ENT Conditions Flashcards

1
Q

Acute mastoiditis (key points, diagnosis, treatment)

A

Key point
● Rare but most common compilation of acute otitis media
Diagnosis:
● Post-auricular inflammatory signs (erythema, oedema, tenderness or fluctuance)
● Protruding auricle/external auditory canal oedema and signs of AOM
Treatment:
● IV Abx
● ENT involvement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Allergic rhinitis (classifications, clinical findings, management approach)

A

Classifications
- Intermittent: <4 days/week or <4 weeks
- Persistent: >4 days/week or >4 weeks
- Mild: sx present but not troublesome
- Mod to severe: present but causing disturbance
Clinical findings
● Nasal mucosa: oedematous and pale.
44
Management tips
● Combining oral and intranasal antihistamine has no added benefit
● Advised to trial new treatments for 4 week prior to re-assessing
● Oral and nasal decongestants have no role in allergic rhinitis
Management approach
● Mild - oral or intranasal antihistamin
● Mod to severe - oral or intranasal antihistamine PLUS intranasal corticosteroid
*Continue to with minimum effective treatment as required

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Allergic rhinitis (treatment options)

A

Oral
Less effective than intranasal sprays, consider alternatives in the group if not adequate response.
● Cetirizine 10mg PO daily
● Fexofenadine 120-180mg PO daily
● Loratadine 10mg PO daily
Intranasal
Antihistamine
More effective than the oral agents, have the fastest onset, can be used for intermittent symptoms.
● Azelastine 1mg/mL 1 spray each nostril BD
● Levocabastine 0.5mg/mL 2 sprays each nostril 2-4 times a day
Corticosteroid alone
● Useful in moderate to severe, more effective than oral antihistamines.
Combined
● Dymista (azelastine + fluticasone 125+50mcg, 1 spray each nostril BD)
Other
● Montelukast - used in combination with antihistamine and intranasal corticosteroid, may be first line in
children with asthma
○ ADRs: nightmares, sleep disturbance, agitation, depression and rarely suicidal thinking and
behaviour
● Ipratropium (intranasal) - use for marked rhinorrhoea, has rapid onset with prolonged effect (4-12 hours)
Eye drops
Use with oral antihistamines or intranasal corticosteroids and saline eye drops are effective.
● Antihistamine + mast cell stabiliser: Olopatadine 0.1% 1 drop both eyes BD
● Antihistamine: Levocabastine 0.05% 1 drop both eyes 2-4 times a day
● Mast cell stabiliser: Lodoxamide 0.1% 1 drop both eyes QID.
Allergen immunotherapy
Used in moderate to severe cases, more successful in patients with single sensitivity.
Course is is around 3 years with sublingual and subcutaneous options.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Audiology - patterns

A

** Refer to page 65-68 of GP Study Notes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Barotrauma (cause, symptoms, clinical findings, natural course)

A

Cause: Rapid changes in atmospheric pressure in the presence of occluded Eustachian tube
Symptoms: Temporary or persisting pain, deafness, vertigo, tinnitus +/- discharge
Clinical findings: Retraction, erythema, haemorrhage, fluid or blood in middle ear, perforation
Natural course: Spontaneous resolution.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Cholesteatoma (presentation, types)

A

Presentation: asymptomatic or hearing loss, dizziness and/or otorrhoea
● Hearing loss occurs late
Types:
● Primary - acquired, posterosuperior of TM
● Secondary - e.g. perforation, pearly mass behind ear behind TM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Thyroglossal duct cysts (location, epidemiology, clinical feature, presentation)

A

Location: Midline at level of thyrohyoid membrane
Epidemiology: Children or adolescents (most), >20 (⅓)
Clinical feature: Painless, moves with swallowing or protrusion of the tongue.
Presentation: Most present when there is a degree of infection or inflammation.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Brachial cleft cyst

A

Key points
● Present at birth
● Located anterior to sternocleidomastoid muscle
● Generally unrecognised until infected - late childhood or early adulthood

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Dacryocystitis (definition, history, type, management)

A
Definition: Infection of the lacrimal sac secondary to obstruction of the nasolacrimal duct.
History: Watery eyes (usually months).
Type:
● Acute
● Chronic
○ Recurring episodes which may be associated with nontender mass
○ Can be superimposed with acute infection
Management:
● Local hot moist compresses
● Analgesia
● Massage (mild cases)
● Systemic antibiotics (acute cases)
○ Cephalexin 500mg PO 6 hourly
● Surgery (if recurrent)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Dacryocystocele (findings, referral)

A

Key point
● Usually noted shortly after birth
Findings: Bluish swelling of the skin overlying the lacrimal sac and superior displacement of the medial canthal tendon.
Referral: Urgent review by ophthalmologist because of the risk of infection and/or nasal obstruction.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Dacryostenosis (prognosis, presentation, examination, diagnosis, management, referral indications)

A

AKA congenital nasolacrimal duct obstruction
Key point
● Most common cause of persistent tearing and ocular discharge in infants and young children.
Prognosis: Spontaneous resolution in more than 90% of cases.
Presentation: Intermittent tearing and debris on the eyelashes.
Examination: Increase in the size of the tear meniscus. Palpation of the lacrimal sac may cause reflux of tears and/or
mucoid discharge onto the eye.
Diagnosis: Clinical.
Management:
● First-line: Lacrimal sac massage and observation.
● If persisting 6-10 months: Lacrimal duct probing.
Indications to refer to ophthalmologist:
● Uncertain diagnosis (concern of infantile glaucoma)
● Symptoms persist past 6 months of age
● Signs and symptoms of acute dacryocystitis
● Clinical findings suggesting dacryocystocele

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Ear Images - exotosis, tympanosclerosis, myringosclerosis plaque

A

** Refer to page 134-135 of GP Study Notes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Otitis externa (cause, aetiology, organism, history, examination, investigations, management, prevention)

A

AKA Swimmer’s ear

Cause: Decreased cerumen → increased water and debris retention → increased bacterial growth

Aetiology: summer due to increased swimming, secondary to atopic dermatitis, irritation secondary to chemicals e.g.
hair dye, trauma due to cleaning

Organism: Staphylococcus aureus, Pseudomonas and fungi are common.

History:
● Ear pain
● Conductive hearing loss
● Blockage/pressure
● Itchiness
● +/- discharge

Examination:
● Pain when moving tragus + pinna
● Ear canal
○ Common - moist + oedematous, filled with serous or purulent debris
○ Possible - erythema, dry, grey/black fungal plaque

Investigations: Nil required

Management: Analgesia + topical treatment (ear drop)
Prevention: 2% acetic acid drops (Aqua ear)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Otitis externa - management

A

Short-term
○ Dry aural toileting 6 hourly until external canal is dry (during and at least 2 weeks after)
○ Followed by ear drops (apply gentle pressure on the tragus for 30 seconds). Can insert wick in ear
canal if it is too blocked to help with administration. Consider oral antibiotics if fever, spread to
pinna or folliculitis.
■ Dexamethasone/Framycetin/Gramicidin 0.05%/0.5%/0.005% ear drops, 3 drops in
affected ear TDS for 7 days ** avoid if perforated tympanic membrane → risk inner ear
damage**
○ Analgesia - paracetamol 15mg/kg (max 1g) PO QID PRN

Long-term
○ Keep ear dry - ear plugs or bathing cap during shower/swimming
○ Use acetic acid plus isopropyl alcohol ear drops following exposure to water to prevent recurrent
otitis externa

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Otitis media - shared decision making

A

● Reassure that it is a self-limiting condition and severe complications are rare
● Explain that initial antibiotic therapy is not essential but there can be two approaches:
○ Analgesia alone → follow up → consideration of ABx
○ Analgesia with initial ABx therapy
● Explain symptoms usually lasts for 2-3 days with or without antibiotics
● Discuss limited effect of antibiotic therapy (i.e. does not improve pain in first 24 hours, ABx only shortens by
12 hours)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Otitis media - treatment

A

Chronic otorrhoea → topical ciprofloxacin 0.3% ear drops, 5 drops in affected ear BD until free of discharge for 3 days.

Delayed non severe/immediate/delayed severe hypersensitivity → trimethoprim + sulfamethoxazole (>1 month old) 4+20mg/kg up to 160+800mg BD for 5 days.

17
Q

Otitis media - ATSI

A

Summary
- High risk = antibiotics

** Refer to page 225-226 of GP Study Notes

18
Q

Parotid gland sialothiasis (presentation, examination, management, when to refer)

A

Key point
● Diagnosis is clinical
Presentation: Pain and swelling with eating or anticipation of eating.
Examination: Small rock hard mass palpable in the salivary gland or duct or visible at the os.

Management: Conservative (hydration, heat, massa, milk duct, suck on tart/hard candies to promote salivary flow).
● Consider Abx if there is concern of secondary infection i.e. sialadenitis (dicloxacillin 500mg QID or
cephalexin 500mg QID for 7-10 days)

Refer: If pain is severe and symptoms are persistent.

19
Q

Sinusitis - Acute rhinosinusitis (ARS)

types, assessment, imaging, complications

A

Types: viral rhinosinusitis, post-viral (symptoms worsen after 5 days, persistent after 10 days), acute bacterial.

Clinical assessment
● Symptoms: nasal obstruction, discharge, changes in smell, facial pain/pressure (worse on bending forward
and can radiate to teeth), cough (children)
● Examination:
○ Nose - discharge, polyposis, swelling and erythema
○ Oral - post-nasal discharge, ? dental disease

Imaging: reserve CT for when complications are suspected. XR provides little information.

Acute bacterial rhinosinusitis (‘double-sickening’)
● Aetiology: Generally preceded by a viral or post-viral ARS.
● Symptoms: Discoloured discharge, severe localised pain, often unilateral predominance.
● Management:
○ Supportive therapies - analgesia, nasal saline irrigations, nasal decongestants, intranasal steroids
(if symptoms persist)
○ Antibiotics - used for bacterial infection (amoxicillin 500mg 8 hourly for 5 days), should not be used routinely for other causes.

Complications of ARS: orbital involvement (ophthalmoplegia, diplopia, proptosis, decreased visual acuity, loss of
green/red colour differentiation) → ENT referral

20
Q

35F, presents with 3/7 of coryza, loss of smell and facial pain. O/E: Temp 37, chest clear. No PHx and regular medications. Allergies: penicillin.

  1. What is the most likely diagnosis?
  2. What further features do you look for on examination?
  3. What are the features of your management of her current condition?
  4. After 8 days she comes back. She initially got better and then worsened. The pain is severe. Temp 38.5. What is your next management step?
A

What is the most likely diagnosis?
● Acute viral rhinosinusitis
What further features do you look for on examination?
● Unilateral sinus tenderness
● Inspection of nasal cavity for polyposis
● Painful ophthalmoplegia
● Dental caries
● Neck stiffness
What are the features of your management of her current condition?
● Nasal saline irrigation
● Decongestant nose spread for up to 3 days
● Paracetamol 1g PO QID
● Advised about red flags requiring early review (neck stiffness)
After 8 days she comes back. She initially got better and then worsened. The pain is severe. Temp 38.5. What is your
next management step?
● Cefuroxime 500mg BD for 5 days

21
Q

Sinusitis - Chronic rhinosinusitis (CRS)

assessment, management, triad

A

Clinical assessment: Anterior rhinoscopy to determine presence or absence of polyps to guide treatment.

Facial pain: *** Diagnosis of sinusitis requires the presence of either nasal congestion or discharge. Facial pain is rarely a significant feature of CRS

GP approach to CRS

  1. Topical steroids (if no polyps), oral steroids (if polyps)
  2. Nasal irrigation (BD for at least 8 weeks)

Surgical management: Functional endoscopic sinus surgery (FESS) → removal of polyposis and ventilation of sinus cells

Samter’s triad: NSAID drug sensitivity, asthma, CRSwNP