ENT Staging Systems Flashcards
ANOMALOUS FACIAL NERVE IN THE MIDDLE EAR (Rohrt & Lorenzen)
- Facial nerve partially obliterates stapes footplate
- Bifurcation of VII
- VII rests on footplate with deformed stapes or oval window
- VII rests on promontory
Anomalies often in association with stapes fixation Stapes may be normal even in association with VII bifurcation
SIGNS OF OPTIC NEUROPATHY (5)
- Visual acuity
- Snellen chart vs finger counting vs hand movement (light perception only) - Pupils
- RAPD (bilateral dilation of pupils) - Colours
- Ishihara chart, check for red desaturation - Fields
- Subjective brightness
- Compare torch brightness between the 2 sides AcuityBrightnessColorDilation(RAPD)Fields
SPHENOID APPROACHES
Approaches:
Transnasal
Transethmoid
Transseptal
Trans-pterygoid root (violates SPA, PPF)
Wilson Grading for Pituitary Adenomas (Based on Extrasellar Extension)
STAGE
DESCRIPTION
0
No suprasellar extension
A
Extension into suprasellar cistern only
B
Extension into anterior recess of third ventricle
C
Obliteration of anterior recess and deformation of floor of third ventricle
D
Intradural extension into ACF/MCF/PCF
E
Extradural invasion into cavernous sinus
pl 147 105
THYROID N STAGING
N0 = no regional LN metastasis
N1a = Level VI nodes
N1b = Metastasis in other unilateral, bilateral or contralateral LNs
Thyroid staging is weighted towards primary site
- Lateral neck disease does not significantly alter prognosis
THYROGLOSSAL DUCT CYST
- Lingual 2%
- Suprahyoid 24%
- Thyrohyoid 61%
- Suprasternal 13%
TNM CANCER STAGING Not NPx Not p16+ OroP SCC
1 = 1
2 = 2
3 = 3 or N1
4a = 4a or N2
4b = 4b or N3
4c = M1
INVERTED PAPILLOMA (Krouse)
Developed by Krouse, Laryngoscope 2000:
- Confined to nasal cavity
- Ethmoids, OMU, medial wall maxillary sinus
- Anterior/lateral/superior/inferior/posterior walls maxillary sinus, frontal, sphenoid
- Extrasinus or any tumour associated with malignancy
Initial paper suggested endoscopic approach for T1/T2/selected T3
- Now routinely resect T3 and T4 with endoscopic approach
Recurrence rate directly related to extent of surgery
FRIEDMAN STAGING
dont forget BMI
Oral Cavity SCC T Staging AJCC 8th ed
T1 tumour <2cm and DOI < = 5mm
T2 Tumour < 2cm and DOI >5 - < =10mm OR 2-4cm and DOI < = 10mm
T3 Tumour >4cm or DOI > 10mm
T4a (oral cavity) Tumour invades adjacent structures- through cortical bone, into deep extrinsic muscles of tongue (genioglossus, hyoglossus, palatoglossus, styloglossus), maxillary sinus, skin of face
T4a (lip)- tumour invades through cortical bone, inferior alveolar nerve, floor of mouth, or skin of face
T4b Tumour invades masticator space, pterygoid plates, skull base or encases carotid
4,4,4 check Front: cohesive or non cohesive? whats this?
ANGLE CLASSIFICATION OF MALOCCLUSION
Assesses relationship between: 1 is to 1
- Upper 1st molar tooth- mesiobuccal cusp (1st cusp): top molar is more posterior
- Lower 1st molar- mesiobuccal groove
Class I:
Maxillary cusp sits within the mandibular groove
- Normal arrangement
Class II:
Maxillary cusp sits in front of mandibular groove — retrognathia
Class III:
Maxillary cusp sits behind mandibular groove — prognathia
FITZPATRICK SKIN CLASSIFICATION
III: sometimes, gradual
STOMAL RECURRENCE (Sisson, 1976)
- Superior 1/2 of stoma without oesophageal involvement
- Superior 1/2 of stoma with oeseophageal extension
- Inferior 1/2 of stoma with direct extension to mediastinum
- Extension laterally and often below clavicles
Survival:
I/II = 45% at 2 years
III/IV = 9% at 2 years
- Aggressive Rx of III/IV usually not indicated
- Risks of surgery: great vessel injury, hypocalcaemia, fistula formation, mediastinitis
Risk factors:
- Advanced T stage — invasion of thyroid gland
- Advanced N stage — paratracheal node involvement
- Pre-op tracheostomy
- Subglottic involvement — failure to obtain clear margin
- Failure to complete PORT
Nasopharynx SCC T Staging
T1
Tumor confined to the nasopharynx or extends to oropharynx and/or nasal cavity
T2
Tumor extends to soft tissues of parapharyngeal space; muscle involvement such as MP, LP, PV muscles (new classification)
T3
Tumor involves bony structures of the base of skull and/or paranasal sinuses
T4
Tumor with intracranial extension and/or involvement of cranial nerves, infratemporal fossa, hypopharynx, orbit, or masticator space (5) air, soft tissue, bone, intracranial/orbit
FRONTAL MUCOCELE
Type 1: limited to frontal sinus (+/- orbital extension)
Type 2: Frontoethmoidal mucocele (+/- orbital extension)
Type 3: erosion of posterior wall
A. minimal or no intracranial extension
B. major intracranial extension
Type 4: erosion of anterior wall
Type 5: erosion of both posterior and anterior walls A. minimal or no intracranial extension
B. major intracranial extension
80% present with some degree of proptosis
REFLUX SYMPTOM INDEX 9 questions
RSI>13 strongly correlates with LPR on dual-probe pH study
Hoarseness, throat clearing, PND, cough x 2, dysphagia, heartburn, globus, aspiration
FUJITA CLASSIFICATION
Type 2 subdivided- A = predominantly palate, B = predominantly tongue
1 retropalatal collapse
2 both
3 retrolingual
MAXILLARY SINUS SCC T STAGING
- Sinus mucosa, no bony destruction
- Bony erosion — HP and middle meatus
- Excludes post wall and PPF - Post wall max sinus, PPF, subcutaneous tissues, ethmoid sinus, floor/med wall orbit (3 for E)
4a. Ant orbit, skin of cheek, ITF, pterygoid plates, frontal/sphenoid sinus, cribriform plate
4b. Orbital apex, dura, brain, MCF, CNs (except V2), NPx, clivus (same as ethmoid/nasal cavity SCC 7)
“4a for frontal”
CHOLESTEROL GRANULOMA
- Confined to one area of mastoid or middle ear system 2. Involves entire middle ear cleft
OLFACTORY ESTHESIONEUROBLASTOMA (Hyams histologic grading)
Graded I-IV
I = well-differentiated, relatively indolent
IV = undifferentiated, extremely aggressive
Homer Wright (pseudorosettes) present in Grade I and II (photo)
- Grouping of cells in a circumferential fashion around neurofibrillary matrix but without basement membrane
Flexner-Wintersteiner (true rosettes) present in Grade III and IV
- Cells align in a glandular fashion around spaces lined by distinct cell membranes
page 112 in Wenig
Major Salivary Gland T Staging
T1- Tumour <2cm in greatest dimension without extraparenchymal spread
T2- Tumour is 2-4cm without extraparenchymal spread
T3- Tumour >4cm and/or has extraparenchymal spread
T4a- Tumour invades skin, mandible, EAC and/or VII
T4b- Tumour invades skull base and/or pterygoid plates and/or encases carotid
*Extraparenchymal extension is clinical or macroscopic evidence of invasion of soft tissues.
T3 and above: N0: SND
EWALD’S LAWS/ ALEXANDER’S LAWS
EWALD’S LAWS:
1st- direction of nystagmus in plane of affected canal
2nd- Lateral canal- ampullopetal flow (towards ampulla) produces greater response
3rd- Vertical canals- ampullofugal flow (away from ampulla) produces greater response
ALEXANDER’S LAWS: degrees of nystagmus
1st- looking in direction of fast phase
2nd- present looking straight ahead
3rd- present looking in all directions
CUTANEOUS SCC T Staging N Staging as for mucosal sites (7th vs 8th edition change?)
T1 < 2cm and < 2 high-risk factors
T2 > 2cm or > 2 high-risk factors
T3 Invasion of maxilla, mandible, orbit, temporal bone (bone)
T4 Skeletal invasion (axial/appendicular) or perineural invasion of skull base
(AJCC 8th Edition: removed the risk factors)
High risk factors:
- Depth/invasion
- >2mm thickness
- Clark level > IV
- Perineural invasion - Anatomic location
- Primary site ear
- Primary site hair-bearing lip - Differentiation
- Poorly differentiated or undifferentiated
RT-INDUCED MALIGNANCY Diagnostic criteria
Cahan 1948:
- Within the treatment field
- Significant latency period
- Different type of cancer to the original
Controversial area- patients with one cancer are at increased risk of another due to lifestyle and genetic factors
Benefits of RT outweigh the risks of induced malignancy — shouldn’t be a factor in deciding Rx
- Maybe in those < 40 — avoid RT
Types of cancer:
- Sarcoma — long lead-time (i.e. 40 years)
- Lymphoma/Leukaemia
- Carcinoma (incl cutaneous SCC)
- (Thyroid PTC)
Congenital Ossicular Anomalies (Cremer)
- Isolated congenital stapes fixation 30.6%
- Footplate fixation
- Superstructure fixation - Stapes ankylosis assoc with another congenital ossicular anomaly 38.2%
- Incus/malleus deformation or aplasia of LPI
- Bony fixations of malleus and/or incus - Congenital anomaly of OC but mobile stapes footplate
- OC Discontinuity 15.3%
- Epitympanic fixation 6.3%
- Tympanic fixation 6.9% - Congenital aplasia/severe dysplasia of OW/RW
- Aplasia 2.1%
- Dysplasia 0.7%
Stapes fixation and nothing else
Stapes fixation and something else
Something else but not stapes fixation
Aplasia of OW/RW
Data from Cremer’s series of 144 operated ears (modified by Tos)
PERINEURAL INVASION in SCC Panizza review article. Williams et al - role in prognosis - signs/sx - investigation
Important in prognosis:
- Incidental perineural invasion has a 80-90% LRC 5yr
- Clinical perineural spread has 25-40% LRC 5yr
Signs / Sx
- Often cutaneous / high sun exposure areas
- V & VII MC affected
- Formication an early sign of PNS in V
- Slowly progressive facial palsy
Investigation
MRI - MR neurography detects extent of disease in 83%
- Inc intensity or enlargement of nerves
- Obliteration of fat in foramina
- Denervation change in supplied muscle (V3 / VII) - muscle hyper enhancement with gad
CT will detect late - bone erosion, foraminal widening, sclerosis
AUSTIN CLASSIFICATION FOR OCR
malleus more important than stapes superstructure ? malleus absent represents worse disease
75-50-25
Only A is a PORP, everyone else is a TORP
NASAL FRACTURE CLASSIFICATION Ondik et al, Arch Facial Plastic Surgery 2009 Read scott brown
(Nasal bones, septum dev?, septum fractured?, septum dislocated?)
- Simple straight
- Without midline deviation - Simple deviated
- With midline deviation - Comminuted nasal bones
- B/L nasal bone comminution + deviated septum
- Septum does not interfere with bony reduction - Severely deviated nasal and septal fractures
- Severe deviation of nasal midline
- Severe septal fracture or septal dislocation
- Comminution of nasal bones/septum may interfere with # reduction - Complex nasal and septal fractures
- Severe injuries, including
- Soft tissue trauma
- Acute saddling
- Open compound injury
- Avulsion of tissue
Management of nasal bone #:
- some controversy regarding closed vs open reduction (CR vs OR)
- Up to 50% will have residual deformity after CR
Management based on stage: see 4th facet
1, 2: closed reduction
3: not impacted — CR, impacted — modified OR with osteotomies
4: less severe — modified OR with osteotomies, more severe — acute open nasal/septal repair
5: acute open nasal/septal repair
Persistent deformity — septorhinoplasty (delayed)
LARYNGEAL WEB Cohen Airway occlusion, web character, function Severity classification
thickness, SG extension, cord visibility
1 and 2: endoscopic laser, CO2, cold steel, serial balloon dilatation, keel
3 and 4: LTR : anterior cricoid split and cartilage graft + stent
Supraglottis SCC T Staging
T1: Tumor limited to one subsite* of supraglottis with normal vocal cord mobility
T2: Tumor invades mucosa of more than one adjacent subsite* of supraglottis or glottis or region outside the supraglottis (e.g., mucosa of base of tongue, vallecula, medial wall of pyriform sinus) without fixation of the larynx
T3: Tumor limited to larynx with vocal cord fixation and/or invades any of the following: postcricoid area, pre-epiglottic tissues or paraglottic space
T4a: Tumor invades through the thyroid cartilage, and/or extends into soft tissues of the neck, thyroid, and/or esophagus
T4b: invades prevertebral fascia, encases carotid artery, superior mediastinum
*Subsites include the following:
- ventricular bands (false cords)
- arytenoids
- suprahyoid epiglottis
- infrahyoid epiglottis
- aryepiglottic folds (laryngeal aspect)
HODGKIN’S DISEASE Ann Arbor Staging
Staging guides treatment and prognosis
I: single LN region OR single extra-lymphatic site
II: 2 or more LN sites, same side of diaphragm OR Localised extra-lymphatic involvement + 1 nodal group
III: LNs on both sides of diaphragm may be accompanied by involvement of extralymphatic site or spleen or both
IV: disseminated involvement of one or more extra lymphatic organs with/without LN enlargement
Each Stage subdivided to A and B based on constitutional symptoms:
- Fever, drenching night sweats, unintentional weight loss of 10% within last 6 months
NEW TNM STAGE P16+ OROPHARYNX SCC NEW N STAGE P16- OROPHARYNX SCC
cT and pT (same)
T1 < 2cm
T2 2-4cm
T3 > 4cm or goes to lingual surface of epiglottis
T4 medial pterygoid, hard palate, mandible, larynx, extrinsic tongue muscles and beyond
(difference is no Tis and no T4b)
Clinical N
N0 no nodes
N1 iplateral 1 or more nodes < 6cm
N2 contralateral or bilateral nodes all < 6
N3 any node > 6cm
Pathological N (difference in outcome)
N0 no nodes
N1 < = 4 + nodes
N2 > 4 + nodes
so 2 stages cTNM and pTNM for p16 + only p16 - New N staging (for all new N staging of all subsites)
same as previous but include ENE (extranodal extension)
Clinical stage
N1 and N2 no ENE
N3a > 6 no ENE
N3b any node size and number but clinically overt + ENE
Essentially: P16 +
Stage I: T0-T2, N1
Stage II: T0-T3 N2
Stage III: T0-T4 N3
Stage IV: M1
CURACAO CRITERIA FOR HHT
Designated in 2000
FIVE criteria:
- Spontaneous, recurrent epistaxis
- Mucocutaneous telangiectasia
- Visceral involvement
- Family history
- Genetic testing. Confirms diagnosis
3 = Definite
2 = Suspected
1 = Unlikely
Congenital Cholesteatoma Diagnostic Criteria (Levenson)
- White mass medial to normal TM
- Normal pars flaccida and tensa
- No history of otorrhea or perforations
- No prior otologic procedures
- Prior bouts of otitis media not grounds for exclusion
CHARGE DIAGNOSTIC CRITERIA (Pagon)
Need 4 out of 6 criteria (at least 1 major):
MAJOR:
- Choanal atresia
- Coloboma (ocular- chorioretinal)
MINOR:
- Heart defects
- Genital anomalies
- Retardation of growth
- Ear abnormalities
1st BRANCHIAL ARCH CYSTS Work
Work Type 1: PPP: parallel to EAC, preauricular, purely ectodermal
- Ectodermal
- Doesn’t communicate with EAC
- Runs parallel to EAC. Involves parotid tissue, superior to VII
- cyst lined with squamous epithelium, no adnexa
- usually present as preauricular swellings
Work Type 2:
- Ectoderm and mesoderm
- Cartilage + skin adnexa (hair follicles, sebaceous glands, sweat glands)
- Duplication anomaly of membranous and cartilagenous EAC (EAC and Pinna)
- More intimately assoc with VII (may run through main trunk)
- Myringeal band
- floor of hypotympanum to umbo
- Pathognomonic
- MRI will demonstrate its tract
- pre, infra, post auricular, parotid swelling, posterior or inferior to angle of mandible, ant to SCM but always above hyoid bone
- cyst sinus or fistula anywhere from EAC to submandibular region
8-10% of all branchial arch anomalies Cysts twice as common as sinuses/fistulae
Esthesioneuroblastoma (Dulguerov)
T1 = Involving nasal cavity and/or sinuses (= Kadish 1 and 2)
- Excludes sphenoid and most superior ethmoid cells
T2 = Involving nasal cavity and/or sinuses with extension to or erosion of cribriform plate
- Includes sphenoid
T3 = Extending into orbit or protruding into ACF without dural invasion (K3ish)
T4 = Involving brain (K3ish)
N0 = No cervical LN metastases
N1 = Any form of cervical lymph node metastasis
M0 = No metastasis
M1 = Distant metastases
NEUROFIBROMATOSIS TYPE 1 CRITERIA
Any 2 of the following: 7
>6 cafe au lait spots (>5mm in prepubertal, >15mm in post-pubertal)
> 2 neurofibromas of any type or one plexiform neurofibroma
Axillary or inguinal freckling
Optic glioma
>2 Lisch nodules of the iris
Distinctive osseous lesion- sphenoid wing dysplasia or pseudoarthrosis
1st degree relative diagnosed with NF1
ENDOLYMPHATIC SAC TUMOURS Bambadikis
pre-op embolisation
post-op radio surgery
PARS TENSA RETRACTION (Sade)
- Minimally retracted TM
- Retracted onto incus
- Retracted onto promontory (atelectatic, not adhesive)
- Adhesive otitis
ENDOSCOPIC CORDECTOMY
- Subepithelial cordectomy- epithelium and SLP
- Subligamental cordectomy- epithelium, SLP and vocal ligament
- Transmuscular cordectomy- proceeds through vocalis muscle
- Total cordectomy- vocal process to anterior commissure
- Extended cordectomy
a. Encompasses contralateral VC and anterior commissure
b. Includes the arytenoids
c. Includes the subglottis
d. Includes the ventricle - Encompasses anterior commissure and anterior part of both vocal folds
MCADAM CRITERIA RELAPSING POLYCHONDRITIS EEENT and a Leg
- Recurrent chondritis of both auricles
- Non-erosive, seronegative polyarthritis
- Chondritis of nasal cartilages
- Ocular inflammation incl conjunctivitis, keratitis, scleritis/episcleritis and/or uveitis
- Chondritis of respiratory tract incl laryngeal/tracheal
- Cochlear and/or vestibular damage manifested by sensorineural HL, tinnitus +/- vertigo
(EE2NT and a Leg: eye, 2 ears, 1 nose, 1 throat)
Using the criteria: Damiani and Levine
- >3 criteria- histologic confirmation not necessary
- >1 criteria + histologic confirmation
- 2 or more separate anatomic locations with response to steroids
LATERAL CANAL FISTULA Dornhoffer
I: erosion of bony labyrinth, intact endosteum
II: open perilymphatic space:
IIa: undisturbed perlymph,
IIB: disturbed perilymph
III: open PL space, distubance of membranous labyrinth
RECURRENT RESPIRATORY PAPILLOMATOSIS (Derkay)
Anatomic Score:
Each site scored with
- Surface lesion
- Raised lesion
- Bulky lesion
Anatomic sites:
Epiglottis (lingual/laryngeal), AE fold (L/R), False cords (L/R), True VCs (L/R), Arytenoids (L/R), Ant comm, post comm, subglottis (8)
Trachea- upper 1/3, middle 1/3, lower 1/3, bronchi (L/R), tracheostomy stoma (5)
Other- nose,palate, pharynx, oesophagus, lungs, other (6)
Functional score: 2 2 3 4
- Voice - 0=normal, 1=abnormal, 2=aphonic
- Stridor - 0=absent, 1=present with activity, 2=present at rest
- Urgency of surgery - 0=scheduled, 1=elective, 2=urgent, 3=emergent
- Level of respiratory distress - 0=none, 1=mild, 2=moderate, 3=severe, 4=extreme
Factors significantly associated with repeated interventions:
- Anatomic score > 20
- Stridor with activity vs no stridor
- Urgency of surgery
THYROPLASTY (Isshiki)
Type 1: Medialisation
Type 2: Lateralisation
- Vertical midline cartilage incision through thyroid lamina
- Anterior lateralization of both vocal folds
- Used for Adductor spasmodic dysphonia (alternative to Botox- permanent, doesn’t require repeat injection, voice is breathy)
Type 3: Shortening cord (relax — lower pitch)
- Limited indications. Female-to-male transexuals
- Vocal fold atrophy/scarring/stiffening
Type 4: Elongating cord (tension — increase pitch)
- Cricothyroid approximation (mimics action of cricothyroid muscle). 2 sutures on each side
- Primarily in SLN palsy or male-to-female transexual patients
NEW TNM STAGE P16+ OROPHARYNX SCC
cT and pT (same)
T1 < 2cm
T2 2-4cm
T3 > 4cm or goes to lingual surface of epiglottis
T4 medial pterygoid, hard palate, mandible, larynx, extrinsic tongue muscles and beyond
(difference is no Tis and no T4b)
Clinical N
N0 no nodes
N1 iplateral 1 or more nodes < 6cm
N2 contralateral or bilateral nodes all < 6
N3 any node > 6cm
Pathological N (difference in outcome)
N0 no nodes
N1 < = 4 + nodes
N2 > 4 + nodes
so 2 stages cTNM and pTNM for p16 + only
REINKE’S OEDEMA (Savic)
- Marginal edge oedema
- Obvious sessile swelling, thrown over vocalis muscle during speech
- Large bag-like swelling, filled with fluid
- Partially obstructing lesion, medial borders in contact along most of length
NECK STAGING IN CUTANEOUS SCC - TNM - O’Brien P/N - N1S3 - ITEM
TNM N staging
N1- single node <3cm
N2a- single ipsilateral node 3-6cm
N2b- multiple ipsilateral nodes <6cm
N2c- bilateral or contralateral nodes <6cm
N3- any node >6cm
O’Brien P/N staging
P0- no parotid disease
P1- node <3cm
P2- node 3-6cm or mutliple nodes
P3- node>6cm or involvement- VII/skull base
N0- no nodes
N1- single ipsilateral node <3cm
N2- ipsilateral node >3cm, multiple nodes, contralateral nodes
Demonstrates independent effect of parotid nodes
P3 or N1/2 disease assoc with reduced survival N1S3
- Single node <3cm
- Multiple nodes <3cm or single node >3cm
- Multiple nodes >3cm
NOTE: incorporates parotid as one of the neck sub sites
Demonstrates predictive value for LR control, disease-specific survival, overall survival
ITEM
I= Immunosuppression
T= Treatment
E= Extranodal spread
M= Margins
Only allows post-operative staging
MICROTIA (Marx)
- Slightly smaller ear with majority of structures still present
- Greater deficiency of ear structures (absent helix/lobule)
- Very small auricular tag present (‘peanut’ deformity)
- Anotia
Note Weerda classification:
3 stages, similar to Marx I-III but includes anotia in III
COCHLEAR OTOSCLEROSIS (Shambaugh) S: Schwartz, stapedial reflex biphasic H: history (family) A: audiogram x 2, age B (C): CT scan
7 Diagnostic criteria for cochlear otosclerosis:
- Positive Schwartz sign in either ear
- Family history of otosclerosis
- Unilateral conductive HL typical of otosclerosis with bilateral SNHL
- Audiogram- flat or ‘cookie-bite’ pattern (of bone conduction)with excellent discrimination
- Progressive pure cochlear loss at usual age for otosclerosis onset
- CT scan showing typical demineralisation
- Stapedial reflex demonstrating biphasic “on-off effect” seen before stapedial fixation
Epidemiology:
- Pure cochlear otosclerosis thought to be very rare
Pathogenesis:
Involvement of the cochlea endosteum with an otosclerotic focus
- Progression — atrophy of spiral ligament and stria vascularis — inability to maintain the endolymphatic potential — hair cell loss
Imaging:
CT scan- distinctive pericochlear hypodense double ring appearance = demineralization of bone around the cochlea
Management:
Medical:
- Fluoride- start dose = 60mg, taper to maintenance of 20mg if show a clinical response
- Reduces bone resorption by inhibiting proteolytic enzymes, converts spongiotic lesions to sclerotic (inactive)
- Bisphosphonates- inihibit osteoclast activation
Hearing aid
Surgical
- CI if not aidable hearing (good candidates as preserved discrimination) — increased risk of cochlear ossification and VII stimulation
2ND BRANCHIAL ARCH CYSTS Bailey
Classification of 2nd arch cysts:
Type 1- superficial at border of SCM
Type 2- between SCM and IJV
Type 3- in the carotid bifurcation
Type 4- in the pharyngeal wall
Type II are most common
PHARYNGEAL POUCH
van Overbeek:
Small <1 vertebral body
- CP myotomy Moderate 1-3 vertebral bodies
- Endoscopic stapling
Large >3 vertebral bodies
- External approach CP myotomy + pouch excision/inversion/pexy
Morton and Bailey: (like a T stage)
Small <2cm
Medium 2-4cm
Large >4cm
Management as per van Overbeek
FESS SURGICAL FIELD GRADING (Boezaart)
0 - No bleeding (cadaveric conditions)
1- Slight bleeding — no suctioning required
2- Slight bleeding — occ frequent suctioning
3- Slight bleeding — frequent suctioning required. Bleeding threatens surgical field a few seconds after suction is removed
4- Moderate bleeding — frequent suctioning required, bleeding threatens surgical field directly after suction is removed
5- Severe bleeding — constant suctioning required. Blood appears faster than can be removed by suction - Surgical field severely threatened and surgery usually not possible
PITUITARY TUMOURS (Hardy and Somma)
Grade 1 and 2- confined to pituitary fossa
Grade 3- localised invasion and destruction of the sella
Grade 4- more extensive invasion and extension beyond the sella
Then use the Wilson grading system for extra-sellar extension
NECK DISSECTION
Neck Dissection Classification Update Arch OHNS 2002
Comprehensive ND: dissects Levels I-V
- Radical ND
- I-V + sacrificing XI, IJV, SCM - Modified Radical ND
- Type I- spares XI
- Type II- spares XI and IJV
- Type III- spares XI, IJV and SCM - Extended ND
- Skin, Carotid, XII, levator scapulae
- Additional LN groups- retropharyngeal, post-auricular, occipital
Selective ND: sparing LN groups
- Now classified based on levels dissected
- Previously- supraomohyoid, lateral, posterolateral, central
- Extended SND: take non-nodal structures in addition to limited nodal levels
TYMPANOSCLEROSIS
Stages:
- Confined to TM without hearing loss
- Confined to TM with hearing loss
- Involves middle ear without hearing loss
- Involves middle ear with hearing loss
- TM and middle ear without hearing loss
- TM and middle ear with hearing loss
MERKEL CELL CARCINOMA TNM STAGING
T stage: (unique)
T1- <2cm
T2- 2-5cm
T3- >5cm
T4- deep structures- bone, muscle, fascia, cartilage
N stage:
N1a- microscopic metastasis
N1b- macroscopic metastasis
N2- in transit metastasis (i.e. between primary and regional nodes or distal to primary site)
M stage: (same as melanoma)
M1a- skin, subcutaneous, non-regional lymph nodes
M1b- Lung
M1c- other sites
Staging:
I = Any T1, N0 (subdivide to A and B based on pathologic vs clinical nodal staging)
IIA = T2-T3, N0
IIB - T4 N0
IIIA = Any T N1a (A and B based on microscopic vs macroscopic nodes)
IIIB - Any T N1b N2
IV = Any M1 (no subgroups)
Simplified:
Local (N0)
I = T1
II = T2 3 4
Regional (N+) III = all N+
Mets IV = M
ORBITAL COMPLICATIONS OF SINUSITIS (Chandler/Wormald)
Chandler:
- Pre-septal cellulitis
- Orbital cellulitis
- Subperiosteal (extraconal) abscess
- Orbital (intraconal) abscess
- Cavernous sinus thrombosis
- Bilateral eye signs, obtundation
Wormald:
- Pre-septal- cellulitis vs abscess
- Subperiosteal- cellulitis vs abscess
- Orbital- cellulitis vs abscess
Advantages of Wormald classification:
- Indicates progression of disease
- Guides management- cellulitis vs abscess
- Recognises CST as an intracranial complication 24% with an orbital complication have a co-existent intracranial complication subperiosteal abscess: lenticular shaped
Ear Barotrauma (Teed)
- No visible damage, normal ear
- Congestion around the umbo
- Congestion of entire TM
- Hemorrhage into the middle ear
- Extensive middle ear hemorrhage with blood bubbles visible behind TM; TM may rupture
- Entire middle ear is filled with dark (deoxygenated) blood