ELECTROLYTE EXCESS AND DEFICITS Flashcards
ELECTROLYTES
- SODIUM—– NA 135-145 mEq/L
- POTASSIUM — K 3.5-5.1 mEq/L
- CALCIUM —- CA2 8.5 - 10.mg/dl
- PHOSPHATE—- -PO4 3.5 -4.5 mg/dl
- MAGNESIUM— -MG 1.5-2.5 mg/dl
NA 135-145mEq/L
HYPONATREMIA
NA LOSS WITH H20 EXCESS
- LOW NA INTAKE
- EXCESS DIURETICS / HYPOTONICS
- GI SECRETION LOSS / DIARRHEA
- WOUND DRAINAGE
- ADERENAL INSUFFICIENCY
- DIAPHORESIS
HYPONATREMIA CLINICAL MANIFESTATIONS
- CONFUSION / MUSCLE CRAMPS
- TACHYCARDIA
- N/V, FATIGUE , APATHY
- DRY MUCUS MEMBRANES
HYPONATREMIA INTERVENTIONS
- ID UNDERLYING CAUSE AND TREAT
- DIETARY NA
- NA IV’S
- CLIENT AND LAB ASSESSMENT
- LIMIT H20 IF PRIMARY PROBLEM
NA –135-145
HYPERNATREMIA
*NA EXCESS / H2O LOSS
*HYPERTONIC TUBE FEEDINGS CAN
LEAD TO DEHYDRATION WITHOUT H2O
SUPPLEMENT
* EXCESS NA IV
CLINICAL MANIFESTATIONS OF HYPERNATREMIA
- THIRST / WT. LOSS
- DRY MUCUS MEMBRANES
- UA OUTPUT DECREASE
- DISORIENTATION / WEAKNESS
HYPERNATREMIA INTERVENTIONS
*ID UNDERLYING CAUSE AND TREAT
*HYPOTONIC IV SOLUTIONS (GRADUAL
INFUSION TO AVOID ICP)
*DIURETICS
*CLIENT AND LAB ASSESSMENT
K 3.5-5.0
HYPOKALEMIA
*K LOSS DUE TO A SHIFT OF K FROM ECF TO ICF *EXCESS LOSS VIA KIDNEYS,AND GI *EXCESS DIURESIS *INCREASE IN ALDOSTERONE *METABOLIC ALKALOSIS K SHIFT FROM ECF TO ICF
HYPOKALEMIA (DIABETIC KETOACIDOSIS)
SHIFT OF K INTO CELLS WITH
INSULIN ADMINISTRATION AND
CORRECTION OF ACIDOSIS.
EKG CHANGES WITH
HYPOKALEMIA
- FLATTENED T WAVE
- ST WAVE DEPRESSION
3 .U WAVE PRESENCE - VENTRICULAR ARRHYTHMIAS,PVC
5 BRADYCARDIA / FATIGUE
6 .ENHANCED DIGOXIN EFFECT - MUSCLE WEAKNESS
Clinical manifestations/HYPOKALEMIA
- MUSCLE WEAKNESS
- ILEUS
- LEG CRAMPS
- NAUSEA/VOMITING
- DECREASED BOWEL SOUNDS
- DECREASED REFLEXES
HYPOKALEMIA INTERVENTIONS
- ID UNDERLYING CAUSE AND TREAT
- DIETARY INTAKE
- K SUPPLEMENTS/ EKG MONITORING
- IV K
* RENAL FUNCTION PRIOR TO IV K
* CLIENT AND LAB ASSESSMENT
K PRECAUTIONS
- NEVER GIVEN IV PUSH
- MUST BE DILUTED
- IRRITATING TO VEINS
- EKG MONITOR PRN
- IV PUMP PRN
- TYPICAL DOSE 20mEq/L of KCL
- ASSESS LABORATORY DATA