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Cardiology > ECG > Flashcards

ECG Flashcards

0
Q

If there are 5 squares between QRS complexes, what is the heart rate

A

60 boom

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1
Q

What is the normal PR interval?

A

120-220 ms

= 3-5 small squares

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2
Q

What does the PR interval represent

A

Time taken for wave of depolarisation to spread from the SAN through the atrial muscle and AVN down the bundle of His and into the ventricular muscle (most of the time is taken up by delay at the AVN)

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3
Q

What is the normal duration of the QRS complex?

A

120ms (3 small squares)

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4
Q

Which leads look at the left lateral surface of the heart?

A

Leads I, II and aVL

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5
Q

Which surface do III and aVF look at?

A

Inferior

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6
Q

Which areas of the heart to V1-V6 look at?

A

V1 and V2= right ventricle
V3 and V4= septum
V5 and V6= left ventricle

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7
Q

Which leads look at the right ventricle?

A

aVR, V1 and V2

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8
Q

Which of the six standard leads is mainly negative?

A

aVR

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9
Q

Which lead(s) are predominantly negative in right axis deviation? What happens to the other lead(s)?

A

Lead I is predominant negative. The deflection in lead III is more positive

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10
Q

Which lead(s) are negative in left axis deviation?

A

Lead 3 and sometimes lead 2

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11
Q

What pathologies may be suggested by right axis deviation?

A

Right ventricular hypertrophy
Pulmonary oedema
Congenital heart disease

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12
Q

What conditions are associated with left axis deviation?

A

Left ventricular hypertrophy
Conduction defect
N.B. Left axis deviation is only significant when the QRS is predominantly negative in lead III AND lead II

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13
Q

Describe the QRS complex in a right ventricular chest lead (V1 and V2)

A

Deflection is first upward (R wave) as the septum is depolarised from left to right

There is then a downward S wave as the main muscle is depolarised. This is because the electrical effects in the LV outweigh those of the smaller RV so the predominant electrical activity is moving away from the RV leads

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14
Q

Describe the QRS complex in a left ventricular chest lead (V5 and V6)

A

The first deflection is downwards (‘septal’ Q wave) as the septum depolarises from left to right, away from the LV leads

The second deflection is upwards (R wave) due to the depolarisation of the left ventricular towards the LV leads

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15
Q

What is the transition point? Why is it important?

A

The transition point is the point at which R=S, which shows the position of the interventricular septum. It is normally at V3/V4.

This is important as if the RV is enlarged it occupies more of the precordium so the septum is shifted to the left and the transition point moves to V4/V5 or even V5/V6

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16
Q

Describe the features of first degree heart block on an ECG

A

One P wave per QRS complex

PR interval is prolonged (>220ms or 6 small squares)

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17
Q

Describe the characteristic ECG seen in Mobitz type I (Wenckebach) heart block

A

Progressive lengthening of the PR interval
One non-conducted P wave
Next conducted beat has a shorter PR interval than the previous conducted beat

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18
Q

Describe the characteristic features see in Mobitz type 2

A

Most beats are conducted with a constant PR interval but occasionally there is atrial depolarization without subsequent ventricular depolarization

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19
Q

Describe the characteristic features see in third degree (complete) heart block

A

No relationship between P waves and QRS complexes
QRS complex rate much less than P wave rate (e.g. 36/min vs 90/min)
Abnormally shaped QRS complexes because of abnormal spread of depolarization from a ventricular focus

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20
Q

What is the characteristic feature of RBBB

A

RSR1 pattern in V1

Also observe deep S waves in V6

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21
Q

What is the characteristic feature of LBBB

A

‘M’ pattern in V6 (‘W’ pattern in V1 may also be seen)

T waves are inverted in lateral leads (I, VL, V5, V6)

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23
Q

Causes of sinus bradycardia

A 
Athletic training
Fainting attacks
Hypothermia
Hypothyroidism
Drugs (beta-blockers, digoxin, amioderone, verapamil)
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23
Q

Which three rhythms can be described as supraventricular?

A

Sinus rhythm
Atrial rhythm
Junctional rhythm

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24
Q

Causes of sinus tachycardia

A 
Exercise
Anaemia
Fever
Sepsis
Sympathomimetic drugs (caffeine, adrenaline, nicotine)
Fear
Pain
Haemorrhage
Hyperthyroidism
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25
Q

What is the major difference between ECGs showing supraventricular rhythms to ECGS showing ventricular rhythms?

What is the exception to this rule?

A

Supraventricular rhythms have narrow QRS complexes
Ventricular rhythms have wide QRS complexes

The exception to this is when there is a supraventricular rhythm with right or left BBB (or Wolff-Parkinson-White WPW syndrome), where the QRS complexes will be wide

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26
Q

What is an escape rhythm?

A

The heart is usually controlled by the SAN as this is the region which spontaneously depolarises the most frequently, at a rate of around 70bpm. If the SAN fails to depolarise control is assumed by a focus either in the atrial muscle or in the region round the AV node (the junctional region), both of which depolarise at a rate of around 50bpm. If these fail or conduction through the bundle of His is blocked, a ventricular focus will take over with a rate of around 30 bpm. These slow, protective rhythms are called escape rhythms.

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27
Q

What are the four categories of abnormal rhythm?

A

Bradycardias- slow and sustained
Extrasystoles- occur as early single beats
Tachycardias- fast and sustained
Fibrillation- activation of the atria or ventricles which is totally disorganised

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28
Q

Describe the ECG seen in atrial escape

A

After one sinus beat the SAN fails to depolarise
After a delay, an abnormal P wave is seen because excitation of the atrium has begun somewhere other than the SAN
The abnormal P wave is followed by a normal QRS complex, because excitation has spread normally down the His bundle
The remaining beats show a return to sinus arrhythmia

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29
Q

What is the characteristic feature of the ECG seen in junctional escape

A

No P waves

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30
Q

Describe three types of ventricular escape

A
  1. Complete heart block: Regular P waves due to normal atrial depolarization occurring at a rate of 145/min. QRS complexes are highly abnormal due to abnormal conduction through ventricular muscle and occur rarely at a rate off 15/min. No relationship between P waves and QRS
  2. Single ventricular escape beat: after x number of normal sinus beats, the SA node fails to discharge. No atrial or junctional escape occurs. After a pause there is a single wide and abnormal QRS complex with an abnormal T-wave. A ventricular focus controls the heart for one beat before sinus rhythm is restored
  3. Accelerated idioventricular rhythm: Ventricular focus has a faster intrinsic frequency than that seen in complete heart block. Often associated with acute MI. Appearence of ECG is similar to that of ventricular tachycardia but it is benign. Following failure of SAN to depolarize, escape focus in ventricle takes over causing a regular rhythm of 75 bpm with wide QRS complexes and abnormal T waves
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31
Q

Describe the appearance of a superventricular extrasystole

A

An atrial extrasystole has an abnormal P wave
A junctions extrasystole has no P wave at all, or the P wave appears immediately before or immediately after the QRS complex
QRS complexes are normal and the same as those of sinus rhythm

A superventricular systole resets the P wave cycle. No P wave is seen at the expected time and the next P wave is late

N.B. The ECG appearance of an extrasystole arising in the atrial muscle or junctional region is the same as that of the corresponding escape beat. The difference is that an extrasystole comes early and an escape beat comes late

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32
Q

Describe the appearance of an ECG showing ventricular extrasystole

A

Abnormal and wide QRS complexes
Abnormal T wave
Common and usually of no importance

BUT
Can occur at the peak of the T wave of the preceding sinus beat causing the R on T phenomenon. This can induce ventricular fibrillation so is potentially dangerous

A ventricular extrasystole does not affect the SA node so the next P wave appears at the expected time

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33
Q

What five questions should be asked to differentiate between atrial, junctional and ventricular extrasystole?

A
  1. Is there an early P wave followed by an early QRS? If so it must be an atrial systole
  2. Can a P wave been seen anywhere? If there is no P wave it must be junctional. However, a junctional extrasystole may also cause the appearence of a P wave very close to and even after the QRS because excitation is conducted both to the atria and to the ventricles
  3. Is the QRS complex the same throughout? Yes= supraventricular extrasystole
    No= ventricular extrasystole
  4. Are the T waves normal or inverted?
    Normal= supraventricular extrasystole
    Inverted= ventricular extrasystole
  5. Does the next P wave after the extrasystole appear at an expected time?

Supraventricular extrasystole= P wave is late (as the normal periodicty of the SAN is upset)

Ventricular extrasystole= P wave comes at the expected time

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34
Q

At what rate to the atria depolarize in atrial tachycardia?

A

≥150bpm

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35
Q

At what rate does atrial tachycardia become atrial flutter?

A

≥250bpm

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36
Q

What is the fastest rate of discharge that can be conducted by the AV node? What happens if the atrial rate is faster than this?

A

200bpm

If the atrial rate is faster than this, atrioventricular block occurs with some P waves not followed by QRS complexes. This is different from 1st/2nd/3rd degree heart block as the AV node is functioning properly and is preventing the ventricles from being activated at a fast and therefore inefficient rate

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37
Q

Describe the ECG seen in atrial tachycardia

A

P waves are superimposed on the T waves of preceding beats

The QRS complexes have the same shape as those of the sinus beats

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38
Q

Describe the ECG seen in atrial flutter

A

No flat baseline between P waves giving a ‘saw-tooth’ appearance. Approx. 4 P waves per QRS complex

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39
Q

Describe the ECG seen in atrial flutter with a 2:1 block

A

Two P waves per QRS. If atrial rate is 250bpm, ventricular rate is 125bpm
The first of the 2 P waves associated with each QRS can be mistaken for the T wave of the preceding beat, but P waves can be identified by their regularity
T waves often cannot be clearly identified

40
Q

Describe the ECG seen in junctional tachycardia

A

No P waves

Regular QRS complexes

41
Q

Why is it useful to apply carotid sinus pressure (carotid sinus massage)

A

This helps to differentiate between supraventricular and ventricular tachycardia. This is because applying pressure to the carotid sinus may either abolish supraventricular tachycardia, or slow the ventricular rate. However, it has no effect on ventricular tachycardia

42
Q

Describe the ECG seen in ventricular tachycardia

A

No P waves
Wide, abnormal QRS complexes seen in all 12 leads of the ECG
T waves are difficult to identify/unidentifiable

44
Q

Give four ways of distinguishing between ventricular tachycardia and supraventricular tachycardia with bundle branch block

A
  1. Compare the QRS complex during tachycardia with that during sinus rhythm. If the patient has bundle branch block when in sinus rhythm, the QRS complex during tachycardia will have the same shape as during sinus rhythm
  2. If the QRS complex is wider than 4 squares (160bpm), the rhythm will probably be ventricular in origin
  3. Left axis deviation during the tachycardia usually indicates a ventricular origin
  4. If during tachycardia the QRS complex is very irregular, the rhythm is probably atrial fibrillation with bundle branch block
45
Q

Describe the characteristic ECG seen in atrial fibrillation

A

No P waves and an irregular base line

QRS complexes occur irregularly (AV node conducts in an ‘all or none’ fashion so that the depolarization waves passing into the His bundle are of constant intensity. However, these waves are irregular so the ventricles contract irregularly)

QRS complexes are normally shaped (as conduction into and through the ventricles is by the normal route)

Normal T waves

Depressed ST segments in leads V5 and V6

46
Q

Describe the characteristic ECG seen in ventricular fibrillation

A

No identifiable QRS
Completely disorganised ECG
Patient is likely to be unconcious

47
Q

What is Wolff-Parkinson-White syndrome? What is its clinical significance?

A

The only normal electrical connection between the atria and ventricles is the His bundle. In WPW, however, there is an extra or accessory conducting bundle which forms a direct connection between the LA and LV, with no AV node to delay conduction. A depolarization wave therefore reaches the ventricle early and ‘pre-excitation’ occurs.

The only clinical importance of WPW is that it can cause paroxysmal tachycardia. This is because depolarization can spread down the His bundle and back up the accessory pathway and so reactivate the atrium. A re-entry circuit is thus set up and a sustained tachycardia occurs.

48
Q

What is the distinguishing feature on the ECG of a patient with WPW

A

The QRS complex shows an early slurred upstroke called a delta wave (best seen in V3 and V4)

There is also a short PR interval

49
Q

What causes a peaked P wave?

A

Right atrial hypertrophy due to tricuspid valve stenosis or pulmonary hypertension

50
Q

What causes a broad, bifid P wave?

A

Left artial hypertrophy due to mitral valve stenosis

51
Q

What are the three causes of a wide QRS complex?

A
  1. Bundle branch block
  2. Depolarization initiated by a ventricular focus
  3. WPW syndrome
52
Q

What effect does right ventricular hypertrophy have on an ECG?

A

QRS complex in V1 becomes upright as the left ventricle no longer has it’s usual dominant effect. There will also be a deep S wave in V6

Right ventricular hypertrophy is usually accompanied by:

  1. Right axis deviation
  2. Peaked P wave (due to concurrent right atrial hypertrophy)
  3. Inversion of T waves in leads V1, V2 and sometimes V3 or even V4 (severe hypertrophy)
53
Q

What signs on an ECG may indicate pulmonary embolism?

A
  1. Peaked P waves
  2. Right axis deviation (inversion of QRS in lead I)
  3. Tall R waves in V1
  4. RBBB
  5. Inverted T waves in V1 (normal) spreading across to leads V2 and/or V3
  6. Shift of transition point to the left, such that R=S in lead V5 or V6 rather than in V3 or V4
  7. Q wave in III
54
Q

What signs of left ventricular hypertrophy are seen on an ECG

A

Tall R wave (>25mm) in V5 or V6
Deep S wave in V1 or V2
Inverted T waves in I, aVL, V5 and V6 (necessary for diagnosis of LV hypertrophy)
May be left axis deviation

55
Q

What causes small Q waves (<2mm) in the left ventricular leads (V6 and sometimes V5)

A

Depolarization of the septum from left to right

56
Q

What is indicated by Q waves >1 small square in width and >2mm in depth?

A

Myocardial infarction

57
Q

What is indicated by Q-waves in leads aVF and III

A

Infarction of the inferior surface of the heart

58
Q

Which leads would show Q waves if there was an infarction affecting both the anterior and lateral surface of the heart?

A

Anterior: V2-V4

Lateral: I, aVL, V5 and V6

59
Q

Describe the characteristics of an ECG showing posterior myocardial infarction

A

Dominant R waves in V1 (i.e. QRS complex is upwards)

N.B. appearance of ECG is therefore similar to that seen in right ventricular hypertrophy but other features of RV hypertrophy (right axis deviation, peaked P waves)

60
Q

What are the causes of and elevated ST segment?

How can you tell from an ECG what has caused an elevated ST segment?

A

An elevated ST segment may be caused by MI or pericarditis

If ST elevation is caused by MI, the leads in which elevation occurs indicate the location of the infarct (V leads= anterior damage; aVF and III= inferior damage.

If ST elevation is caused by pericarditis, symptoms will not be localised so ST elevation is seen in most leads.

61
Q

What are the causes of a horizontal depressed ST segment?

A
  1. Ischaemia
  2. If the ECG is normal at rest, ST horizontal depression may appear during exercise, particularly when effort induces angina
62
Q

What is the main cause of a downward sloping ST segment?

A

Digoxin use

63
Q

In which leads is the T wave normally inverted?

A

Normally inverted in leads aVR and V1

Sometimes inverted in leads III and V2

May be inverted in lead V3 in some black people

64
Q

When might T wave inversion be seen abnormally?

A
  1. Ischaemia
  2. Ventricular hypertrophy
  3. Bundle branch block
  4. Digoxin treatment
65
Q

What is the first sign to appear following an STEMI? Is this sign permanent? What happens subsequently?

A

ST elevation. This sign is not permanent- return to baseline within 24-48 hours

Subsequently, Q waves develop and T wave inversion is seen. These changes are often permanent

66
Q

How does a NSTEMI ECG differ from an STEMI ECG?

A
  1. ST segment is not elevated.
  2. No Q waves

T wave inversion is seen in V3-V4 and may be seen (or biphasic T waves may be seen) in V2 and V5

67
Q

Which leads show T wave inversion in left ventricular hypertrophy?

A

The leads looking at the left ventricle- I, II, aVL, V5 and V6

68
Q

Which leads show T wave inversion in right ventricular hypertrophy?

A

The leads looking at the RV- aVF, III, V1, V2 and V3. N.B inversion is normal in V1 and may be normal in V2 but is abnormal in V3 in white people

69
Q

What changes to the ECG are caused by digoxin?

A

T wave inversion. Sloping of ST segment

70
Q

What changes to the ECG are caused by hypokalaemia?

A

T wave flattening with the appearance of a hump on the end of the T wave called a U wave

71
Q

What changes to the ECG are caused by hyperkalaemia?

A

Peaked T wave with disappearance of ST segment

72
Q

What is the effect of hyper/hypocalcaemia on the ECG?

A

Hypercalcaemia shortens QT interval

Hypocalcaemia prolongs QT interval

73
Q

Below what rate is the heart said to be in sinus bradycardia?

A

60bpm

74
Q

Above what rate is the heart said to be in sinus tachycardia

A

100bpm

75
Q

What four things may be indicated by total absence of P waves?

A
  1. Sinus arrest
  2. Junctional escape
  3. Atrial fibrillation
  4. Hyperkalaemia
76
Q

What benign feature may be seen in the ECG of a tall, thin individual

A

Right axis deviation

77
Q

What is meant by partial RBBB

A

RSR1 pattern in V1 but QRS duration is normal. Very common and of no significance

78
Q

What variations may be seen in the ECG of a healthy athlete?

A 
Variations in rhythm:
Sinus bradycardia
Junctional rhythm
First degree block
Mobitz type 1- second degree block
Other variations in the ECG:
Tall P waves and QRS complexes
Prominent septal Q waves
Counterclockwise rotation
Tall symmetrical T waves
Biphasic T waves
T wave inversion in lateral leads
Prominent U waves
79
Q

What is meant by bifascicular block?

A

Left axis deviation + RBBB

Due to RBBB + left hemiblock (block of left bundle branch anterior fascicle)

80
Q

What is meant by trifascicular block?

A

LAD (due to left anterior hemiblock) + RBBB + 1st degree heart block

81
Q

How should you report/interpret an ECG?

A
  1. Rate and rhythm
  2. Cardiac axis and transition point
  3. Conduction intervals- PR, QRS, QT
  4. P waves (present/absent; peaked/bifascicular)
  5. QRS complexes (broad or narrow)
  6. ST segments and T waves
82
Q

An ECG shows a fast heart rhythm with absent P waves and narrow QRS complexes. What is the diagnosis?

A

Supraventricular tachycardia

83
Q

Which, of RBBB and LBBB is almost always pathological.

A

LBBB- often due to MI or LV pathology e.g. LV hypertrophy

84
Q

Causes of left axis deviation?

A 
Left anterior hemiblock
Left ventricular hypertrophy
Inferior MI
Ventricular tachycardia from LV focus
WPW syndrome (some types)
85
Q

Causes of right axis deviation?

A

PE
Anterolateral MI
Left posterior hemiblock (rare)
WPW syndrome (some types)

86
Q

What drugs may prolong QT interval?

A 
Sotalol
Quinidine
Antihistamines
Macrolides
Amiodarone
Tricyclics
87
Q

When might a J wave be seen?

A

Hypothermia
Subarachnoid haemorrhage
Hypercalcaemia

88
Q

What defines a pathological Q wave?

A

> 0.04s wide and >2mm deep

89
Q

In which leads may non-pathological Q waves occur?

A

I, aVL, V5 and V6

90
Q

What is seen on an ECG showing pericarditis?

A

Global ST elevation
“Saddle shaped”
ST depression in aVR

91
Q

How is QTc calculated?

A

QT/rootRR (because QT varies with heart rate

92
Q

What are abnormal QTc values in males and females?

A

QTc>450 in males

QTc>470 in females

93
Q

What treadmill protocol is used for ECG testing?

A

Bruce protocol

94
Q

When is exercise ECG testing used?

A
  1. To help confirm a suspected diagnosis of IHD. Should only be used when their is diagnostic uncertainty in peoples with known coronary artery disease e.g. previous MI or angioplasty, not to make a primary diagnosis.
  2. Assessment of exercise induced arrhythmias
95
Q

What are the six main contraindications for an exercise ECG?

A
  1. Recent Q wave MI <5 days ago, or unstable angina
  2. Severe aortic stenosis
  3. Uncontrolled arrhythmia, hypertension or heart failure
  4. Acute myocarditis or pericarditis
  5. Acute aortic dissection
  6. Acute PE
96
Q

How do you calculate 90% of maximal heart rate?

A

(220-age)+/- 10

97
Q

What is a Holter monitor?

A

An ECG recorder given to patients to take home which they can activate themselves when they feel symptoms. Useful as in >70% of cases, 24h ECGs are not useful as patients do not experience symptoms during the period of monitoring.

98
Q

What is bigeminy?

A

A cardiovascular phenomenon characterized by groups of two heartbeats close together followed by a longer pause. The second pulse is weaker than the first. Look for a pattern of what appears to be a relatively normal QRS complexes, each followed by a smaller, abnormal one.

An ECG can also show trigeminy, whereby there is an abnormal QRS followed by two normal beats

Cardiology (18 decks)

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