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Fitness management 386 > EKG > Flashcards

EKG Flashcards

1
Q

What is an EKG?

A

An electrocardiogram (ECG or EKG) is a recording of the electrical activity of the heart over time produced by an electrocardiograph, usually in a noninvasive recording via skin electrodes.

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2
Q

EKG word origin

A

electro, because it is related to electrical activity
cardio, Greek for heart, (German Kardio)
gram, a Greek root meaning “to write”.

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3
Q

5 components of basic EKG

A 
RATE
RHYTHM
HYPERTROPHY
INFARCTION
AXIS
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4
Q

Rate is it what or what?

A

bradycardia or tachycardia

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5
Q

What sets the rate at which the heart beats?

A

SA Node - Sinus Rhythm NORMAL SINUS Rhythm

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6
Q

Right Ventricular Hypertrophy -

A

large R wave in V1

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7
Q

Left Ventricular Hypertrophy -

A

S in V1 and R in V5 > 35 mm

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8
Q

ST segment elevation

A
  • means acute or recent
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9
Q

ST segment depression >

A

2mm older injury, ischemia

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10
Q

Axis – refers to

A

the diection of depolarization wave

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11
Q

What is an EKG Used for?

A

The display indicates the overall rhythm of the heart and weaknesses in different parts of the heart muscle.

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12
Q

What is the best way to measure and diagnose abnormal rhythms of the heart?

A

The best way to measure and diagnose abnormal rhythms of the heart through EKG

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13
Q

particularly abnormal rhythms caused by

A

damage to the conductive tissue that carries electrical signals

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14
Q

abnormal rhythms caused by levels of

A

dissolved salts (electrolytes), such as potassium, that are too high or low.

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15
Q

In myocardial infarction (MI), the EKG can identify

A

damaged heart muscle.

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16
Q

What can’t the EKG do?

A

It can only identify damage to muscle in certain areas, so it can’t rule out damage in other areas.

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17
Q

The ECG cannot reliably measure

A

the pumping ability of the heart.

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18
Q

Since an ECG cannot reliably measure pumping ability of the heart, what should be used?

A

ultrasound-based (echocardiography)

nuclear medicine tests

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19
Q

The electrical impulse starts in the _____ ______and moves through _____ to the ______ _______

It then moves through the left bundle branch (LBB) and right bundle branch (RBB) and finally to the purkinje fibers to contract the ventricles.

A

SA node

the atria to the AV node.

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20
Q

Bachman’s bundle electrically connects

A

the left and right atria.

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21
Q

There is a pause at the __ _____ when? the _____ the ____ __ ___

A

AV node before the signal hits the Bundle of His (pronounced hiss).

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22
Q

How many electrode placed on the body and where? What are those leads called?

A

10 electrodes placed on the body . . . Yet called a 12 lead?

3 limb leads (I, II, III)
3 augmented limb leads (aVR, aVL, aVF)
6 chest leads
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_
	12 leads
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23
Q

Why is it called 12 lead when their are only 10 leads?

A

*The EKG machine does the work to “create” the other leads, you just put the 10 stickers on.

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24
Q

What is a lead?

A

a combination of electrodes that form an imaginary line in the body along which the electrical signals are measured.

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25
Q

Frontal Plane View

A 
Leads I, II, III
\+
Augmented limb leads aVR, aVL, aVF
\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_\_
Frontal plane view of the heart
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26
Q

Sensing the heart’s electrical activity

A

via electrodes (contacts placed on the surface of the body )

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27
Q

For EKG, anatomical orientation is from the

A

subject’s perspective

right = left

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28
Q

The basic four limb electrodes:

A

electrical polarity:
neutral or ground
negative
positive

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29
Q

(manipulated by the EKG machine)

A

polarity

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30
Q

Lead I goes

A

toward left arm

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31
Q

Lead II goes

A

toward left foot

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32
Q

Lead III goes

A

(down & rightward)

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33
Q

Leads I, II, & III together create what

A

“Einthoven’s triangle”

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34
Q

“Einthoven’s triangle” is what kind of triangle whose vertices lie to

A

an equilateral triangle whose vertices lie at the left and right shoulders and the pubic region and whose center corresponds to the vector sum of all electric activity occurring in the heart at any given moment, allowing for the determination of the electrical axis.

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35
Q

Einthoven’s triangle is approximated by the

A

triangle formed by the axes of the bipolar electrocardiographic (ECG) limb leads I, II, and III.

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36
Q

The center of the Einthovens’s triangle offers

A

a reference point for the unipolar ECG leads.

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37
Q

Plus “augmented” leads, e.g.

A

aVR (augmented vector right)

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38
Q

Plus “augmented” leads, e.g.

A

aVL (augmented vector left

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39
Q

Plus “augmented” leads, e.g.

A

aVF (augmented vector foot)

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40
Q

Chest leads

A

V1 - V6
provides cross sectional view of the heart
horizontal plane

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41
Q

Limb leads

A

I, II, III

aVR, aVF, and aVL

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42
Q

Limb Lead I

A

I, from the right arm (-) toward the left arm (+)

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43
Q

II, from the right arm toward the left leg

A

Limb Lead II

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44
Q

LIMB LEAD III

A

III, from the left arm toward the left leg

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45
Q

aVR,

A

augmented lead toward the right (arm)

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46
Q

aVL,

A

augmented lead toward the left (arm)

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47
Q

aVF,

A

augmented lead toward the foot

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48
Q

aVR is approx opposite of

A

of I and should essentially mirror the shape of I vertically

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49
Q

Chest leads –

A

V1 through V6, starting over the right atrium with V1, and placed in a semi-circle of positions leftwards, to the left side of the left ventricle

50
Q

The normal progression of muscular contractions, hence, electrical activity, travels from

A

the upper right part of the atria downward and leftwards to the ventricles, with the left ventricle being the strongest.

51
Q

Various combinations of limb leads and chest leads taken together provide

A

a three-dimensional view into the electrical activity and workings of the heart for anyone who knows how to read an EKG.

52
Q

Interpreting the view from an electrode

A

An approaching train of muscle fiber depolarizations (or repolarizations moving away)is seen as an upward trace on the recording

(opposite movement = downward trace)

53
Q

the normal average direction for the heart’s electrical activity is from

A

the upper right, in the right atrium, to the lower left. (like cpr)

54
Q

Typical waves of an EKG.

A

PQRST

55
Q

P wave

A

ATRIA: depol-pause-repol

56
Q

atrial repolarization is obscured by

A

ventricular depolarization

57
Q

VENTRICLES: depol-pause-repolarize

A

QRS complex

58
Q

Pacemakers of the Heart

A

SA Node -

AV Node -

Ventricular cells -

59
Q

SA NODE

A

Dominant pacemaker with an intrinsic rate of 60 - 100 beats/minute.

60
Q

AV Node

A

Back-up pacemaker with an intrinsic rate of 40 - 60 beats/minute.

61
Q

Ventricular cells

A

Back-up pacemaker with an intrinsic rate of 20 - 45 bpm.

62
Q

Ectopic Focus (foci)

A

An excitable group of cells that causes a premature heart beat outside the normally functioning SA node.

63
Q

Acute occurrence of ectopic focus is usually

A

non-life threatening, but chronic occurrence can progress into arrhythmia.

64
Q

In a normal heart beat rhythm the SA node usually suppresses

A

the ectopic pacemaker activity due to the higher impulse rate of the SA node.

65
Q

However, if there is a malfunctioning SA node

A

it’s inactivity allows the ectopic pacemakers to generate their rhythm

66
Q

How to analyze EKG

A 
Step 1:	Calculate Heart rate.
Step 2:	Determine regularity.
Step 3:	Assess the P waves.
Step 4:	Determine PR interval.
Step 5:	Determine QRS duration.
67
Q

Step 1: Determine HR

A

Heart rate (bpm)
Find an R wave on a heavy black line
Count the number of 1mm lines between two R waves
Divide 1500 by the number of 1mm lines between 2 R waves
Need to determine rate quickly to assess presence of abnormalities
Tachycardia, bradycardia

68
Q

Step 2: Determine regularity

A

Look at the R-R distances (using a caliper or markings on a pen or paper).
Regular (are they equidistant apart)? Occasionally irregular? Regularly irregular? Irregularly irregular?

69
Q

A variety of factors affect heart rate including:

PR Interval – comprises the time period from the onset of atrial depolarization (beginning of P wave) until the onset of ventricular depolarization (beginning of QRS complex) – normal = 0.12 – 0.20 seconds or 3-5 little boxes. A substantial portion of this time period is taken up by delay in AV node; also includes the bundle and bundle branches
QRS complex normal duration = no more than 0.10 second or 2.5 little boxes
(if this takes longer usually means some sort of intraventricular conduction delay (bundle branch block)

A

age (declines with age)
gender (females generally have higher resting heart rates)
physical stature (small animals have higher heart rates)
emotion (stress can elevate HR)
Type of food consumed (caffeine increases HR)
Body temperature (rise in temp increases HR)
Environmental factors (smoking increases heart rate)
Highly trained endurance athletes (have low resting HRs)
Some abnormalities occur naturally, while others may be more serious, such as sick sinus syndrome, bundle branch block and may require medical therapy.

70
Q

We can see P waves for each Q wave, but some QRS complexes are closer together than others.
Using ECG calipers, you can measure the first QRS then spin the points. You’ll notice they gradually increase.
Although it is not shown, the rate will return to normal; this fluctuation usually occurs with breathing patterns.
The rate is 8*10 here so it is sinus and within normal range.
This is mostly due to vagus (CNX) nerve innervation, which controls the parasympathetic system.
Healthy and young individuals have this and it is considered normal.
Loss of sinus arrhythmia may signify the beginnings of heart failure.

A

s

71
Q

Step 3: Assess the P waves

A 
Normal P waves with 1 P wave for every QRS
 
Are there P waves?
Do the P waves all look alike?
Do the P waves occur at a regular rate?
Is there one P wave before each QRS?
Interpretation?
72
Q

Step 4: Determine PR interval

A

Normal: 0.12 - 0.20 seconds.

(3 - 5 boxes)

73
Q

Step 5: QRS duration

A

Normal: 0.04 - 0.12 seconds.

(1 - 3 boxes)

74
Q

Normal Sinus Rhythm

A 
Rate				90-95 bpm	
Regularity			regular
P waves				normal
PR interval			0.12 s
QRS duration			0.08 s
75
Q

Normal Sinus Rhythm (NSR) cause

A

Etiology: the electrical impulse is formed in the SA node and conducted normally.

76
Q

This is the normal rhythm of the heart; other rhythms that do not conduct via the typical pathway are called arrhythmias.

A

NSR

77
Q

arrhythmias

A

other rhythms that do not conduct via the typical pathway

78
Q

NSR Parameters

A 
Rate				60 - 100 bpm	
Regularity			regular
P waves			normal
PR interval			0.12 - 0.20 s
QRS duration		0.04 - 0.12 s

Any deviation from above is sinus tachycardia, sinus bradycardia or an arrhythmia

79
Q

Ways the ECG can change include: for MI

A

ST elevation & depression

80
Q

Significant Q wave = for MI

A

Necrosis

81
Q

FOR MI

ST elevation =

A

Injury

82
Q

FOR MI

T wave inversion =

A

Ischemia

83
Q 
Myocardial Infarction (MI)
An old MI
A

(“age-indeterminate”) will likely have significant Q waves as well as T wave inversion.

84
Q

Left Ventricular Hypertrophy

A

The QRS complexes are very tall (increased voltage)

85
Q

Why is left ventricular hypertrophy characterized by tall QRS complexes?

A

As the heart muscle wall thickens there is an increase in electrical forces moving through the myocardium resulting in increased QRS voltage.

86
Q

Left Ventricular hypertrophy

A

S wave in V1 (mm)
+ R wave in V5 (mm)
——————————
Sum is > 35mm = L.V.H.

87
Q

depolarization of the Bundle Branches and Purkinje fibers are seen as

A

the QRS complex on the ECG

88
Q

a conduction block of the Bundle Branches would be

A

reflected as a change in the QRS complex.

89
Q

With Bundle Branch Blocks you will see two changes on the ECG:

A

QRS complex widens (> 0.12 sec).

QRS morphology changes (varies

90
Q

Why does the QRS complex widen?

A

When the conduction pathway is blocked it will take longer for the electrical signal to pass throughout the ventricles.

91
Q

What QRS morphology is characteristic? bbb

A

For RBBB the wide QRS complex assumes a unique, virtually diagnostic shape in those leads overlying the right ventricle (V1 and V2).

92
Q

Graded Exercise Test

A

GXT

93
Q

GXT what is it is?

A

A maximal (or submaximal) exercise test with planned and controlled increases in intensity.

94
Q

What is the GXT used for?

A

Diagnose overt or latent heart disease
To evaluate cardiorespiratory functional capacity
To evaluate responses to conditioning or rehabilitative programs
To increase motivation for entering and adhering to exercise programs
Dt3

95
Q

When to use GXT?

A

Older men (45 years) and women (55 years)
Individuals of any age with moderate risk (two or more CHD risk factors)
High – risk individuals with one or more signs/symptoms of cardiovascular/pulmonary disease
High – risk individuals with known CV, pulmonary, metabolic disease

96
Q

Maximal test before (GXT)

A

starting a vigorous (>60%VO2max

97
Q

When not to use GXT?

A

When a person meets any absolute contraindications for the test
OR
Relative contraindications (Situation specific)

98
Q

Use Submaximal Exercise Test for: GXT

A

low – risk individuals
Moderate – risk individuals,
if starting a moderate exercise program
(40 – 60%VO2max)

99
Q

Contraindications to Clinical Exercise Testing abosulute

A 
A recent significant change in resting EKG
Unstable angina
Uncontrolled cardiac dysrhythmias
Symptomatic severe aortic stenosis
Uncontrolled symptomatic heart failure
Acute pulmonary embolus or pulmonary infarction
Acute myocarditis or pericarditis
Suspected or known dissecting aneurysm
Acute systemic infection
100
Q

Recent EKG suggesting significant ischemia, recent MI (within 2 days) or other acute cardiac event
Uncontrolled cardiac dysrhythmias causing symptoms or hemodynamic compromise
Heart failure - is a condition that can result from any structural or functional cardiac disorder that impairs the ability of the heart to fill with or pump a sufficient amount of blood through the body

A

s

101
Q

Common GXT Protocols Decision based on

A

subject/patient and purpose of the test

102
Q

Common GXT Protocols names

A 
Bruce – walk to run up a hill
Balke
Naughton
Ellestad
Modified Astrand
103
Q

Bruce Protocol

A
  • Each stage is 3 mins in duration
104
Q

Patient Monitoring

A

Electrocardiography (EKG)
Blood Pressure
Ratings of Perceived Exertion (RPE)

105
Q

Electrode Preparation Goal:

A

improve electrical conductivity; reduce electrical impedance

106
Q

Electrode Prep Goal

Steps to follow:

A

Shave hair, if necessary
Remove superficial layer of skin
Oil should be removed by a fat solvent (isopropyl alcohol)
Abrade with fine-grain emery paper, gauze, other appropriate material (I.e. scrubby pad)

107
Q

Electrode Preparation reduce

A

Reduce excessive motion artifacts that result from movement of chest electrode during exercise
reduce oily skin
tincture of bezoin (for excess sweat) increases the stickiness of electrodes

108
Q

Electrode Preparation

Obese individuals

A

may need to move V4 and V5 to sixth intercostal space

109
Q

Blood Pressure during GXT

A

You now know how to do this
Listen for phase IV
Diastolic may drop to 0 mmHg
Once the subject starts to run – remove BP cuff
Not safe to take while running in the lab

110
Q

Rating of Perceived Exertion

A

Borg, 1981

Measured near the end of each exercise stage

111
Q

Test Sequence & Measurements Before Exercise

A

EKG tracings taken w/ patient supine, standing and standing 15sec hyperventilation

112
Q

Test Sequence & Measurements During Exercise Test

A

HR & BP measured during each stage (typically every 2 or 3 minutes
HR & BP monitored every 1 or 2 minutes during recovery period (walking 2-3 mi/hr or 75-150 kgm/min) until HR & BP stabilize (3-5 minutes)

113
Q

Attainment of Maximal Capacity

A

Failure of heart rate to increase with increases in exercise intensity
Venous lactate concentration exceeding 8 mmol/L
Respiratory exchange ratio (RER) greater than 1.15
Rating of perceived exertion > 17 (using original Borg 6-20 scale)

114
Q

Active or passive recovery

Diagnostic purpose

A

– supine position immediately after exercise

ST Segment

115
Q

Supine position does what?

A

Increases venous return, myocardial O2 demand, ventricular wall stress

116
Q

Absolute Indications for Test Termination

A

Moderate-to-severe angina
Drop in SBP of 10 mm Hg from baseline BP despite an increase in workload, when accompanied by other evidence of ischemia
Increasing nervous system symptoms (ataxia, dizziness or near syncope)
Signs of poor perfusion
Technical difficulties monitoring EKG
Client’s desire to stop
Sustained ventricular tachycardia
ST elevation ( 1 mm) in leads without diagnostic Q waves

117
Q

Ataxia –

A

inability to perform coordinated muscular movements

118
Q

Signs of poor perfusion –

A

cyanosis, pallor

119
Q

Relative Indications for Termination of GXT (GETP8-Box 5-2)

A

Drop in SBP of  10 mm Hg from baseline BP despite an increase in workload, in absence of other evidence of ischemia

Increasing chest pain

Fatigue, shortness of breath, wheezing, leg cramps, or claudication

Hypertensive response (SBP > 250 mm Hg and/or DBP > 115 mm Hg
 Development of bundle-branch block or intraventricular conduction delay that cannot be distinguished from ventricular tachycardia

Arrhythmias including multifocal PVCs, triplets of PVCs, heart block, supraventricular tachycardia or bradyarrhythmias

ST or QRS changes – Excessive ST depression (2 mm horizontal or downsloping ST-segment depression)

120
Q

claudication

A

limping

121
Q

General Indications for Stopping an Exercise Test - Low Risk Adults

A

Onset of angina or angina-like symptoms

Significant drop (20 mm Hg) in SBP or failure of SBP to rise w/increase in exercise intensity

Excessive rise in BP: SBP > 260 mm Hg or DBP > 115 mm hg

Signs of poor perfusion: light-headedness, confusion, ataxia, pallor, cyanosis, nausea or cold and clammy skin

Failure of heart rate to  with increasing intensity

Noticeable change in heart rhythm

Subject requests to stop = volitional exhaustion

Physical or verbal manifestations of severe fatigue
Failure of testing equipment

Fitness management 386 (6 decks)

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