Electrocardiogram Flashcards

1
Q

what results in positive deflection

A

the electrical impulse travelling towards the electrode

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2
Q

what results in negative deflection

A

the electrical impulse travelling away from the electrode

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3
Q

6 chest electrodes

A

v1-6 or c1-6

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4
Q

right arm electrode

A

red

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5
Q

left arm electrode

A

yellow

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6
Q

left leg electrode

A

green

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7
Q

right leg electrode

A

black - neutral/dummy

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8
Q

types of coronal leads (limb leads)

A

bipolar leads - 1, 2, 3
unipolar leads - aVL, aVR, aVF

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9
Q

the qrs axis

A

represents the net overall direction of the hearts electrical activity

defined as ranging from -30 to +90 degrees

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10
Q

what can abnormalities in the qrs axis hint at

A

ventricular/structural abnormality
conduction abnormality

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11
Q

left axis deviation

A

-30 to -90

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12
Q

right axis deviation

A

+90 to +180

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13
Q

3 waves produced during cardiac cycle

A

p wave
qrs complex
t wave

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14
Q

p wave

A

caused by atrial depolarisation

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15
Q

qrs complex

A

caused by ventricular depolarisation

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16
Q

t wave

A

from ventricular repolarisation

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17
Q

pr interval

A

time to conduct through av node/bundle of His

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18
Q

qrs duration

A

time for ventricular depolarisation

19
Q

st segment

A

start of ventricular repolarisation

20
Q

st elevation

A

acute infarction

21
Q

st depression

A

ischaemia
LV strain

22
Q

woldd-parkinson-white ecg

A

a to v conduction through accessory pathway on rest ecg
delta wave with short pr interval

23
Q

when is there a risk of ventricular fibrillation

A

if patient develops atrial fibrillation and if accessory pathway is capable of rapid conduction

24
Q

left ventricular hypertrophy

A

r wave in v5 or v6
plus
s wave in v1 > 35 mm

25
normal ventricular conduction
fibres of left bundle branch begin conduction impulse travels across interventricular septum from left to right - small r wave v1 - small q wave v6 travels across ventricles causing depolarisation of both RV and LV LV contributed most to complex - deep s wave v1 - tall r wave v6
26
right bundle branch block in v1
no change in initial impulse travel - small r wave impulse depolarises LV by itself sing RBBB (s wave) RV depolarises late by impulse through muscle (r' wave) hence rsr' pattern - M shape 'MaRRoW' pattern
27
left bundle branch block in v1
initial deflection altered since travels right to left - q wave/negative deflection RV depolarises unopposed - may produce small r wave travels across septum to depolarise LV - deep s wave w pattern in v1 WiLLiaM pattern **ST segment uninterpretable in LBBB
28
st segment
begins at end of qrs complex ends at beginning of t wave
29
what can st segment elevation or depression indicate
myocardial ischaemia or infarction
30
anterior st elevation
left anterior descending occlusion
31
inferior st elevation
right coronary artery 80% left circumflex 20%
32
widespread ischaemia
probably LMS
33
calculating HR - regular cardiac rhythm
count number of large squares between r waves rate = 300/number
34
calculating HR - irregular cardiac rhythm
use rhythm strip at bottom - 10 second recording rate = number of qrs complexes x6
35
bradycardia
any abnormality of cardiac rhythm resulting in a slow heart rate HR < 60 bpm
36
tachycardia
any abnormality of cardiac rhythm resulting in a fast HR HR > 100bpm
37
bradyarrhythmias
heart block - first degree - second degree - third degree/complete HB
38
1st degree av block
regular rhthm pr interval > .2 seconds and is constant causes - IHD, conduction system disease, seen in healthy children or athletes usually doesnt require treatment
39
2nd degree av block - mobitz 1/wenckebach
irregular rhythm pr interval continues to lengthen until a qrs is missing (non-conducted sinus beat) pr intervant not constant rhythm usually benign unless associated with underlying pathology
40
2nd degree av block - mobitz 2
irregular rhythm qrs complex may be wide non-conducted sinus impulses appear at irregular intervals rhythm is somewhat dangerous as the block is lower in the conduction system may cause syncope of may deteriorate into complete HB appearance in setting of an acute MI identifies high risk patients cause - IHD, fibrosis of conduction system treatment - pacemaker
41
complete heart block
atria and ventricles beat independent of one another - av dissociation QRS's and P waves each have own rhythm may be caused by inferior MI and presence worsens the prognosis may cause syncopal symptoms or angina, especially is ventricular rate is low treatment - usually pacemaker +/- temporary pacing/isoprenaline
42
narrow complex tachyarrhythmias
uncontrolled - ie fast - atrial fibrillation or flutter atrial tachycardia avnrt/avrt
43
broad complex tachycardia
ventricular tachycardia ventricular fibrillation