness of calf
      • Localised pain/tenderness Increased temperature
      • Positive Homan’s sign (calf pain on passive DF)
    • Ankle Circles
    • Compartment syndrome, bleeding into connective tissue
      • palor, pain, paraesthesia, pulses, paralysis
  • Neurological
    • Dermatome sensation
    • L2-L5
  • Musculoskeletal
    • Specific to affected region

Do functional assessments

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17
Q

5 P’s of compartment syndrome

A
  • palor, pain, paraesthesia, pulses, paralysis
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18
Q

Important TKA management goals

A
  • Knee flexion >/= 90
  • Knee ext 0 (critical for gait and shock absorption)
  • Single leg raise with no lag
  • Independence with HEP
  • Independent mobility on stairs
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19
Q

Physio management overview TKA

A

Day 1

  • Circuloresp and qauds exercises
  • Knee flex (90)
  • SLR
  • FWB so mobilise with rollator
Day 2 (discharge)
-Swelling management, more quads (no lag), GAIT edu and knee flex

Discharge criteria
-SLR (<5 lag), knee flex >80, independent mobility including stairs, home exercises

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20
Q

Hip replacement dislocation positions

A

Post - flex >90, adduction past neutral, IR past neutral (knee to elbow) possible during sitting

Ant - force ext. Flex or ext with add and ER (block ant)

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21
Q

THA overview

A

Day 0

  • Mobilise 2-3 post in WBAT
  • Hip ROM, quads, circuloresp, inner range quads, bridging and bed mobility

Day 1
-progress mobility

Day 2 - discharge

  • progress ROM and strength functionally (mini squats)
  • balance, mobility, stairs

Discharge education
- avoid diss positions, no low chairs, no cross lgs, dont lie on affected side, dont squat to ground, bend to pick up, twisting, no driving in first 6 weeks

22
Q

Meniscectomy

A

Management - FWB, rehab include ROM, SLR, IRQ and limit walking to manage swelling

23
Q

Meniscal repair

A

Mobilised NWB crutches, ROM restriction to limit shear forces, still have ROM exercises, SLR, IRQ and manage pain and swelling

24
Q

ACL repair

A 
ROM brace (Richards splint) to 90 
Slow return to flexion, 110 over first 2 weeks and full by 6
25
Q

Pelvic fracture management

A

Stable normally conservatively

  • system maintenance (cardio, resp, neuro)
  • teach bridge and block rolling
  • WBAT on aid, hip and knee strength with decreasing pain
  • Address balance for further falls
  • Discharge when independent with aid
Unstable 
-Normally surgical not traction
-Ant ex fix is temporary 
Post op rehab variable due to high impact trauma, need notes
RIB 48 hrs -> TWB 6-8/52
26
Q

Acetabular fractures

A

Often need THA

27
Q

Wound closer for burns

A

Superficial = 7-10 days
Superficial partial thickness = 14 days
Deep partial thickness = >14 days
Full thickness = 3 weeks

28
Q

List of physical impairments

A 
Limb shortening
▪Weight transmission
▪Muscle loss
▪Muscle imbalance
▪Loss of joints
▪Loss of sensation
▪Changed base of support
▪Change to circulation
▪Impaired heat loss
▪Increased energy cost of ambulation
29
Q

5 stages of fracture healing

A

Tissues destruction and hematoma formation -

Inflammation and cellular proliferation (up to 2 weeks)-

Callus formation (2-4 weeks) - Woven bone formation

Consolidation (8 weeks) - Woven bone to lamellar bone, clinically and radiographically united

Remodelling (2 months - 2 years) - resorption and formation

30
Q

Discectomy and microdiscectomy

A

Discectomy

Often done with partial laminectomy

Microdiscetomy

Just a smaller incision, meaning the recovery time is smaller and surgical trauma is minimised just to less soft tissue effected.
Inadequate decompression or Dural tearing

31
Q

Lumbar spine stenosis and symptoms

A

Lumbar spine stenosis

IV disc loose fluid with age (less resistance to compression), loosing height meaning disc can bulge into vertebral canal. Spinal facet joints can thicken and enlarge with arthritis, further limiting the canal. Spinal stenosis = narrowing the canal

Symptoms:

  • Lower back pain
  • Pine, needles, numbness in legs
  • Cramping and weakness in legs
  • mediating pain with forward flexion

(need to decompress)

32
Q

Laminectomy

A

Laminectomy

Removal of piece of lamina to decrease pressure on spinal cord or nerve root. Cannot do more than 2 due to decrease in stability, this case would require fusion.

33
Q

Management of decompressive surgery (discectomy/laminectomy)

A
  • Circuloresp, neurological checks, log roll for comfort
  • Regular changes of positions
  • Transverse and multifidus activation
  • Mobilise day 0-1 on orders
  • Need to be intendent on stairs and HE for discharge (day 0/1)
34
Q

Spinal fusion techniques

A

Instability, deformity (scoliosis, kyphosis, spondylolisthesis) or other conditions (osteomyelitis, TB)

Techniques:

  • Bone grafts
    • Unable to correct spinal deformity
    • Used before fusion
    • Requires bracing after if done alone
  • Posterior lumbar fusion
    • Screws
  • Anterior lumbar interbody fusion
    • For high grade spondylolisthesis
35
Q

Spinal fusion management

A

Management:

  • +/- Bracing (need for just bone graph)
  • Circuloresp, neuro checks
  • Don’t sit for long periods, log roll for comfort
  • Exercise day 1
  • Multifidus and transverse
  • Ind3pdent on stairs and HEP for discharge (day 2-3)
36
Q

Scoliosis treatment and management

A

Treatment:

Depends on angle of curve

<15 - advice on exercise, muscle strengthening and posture

15-30 - bracing

> 30-40 - surgery

Post OP:

  • Post op immobilisation, balance and movement will feel different
  • Circuloresp exercises, log roll, exercises dat one, mobilise day 1-2 (one stage)
  • If two stage then after first circuloresp, log roll, no SLR, hip/knee flexion then after stage 2 same as one stage
  • Discharge when independent on stairs and HEP
37
Q

Neural mobiltiy exercises

A

Program for after spinal surgery to mobilise neural structures, limit scarring and relieve pain and symptoms.

  • stage 1 - DF and PF ROM, hip abd/add, hip rotations in neutral → DF/PF in small SLR
  • stage 2 - knee ext in some hip flex (some gentle SLR)
  • stage 3 - progress with increasing hip flexion
38
Q

Spinal fractures

A

Cervical

Stable - collar for 6 weeks. System ass/maintence (neuro!), log roll

Unstable - skull tongs or stabilisation +/- halo brace (impacts balance, log roll in/out bed). Systems ass/maintenance (neuro!), chest maintenance

Thoracic

Often stable - brace, systems, log roll (avoide rotation/flexion), mobilise with aid

Lumbar has same physio

39
Q

What is OA

A
  • Disorder that involves multiple joints
  • ECM degeneration and cell stress along with maladaptive repair processes
  • Begins first as a molecular issue (abnormal joint tissue metabolism) this then progresses to have anatomic and/or physiologic abnormalities such as cartilage degeneration, bone remodelling, osteophyte formation, joint inflammation all of which contribute to loss of normal joint function.
  • Important to remember that these issue can but don’t always relate to symptoms

A common misconception is that it is just wear and tear of articular cartilage but OA is a WHOLE joint disease (meniscus, labrum, cartilage, synovium, muscles, subchondral bone). Not just mechanical, also contains inflammatory and metabolic aspects

40
Q

Dfferences between OA and R

A

OA:

  • Gradual onset of symptoms, +/- joint stiffness that is no longer than 30 mins, Activity related joint pain, >45 age, Imaging (osteophytes, reduced joint space, bony contour deformity)

Rheumatoid:

  • Faster onset, Hot swollen joint, Stiffness longer than 30 mins, Systemic features, More common bilaterally
41
Q

Specific features of OA in hip and knees

A

Knee:

  • Pain in knee (either whole or just joint lines)
  • Instability and giving way (pain inhibition of quads or from structural deformities)
  • Crepitus
  • Loss of full ROM (osteophytes, joint shape)
  • Varus or valgus
  • Muscle atrophy and weakness
  • Synovial thickening

Hip:

  • Pain in variety of areas around hip and thigh
  • Loss of ROM
  • Muscle atrophy
42
Q

When should surgery be considered for OA

A

After first and second line treatment have been trialled and the patient has true mechanical locking.

43
Q

Stages of wound healing

A

Inflammation (1-5) -

Cellular proliferation (3-5 - 3w)
Fibroblast secrete collagen and ground substance, fibroblasts differentiate into myofibroblasts.
Myo cause wound contraction to pull edges of wound together.
Underlying cause of contracture. Wounds is closed at the end of this phase, scar tissue created.

Remodelling (3w- 12-18m) -
Collagen forms cross links to increase tensile, ground substance more dense, collagen orientation less random. Scars are adaptable during this phase.

44
Q

Contracture development

A

Scar tissue, contractures due to myofibroblasts. These are first seen 5-7 days but peak at 4-5 months. Scare contracture force left unopposed by resistance.

45
Q

Conservative burn management

A

Silver based dressings

46
Q

ASIA impairments UL motor

A 
C5 - elbow flexors 
C6 - wrist extensors 
C7 -  elbow extensors 
C8 - finger flexors 
T1 - 5th abduction
47
Q

ASIA impairments LL motor

A 
L2 - hip flex 
L3 - knee ext
L4 - Ankle DF
L5 - toe ext 
S1 - Ankle PF
48
Q

Grades for ASIA

A

A - Complete (no sensory or motor in sacral)

B - Sensory Incomplete (Sensory but no motor below level and includes sacral)

C - Motor incomplete (Motor below level and more than half muscles grade <3)

D - Motor incomplete (Motor below level and more than half muscles grade >3)

E - Normal

49
Q

Sensory for ASIA

A

28 dermatomes tested using 3 point scale:

0 - absent

1 - impaired (feels different)

2 - normal (same as reference)

This scale is used for light touch and then for differentiation between sharp and blunt

50
Q

Level of neurological injury

A

Lowest segment with normal sensory and motor on both sides

51
Q

Early SPI rehab

A

Pressure redistribution is important:

  • Pressure lifts, pressure leans or tilt in space equipment. Do this every 20-30 mins
  • Balance
  • Bed mobility
  • Transfers
52
Q

Functional electrical stimulation

A

Functional electrical stimulation

This is the application of electrical current to paralysed muscle to restore or improve function. It is applied to achieve a functional task.

30-60 mins of exercise 3 times per week is requirede

PHTY2101 (7 decks)

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