ECG interpretations Flashcards
Right bundle branch block - characteristic ECG findings
WIDE QRS COMPLEX (>120ms)
M wave in V1
W wave in V6
remember - William Marrow
Left bundle branch block - characteristic ECG findings
WIDE QRS COMPLEX (>120ms)
W wave in V1
M wave in V6
remember - William Marrow
True or false - a new left bundle branch block is always pathological
TRUE
What are the possible causes of left bundle branch block?
- aortic stenosis
- ischaemic heart disease
- hyperkalaemia
- digoxin toxicity
- myocardial infarction
- hypertension
- cardiomyopathy
What are the possible causes of right bundle branch block?
normal (especially with increasing age) right ventricular hypertrophy cor pulmonale pulmonary embolism ischaemic heart disease myocardial infarction atrial septal defect cardiomyopathy or myocarditis
Atrioventricular block - characteristic ECG findings in first degree AV block AND what needs to be done to manage it
Consistent prolonged PR interval (>200ms/5sqs)
No management
Atrioventricular block - characteristic clinical/ECG findings in second degree mobitz I AND what needs to be done to manage it
Progress prolonged PR interval, until QRS is missed (dropped beat) – PR interval longest before dropped beat
(Long, long, drop)
Regularly irregular rhythm
No management (unless bradycardic then atropine)
Atrioventricular block - characteristic clinical/ECG findings in second degree mobitz II AND what needs to be done to manage it
QRS regularly missed, consistent PR interval duration, regularly irregular rhythm
(Normal, normal, drop)
Regularly irregular rhythm
Consider interim atropine
Permanent pacemaker
Atrioventricular block - characteristic clinical/ECG findings in complete heart block
Severely bradycardic
May also have cannon A waves
Complete AV dissociation
(most commonly due to RCA occlusion)
Permanent pacemaker
Atrial fibrillation - characteristic ECG findings
Irregularly irregular pulse
Absent A wave in JVP
Absent P waves
Irregular QRS complex
Chaotic baseline
Paroxysmal SVT - characteristic ECG findings
HR 140-280bpm
P waves often unidentifiable/not visible
Regular narrow QRS complex (QRS<120ms)
UNLESS SVT w/BBB then wide QRS complex (QRS>120ms)
Ventricular tachycardia - characteristic ECG findings
wide complex tachycardia (>120ms) at a rate of ≥100bpm
monomorphic - commonly caused by MI
polymorphic + subtype torsades de pointes
cannon A waves in JVP
Ventricular fibrillation - characteristic ECG findings
Chaotic irregular deflections of varying amplitude – polymorphic and irregular
No identifiable/clear P waves, QRS complexes or T waves
Rate 150 – 500 bpm
Amplitude decreases with duration
Paroxysmal SVT - how do patients classically present
Palpitations is the MAIN symptom - usually they are otherwise well (haemodynamically stable)
Subsequently usually find a high HR (140-280bpm)
Hypokalaemia - causes
- Excess alcohol
- Excessive laxative use +/- Diarrhoea
- Diuretics use (especially potent diuretics like furosemide)