ECG Basics my Brain Rejects x Flashcards
What are the two things you need to be in sinus
Every p wave is followed by a QRS
The PR interval is less than 200ms
What is normal axis
Net + QRS in I and II/aVF
What is right axis deviation
Net - QRS in I and + QRS in II/aVF
What is left axis deviation
Net + QRS in I and - QRS in II/AVF
Normal p wave duration
0.12s/120ms
Normal p wave amplitude
<2.5mm (2 lil squares)
Normal PR interval
0.12-0.22s
Normal QRS duration
<0.12
How does the R wave progress
Should be small in V1 and get bigger through precordial leads
Normal QTc interval:
Men: <0.45
Women: <0.46
What is a shortened QTc interval
<0.32
Where can you most easily see a U wave
V3/4
Normal U wave characteristics
<1-2mm amplitude or <25% t wave’s amplitude
1st Degree Heart Block
PR interval >0.22
Causes:
- Degenerative fibrosis!
- Ischaemia
- Beta Blockers
Prognosis:
- Benign
2nd Degree Heart Block Mobitz Type 1 (Wenckebach)
Longer, longer, longer drop
Gradually increasing PR interval until the atrial impulse (p wave) is blocked at the AV node and no QRS happens
Prognosis:
- Usually benign and asymptomatic
- Pts respond well to atropine
- Don’t usually need pacing
2nd Degree Heart Block Mobitz Type 2
If some Ps don’t get through then you have mobitz 2
Intermittent blockage of the atrial impulse (no QRS) but with constant PR interval
Causes:
- Mostly due to structural damage to the conduction system (infarction, fibrosis, necrosis)
Prognosis:
- Likely symptomatic bradycardia
- Possible progression to CHB
3rd Degree Heart Block
If Ps and Qs don’t agree then you have a 3rd degree = Complete Heart Block
All atrial impulses (p waves) are blocked by the AV node until an escape rhythm happens which is either wide or narrow QRS –> NO relation between p waves and QRS complexes
Causes:
- Complete blockage at the AV node
- Inferior AMI
- AV nodal blocking drugs: Bblockers, digoxin
Prognosis:
- High risk of symptomatic bradycardias and death
- Requires a pacemaker
Inferior limb leads
III, aVF, II
Lateral limb leads
I, aVL, -aVR
Septal chest leads
V1 V2
Anterior chest leads
V3 V4
Anterolateral chest leads
V5 V6
RBBB
QRS >0.12
MaRRoW
M in V1
W in V6
Secondary ST changes: V1-2 have inverted t waves
LBBB
QRS >0.12
WiLLiaM
W in V1 because R wave small or missing so can have just QS
M in V6
Secondary St changes: left sided (V5,V6) t wave inversions and ST depressions