ECG Flashcards
(42 cards)
Types of cardiac cells
1) myocardial = working cells(contraction)
2) pacemaker cells = specialized for electrical conduction system
- spontaneously generate and conduct impulses
phases of cardiac action potential
Phase 0 = Na+ rushing into cell(depolarization)
- responsible for QRS complex
Phase 1 = K rushing out of cell(brief early repolarization)
Phase 2 = Ca moving into cell(plateau phase)
- ST segment
Phase 3 = repolarization(Na-K pump)(Final rapid repolarization)
- T wave
Phase 4 = returns to polarization(resting)
Hypercalemia
too much K+
- bradycardia
hypocalemia
low K+
- dysrythmia, changes in ST(depression)
- from diuretic medication
absolute refractory period
cells cannot be stimulated to conduct an electrical impulse
- period from onset of QRS complex to peak of T wave
relative refractory period
cardiac cells can be stimulated if stimulus is strong enough
- downslope of T wave
supernormal period
weaker than normal stimulus can cause cardiac cells to depolarize
- end of T wave
Pacemaker sites
SA node = 60-100 bpm(normal sinus rhythm)
AV(junctional ectopic focus = 40-60 bpm
Ventricular ectopic focus = 20-40 bpm(purkinje fibers and bundle branch)
P wave
atrial depolarization
QRS complex
ventricle depolarization and atrial repolarization
- less than 0.1 seconds
ST segment and T wave
repolarization
- ST elevation = MI or injury
- ST depression = ischemia
PR interval
less than 0.2 seconds
- time for impulse from SA node through AV node
- atrial and atrioventricular node depolarization
QT interval
systolic phase of heart
- electrical systole
7 steps for basic EKG interpretation
1) general rhythm = regular or irregular
2) rate = both general and specific
3) P wave = present or absent(are they identical)
4) Interval = PR interval(.12-.2 secs), QRS interval(.06-.1 secs), QT interval(<.4 secs)
5) origin = supraventricular(SA&AV nodes) or ventricular
6) rhythm interpretation = name of the rhythm and location of any abnormalities
7) hemodynamic compromising?
Sinus Tachycardia
rate >100bpm
- caused by anxiety, thyroid toxicosis, fever
sinus bradycardia
rate < 60 bpm
- athletes, B-blockers, vomiting, valsalva stimulation, hypotension
sinus arrhythmia
irregular rhythm with impulses originating in the SA node(supraventricular)
premature atrial contraction
PAC
- regular with premature beats(premature P waves)
- peaked P waves
paroxysmal atrial tachycardia
sudden onset tachycardia(150-250 bpm)
Wandering atrial pacemaker
“wandering atrial pacemaker”
- P wave changes in size, shape, and direction from beat to beat(at least three different P wave configurations)
- rhythm is irregular
atrial flutter
250-350 bpm(atrial rate)
- multiple activations of P wave, but does not cause QRS
- AV node prevents ventricle from contracting and reducing output
Atrial fibrillation
350-450 bpm(atrial rate)
- atrium quivers and doesn’t contract fully so all blood does not go into ventricle = decreased Q
- tend to develop clots so pt’s on blood thinner
Paroxysmal junctional tachycardia
- sudden onset tachycardia(150-250 bpm)
- P wave absent or inverted
- from emotional stress, hypoxia
premature ventricular contraction
PVC = originates suddenly in an ectopic focus in a ventricle producing a giant ventricle complex
- bigeminy = PVC every 2 beats
- trigeminy = PVC every 3 beats
- quadrigeminy = PVC every 4 beats
- couplet = 2 PVC’s in a row
- triplet = 3 PVC’s in a row