ECG Flashcards

(42 cards)

1
Q

Types of cardiac cells

A

1) myocardial = working cells(contraction)
2) pacemaker cells = specialized for electrical conduction system
- spontaneously generate and conduct impulses

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2
Q

phases of cardiac action potential

A

Phase 0 = Na+ rushing into cell(depolarization)
- responsible for QRS complex
Phase 1 = K rushing out of cell(brief early repolarization)
Phase 2 = Ca moving into cell(plateau phase)
- ST segment
Phase 3 = repolarization(Na-K pump)(Final rapid repolarization)
- T wave
Phase 4 = returns to polarization(resting)

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3
Q

Hypercalemia

A

too much K+

- bradycardia

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4
Q

hypocalemia

A

low K+

  • dysrythmia, changes in ST(depression)
  • from diuretic medication
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5
Q

absolute refractory period

A

cells cannot be stimulated to conduct an electrical impulse

- period from onset of QRS complex to peak of T wave

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6
Q

relative refractory period

A

cardiac cells can be stimulated if stimulus is strong enough

- downslope of T wave

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7
Q

supernormal period

A

weaker than normal stimulus can cause cardiac cells to depolarize
- end of T wave

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8
Q

Pacemaker sites

A

SA node = 60-100 bpm(normal sinus rhythm)
AV(junctional ectopic focus = 40-60 bpm
Ventricular ectopic focus = 20-40 bpm(purkinje fibers and bundle branch)

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9
Q

P wave

A

atrial depolarization

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10
Q

QRS complex

A

ventricle depolarization and atrial repolarization

- less than 0.1 seconds

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11
Q

ST segment and T wave

A

repolarization

  • ST elevation = MI or injury
  • ST depression = ischemia
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12
Q

PR interval

A

less than 0.2 seconds

  • time for impulse from SA node through AV node
  • atrial and atrioventricular node depolarization
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13
Q

QT interval

A

systolic phase of heart

- electrical systole

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14
Q

7 steps for basic EKG interpretation

A

1) general rhythm = regular or irregular
2) rate = both general and specific
3) P wave = present or absent(are they identical)
4) Interval = PR interval(.12-.2 secs), QRS interval(.06-.1 secs), QT interval(<.4 secs)
5) origin = supraventricular(SA&AV nodes) or ventricular
6) rhythm interpretation = name of the rhythm and location of any abnormalities
7) hemodynamic compromising?

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15
Q

Sinus Tachycardia

A

rate >100bpm

- caused by anxiety, thyroid toxicosis, fever

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16
Q

sinus bradycardia

A

rate < 60 bpm

- athletes, B-blockers, vomiting, valsalva stimulation, hypotension

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17
Q

sinus arrhythmia

A

irregular rhythm with impulses originating in the SA node(supraventricular)

18
Q

premature atrial contraction

A

PAC

  • regular with premature beats(premature P waves)
  • peaked P waves
19
Q

paroxysmal atrial tachycardia

A

sudden onset tachycardia(150-250 bpm)

20
Q

Wandering atrial pacemaker

A

“wandering atrial pacemaker”

  • P wave changes in size, shape, and direction from beat to beat(at least three different P wave configurations)
  • rhythm is irregular
21
Q

atrial flutter

A

250-350 bpm(atrial rate)

  • multiple activations of P wave, but does not cause QRS
  • AV node prevents ventricle from contracting and reducing output
22
Q

Atrial fibrillation

A

350-450 bpm(atrial rate)

  • atrium quivers and doesn’t contract fully so all blood does not go into ventricle = decreased Q
  • tend to develop clots so pt’s on blood thinner
23
Q

Paroxysmal junctional tachycardia

A
  • sudden onset tachycardia(150-250 bpm)
  • P wave absent or inverted
  • from emotional stress, hypoxia
24
Q

premature ventricular contraction

A

PVC = originates suddenly in an ectopic focus in a ventricle producing a giant ventricle complex

  • bigeminy = PVC every 2 beats
  • trigeminy = PVC every 3 beats
  • quadrigeminy = PVC every 4 beats
  • couplet = 2 PVC’s in a row
  • triplet = 3 PVC’s in a row
25
Ventricular tachycardia
greater than 40 bpm - all signals from the ventricle - no P wave, origin from ventricle - emergency situation
26
ventricular flutter
250-350 bpm - no effective cardiac output - no relaxation stage
27
ventricular fibrillation
greater than 350 bpm
28
1st degree AV block
PR interval > 0.2 sec and constant - generally benign - one P wave per QRS
29
2nd degree AV block
Mobitz 1 or Wenckebach(type 1) | - PR interval progressively lengthens until missing QRS
30
2nd degree AV block(type 2)
Mobitz 2 - PR interval are constant and within normal limits for those conducted beats - some P waves are not followed by QRS complex - need pacemaker to correct otherwise will progress to 3rd degree
31
3rd degree AV block
AV dissociation, complete heart block | - atrial rate and ventricular rate are regular but not matched(independently)
32
Bundle branch block
one ventricle depolarizes slightly later than the other, causing two joined QRS's(one for right ventricle and one for left ventricle)
33
ischemia
T wave inversion
34
Injury(acute)
ST segment elevation - returns to baseline with time - ST elevation associated with Q waves indicates an acute infarct
35
infarction
significant Q wave | - 1mm wide or 1/3 amplitude of QRS
36
sinus rhythm with ST segment depression
depression > 2mm boxes
37
Torsades de pointes
complexes gradually increase then gradually decrease causing a classical spindle shaped progression of waves
38
Rhythm
1) regular 2) regular but interrupted by premature beats 3) regular but interrupted by pauses 4) irregular 5) regularly irregular
39
Rate
1) regular = one HR 2) irregular = range from slowest to fastest, plus mean rate 3) regular but interrupted by premature beats = one HR(ignore premature rates) 4) regular by interrupted by pauses = range from slowest to fastest, plus mean rate 5) if it has two origins, calculate the rate from each origin separately
40
P wave
1) present 2) absent 3) shape 4) relationship with QRS complexes 5) is each followed by a QRS 6) do all P waves look the same
41
Intervals
1) PR < .2 sec 2) QRS < .1 sec and same shape 3) QT < .4 sec
42
origin
1) supraventricular = SA node, atrium, junction | 2) ventricular