Emergency Medicine Flashcards

1
Q

Major risk factor for coronary artery disease or MI in teenage population.

A

Cocaine use

-leads to myocardial oxygen deprivation thru coronary vasoconstriction

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2
Q

When a patient presents to the ER with chest discomfort when should an EKG be performed?

A

Hopefully within ten minutes of arrival.

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3
Q

What type of test checks for a right ventricle MI?

A

Right sided EKG

-look specifically at lead rV4 for ST elevation

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4
Q

In the ER, which symptoms should NEVER exclude acute MI?

A

Reproducible chest pain

most of the time it indicates MSK etiology but in rare cases it can be indicative of MI

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5
Q

What is MONA therapy.

A

Therapy used in patients suspected of any acute coronary syndrome.
Morphine, Oxygen, Nitrates, ASA

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6
Q

What are contraindications for nitrate therapy?

A

Hypotension (<90 mmHg sbp)

-possible caused by ED meds, the nitrates dilate coronary vasculature and further drop bp

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7
Q

When is the ideal time for fibrinolytic therapy?

A

For acute STEMI within a 30 minute time frame of admission to the ER
(if a cath lab is available, that should be the first choice)

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8
Q

What is the ideal time frame for PCI (like balloon therapy) in patients presenting with ACS?

A

Within 90 minutes of admission

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9
Q

What is the use of cardiac markers in the ER?

A

Used to “rule in” MI, however, the absence of a positive marker test does NOT “rule out” MI.

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10
Q

What is the population distribution that most commonly develops abdominal aortic aneurysms (AAA)?

A

Elderly Males

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11
Q

Which layer of the blood vessel is weakened in AAA?

A

Media Layer

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12
Q

What diameter measurement of the aorta classifies it as an AAA?

A

> 3cm

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13
Q

What does Laplace’s Law state?

A

The size of the aneurysm directly correlates to the speed of its progression. Basically as they get larger, the rate of growth also increases.

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14
Q

What is the classic presentation of AAA?

A

Syncope without warning followed by abdominal and lower back pain. PE will show hypotension and pulsatile abdominal mass.

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15
Q

What are Cullen and Grey Turner signs?

A

Cullen: periumbilical ecchymoses
GT: flank ecchymoses
-both indicate retroperitoneal hematoma

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16
Q

What is a GI complication of AAA?

A

Aortoenteric fistula causing GI bleeding.

17
Q

What diameter measurement puts an AAA at highest risk of rupture?

A

> 5cm

18
Q

Test to confirm diagnosis of AAA in stable patients vs. unstable.

A

Stable: CT
Unstable: US
(imaging is not required to begin treatment for suspected AAA)

19
Q

What is treatment for AAA in the ER.

A

Early blood transfusion but care not to overtransfuse (can lead to excessive bleeding).
-patient should then be taken to surgery for endovascular repair

20
Q

Population distribution for aortic dissection (AD).

A

Bimodal

  • young usually with congenital vascular pathology
  • elderly with lifestyle risk factors
21
Q

Other than congenital conditions like Marfanism, what can illicit AD in a younger population?

A

Cocaine or Amphetamine use

22
Q

Which layer of the blood vessel is torn in AD?

A

Intima layer

23
Q

What is a Type A vs. Type B AD?

A

Type A: involves ascending aorta
Type B: does not

Other classification:
Type I: involves ascending and descending
Type II: just ascending
Type III: just descending

24
Q

What is the presentation of AD?

A

Sudden sharp chest pain that may radiate between the shoulder blades.

25
Q

What type of medications are absolutely contraindicated to treat AD?

A

Thrombolytics

26
Q

What can be seen on CXR that might indicate AD?

A

Mediastinal widening, aortic knob abnormalities (calcification), trachea deviation, left pleural effusion

27
Q

What is the gold standard for AD diagnosis?

A

Dx: angiography

-imaging is necessary to diagnose and treat AD

28
Q

What is the first line ER treatment for confirmed AD?

A

Beta Blockers to lower HR (CCB can be used if allergy or COPD)

Nitrates can also be used to lower BP.

29
Q

How is Type A treated vs. Type B?

A

Type A: surgically

Type B: with meds