EM Trauma 4 (head trauma) Flashcards

1
Q

remarks on head trauma

A

young adults and chidren - mvc
elderly - fall

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2
Q

lower limit of autoregulation of CPP in humans

A

<60 mm Hg
below this, local control of cerebral blood flow cnnot be adjusted to maintain flow adequate for function

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3
Q

In the absence of an ICP monitor, it is important to maintain

A

MAP ≥80 mm Hg
because low blood pressure in the setting of elevated ICP will result in a low CPP and brain injury

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4
Q

intracranial pressure by age group

A

adults: <10-15 mmHg
young children: 3-7 mm Hg
infants: 1.5-6 mmHg

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5
Q

types of brain herniation

A

uncal herniation
central transtentorial herniation
cerebellotonsillar herniation
upward transtetorial herniation

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6
Q

uncal herniation

A

most common
uncus of temporal lobe is displaced inferiorly through the medial edge of the tentorium
usually caused by an expanding lesion in the temporal lobe or lateral middle fossa

ipsilateral fixed and dilated pupil
contralateral motor paralysis

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7
Q

central transtentorial herniation

A

occurs with midline lesions, such as lesions of the frontal or occipital lobes, or vertex

bilateral pinpoint pupils
bilateral Babiinski’s signs
increased muscle tkne
decorticate posturing

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8
Q

cerebellotonsillar herniation

A

occurs when the cerebellar tonsils herniate through the forament magnum

pinpoint pupils
flaccid paralysis
sudden death

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9
Q

upward transtentorial hearniation

A

Posterior fossa lesion

conjugate downward gaze with absence of vertical eye movements
pinpoint pupils

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10
Q

regards on GCS

A

get the patient’s best score

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11
Q

decorticate

A

upper extremity flexion
lower extremity extension

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12
Q

decerebrate posturing

A

“we don’t celebrate”

arm extension and internal rotation
wrist and finger flexion
lower extremity extension and internal rotation

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13
Q

means to control agitated patients with TBI

A

Midazolam 1-2 mg IV
Propofol 20 mg every 10 seconds

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14
Q

New Orleans criteria

A

for GCS 15
Age >60y

Seizure
Headache
Intoxication
Vomiting
Anterograde Amnesia, persistent

evidence of trauma above the clavicles

100% sensitive, but 5% specific in identifying patients who have an intracranial lesion on CT

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15
Q

Canadian CT head rule

A

gcs 13-15
GCS <15 at 2h
Age ≥65y

> /=2 eps of vomiting
retrograde amnesia >30 m

suspected open or depressed skull fracture
any sign of basal skull fracture

dangerous
mechanism
-fall >3ft
-struck as pedestrian
-fall >5 stairs

83% snesitive, but 38% specific in identification of patients who have an intracranial lesion on CT

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16
Q

primary goals in head trauma

A
  1. maintain cerebral perfusion and oxygenation by optimizing intravascular volume and ventilation
  2. prevent secondary injury by correcting
    -hypoxia
    -hypercapnia
    -hypoperfusion
    -anemia
    -hyperglycemia
    -hyperthermia
  3. recognize and treat elevated ICP
  4. arrage for neurosurgical intervation to evacuate intracranial mass lesions
  5. treat other life-threatening injuries
17
Q

remarks on hypotension and hypoxemia in head trauma

A

SBP <90 mmHg and hypoxemia PaO2 <60 mmHg are associated with a 150% increased risk in mortality

goals:
50-69y: SBP ≥100 mmHg
15-49, >70y: SBP ≥110 mmHg
CPP 60-70 mmHg

SpO2 >90%
PaO2>60 mmHg
PCO2 35-45 mmHg
Hgb ≥8 g/dL

normal saline for volume resuscitation

18
Q

remarks on sedatation in head trauma

A

sedation and analgesia may decrease baseline ICP and prevent transient rises in ICP attributed to agitation, coughing, or gagging from the ET tube

ketamine is not recommended as preintubation agent bec it can cause agitation in patients after trauma

19
Q

remarks on seizures in head trauma

A

administer prophylactic antiseizure for GCS ≤10, if patient has abn head CT, or if the patient had an acute seizure after the injury

phenytoin 18mg/kg IV at 25mg/min
levetiracetam

20
Q

remarks on mannitol in head trauma

A

1 - Mannitol by repetitive bolus
2 - (0.25-1 g/kg)
3 - no dose-dependent effect, so it’s okay to begin at lower range of the suggested dose

4 - in the setting of acute herniation, mannitol has been demonstrated to effectively reduce life-threatening elevations of ICP

5 - alt:
hypertonic saline
3% sodium chloride, 250 mL over 30 mins
23.4% sodium chloride, 30 mL over 30 mins

21
Q

remarks on skull fractures

A

skull fractures that are open or depressed,
involve a sinus, or are
associated with pneumocephalus

should be given antibiotics:
vancomycin 1g IV, ceftriaxone 2g IV

22
Q

remarks on basilar skull fracture

A

most common:
PET-7 (pito)
PETrous portion of the temporal bone,
External auditory canal, and
Tympanic membrane

may involve 7th nerve palsy

antibiotic prophylaxis:
ceftriaxone 2g IV, vancom 1g iv

23
Q

contusion most commonly occur in the

A

subfrontal cortex
frontal and temporal lobes
occ in occipital oolobes

often associated with SAH

24
Q

Most common CT abnormality in patients with mod-to-severe TBI

A

SAH

CT scans performed 6-8 hours after injury are sensitive for detecting traumatic SAH

25
Q

remarks on subdural hematoma

A

1) hematoma formation bet dura mater and arachnoid

2) brains with extensive atrophy, such as in the elderly or in chronic alcoholics, are more susceptible to the development of acute subdural hematoma
children <2 y are also at increased risk of subdural hematoma

3) acute symptoms usually develop within 14 days of the injury
often the patient is unconsicous

4) crescent shape lesions that cross the suture line

26
Q

remarks on epidural hematoma

A

classic history:
significant blunt head trauma with LOC or altered sensorium, followed by a lucid period and subsequent rapid neurologic demise

27
Q

remarks on diffuse axonal injury

A

CT scan may appear normal, but classic CT findings include punctuate hemorrhagic injury along the gray-white junction of the cerebral cortex and within the deep structures of the brain

28
Q

remarks on penetrating head injury

A

GCS>8 with reactive pupils have a 25% mortality risk

gcs <5, mortality approaches 100%

29
Q

mild tbi

A

concussion
confusion
amnesia
vomiting, headache
loss of consciouness

primary intervention is rest
“splinting the brain”
physical and neurologic rest past the acute 48-hour period shortens the duration of symptoms

30
Q

second impact syndrome

A

mortality 60-80%
Rapid onset of cerebral edema due to
- Autoregulation, loss of
- Ion imbalance (ion channel UPregulation)