Endodontics Flashcards
dental pulp
low compliant CT lined by odontoblasts
causes of pulpal damage
caries
microleakage
trauma
traumatic occlusion
attrition, abrasion, erosion
ortho
radiotherapy
periodontal pathology
causes of pulpal damage in relation to cavity prep
heat generation
dehydration of dentine
cutting of odontoblast processes
direct injury to pulp
remaining dentine thickness
causes of pulpal damage in relation to restoration material
toxicity
pH of water
water absorption
heat of reaction
poor marginal adaptation
cementation
impression making
agents for treating dentine
dentine permeability
dentinal tubules increase in number & diameter as they get closer to the pulp
there are more tubules at the coronal aspect as opposed to roots which means deep occlusal cavities will have a lot of communication with the pulp
dental pain
in sub odontoblastic layer there is an extensive nerve plexus called rachow’s plexus - will transmit only pain from teeth
when there is inflammation these nerves will be stimulated & produce pain
nerves in this plexus inc
- a delta fibres (myelinated & stimulation causes sharp pain)
- c fibres (cause increased pulpal blood flow & pressure in pulp so dull aching pain)
EPT stimulates what fibres in pulp
a delta nerve fibres
healthy pulp
vital & free from inflammation
traumatic exposure treated within 30hrs will also be healthy
reversible pulpitis
a vital pulp that is inflamed
chance it can revert back to healthy pulp
diagnosed with pain to cold which lasts a short time, mediated by a delta fibres & no change in pulpal blood flow
irreversible pulpitis
pulp inflamed & vital but it cannot heal so only tx options are pulpectomy / RCT
diagnosed with spontaneous pain which is intermittent & may keep ptx awake at night, negative to cold but pain to hot with C fibre activation & there is increase in pulpal blood flow
necrotic pulp
pulp is non vital
RCT indicated
signs inc - discolouration, sinus present, gross caries, large restoration, radiographic evidence
infected pulp
radiographically diagnosed chronic radicular periodontitis
signs of inflammation will be absent due to lack of vitality
sometimes infection can be in only 1 root of multirooted tooth
when doing sensitivity tests
compare with adjacent & contralateral teeth
EPT (electric pulp test)
stimulates sensory nerves at pulp-dentine junction
a delta fibres tested
tooth should be dried, probe tip placed on incisal edge / cusp tip adjacent to pulp horn with conducting medium
circuit complete by holding handle
tingling or heat sensation should be felt
+ test = vital pulp tissue present but no indication of reversibility of inflammation
- test = reliable indicator for pulpectomy in 98% cases apart from immature pulps where it is unreliable
thermal tests
work by hydrodynamic forces where fluid movement in dentinal tubules will activate sensory nerve receptor units in pulp - either by hot or cold tests
cold = spraying ethyl chloride onto cotton pellet & placing on tooth; - response indicative of pulp necrosis
hot = vaseline on tooth then hot GP; - response indicates pulp necrosis but if too hot this can cause irreversible pulpitis
removal of necrotic pulp
only in incomplete root development should necrotic pulp be removed to a non-inflamed level
impact of age on dentine & pulp
dentine encroaches on pulp throughout life thus reducing size & volume of pulp
number of cellular components, blood vessels and nerves will also be decreased
increased calcification & fibrous components
all of this leads to pulp that is less likely to reverse in inflammatory response
cavity sealers
e.g. varnish, liner, base material
can protect cut dentine & underlying pulp from bacteria
may also protect from toxic effects of restorative material in setting phase of reaction
whole exposed pulpal dentine must be covered & it should adhere to dentine rather than restorative material but not reduce strength of restorative material or dissolve in biological liquids
varnish
material dissolved in organic solvent which will evaporate on application to dentine
must be applied twice
leaves a layer of synthetic resins that reduce permeability of dentine by 69% but is now rarely used
liners
varnish that contains fillers & additives & will be applied as thin layer
intended to have beneficial effect on pulp so will contain additives i.e. CaOH
cavity base
thicker sealer that will protect pulp from thermal effects from restorative material
usually CaOH or RMGI
CaOH
bacteriocidal & bacteriostatic
produces high pH that stimulates fibroblasts & reparative dentine formation; will also stimulate recalcification of any demineralised dentine & neutralises low pH from acidic restorative materials
*** weak cement & very soluble if not protected so if used close to pulp then it should be covered with RMGI then restorative material of choice
DBA
both dentine primers with or without adhesives are tolerated by pulp & there will be marked reduction in microleakage when DBA used; very technique sensitive though
periradicular disease
disease around root of tooth, not in the root
aetiology = gross caries // severe perio disease // trauma // cracks or fractures
this can cause:
- bacterial infiltration of root canal
- leading to pulpal necrosis
bacteria involved in periradicular disease
- necrotic untreated cases - gram neg anaerobic
- failing RCT & persistent infection - gram pos anaerobic
- non healing cases - enterococcus faecalis
3 processes to successful endo tx
- shaping
- irrigation
- 3D obturation
what information will a PA give
- extent of caries
- periapical pathology
- periodontal pathology
- no, morphology, length of roots & canals
- calcification of pulp chamber & root canals
- proximity of anatomical structures
common variations of root canal
upper molars - 90% have 4 canals
lower incisors - 40% have 2 canals
lower canines - 14% have 2, sometimes 3 canals
premolars - canal will frequently divide
radiographic clues - if canal very noticeable but suddenly disappears then it probably divides into 2 or 3