Emergency Medicine Flashcards

1
Q

Major risk factor for coronary artery disease or MI in teenage population.

A

Cocaine use

-leads to myocardial oxygen deprivation thru coronary vasoconstriction

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2
Q

When a patient presents to the ER with chest discomfort when should an EKG be performed?

A

Hopefully within ten minutes of arrival.

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3
Q

What type of test checks for a right ventricle MI?

A

Right sided EKG

-look specifically at lead rV4 for ST elevation

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4
Q

In the ER, which symptoms should NEVER exclude acute MI?

A

Reproducible chest pain

most of the time it indicates MSK etiology but in rare cases it can be indicative of MI

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5
Q

What is MONA therapy.

A

Therapy used in patients suspected of any acute coronary syndrome.
Morphine, Oxygen, Nitrates, ASA

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6
Q

What are contraindications for nitrate therapy?

A

Hypotension (<90 mmHg sbp)

-possible caused by ED meds, the nitrates dilate coronary vasculature and further drop bp

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7
Q

When is the ideal time for fibrinolytic therapy?

A

For acute STEMI within a 30 minute time frame of admission to the ER
(if a cath lab is available, that should be the first choice)

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8
Q

What is the ideal time frame for PCI (like balloon therapy) in patients presenting with ACS?

A

Within 90 minutes of admission

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9
Q

What is the use of cardiac markers in the ER?

A

Used to “rule in” MI, however, the absence of a positive marker test does NOT “rule out” MI.

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10
Q

What is the population distribution that most commonly develops abdominal aortic aneurysms (AAA)?

A

Elderly Males

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11
Q

Which layer of the blood vessel is weakened in AAA?

A

Media Layer

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12
Q

What diameter measurement of the aorta classifies it as an AAA?

A

> 3cm

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13
Q

What does Laplace’s Law state?

A

The size of the aneurysm directly correlates to the speed of its progression. Basically as they get larger, the rate of growth also increases.

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14
Q

What is the classic presentation of AAA?

A

Syncope without warning followed by abdominal and lower back pain. PE will show hypotension and pulsatile abdominal mass.

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15
Q

What are Cullen and Grey Turner signs?

A

Cullen: periumbilical ecchymoses
GT: flank ecchymoses
-both indicate retroperitoneal hematoma

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16
Q

What is a GI complication of AAA?

A

Aortoenteric fistula causing GI bleeding.

17
Q

What diameter measurement puts an AAA at highest risk of rupture?

18
Q

Test to confirm diagnosis of AAA in stable patients vs. unstable.

A

Stable: CT
Unstable: US
(imaging is not required to begin treatment for suspected AAA)

19
Q

What is treatment for AAA in the ER.

A

Early blood transfusion but care not to overtransfuse (can lead to excessive bleeding).
-patient should then be taken to surgery for endovascular repair

20
Q

Population distribution for aortic dissection (AD).

A

Bimodal

  • young usually with congenital vascular pathology
  • elderly with lifestyle risk factors
21
Q

Other than congenital conditions like Marfanism, what can illicit AD in a younger population?

A

Cocaine or Amphetamine use

22
Q

Which layer of the blood vessel is torn in AD?

A

Intima layer

23
Q

What is a Type A vs. Type B AD?

A

Type A: involves ascending aorta
Type B: does not

Other classification:
Type I: involves ascending and descending
Type II: just ascending
Type III: just descending

24
Q

What is the presentation of AD?

A

Sudden sharp chest pain that may radiate between the shoulder blades.

25
What type of medications are absolutely contraindicated to treat AD?
Thrombolytics
26
What can be seen on CXR that might indicate AD?
Mediastinal widening, aortic knob abnormalities (calcification), trachea deviation, left pleural effusion
27
What is the gold standard for AD diagnosis?
Dx: angiography -imaging is necessary to diagnose and treat AD
28
What is the first line ER treatment for confirmed AD?
Beta Blockers to lower HR (CCB can be used if allergy or COPD) Nitrates can also be used to lower BP.
29
How is Type A treated vs. Type B?
Type A: surgically | Type B: with meds